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damaged group
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  损伤组
     Compared with the Aβ damaged group,at 10~(-10) mol/L concentration of 17βE_2,the values η of the 17βE_2 intervention group showed no significant difference(P>0.05);
     17βE2干预组在10-10mol/L浓度的条件下各时间点η值与Aβ损伤组比较差异无显著性(P>0.05);
短句来源
     RESULTS: ① The activities (A values) of the islet cells in the IL-1β damaged group, IL-1β+1, 25, 50 mmol/L FDP groups were obviously lower than that in the normal control group (0.116±0.012, 0.129±0.008, 0.125 ±0.015, 0.120±0.016, 0.252±0.020, P < 0.01).
     结果:①白细胞介素1β损伤组、白细胞介素1β+1,25,50mmol/L1,6-二磷酸果糖组胰岛细胞活性(A值)明显低于正常对照组(分别为0.116±0.012,0.129±0.008,0.125±0.015,0.120±0.016,0.252±0.020,P<0.01);
短句来源
     Results Compared with the blank group,after adding 5 μmol/L of soluble Aβ25-35,the microviscosity values η of Aβ damaged group increased significantly(P<0.01) at each time point.
     结果Aβ损伤组在加入5μmol/L可溶性Aβ25-35后各时间点,与空白对照组比较,其微黏度η值均明显升高(P<0.01);
短句来源
     The activities (A values) of the islet cellswere not significantly different from that in the IL-1β damaged group (P > 0.05) when the FDP dosage was too low (1 mmol/L) or too high (25 mmol/L).
     1,6-二磷酸果糖浓度过低(1mmol/L)或过高(25,50mmol/L)时胰岛细胞活性(A值)与白细胞介素1β损伤组比较差异无显著性意义(P>0.05)。
短句来源
     It is much lower than that in the liver damaged group (188. 7±21. 4u p<0. 001). The blood MDA in Zhujie Gingseng groups,7. 59±1. 1nmol/ml6. 55±1. 09n mol/ml.
     过氧化物丙二醛(MDA)含量分别为7.59±1.1 nmol/ml和6.55±1.09 nmol/ml,明显低于肝损伤组(13.10±1.85 nmol/ml,p<0.001)。 而且这种作用成剂量效应关系。
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  “damaged group”译为未确定词的双语例句
     Result: PL in N 2 and P 300 of brain damaged group was significantly longer than that of control group ( P <0 01),and amplitude of P 3 was significantly decreased ( P <0 05).
     结果 :脑损害各组患者的N2 及P3波峰潜伏期 (PL)较对照组显著延长 (P <0 0 1) ,P3波幅显著降低 (P <0 0 5) .
短句来源
     11 cases of the moderately damaged group (8-12 points) improved on 75%;
     中度(8~12分)11例,术后改善率75%;
短句来源
     Results The MTT and protein levels in cells in normal control group and VC or VE treated groups were higher than that in H2O2 damaged group without VC or VE(P<0.05).
     结果:正常对照组及VC、VE各剂量组细胞活力、蛋白质含量均高于H2O2损伤对照组,差异有显著性(P<0.01);
短句来源
     The activities of SOD? GSH-PX and the concentrition of NO in ox-LDL damaged group were lower than that in the other groups(P<0.05).
     而ox -LDL组SOD、GSH -PX活性及NO浓度均低于control组和VE +ox -LDL组、(SI-L、M、H) +ox -LDL组 ,差异有显著性 (P<0.01) ;
短句来源
     3 cases of the severely damaged group (< 7 points) improved on 78%.
     重度(< 7分)3例,术后改善率78%。
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  相似匹配句对
     B.C and group D.
     B组:10例肾衰竭非透析组;
短句来源
     Group C.
     实验动物随机分为3组,每组9只。
短句来源
     Results Organs function was damaged in group E.
     结果注ET后24h器官功能受损,病理学检查有器质性损害。
短句来源
     And the tumor cells of group A were mildly damaged.
     A组的肿瘤细胞出现轻度损害 ;
短句来源
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  damaged group
Low IQ patients performed like the brain-damaged group; high IQ children failed to benefit from reduction in stimulus competition and produced more bizarre and irrelevant responses.
      
