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   chronic hyperglycemia 在 内分泌腺及全身性疾病 分类中 的翻译结果: 查询用时:0.129秒
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chronic hyperglycemia
相关语句
  长期高血糖
    Effect of chronic hyperglycemia on islet β cells function in type 2 diabetes mellitus with secondary sulfonylureas failure
    长期高血糖对继发磺脲类药物失效患者胰岛β细胞功能的影响
短句来源
    Conclusion Chronic hyperglycemia can induce increased LDL oxidation. Ox LDL may promote the development of carotid atherosclerosis and cerebral infarction.
    结论 长期高血糖可促进低密度脂蛋白 (LDL)的氧化修饰 ,而Ox LDL可加速颈动脉粥样硬化 ,促进脑梗死的发生
短句来源
    The occurrence of erectile dysfunction is closely correlated with diabetic duration and chronic hyperglycemia in male patients with type 2 diabetes,so control of blood glucose is an important foundational treatment for them.
    结论:2型糖尿病患者勃起功能障碍的发生与病程、长期高血糖密切相关。 对男性2型糖尿病合并ED的患者,控制血糖为重要的基础治疗措施,在治疗方面应为以控制血糖为主的综合治疗。
短句来源
  “chronic hyperglycemia”译为未确定词的双语例句
    OBJECTIVESDiabetes mellitus (DM) is a complex metabolic disorder characterized with chronic hyperglycemia, type 2 diabetes (T2DM) is resulted from both genetic and environmental risk factors such as physical inactivity, unhealthy dietary habits and obesity.
    糖尿病(Diabetes Mellitus,DM)是由多种病因引起以慢性高血糖为特征的代谢紊乱,2型糖尿病(Type 2 Diabetes Mellitus,T2DM)是由遗传和环境因素如缺乏体力活动、不健康的饮食习惯、肥胖等共同作用的多因素遗传性疾病,占糖尿病的90%-95%。 近年来T2DM在全世界广泛流行,已成为全球性的公共卫生问题。
短句来源
    The results showed that chronic hyperglycemia could decrease the protein expressions of GLUT 1 and GLUT3.
    结果发现慢性高血糖可引起脑内GLUT1和GLUT3蛋白表达降调节。
短句来源
    Diabetes Mellitus (DM), a multi-etiopathogenisis metabolic disease, characterized with chronic hyperglycemia and metabolic disorder of sugar, fat and protein induced by the malfunction of insulin secretion or utilization. It is a worldwide serious health problem and can cause severe complications including kidney failure, acroisa, leg and foot damage, heart attack and stoke.
    糖尿病(DM,Diabetes,Mellitus)是一种多病因的代谢疾病,特点是慢性高血糖,伴随因胰岛素分泌及/或作用缺陷引起的糖、脂肪和蛋白质代谢紊乱,可引起严重的并发症,包括肾衰竭、失明、需要截肢的严重腿足损伤,心脏病发作与中风,是目前世界范围内严重的健康问题。
短句来源
    Age, chronic hyperglycemia exposure, 24-h UAE and the duration of DM are independent risk factors for DPN, That DPN is associated with diabetic microangiopathy (DN and DR ),which suggested microangiopathy is involved in the development of DPN.
    考虑年龄、身高等对参考值的影响后,明确高血糖,病程,24小时尿白蛋白是DPN发生的独立危险因素。 DR、DN与DPN密切相关提示糖尿病微血管病变参与DPN的发生。
短句来源
    Pathophysiology characteristics of T2DM are insulin resistance of liver, adipose tissue ,skeletal muscle and decreased secretion of islet βcell( namely β cell’s function abnormality). Comprehensive action of these abnormal factors causes chronic hyperglycemia and serious chronic complications.
    2型糖尿病的病理生理学特征是肝脏、脂肪组织和骨骼肌对胰岛素的抵抗,以及胰岛β细胞分泌胰岛素减少(即β细胞功能异常),这些异常因素的综合作用导致慢性高血糖和严重的慢性并发症。
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  chronic hyperglycemia
The effect of diabetes in rats on lipid composition and order of synaptosomal membranes (SM) was determined in streptozotocin-induced diabetic rats after 6 weeks of chronic hyperglycemia.
      
These data suggest that down-regulation of facilitated glucose transport into the brain does not occur during chronic hyperglycemia.
      
Acute hyperglycemia was produced by intraperitoneal injection of glucose, and chronic hyperglycemia was produced by treatment with streptozotocin.
      
Because of this discrepancy the brain capillary permeability-surface area product (PA) was measured in awake-restrained rats during acute and chronic hyperglycemia.
      
However, available measurements of brain glucose content during chronic hyperglycemia are consistent with an unaltered transport system.
      
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The thiobarbituric acid (TBA) assay was used to determine non-enzymatic glycosylation of hair and fingernail keratin in non-diabetics and diabetics. Some measuring conditions including the amount of sample, the duration of hydrolysis, etc were described. The concentration of glycosylated hair protein (GHP) and glycosylated nail protein (GNP) in diabetics were both significantly higher than that in non-diabetics. GHP and GNP might be used as a monitor marker in retrospecfive assessment of chronic hyperglycemia....

