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conclusion
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  “conclusion”译为未确定词的双语例句
    Different concentration of m-Nis(1×10-5,1×10-6,1×10-8,1×10-10 mol·L-1) could antagonize the contractile responses of isolated rabbit coronary artery branch induced by ET-1 and reduce both efficacy and potency of ET-1.CONCLUSION m-Nis antagonizes the contraction responses of rabbit coronary artery smooth muscle induced by KCl and ET-1.
    不同浓度的m-Nis(1×10-5、1×10-6、1×10-8、1×10-10mol.L-1)均可以抑制ET-1引起的离体家兔冠状动脉分支收缩; m-Nis使ET-1收缩冠状动脉的效能和效价均降低,随着剂量的增大,抑制作用增强。
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    Results and Conclusion:Their structures were characterized by elemental analysis,IR,1H NMR and MS spectra.
    结果:合成的化合物Ⅰ、Ⅱ结构经IR、1HNMR、MS和元素分析等确定;
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    Results:The av- erage recovery was 99.97 %,with RSD of 0.17 %(n=5),the linear range was 0.1~0.5mg/ml,r=0.9995.Conclusion: Assayed of lbuprofen tablets by UV-spectrophotometry was simple and quick.
    结果:该法测定平均回收率为99.97%,RSD为0.17%,线形范围为0.1~0.5mg/ml,r= 0.9995。
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    Conclusion:the average recovery percentage:methanol 98.6%-102.6%,acetic ether 100.9%-103. 8%,and isopropyl alcohol 98.3%-103.3%.
    乙酸乙酯100.9%-103.8%;
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    The DUI of 77% of the statistical antimicrobials was less than 1,and that of the 23% of the statistical antimicrobials was more than 1. Conclusion The application of antimicrobials in the pharmacy was rational.
    被统计的抗生素77%的品种DUI<1,23%的品种DUI>1。
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  conclusion
The conclusion that the equilibrium is the continuous function of the dilution rate and substrate concentration in medium in a certain range is concluded.
      
In this paper, a new nonlinear closed graph theorem is established, whose result improves substantially a recent-known important conclusion.
      
The conclusion that this algorithm will definitely converge to the optimal solution under the condition of 0>amp;lt;q0>amp;lt;1 was proved true.
      
An important conclusion of wood for musical instruments with proper anisotropy, fine toughness, and weak shear of longitudinal and radial vibration was inducted.
      
However, the change was not so obvious as to draw any further conclusion concerning the influence of NW and NS treatments on the surface energy of wood.
      
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Although the occurence of a coenzyme I-independant, particle-bound α-glycerophosphate dehydrogenase in skeletal muscle of higher animals has long been recognized, little is known about its relation to the cytochrome system. Green has found that it is linked to cytochrome c but details of the electron transporting pathway has remained obscure. This problem has now been studied using the method of simultaneous action of two or more enzyme systems as described previously. Enzyme preparation obtained from thoroughly...

Although the occurence of a coenzyme I-independant, particle-bound α-glycerophosphate dehydrogenase in skeletal muscle of higher animals has long been recognized, little is known about its relation to the cytochrome system. Green has found that it is linked to cytochrome c but details of the electron transporting pathway has remained obscure. This problem has now been studied using the method of simultaneous action of two or more enzyme systems as described previously. Enzyme preparation obtained from thoroughly washed rabbit muscle mince has been employed in the present investigation. It has been found that in the presence of the rabbit muscle enzyme preparation, succinate and α-glycerophosphate each interferes with the rate of oxidation of the other when they are oxidized simultaneously. The inhibition of α-glycerophosphate oxidase by succinate can be reversed by the addition of pyrophosphate, a powerful inhibitor of succinic dehydrogenase. With cytochrome c as electron acceptor, the overall rate of simultaneous oxidation of α-glycerophosphate, succinate and reduced coenzyme I (CoIH) does not represent the sum of the rates of their separate oxidation, but corresponds only to the highest of the three rates, i.e. the rate of oxidation of CoIH. It is, therefore, believed that the α-glycerophosphate-, succinate- and CoIH-cytochrome c reductase systems have a common, velocity limiting electron carrier which is most probably the linking factor first proposed by Slater. In agreement with this conclusion, the α-glycerophosphate oxidase of rabbit muscle preparation has been found to be sensitive to the action of 2,3-dimercaptopropanol. Using 2,6-dichlorophenolindophenol, as acceptor, the overall rate of the simultaneous oxidation of succinate and α-glycerophosphate equals exacdy to the sum of the rates of their separate oxidation. Similar results have also been obtained even in presence of phenylurethane, which markedly inhibits the activity of succinic dehydrogenase and does not affect the activity of α-glycerophosphate dehydrogenase. These facts suggest that cytochrome b is not involved in the oxidation of α-glycerophosphate in rabbit muscle preparation. The pathway of hydrogen or electron transfer of the particulate α-glycerophosphate oxidase system may, therefore, be represented as follow: (See also Fig. 4)

(一) 在經徹底冲洗的兔骨骼肌製劑中,[L-α]甘油磷酸和琥珀酸的氧化彼此干涉。琥珀酸對[L-α]甘油磷酸氧化的抑制作用能因加入抑制琥珀酸脫氫酶的焦磷酸而解除。 (二) 當用細胞色素c作受體時[L-α]甘油磷酸,還原輔酶I和琥珀酸三者同時氧化時總氧化速度僅相當其中氧化速度最高者即還原輔酶I單獨氧化的速度。[L-α]甘油磷酸氧化酶系也因[2,3]二氫硫基丙醇的處理而失效。 (三) 當用[2,6]二氯酚靛酚作受體時[L-α]甘油磷酸和琥珀酸同時氧化時速度完全等於二底料單獨氧化時速度的和。[L-α]甘油磷酸的氧化不受苯代氨甲酸乙酯的影響。 (四) 本文結果說明[L-α]甘油磷酸的氧化不通過細胞色素b而通過中間因子和細胞色素c連接。

In phosphate buffer the optimal pH for the activity of the particle-bound CoIH cytochrome c reductase system of heart muscle preparation is between pH 7 and 8, whereas in glycylglycine buffer it is pH 8.0. The activity of this enzyme system in the latter buffer is considerably lower than that in the former (Fig. 1). Addition of ethylenediamine tetraacetic acid caused considerable stimulation of its activity in glycylglycine buffer, indicating that the lowered activity in the latter buffer is probably due to...

