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all the rats
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  各组大鼠
     All the rats received a intraplantar injection of 0.1 ml of 5% formalin solution and those in GroupⅡ-Ⅴ respectively received a intraperitoneal injection of 2 ml of normal saline solution and 2 ml of 2 mg/kg,4 mg/kg and 8 mg/kg lornoxicam solution 25 min prior to injection of formalin solution.
     各组大鼠均在右后脚趾部皮下注射5%福尔马林0.1 ml,其中Ⅱ~Ⅴ组大鼠在注射福尔马林前25 min分别向腹腔内注射生理盐水2 ml、含有2 mg/kg,4 mg/kg和8 mg/kg的氯诺昔康2 ml。
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     RESULTS All the rats were analyzed in the result.
     结果:各组大鼠全部进入结果分析。
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     There was no linear correlativity between the plasma resistin and the rate of decline of blood sugar,blood glucose in all the rats(P>0.05).
     各组血浆抵抗素之间方差分析未显示统计学差异(P>0.05); 各组大鼠血浆抵抗素与降糖率、血糖等无直线相关关系(P>0.05)。
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     ③ Test of learning and memory: All the rats in each group were given learning and memory praxiology test 35 days after operation.
     ③学习记忆测试:各组大鼠于术后第35天进行学习记忆行为学测试。
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     Six weeks later, all the rats were killed and sodium pump α 1 , α 2 , and α 3 subunit were detected in the pituitary with RT PCR method and immunohistochemical assay at mRNA and protein levels, respectively.
     分别应用分子生物学RT PCR及免疫组织化学技术 ,探讨各组大鼠垂体钠泵α1、α2 及α3 亚单位mRNA及蛋白水平基因表达的改变。
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  “all the rats”译为未确定词的双语例句
     Results: All the rats in the groups of pcDNA3. 1 /TCR Vβ8, pcDNA3.1/ TCR Vβ8 + IL-2, and pcDNA3.1/ TCR Vβ8 + CpG+liposome produced special antibody against idiotype antigen in serum. The titer of antibody began to rise in the 4th week and reached the peak in the 6th week .
     结果:pcDNA3.1/TCR Vβ8组、pcDNA3.l/TCR Vβ8+CpG+脂质体组、pcDNA3.1/TCR Vβ8+IL-2组小鼠血清中均产生了特异性抗独特型抗体,抗体滴度在第4周开始增高,第6周时达到高峰。
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     The plasma levels of IL-10,IL-6,TNF-α and amylase were determined in all the rats. Results IL-10 could not be measured in the control group and the plasma levels of IL-6,TNF-α and amylase were 17.5±1.3 pg/ml,71.25±5.75 pg/ml and 885.4±136.3 U/L,respectively.
     结果假手术组大鼠血浆IL-10测不出,IL-6、TNF-α和淀粉酶分别为(17.5±1.3)pg/ml、(71.25±5.75)pg/ml和(885.4±136.3)U/L,AP大鼠血浆IL-6、TNF-α明显升高,与假手术组对比有显著差异(P<0.05);
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     Methods:All the rats used in the experiments were randomized into 4 groups: NS group; TP25mg/kg, TP50mg/kg and TP100mg/kg group.
     方法:40只大鼠随机分成4组:NS组,TP25mg/kg,TP50mg/kg和TP100mg/kg组,用麻醉大鼠在体心脏法测定血流动力学参数。
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     After eight weeks, all the rats were executed and the blood were kept for detecting ALT, AST、 HA、 PC-Ⅲ、 TGF-β、 TNF-α.
     8周后处死大鼠,留取全血检测ALT、AST、HA、PC-Ⅲ、TGF-β、TNF-α;
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     Results All the rats in groupⅠand Ⅳdied from malnutrition on (14.8±1.3) and (9.2±1.6)d respectively.
     结果切除Ⅰ组和移植Ⅰ组大鼠分别于术后(14.8±1.3)d和(9.2±1.6)d死于营养不良。
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  相似匹配句对
     All rats
     ②手术对照组:给大鼠施予与实验组大鼠相同
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     All rats were sacrificed by decapitation.
     断头处死各组大鼠,取出脑组织,进行冰冻切片。
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     V(I) for all f.
     V(I)对所有f.
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     All.
     建议All.
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     All of the rest rats were ovariectomized.
     其余大鼠均予摘除双侧卵巢。
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  all the rats
All the rats except one group of sedentary controls (n = 10) were subjected to 90 minutes of downhill treadmill running.
      