Figure 9 shows that the severely damaged group has two subgroups, A and B.
      


In the present paper the effects of septel electrolytic lesions on theECoG orienting reflex in rabbits are reported. The experiment shows that inthe septal damaged group the rate of the arousal reaction of the cortexand the hippocampus to the novel stimuli were significantly lower than thatof the control group. The result is identical with that of our previousinvestigation of the hippocampal damaged group. A discussion concerning the results of the experiments are given by theauthors.

本实验观察电损毁家兔隔区对新异刺激的探究反应的影响。实验结果表明:损毁隔区的动物对新异刺激作用时脑皮层电图(ECoG)惊醒反应的出现率,在皮层与海马都明显低于对照组,与损毁海马动物的特点一致。作者对这一实验结果进行了讨论。

The present report is a summary ofthe study designed to revise the LNNB.68 items of the original battery,whichdid not suit the background of Chineseculture,were changed.50 brain-damagedpatients and normals were tested assamples.The cutoff points of eachsubtest were yeilded according to themeans and standard deviations of the twogroups.They were considered as thenorms of the revised battery.To classifythe normal or the brain-damaged by them,the accuracy rates ranged from 86% to98% in the normal group and 60%...

The present report is a summary ofthe study designed to revise the LNNB.68 items of the original battery,whichdid not suit the background of Chineseculture,were changed.50 brain-damagedpatients and normals were tested assamples.The cutoff points of eachsubtest were yeilded according to themeans and standard deviations of the twogroups.They were considered as thenorms of the revised battery.To classifythe normal or the brain-damaged by them,the accuracy rates ranged from 86% to98% in the normal group and 60% to 88%in the brain-damaged group.The hit-rateof the brain-damaged group computedwith the method of the discriminantanalysis was 94%,and the hit-rate ofdamaged lateralization was 90%.Thosewere regarded as the validity of therevised battery.The reliability wascomputed by the coefficients of split-half correlation of subtests.Therer's were.82 to.97.

本文是对Luria-Nebraska 神经心理成套测验初步修订的总结。修订了原测验的一些项目,制作了测验器材,共测试了50例脑病患者和50例正常人,确定了划界分,对该测验的信度、效度作了初步分析。劈半相关系数为.82—.97。应用判别分析法,获得样本病人脑损害诊断符合率为94%,定侧诊断符合率为90%。

Examination was performed to study the mechanism concerning the protective effect of somatostatin on isolated islet cells damaged by streptozotocin (STZ). It was found that somatosta-tin (0.1 μg/ml) could hinder the incorporation of 3H-STZ into the islet cells, therefore the depletion of NAD in islet cells induced by STZ was prevented. The incorporation percentage was decreased from 100 + 8.9% in damaged group to 44.90+7.09% in protected group ( p<0.001); the NAD content was increased from 0.190...

Examination was performed to study the mechanism concerning the protective effect of somatostatin on isolated islet cells damaged by streptozotocin (STZ). It was found that somatosta-tin (0.1 μg/ml) could hinder the incorporation of 3H-STZ into the islet cells, therefore the depletion of NAD in islet cells induced by STZ was prevented. The incorporation percentage was decreased from 100 + 8.9% in damaged group to 44.90+7.09% in protected group ( p<0.001); the NAD content was increased from 0.190 ± 0.082 pmol/105 cell in damaged group to 0.442± 0.071 pmol/105 cell in protected group (p<0.05).The results indicate that the inhibition of the intracellular entry of STZ might be one of the protective mechanisms of somatostatin on pancreatic islet cells.

本工作在细胞水平研究生长抑素保护胰岛细胞,防止链佐霉素损伤的可能机制,结果表明,生长抑素很可能通过抑制链霉素进入胰岛细胞,进而阻止细胞内尼克酰胺腺嘌呤二核苷酸(NAD)的耗竭而实现其保护作用的。

 
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