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  另一组
     The reaction of[Fe-(CO)_2Cp]_2Sn(Ar)Cl with a series of nucleophiles,Y,gave another group of new compounds,[Fe(CO)_2Cp]_2Sn (At) Y,among which[Fe(CO)_2Cp]_2Sn(p-MeC_6H_4)Br was characterized by X-ray structural analysis.
     [Fe(CO)_2Cp]_2Sn(Ar)Cl与各种亲核试剂Y发生取代反应给出另一组新的化合物[Fe(CO)_2Cp]_2Sn(Ar)Y,其中[Fe(CO)_2Cp]_2Sn(p-MeC_6H_4)Br经X射线衍射分析确定结构.
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     The abnormalities of FEV_1%, FMF, deduced cv/vc% and measured cv/vc% in another group (175 cases)were analysed.
     对另一组175例COPD患者的FEV_1%、FMF及推算和实测CV/VC(%)的异常率也进行了分析。
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     Another group was given 0.5U% oxytocin intravenously.
     另一组给予 0 .5U %缩宫素静脉点滴。
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     The viscosity of intraocular silicone oil in one group was 3 700 cst and was 5 000 cst in another group.
     一组眼内填塞硅油的粘度为3 700 cst,其为法国产,另一组硅油的粘度为5 000 cst,其为德国产。
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     Methods 20μmol/L of one non-sense oligonucletide(N-ODN) and three ASODNs against different IGF-IR mRNA sites were transfected into EC9706 cells, another group was non-transfected.
     方法将4组终浓度为20μmol/L的3条封闭IGF-IR不同基因位点的反义寡核苷酸(ASODN1~3)及1条无关寡核酸(N-ODN)分组转染培养的食管癌EC9706细胞,另一组不转染;
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  “another group”译为未确定词的双语例句
     kg-1 .d-1* 2~3d. Another group,22cases,were t reated with IVIG 2g .
     kg-1.d-1*2~3d和22例IVIG2g.
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     The reserving anus rates in first groups was 32.35%(11/34),more significantly as compared with that in another group(8.11%,3/37)(P<0.05).
     放疗组保肛率32.35%(11/34),对照组为8.11%(3/37),放疗组的保肛率高于对照组,差异有统计学意义(P<0.05)。
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     MVD in p53 positive stained group was 31.03±10.27,in another group 19.32±6.64,there was significant difference (P<0.001).
     p53阳性组MVD值为31.03±10.27,而p53阴性组MVD值为19.32±6.64,有显著性差异(P<0.001)。
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     Another group were treated with de xamethasone(DXM)0 3~0 5mg/kg.
     组应用地塞米松 0 .3~ 0 .5 mg/ kg.
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     The expression of PCNA,P21and P53were significantly higher in Hp—L infection group than in the another group (P<0.01,P<0. 05).
     Hp-L型阳性组的PCNA、P21、P53表达也明显高于Hp-L型阴性组(P<0.01,P<0·05)。
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     ] group.
     ]组。
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     . group's.
     而且实验组的增量显著高于控制组的增量。
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     Another blank group was controled.
     另设空白对照组。
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     another group was used as control.
     另 1组为常规细胞组接种组 ,设为对照 ,不作任何处理。
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  another group
Another group was treated with the vaccine after tumor implantation.
      
Another group of marine and (halo)alkaliphilic heterotrophic bacteria capable of complete oxidation of sulfur compounds to sulfate mostly includes representatives of the alphaproteobacteria which are most closely related to nonsulfur purple bacteria.
      
Conduction along block boundaries is two-dimensional (2D) in one group of samples and three-dimensional (3D) in another group.
      
The problems and results of another group of methods for obtaining artificial substrates which gained favor recently and become known as "compliant" or "soft" substrates are discussed.
      
The phylogenetic tree also showed that Pneumocystis from human and monkey formed one group and four rodent Pneumocystis formed another group.
      
