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damaged group
相关语句
  损伤组
    Methods: 52 Sprague-Dawley rats were devided into three groups at random, containing cornia damaged group, ischemia-reperfusion group(IR), ischemia-reperfusion and PNS group (IR+PNS).
    方法:SD大鼠52只,随机分为对照角膜损伤组,缺血再灌注组(IR),缺血再灌注十三七总皂甙(IR+PNS)治疗组。
短句来源
    It is much lower than that in the liver damaged group (188. 7±21. 4u p<0. 001). The blood MDA in Zhujie Gingseng groups,7. 59±1. 1nmol/ml6. 55±1. 09n mol/ml.
    过氧化物丙二醛(MDA)含量分别为7.59±1.1 nmol/ml和6.55±1.09 nmol/ml,明显低于肝损伤组(13.10±1.85 nmol/ml,p<0.001)。 而且这种作用成剂量效应关系。
短句来源
    Results:Compared with the damaged group,the lactate dehydrogenase (LDH) release decreased significantly (P<0. 01) and the neuronal survival rate increased significantly (P<0. 01 ) in the protective group of ginsenosides.
    结果:与损伤组比较,人参皂甙保护组的乳酸脱氢酶(LDH)释放量显著减少(P<0.01); 而细胞存活率显著增高(P<0.01)。
短句来源
    Results: Compared with the damaged group, the LDH release decreased significantly (P<0.01) and the MTT metabolism increased significantly(P<0.01) in the protective group of SSTF.
    结果:与H2O2损伤组比较,黄芩茎叶总黄酮保护组的LDH释放量显著减少(P<0.01),而MTT代谢率显著增高(P<0.01)。
短句来源
    Human neuroblastoma cells SY5Y were segregated into 3 groups: normal control group; Aβ 25-35(25 μmol/L) damaged group; and Aβ 25-35(25 μmol/L) + Lishi No.
    将人神经母细胞瘤株SY5Y细胞接种后分为正常对照组、Aβ2 5 352 5 μmol/L损伤组和Aβ2 5 352 5μmol/L加李氏 5号方水提液保护组。
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  “damaged group”译为未确定词的双语例句
    The mice were divided randomly(n=12) into five groups:normal control group(A),PAF damaged group(B),SY high dose group(C),SY middle dose group(D)and SY low dose group(E).
    小鼠随机分为正常动物空白对照组(A),模型动物对照组(B) ,模型动物大剂量(C) ,中剂量(D) ,低剂量(E) 药物组共五组(n = 12) 。
短句来源
    Compare with normal control group, Aβ 25-35 damaged group showed reduced MTT metabolic rate, increased LDH leakage rate, and diminished axonal length and area of cell body, but the addition of Lishi No. 5 Formula normalized the foregoing changes. Lishi No.
    结果 :与正常对照组相比 ,Aβ2 5 35组SY5Y细胞的轴突长度和胞体面积均较小 ,MTT代谢率降低 ,LDH漏出率升高 ,而加入李氏 5号方保护后 ,可使上述指标恢复或接近正常。
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  damaged group
Low IQ patients performed like the brain-damaged group; high IQ children failed to benefit from reduction in stimulus competition and produced more bizarre and irrelevant responses.
      
Figure 9 shows that the severely damaged group has two subgroups, A and B.
      


In TCM-WM therapy of severe viral hepatitis,certain therapeutic effects had been shown by some decoctions,i.e.a reinforced decoct.of compo- site prescription Herba Artemisiae scopariae,decoct.Radix et Rhizoma Rhei,cooling blood & clearing heat with heavy Radix Paeoniae rubra, as well as a composite prescription-decoct.of Pr-FG-Rp(P.r.,Fructus Gardenia jasminaides,and Rheum palmatum).In this present experiment, as a continuation study following the authors'rat experiment with comp- osite prescription Pr-FG-Rp(Shu,et...

In TCM-WM therapy of severe viral hepatitis,certain therapeutic effects had been shown by some decoctions,i.e.a reinforced decoct.of compo- site prescription Herba Artemisiae scopariae,decoct.Radix et Rhizoma Rhei,cooling blood & clearing heat with heavy Radix Paeoniae rubra, as well as a composite prescription-decoct.of Pr-FG-Rp(P.r.,Fructus Gardenia jasminaides,and Rheum palmatum).In this present experiment, as a continuation study following the authors'rat experiment with comp- osite prescription Pr-FG-Rp(Shu,et al,1988),repairing effects of de- coctions Pr,FG and Rp were compared with each other in 28 CCI_4-induced liver necrosis Wistar rats,in order to provide further cytomorphological basis for clinical trials and probing into their pharmacodyamics. Altogether 43 experimental Wistar rats were divided into five groups. The liver damage group(10 rats)was poisoned via hypodermal injectioils (hdi) with CCI_4;as for the three treatment groups,besides CCI_4 hdi, Pr,FG or Rp was administrated daily through gastric intubation(gt) for 7 days;while the control group(5 rats)was administrated with normal saline hdi and tap water gt only.Liver histopathological and ultrastructure speci mens following decapitation of the animals at the 8th day showed that,of the three treated groups,besides the still more or less severe damages in group FG,the other two groups both displayed only mild damages,in hepatic lobule,the necrotic area and the liver cellul- arfat droplets decreased,while RNA,glycogen increased,nucleole and SDH- ase,G-6-Pase activities increased as well,and the roughendopl asmic reti- culumrecovered to normal range,the mitochondria regained the normal str- ucture and increased in number.It is suggested that both Rp and Pr are able to prevent liver damage,and Rp better than Pr.Their mechanism mi- ght be that they prevent and repair the damage of endoplasmic reticulum, promoting protein synthesis and detoxication,thus restore and recover the liver functions.