In phosphate buffer the optimal pH for the activity of the particle-bound CoIH cytochrome c reductase system of heart muscle preparation is between pH 7 and 8, whereas in glycylglycine buffer it is pH 8.0. The activity of this enzyme system in the latter buffer is considerably lower than that in the former (Fig. 1). Addition of ethylenediamine tetraacetic acid caused considerable stimulation of its activity in glycylglycine buffer, indicating that the lowered activity in the latter buffer is probably due to disturbance in the physical state of the enzyme preparation. The optimal pH for the particle-bound CoIH cytochrome c reductase system of heart muscle preparation, whether in phosphate or in glycylglycine buffer, differs markedly from the value of 8.7 reported by Mahler et al for their soluble CoIH cytochrome e reductase in glycylglycine buffer. It has been found that addition of a relatively large amount of the soluble CoIH cytochrome c reductase to heart muscle preparation does not result in any increase in activity of the CoIH oxidase system. The amount of soluble CoIH cytochrome c reductase added had an activity about 8 times that of the reductase system already present. The addition of this amount of the soluble enzyme also fails to restore to any appreciable extent the CoIH oxidase activity of heart muscle preparations previously treated with 2,3-dimercaptopropanol. It is known that 2, 3-dimercaptopropanol treatment does not affect cytochrome c or cytochrome oxidase. These show that the soluble CoIH cytochrome c reductase of IV[abler et al reacts only with soluble cytochrome c but not with the cytochrome c firmly bound to the particulate matter of enzyme preparations. The cytochrome c reductase activity of the Keilin-Hartree heart muscle preparation can be easily extracted by 9% alcohol at pH 5.4 using the procedure of Mahler et al. However even when the CoIH cytochrome c reductase activity of the heart muscle preparation has been completely destroyed by 2, 3-dimercaptopropanol treatment, the alcohol extract still contains the soluble reductase with an activity comparable to that extracted from an untreated control (Table 5). This seems to indicate that the soluble reductase of Mahler et al is formed during the extraction procedure and is therefore different from the enzyme originally present in the heart muscle preparation. The evidence presented above together with those previously obtained by us and by other workers all point to the conclusion that the soluble reductase of Mahler et al is an artifact.

(一)心肌上的輔酶Ⅰ細胞色素c還原酶系活力的最適pH和水溶性輔酶Ⅰ細胞色素c還原酶活力的最適pH顯著不同,酶系物理狀態對酶活力影響頗大。 (二)水溶性輔酶Ⅰ細胞色素c還原酶不能和心肌製劑顆粒上的細胞色素c作用。心肌製劑在經過[2,3]二氫硫基丙醇處理完全破壞原有輔酶Ⅰ細胞色素c還原酶系活力以後仍能抽提出活力很強的水溶性輔酶Ⅰ細胞色素c還原酶。這些以及我們過去曾經討論過的一些事實都說明Mahler等所獲得的水溶性輔酶Ⅰ细胞色素C還原酶是一個矯作物。 (三)本研究指出根據用人為的方法從複雜酶系中抽出的酶的性質簡單地判斷該酶在整個酶系中的作用不一定是可靠的。

Seven central depressants,including chloral hydrate,potassium bromide,sodium bromide,cal- cium bromide,alcohol,amytal and chloretone, were studied for their effects on the intraperito- neal toxicity of tartar emetic and sodium stibo- gluconate in mice. When chloral hydrate & sodium stibogluco- nate were given in suitable doses,significant protection,especially against tartar emetic,was observed.The lethal effect of both antimony compounds was somewhat increased by amytal, alcohol and chloretone. In our experiments...

Seven central depressants,including chloral hydrate,potassium bromide,sodium bromide,cal- cium bromide,alcohol,amytal and chloretone, were studied for their effects on the intraperito- neal toxicity of tartar emetic and sodium stibo- gluconate in mice. When chloral hydrate & sodium stibogluco- nate were given in suitable doses,significant protection,especially against tartar emetic,was observed.The lethal effect of both antimony compounds was somewhat increased by amytal, alcohol and chloretone. In our experiments no definite influence was observed with the 2 central stimuiants,caffeine and strychinine,but as the number of animals used was rather small and the range of their dosage rather narrow no final conclusion can be drawn.

1.关于吐酒石与葡萄糖酸锑 V 钠对小白鼠的致死作用,我们探索了一些中枢抑制药与兴奋药的影响。所试的抑制药有水合氯醛,溴化钾,溴化钠,溴化钙,酒精,安眠妥与三氯叔丁醇七种。兴奋药有苯甲酸钠咖啡硷与士的宁二种,以及溴咖合剂。2.在所试的药物中,水合氯醛与溴化物在适当剂量时,对于二种锑剂尤其是吐酒石中毒呈现比较显著的保护作用。安眠妥,酒精与三氯叔丁醇的数种剂量,似能增加锑剂的毒性。咖啡硷与士的宁在我们的实验条件下,并不表现明显的影响,但因所用动物数较少,剂量范围较窄,难于作出结论。

 
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