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The present study aims to observe (1) sex influence of the host on the development of male and female worms and (2) the effect of the normally developed and copulated males and females on the development of the unpaired worms. For this purpose, mice of different sexes were infected with either male or female cercariae and were killed at definite intervals after infection. The size of worms collected was measured and the condition of the development of sex organs was also observed. Another group of mice...

The present study aims to observe (1) sex influence of the host on the development of male and female worms and (2) the effect of the normally developed and copulated males and females on the development of the unpaired worms. For this purpose, mice of different sexes were infected with either male or female cercariae and were killed at definite intervals after infection. The size of worms collected was measured and the condition of the development of sex organs was also observed. Another group of mice was infected with more female cercariae and less male cercariae while a third group, vice versa. Similar observations were made at definite intervals as in the first group.It was found that the sex difference in the host bore definite influence on males in the portal circulation, but this was not so evident on females. The development of the unisexual males in the male host was more rapid than in the female host. Female worms in the female host were only slightly bigger than in the male host.Mature males and females in copula did not hasten the normal development of the unpaired males which were only slightly larger than those in unisexual infection, but definitely smaller than those in copula. On the contrary, worms in copula seemed to exert an inhibitory action on the unpaired females which were definitely smaller than the females in unisexual infection.The copulation of male and female worms was permanent in character. It did not seem that male schisrosomes would change their partners. One male could only hasten the development and matura:ion of only one female.

1.单性感染之雌性日本血吸虫平均不超过5.35±0.07毫米,只为复性感染中成熟雌虫长度之1/3左右,且其生殖器官始终呈童雏状态。单性感染之雄虫能发育成熟,贮精囊内也有活动精子,但其大小与成熟合抱之雄虫平均相差3.05毫米左右。 2.宿主性别之不同对于血液中的雄性血吸虫有比较显著的影晌。在雄性宿主体内单性雄虫比雌性宿主体内之单性雄虫发育快。宿主性别之不同对于单性雌虫之影响则不甚明显,雌性宿主体内发育之雌虫仅略大于在雄性宿主体内之单性雌虫。 3.血液中合抱成熟之雌虫及雄虫的分泌物质不能促使未合抱之雄虫发育正常。未合抱之雄虫似略大于单性感染之雄虫,但是显著小于合抱雄虫。 4.合抱成熟之雌虫及雄虫的分泌物质对于未合抱之雌虫完全没有促使发育的作用。相反地,有一定的抑制作用而使未合抱之雌虫显著地小于单性感染之雌虫,二者之生殖器官皆停留于雏形状态。 5.复性感染中每对雌雄成虫之抱合是永久性的。每一条雄虫只能促使一条雌虫发育成熟而不再转换抱合其余未成熟之雌虫。 6.鉴于雌雄合抱对于日本血吸虫正常发育的必要性,故就生物进化之观点讨论了日本血吸虫雌雄异体的原始性。

As has been generally accepted, the ventricular fibrillation (VF) occasionally occuring during antimonial therapy may arise from the combined effects of antimonials on the heart muscle itself and on the vegetative nerves. Basing upon the experimental data accumulated in years, two hypotheses have been proposed to explain the nervous mechanism of the pathogenesis of this toxic arrhythmia, one emphasizing the vagal role therein and the other the sympathetic. It was reported in our previous paper that after repeated...