将43只实验大鼠分成5组:肝损伤组给CCI_4皮下注射;大黄组、赤芍组、栀子组除给CCI_4皮下注射外,分别给以上述各药煎剂灌胃各共7日;对照组皮下注射生理盐水,并以自来水灌胃。结果,三个中药治疗组与肝损伤组相比,除栀子组外,大黄组及赤芍组均使肝损伤区域缩小,肝细胞中脂滴减少,细胞核增大,RNA、核仁增多,糖原恢复,琥珀酸脱氢酶和葡萄糖-6-磷酸酶活性增强,粗面内质网恢复平行排列,滑面内质同增加,线粒体形态结构恢复、数量增加,表明大黄及赤芍对CCI_4所致肝损伤均有修复作用(大黄更佳),其机制可能是通过内质同损伤的阻止,促进蛋白质合成及解毒,恢复了肝功能。

Multiple injections of streptozotocin (STZ) not only damaged the islet β cells but also causedatrophy of thymus, increased macrophage in spleen, decreased lymphocytes in blood, and causedlymphocytes infiltration in islets. All the above observations could be partially prevented whensomatostatin (10μg/kg) was given subcutaneously 10 minutes prior to each STZ injection. Thedifferences between STZ damage group and somatostatin protective group were significant. Theseresults suggest that immunological...

Multiple injections of streptozotocin (STZ) not only damaged the islet β cells but also causedatrophy of thymus, increased macrophage in spleen, decreased lymphocytes in blood, and causedlymphocytes infiltration in islets. All the above observations could be partially prevented whensomatostatin (10μg/kg) was given subcutaneously 10 minutes prior to each STZ injection. Thedifferences between STZ damage group and somatostatin protective group were significant. Theseresults suggest that immunological mechanism might be involved to some degree in the protectiveaction of somatostatin against the damage of islets by STZ in mice.

连续多次小剂量注射链佐霉素(STZ)后,小鼠产生严重糖尿病,同时出现胸腺萎缩,脾内巨噬细胞增加,脾小结减少,血中淋巴细胞百分数明显下降,胰岛局部淋巴细胞浸润。在每次给STZ前10min注射生长抑素,小鼠血糖及血清胰岛素基本正常,STZ对全身主要免疫器官的破坏程度均减轻,胰岛局部淋巴细胞浸润消失。实验结果提示,免疫机制在一定程度上参与了生长抑素对胰岛β细胞的保护。

We studied the anti-effects of the OLFHRJ to the radiation damage in the subcellular degree and compared the anti-effects of the OLFHRJ to the radiation damage with the vitamine E. The results showed that the level of the liquid peroxide in the hepetic mitochondria in the OLFHRJ group mice were significantly lower than the vitamine E group mice on the third day after the whole body exposure with 8.5 Gy of 60Co r-ray (P<0.01) . The activities of Mn-SOD and Cu Zn-SOD in the OLFHRJ group...

We studied the anti-effects of the OLFHRJ to the radiation damage in the subcellular degree and compared the anti-effects of the OLFHRJ to the radiation damage with the vitamine E. The results showed that the level of the liquid peroxide in the hepetic mitochondria in the OLFHRJ group mice were significantly lower than the vitamine E group mice on the third day after the whole body exposure with 8.5 Gy of 60Co r-ray (P<0.01) . The activities of Mn-SOD and Cu Zn-SOD in the OLFHRJ group mice both were significantly higher than the radiation damage group mice (P<0.01). But their activities weren't different from the vitamine E group mice (P>0.05).The activity of GSH-Px in the OLFHRJ group mice were significantly higher than the radiation damage group mice and the vitamine E group mice ( P<0.01).

从亚细胞水平探讨了沙棘蜂王浆口服液抗辐射损伤的作用,从抗自由基损伤角度阐述了其作用机制,并与维生素E抗辐射作用进行了对比性研究.结果显示小鼠经8.5Gyγ射线一次性全身照射后,本口服液小组鼠肝线粒体脂质过氧化物含量明显低于辐射损伤组(P<0.01)和维生素E对照组(P<0.01)F Mn-SOD和CuZn-SOD活力均明显高于辐射损伤组(P<0.01),而与维生素E组无显著差别(P<0.05);GSH-P_x活力明高显于辐射损伤组和维生素E组(P<0.01).

 
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