The kidneys of all the rats appeared normal on histological examination and showed no signs of nephrocytomegalia, which has been found in the kidneys of rats given soya protein subjected to severe alkali treatment.
      
Survival of all the rats after the induction of ANP was observed for 24 h.
      
Mucus production was noted in two rats by light microscopy and in all the rats by transmission electron microscopy, and so it was demonstrated that stagnant bile could give rise to this phenomenon in the rat choledochal epithelium.
      
According to the survival time in a pressure chamber simulating an altitude of 11 km all the rats were divided into groups of high resistance, medium resistance, and low resistance to hypoxia.
      
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The LD_(50) in mice was found to be 19.4 g/kg and 0.98±0.075 g/kg respectively for the infusion and crude extract of Chou-wu-tung, given by intravenous injections. The subacute toxicity is negligible when the infusion was crally administered to rats (0.25—2.5 g/kg/day) for 60 days, except that some animals showed quietness, slight fall of systolic pressure and soft stool. No apparent toxic effects were observed. A crude extract and infusion of Chou-wu-tung, when injected intravenously at respective dosages...

The LD_(50) in mice was found to be 19.4 g/kg and 0.98±0.075 g/kg respectively for the infusion and crude extract of Chou-wu-tung, given by intravenous injections. The subacute toxicity is negligible when the infusion was crally administered to rats (0.25—2.5 g/kg/day) for 60 days, except that some animals showed quietness, slight fall of systolic pressure and soft stool. No apparent toxic effects were observed. A crude extract and infusion of Chou-wu-tung, when injected intravenously at respective dosages of 50—100 mg/kg and 150 mg/kg, produced primary and secondary falls of blood pressure on anaesthetized rats and dogs, but with intramuscular injection or oral administration, only the secondary fall of blood pressure was apparent. This hypotensive effect might last for 2—3 hours. Intravenous injection of the decoction to anaesthetized animals produced only primary fall of blood pressure, the infusions of alcohol, ether and ,chloroform, given by the same route, did not lower the blood pressure. Twenty-eight renal hypertensive rats were divided into three groups, the first (11 rats) and the second (7 rats) groups were treated with the infusion at respective dosages of 0.5 g/kg/day and 5 g/kg/day, and the third (10 rats) with the crude extract at 50 mg/kg/day. The drug was given to all the rats for 2 weeks by oral administration. Hypotensive effect was observed in 3—10 days after medication, and became more remarkable in the second week of the therapeutic period or the first week after cessation of medication; the blood pressure dropped 57.4%. The blood pressure in most rats returned in 2 weeks, and in some rats in 2—4 days or 4 weeks after the stopping of medication.

1.本文就臭梧桐的一般性质作了实验性研究,结果证明其降血压成分易溶于水,难溶或不溶于乙醚、乙醇和氯仿,对热稳定,在碱性溶液中可被氯化钙沉淀出来。臭梧桐降血压效果可因产地而不同。开花前的和新鲜的臭梧桐降血压作用分别较开花后的和经长时间保存的要强。 2.臭梧桐毒性甚小,其热浸剂和提出物给小鼠静脉注射时半数致死量分别为19.4克/公斤和0.98±0.075克/公斤。给大鼠每天用热浸剂(0.25—2.5克/公斤)灌胃经60天,除少数动物出现安静、轻度收缩压下降和大便变稀外,未发现其他毒性反应。 3.臭梧桐提出物(50—100毫克/公斤)和热浸剂(150毫克/公斤)给麻醉大鼠和狗静脉注射时,可引起两度血压下降,但肌肉注射或经口给药,仅引起第二度降血压作用,其作用可维持2—3小时。静脉注射煎剂(麻醉大鼠和狗实验)仅出现第一度降血压作用,经口给药时无效。乙醚、乙醇和氯仿的浸出液不论静脉注射或经口给药,均不引起麻醉动物的血压下降。给肾型高血压大鼠每天经口投予臭梧桐热浸剂(0.5—5克/公斤)和提出物(50毫克/公斤)时,给药的第3—10天卽口出现血压下降,在给药的第二周和停药后的第一周,血压下降最明显,最大降血压作用可达原值...