As has been generally accepted, the ventricular fibrillation (VF) occasionally occuring during antimonial therapy may arise from the combined effects of antimonials on the heart muscle itself and on the vegetative nerves. Basing upon the experimental data accumulated in years, two hypotheses have been proposed to explain the nervous mechanism of the pathogenesis of this toxic arrhythmia, one emphasizing the vagal role therein and the other the sympathetic. It was reported in our previous paper that after repeated challenges with ascending doses of potassium antimony tartrate (PAT) combined with fibrillating electric shocks, a large dose of PAT per se could usually induce VF, and this PAT-induced VF could be prevented by prior sympathectomy or reserpinization. In the present investigation the catecholamine (CA) content of rabbit ventricles was determined biologically under the same experimental conditions as previously described. The mean CA content of the ventricles of the saline control group was found to be 1.13±0.06μg/g wet tissue, and that of the electric shock control group 1.72±0.45μg/g, which was characterized by very wide fluctuations among different members in the group, the difference between these two control groups being, however, statistically insignificant. In the group of PAT combined with electric shocks, we noticed the same effects of PAT as previously described, namely, the lowering of VF electric threshold, acceleration of heart rate and the occurrence of PAT-induced VF, and in addition, we found that the CA content of this group significantly dropped to 0.65±0.05μg/g. The CA content of another group, in which ascending doses of PAT were similarly administered without accompaniment of electric shocks averaged 0.47±0.08μg/g, a value also significantly lower than that of the saline control. Reserpinization totally abolished the PAT-induced VF and almost completely depleted the heart's CA store. These data give direct support to our previous postulate that cardiac catechol amines play an important role in the development of the PAT-induced VF. Besides CA content, the acetylcholine (ACh) content of the ventricles of groups of rabbits were also assayed biologically. The saline control group gave a mean value of 0.323±0.013 μg/g, which could neither be changed by PAT in a single large dose or in a course of "3 days treatment", nor by prior vagotomy or atropinization in addition to a large dose of PAT. Moreover, reserpine, in doses effectively protecting rabbits against the PAT-induced VF, did not materially change the ACh content of rabbit ventricles, while larger doses of it markedly elevated that of guinea pig ventricles. From these results, another postulate of ours that the vagus does not take an essential part in this experimental arrhythmia seems to be further substantiated.

前文报导,在麻醉兎观察酒石酸銻鉀(簡称銻鉀)对电致心室顫动的影响实驗中,发現在实驗后期,大剂量銻鉀本身也能产生心室顫动,而这种銻致性心室顫动能被預先切断交感神經或利血平化所防止。本文采用与前文相同的实驗模型和条件,对家兎心室內儿茶酚胺含量进行了生物测定。发現盐水对照组心室儿茶酚胺含量平均为1.13±0.06微克/克。电刺激对照組为1.72±0.45,虽与盐水对照组相比在統計学上差別不显著,但波动較大。在电刺激加銻鉀组中,銻鉀能显著降低全部动物的电致顫阈,稍加速心率,并使6/11兎出現銻致性心室顫动,心室儿茶酚胺含显著减少至0.65±0.05。单注射銻钾組心肌儿茶酚胺含量平均为0.47±0.08,較盐水对照組显著减少,但与电刺激加銻鉀组差別不显著。利血平化(0.1毫克/公斤/天,共2次)后,兎心儿茶酚胺充份地被耗竭,含量降至0.032±0.003,耗竭程度达97%。利血平化后电刺激加銻鉀组的心室儿茶酚胺含量为0.041±0.010,銻致性心室顫动并不出現。上述資料对前文的推論提出直接証明,卽儿茶酚胺在該种心律紊乱发病机制中具有重要意义。除儿茶酚胺含量外,我們又进行了心室乙酰胆碱含量的测定,发現正常盐水对照家兎心...

前文报导,在麻醉兎观察酒石酸銻鉀(簡称銻鉀)对电致心室顫动的影响实驗中,发現在实驗后期,大剂量銻鉀本身也能产生心室顫动,而这种銻致性心室顫动能被預先切断交感神經或利血平化所防止。本文采用与前文相同的实驗模型和条件,对家兎心室內儿茶酚胺含量进行了生物测定。发現盐水对照组心室儿茶酚胺含量平均为1.13±0.06微克/克。电刺激对照組为1.72±0.45,虽与盐水对照组相比在統計学上差別不显著,但波动較大。在电刺激加銻鉀组中,銻鉀能显著降低全部动物的电致顫阈,稍加速心率,并使6/11兎出現銻致性心室顫动,心室儿茶酚胺含显著减少至0.65±0.05。单注射銻钾組心肌儿茶酚胺含量平均为0.47±0.08,較盐水对照組显著减少,但与电刺激加銻鉀组差別不显著。利血平化(0.1毫克/公斤/天,共2次)后,兎心儿茶酚胺充份地被耗竭,含量降至0.032±0.003,耗竭程度达97%。利血平化后电刺激加銻鉀组的心室儿茶酚胺含量为0.041±0.010,銻致性心室顫动并不出現。上述資料对前文的推論提出直接証明,卽儿茶酚胺在該种心律紊乱发病机制中具有重要意义。除儿茶酚胺含量外,我們又进行了心室乙酰胆碱含量的测定,发現正常盐水对照家兎心室乙酰胆碱含量平均为0.323±0.013微克/克。銻鉀一次大剂量注射或銻鉀三日給药法均不明显改变其含量。預先切断迷走神經或注射大剂量阿托品后再給大剂量銻鉀,也对心室乙酰胆碱含量无明显影响。因此完全不能証实黄铭新等关于这方面的报导。利血平在对家兎銻致性顫动具有防止作用的剂量下并不影响心室乙酰胆碱含量,較大剂量却能显著提高豚鼠心室乙酰胆碱的含量(P<0.01)。这些資料也完全支持前文所作出的推論,卽迷走神經在这种实驗性心律紊乱发病机制中的意义不大。

The influences of 15-methyl-prostaglandin F_(2α)(15-methyl-PGF_(2α))and 13-dehydro-ω-ethyl-prostaglandin F_(2α)(13-dehydro-ω-ethyl-PGF_(2α))on plasma pro-gesterone(P)and their effects on the termination of early pregnancy were studiedin pregnant rats by the same doses of 0.5mg/rat i.m.b.i.d on Day 7 of gestation.Theplasma Plevels at 4,8,24 hours after 15-methyl-PGF_(2α)were lowered down to 56.6%,43.7% and 13.3% of the initial values respectively.Pregnancy was terminated72 hours after 15-methyl-PGF(2α)administration...

The influences of 15-methyl-prostaglandin F_(2α)(15-methyl-PGF_(2α))and 13-dehydro-ω-ethyl-prostaglandin F_(2α)(13-dehydro-ω-ethyl-PGF_(2α))on plasma pro-gesterone(P)and their effects on the termination of early pregnancy were studiedin pregnant rats by the same doses of 0.5mg/rat i.m.b.i.d on Day 7 of gestation.Theplasma Plevels at 4,8,24 hours after 15-methyl-PGF_(2α)were lowered down to 56.6%,43.7% and 13.3% of the initial values respectively.Pregnancy was terminated72 hours after 15-methyl-PGF(2α)administration in all the rats of this group.Theplasma P levels at 4,8,24 hours after 13-dehydro-ω-ethyl-PGF_(2α)were reducedto 63.5%,34.4% and 51.9% of the initial values respectively.It was found inthis group that foetuses still remained in the uteri in 5 out of 6 rats 72 hours afterdrug administration,but they were smaller and free from the uteri as comparedwith controls.Concomitant treatment with 20 i.u.of HCG prevented the decrease in P in-duced by 15-methyl-PGF_(2α).The plasma P levels at 4,8,24 hours after HCGand 15-methyl-PGF_(2α)were not significantly different from those of the controls,Pregnancy was not interrupted in all the rats of this group.The size and the numberof the foetuses were similar to those of the controls.In another group,15-methyl-PGF_(2α)(20μg) was given as intravenous infusionto anaethetized pregnant rats,30 minutes after the infusion,the amount of P inutero-ovarian venous blood decreased from 1.271±0.154μg/10 min to 0.279±0.83μg/10 min.No significant change was observed in the blood flow of utero-ovarian vein.When HCG(20 i.u.)was administered intravenously,it stimulated the corpusluteum and produced a rise in the amount of P in utero-ovarian venous blood frominitial value 1.123±0.162μg/10 min to 1.496±0.018μg/10 min.Concomitanttreatment with HCG brought the P content in the utero-ovarian blood back to1.179±0.042μg/10 rain,a value similar to the initial one.The result demonstrated that 15-methyl-PGF_(2α)has a longer suppressing effecton plasma P level of the pregnant rats than 13-dehydro-ω-ethyl-PGF_(2α).Theeffect in terminating early pregnancy in rats seems related to the duration of thereduction of plasma P and the plasma P level 24 hours after PGs administration.The mechanism of the luteolytic effect of 15-methyl-PGF_(2α)appears to be littlerelated to the utero-ovarian venous blood flow, but probably related to the inhibi-tion of the effect of intrinsic gonadotropic hormone at the ovarian level.