1.本文就臭梧桐的一般性质作了实验性研究,结果证明其降血压成分易溶于水,难溶或不溶于乙醚、乙醇和氯仿,对热稳定,在碱性溶液中可被氯化钙沉淀出来。臭梧桐降血压效果可因产地而不同。开花前的和新鲜的臭梧桐降血压作用分别较开花后的和经长时间保存的要强。 2.臭梧桐毒性甚小,其热浸剂和提出物给小鼠静脉注射时半数致死量分别为19.4克/公斤和0.98±0.075克/公斤。给大鼠每天用热浸剂(0.25—2.5克/公斤)灌胃经60天,除少数动物出现安静、轻度收缩压下降和大便变稀外,未发现其他毒性反应。 3.臭梧桐提出物(50—100毫克/公斤)和热浸剂(150毫克/公斤)给麻醉大鼠和狗静脉注射时,可引起两度血压下降,但肌肉注射或经口给药,仅引起第二度降血压作用,其作用可维持2—3小时。静脉注射煎剂(麻醉大鼠和狗实验)仅出现第一度降血压作用,经口给药时无效。乙醚、乙醇和氯仿的浸出液不论静脉注射或经口给药,均不引起麻醉动物的血压下降。给肾型高血压大鼠每天经口投予臭梧桐热浸剂(0.5—5克/公斤)和提出物(50毫克/公斤)时,给药的第3—10天卽口出现血压下降,在给药的第二周和停药后的第一周,血压下降最明显,最大降血压作用可达原值的57.4%。多数高血压大鼠的血压在停药后的第二周恢复,少数在停药的2—4天或4周后恢复。

The LD_(50) in mice was found to be 38.5g/kg for the infusion of Kwang-Ti-lung, given by intravenous injection. The infusion has been orally administered to rats (0.1g/kg/day) for 45 days without any toxic effect. The infusion and extract with 70% alcohol by intravenous injections to anaesthetized dogs (0.1g/kg) or by oral administrations to normal rats (10g/kg) produced a fall of blood pressure. This hypotensive effect might last for 2—3 hours. Ten renal hypertensive rats were treated with the extract at a...

The LD_(50) in mice was found to be 38.5g/kg for the infusion of Kwang-Ti-lung, given by intravenous injection. The infusion has been orally administered to rats (0.1g/kg/day) for 45 days without any toxic effect. The infusion and extract with 70% alcohol by intravenous injections to anaesthetized dogs (0.1g/kg) or by oral administrations to normal rats (10g/kg) produced a fall of blood pressure. This hypotensive effect might last for 2—3 hours. Ten renal hypertensive rats were treated with the extract at a dosage of 0.1 g/kg/day. The drug was given to all the rats for 2 weeks by oral administration. Hypotensire effect in most rats was observed in 3—7 days after medication, and became more remarkable in the second week of the therapeutic period or the first week after cessation of medication. The blood pressure in most rats returned in 2 weeks, and some rats in 4—5 weeks after the stopping of medication. The fall of blood pressure produced by Kwang-Ti-lung was neither due to the effect of the vagus, nor due to the direct dilatation of the blood vessels and the blocking of the ganglia of the vegetative nerves. The mechanism of its hypotensive effect is thought to be chiefly due to the dilatation of splanchnic vessels produced by the direct or reflex effect of Kwang-Ti-lung on the central nervous system above spinal cord.

本文对广地龙毒性、降压作用和降压机制作了初步探討。广地龙热浸剂给小鼠靜脉注射时,LD_(50)=38.5克/公斤。热浸剂(0.1克/公斤)連續給大鼠灌胃45天,未发現毒性反应。 給麻醉狗按0.1克/公斤靜脉注射热浸剂或乙醇浸出液,在給药30—45分钟出現血压下降,一般可維持2—3小时。正常大鼠一次用大剂量(10克/公斤)灌胃或腎型高血压大鼠用个剂量(50毫克/公斤)长时間(2周)灌胃,均有明显的降血压作用。在后一种情况下,多数大鼠血压下降出現于給药的第3—7天,并于停药后第2周回升到用药前水平。广地龙降压机制可能是由于它直接作用于脊髓以上的中枢神經系統或通过某些內感受器反射地影响中枢神經系統,引起部分內脏血管的扩张而使血压下降。

Fifty-one male rats (body weights:200—350gm) were raised with laboratory diet, 39 of them were injected once a week with CCl_4 (0.015cc/100gm body weight) in li- quid paraffin through the mesenteric veins and 12 rats without treatment as controls. Rats with 1,2,3,and 4 injections were killed 7 days afterwards.Pieces of liver tissue were taken from the left anterior lobe of the rats from both experimental and control groups.Paraffin sections were prepared from materials fixed in:(1)Susa or Stieve's for Heidenhain's...

Fifty-one male rats (body weights:200—350gm) were raised with laboratory diet, 39 of them were injected once a week with CCl_4 (0.015cc/100gm body weight) in li- quid paraffin through the mesenteric veins and 12 rats without treatment as controls. Rats with 1,2,3,and 4 injections were killed 7 days afterwards.Pieces of liver tissue were taken from the left anterior lobe of the rats from both experimental and control groups.Paraffin sections were prepared from materials fixed in:(1)Susa or Stieve's for Heidenhain's Azan staining;(2)Regaud's for Bensley's acid fuchsin-methyl green staining of mitochondria;(3)cold Carnoy's for methyl green-pyronin staining of ribo- nucleic acid (RNA),with control sections extracted with 20% perchloric acid or 0.17 M NaCl at 56℃ for 2 hrs.;(4)Carnoy's for Yakovlev's ((?)) and Barrnett- Seligman's methods for protein-bound SH-group,with control sections blocked with 0.1M monoiodoacetic acid;(5)Formol-alcohol for Hotchkiss's periodic acid-Schiff's (PAS) reaction of glycogen,with control sections treated with salivary digestion;(6)cold ace- tone for alkaline phosphatase (ALP);and (7) 10% neutral formalin for frozen sections and Sudan Ⅲ testing for fats. After CCl_4-intoxication,during the development of cirrhosis,most of the parenchymal cells degenerated and finally became necrotic.Free fat droplets and hyperplastic fibrous tissues accumulated in the necrotic regions.The liver lobules thus were destroyed and blood sinuses were engorged with blood cells.Surviving cells showed compensative hyperplasia and were isolated into nodules by the developing fibrous tissues.Cirrhosis developed in all the rats treated with CCl_4. The parenchymal cells showed a progressive changes during the development of cirrhosis.There was an increase of fat droplets and a decrease of the number of mito- chondria in the cytoplasm.The mitochondria fragmented into granules or fused in a mass.There was a progressive decline in the content of RNA and glycogen,but an increase of the ALP activity.Those cells showing compensative hyperplasia enlarged in size,and their number of mitochondria,RNA and glycogen content progressively in- creased. Small-sized cells appeared in the necrotic regions among the fat droplets,necrotic cells and fibrous tissues.They decreased in number as the fibrous tissue developed. These cells contained mitochondria and showed positive reactions of PAS,RNA and ALP.Their origin was discussed and their relation to the formation of collagen fibers needs further investigation.

大白鼠肝在CCl_4中毒所引起肝硬化过程中,肝小叶结构被破坏。肝细胞中毒,坏死,脂肪游离增多。血窦因血流受阻而涨大。大部分肝细胞退变或坏死;部分残存而代偿性增生。结缔组织在坏变部分增生并包围肝小结而形成肝硬化。在肝硬化过程中,退变的肝细胞内脂滴增多。线粒体减少,成粒状或溶成一团。核糖核酸,糖元含量也随病变进展而减少。碱性磷酸酶反应增强。代偿性增生的肝细胞体积增大;肝细胞内线粒体增多,核糖核酸,糖元含量逐渐增多。在病变过程中出现冼多小型细胞。小型细胞出现在坏变的肝细胞区域,脂肪空泡附近及结缔组织内并随纤维的增加而减少。小型细胞含线粒体,核糖核酸,PAS反应及碱性磷酸酶反应均为阳性。文中讨论了肝细胞内细胞学和组织化学变化的意义;小型细胞的来源,与纤维形成的关系及纤维增生机制等问题。

 
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