15-甲-PGF_(2α)和13-去氢-ω 乙-PGF_(2α)在相同剂量时均能使妊娠7天的大鼠血浆孕酮浓度下降。15-甲-PGF_(α2)组于用药后4、8和24小时血浆孕酮浓度下降,分别为用药前的56.6%、43.7%和13.3%。在给药后72小时所有动物子宫中胚胎已被吸收。13-去氢ω-乙-PGF_(2α)组于用药后4、8和24小时血浆孕酮浓度分别为用药前的63.5%、34.4%和51.9%,给药后72小时大多数动物子宫中仍有胚胎,但胚胎比对照组显著为小,且多游离于子宫中。在给15-甲-PGF_(2α)前30分钟和第二次给15-甲-PGF_(2α)的同时,肌注 HOG 20国际单位,能完全对抗15-甲-PGF_(2α)的降低妊娠大鼠血浆孕酮浓度和抗早孕作用,给药后24小时内血浆孕酮浓度与对照组相似,全部动物维持妊娠,胚胎大小和数目也与对照组相似。恒速静注15-甲-PGF_(2α)(20微克)于麻醉妊娠大鼠,30分钟后已使子宫卵巢静脉血中孕酮含量由用药前1.271±0.154微克/10分钟下降到0.279±0.083微克/10分钟,给药后60分钟仍维持于低水平。如预先静注 HOG 20国际单位,可使子宫卵巢...

15-甲-PGF_(2α)和13-去氢-ω 乙-PGF_(2α)在相同剂量时均能使妊娠7天的大鼠血浆孕酮浓度下降。15-甲-PGF_(α2)组于用药后4、8和24小时血浆孕酮浓度下降,分别为用药前的56.6%、43.7%和13.3%。在给药后72小时所有动物子宫中胚胎已被吸收。13-去氢ω-乙-PGF_(2α)组于用药后4、8和24小时血浆孕酮浓度分别为用药前的63.5%、34.4%和51.9%,给药后72小时大多数动物子宫中仍有胚胎,但胚胎比对照组显著为小,且多游离于子宫中。在给15-甲-PGF_(2α)前30分钟和第二次给15-甲-PGF_(2α)的同时,肌注 HOG 20国际单位,能完全对抗15-甲-PGF_(2α)的降低妊娠大鼠血浆孕酮浓度和抗早孕作用,给药后24小时内血浆孕酮浓度与对照组相似,全部动物维持妊娠,胚胎大小和数目也与对照组相似。恒速静注15-甲-PGF_(2α)(20微克)于麻醉妊娠大鼠,30分钟后已使子宫卵巢静脉血中孕酮含量由用药前1.271±0.154微克/10分钟下降到0.279±0.083微克/10分钟,给药后60分钟仍维持于低水平。如预先静注 HOG 20国际单位,可使子宫卵巢静脉血中孕酮含量由用药前1.123±0.162微克/10分钟升高到1.496±0.018微克/10分钟,在 HCG作用的基础上再静脉恒速注入15-甲-PGF_(2α),虽可使子宫卵巢静脉血中孕酮含量下降到1.179±0.042微克/10分钟,但不能降低到

 
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