The thiobarbituric acid (TBA) assay was used to determine non-enzymatic glycosylation of hair and fingernail keratin in non-diabetics and diabetics. Some measuring conditions including the amount of sample, the duration of hydrolysis, etc were described. The concentration of glycosylated hair protein (GHP) and glycosylated nail protein (GNP) in diabetics were both significantly higher than that in non-diabetics. GHP and GNP might be used as a monitor marker in retrospecfive assessment of chronic hyperglycemia.

本文用TBA显色法对头发及指甲糖化角蛋白(GHP,GNP)测定的水解时间、 标本用量等条件进行了探索,确定了较为合理的实验条件。正常人与糖尿病病人GHP测定批内CV分别为1.61%和1.98%,批间分别为1.83%和1.11%。GNP批内CV分别为3.19%和6.72%。GHP与GHb相关为r=0.66(P<0.01)。TBA法测定GHP和GNP方法简便,可作为糖尿病病人血糖控制情况的一回顾性观察指标。

The study was to discuss the changes of erythrocyte membrane ATPase activities.Methods Erythrocyte membrane Na+-K+-ATPase and Ca2+-ATPase activities were determined by modified Hanahan's and Luthra's methods tespectively(CV 7.2%and 3.8%,respectively)in17 IDDM and 48 NIDDM patients,compared with correspondent controls.t test and linear relationship assay were used as statistical method.Results Erythrocyte 5.membrane Na+-K+-ATPase and Ca2+-ATPase activities decreased 24.33%and 17.ll%in IDDM patients respectively,2l.24%and...

The study was to discuss the changes of erythrocyte membrane ATPase activities.Methods Erythrocyte membrane Na+-K+-ATPase and Ca2+-ATPase activities were determined by modified Hanahan's and Luthra's methods tespectively(CV 7.2%and 3.8%,respectively)in17 IDDM and 48 NIDDM patients,compared with correspondent controls.t test and linear relationship assay were used as statistical method.Results Erythrocyte 5.membrane Na+-K+-ATPase and Ca2+-ATPase activities decreased 24.33%and 17.ll%in IDDM patients respectively,2l.24%and l6.42%in NIDDM patients respectively.Two ATPase activities vvere negatively correlated with HbA1c and were not correlated with the age, free blood glucose or serum insulin in IDDM and NIDDM patients,Conclusion Erythrocyte membrane Na+-K+-ATPase and Ca2+-ATPase activities were decreased in both IDDM and NIDDM patients and were probably due to chronic hyperglycemia.

目的探讨糖尿病患者红细胞膜ATPase活性的变化。方法17例胰岛素依赖型(IDDM)和48例非胰岛素依赖型糖尿病(NIDDM)患者红细胞膜Na~+-K~+-ATPase、Ca~(2+)-ATPase活性分别用改良的Hanahan法和Luthra法测定,并设相应年龄对照组。统计方法用t检验和直线相关分析。结果NIDDM患者红细胞膜Na~+-K~+-ATPase、Ca~(2+)-ATPase活性分别下降了24.33%和17.11%,NIDDM患者则分别下降21.24%和16.42%。两组糖尿病患者ATPase活性均与糖化血红蛋白呈负相关,与年龄、空腹血糖或胰岛素水平无相关。结论糖尿病患者均有红细胞膜Na~+-K~+-ATPase、Ca~(2+)-ATPase活性下降,其原因与慢性高血糖有关。

Objective To investigate the effect of the exercise on glucose transporter 4 (GLUT4) gene expression in skeletal muscles of rats with streptozotocin induced diabetes.Methods Three experiment groups of rats were investigated: diabetic non exercise group; diabetic exercise group; and normal controls. Diabetic exercise rats were swim trained for 6 weeks. The GULT4 mRNA of skeletal muscle cells was determined with dot blot.Results There was weak gene expression of GULT4 in diabetic rats, and dot blot revealed...

Objective To investigate the effect of the exercise on glucose transporter 4 (GLUT4) gene expression in skeletal muscles of rats with streptozotocin induced diabetes.Methods Three experiment groups of rats were investigated: diabetic non exercise group; diabetic exercise group; and normal controls. Diabetic exercise rats were swim trained for 6 weeks. The GULT4 mRNA of skeletal muscle cells was determined with dot blot.Results There was weak gene expression of GULT4 in diabetic rats, and dot blot revealed that the GLUT4 mRNA decrease 54.9% in skeletal muscle cells compared with the normal control. After 6 weeks exercise training, the hyperglycemia of diabetic exercise rats decreased significantly (from 18.5±1.9 mmol/L to 14.0±3.3 mmol/L, t =4.64 P <0.01). In muscle cells of diabetic exercise rats, the GLUT4 mRNA increased 56% as compared with the diabetic rats ( t =15.56, P < 0.01). Conclusion Chronic hyperglycemia may inhibit the gene expression of GLUT4 in diabetic rats, which caused the post receptor insulin resistance in peripheral tissue. Exercise training can improve the impaired GLUT4 expression in diabetic rats, which may contribute to the mechanisms of enhanced insulin sensitivity and decreased hyperglycemia in diabetes mellitus.

目的研究运动对链脲佐菌素引起的糖尿病大鼠骨骼肌细胞内葡萄糖运载体4(glucosetrans-porter,GLUT4)基因表达的作用。方法实验大鼠分为3组:(1)糖尿病非运动组;(2)糖尿病运动组;(3)正常对照组。每组6只大鼠。糖尿病运动组大鼠进行6周游泳训练。用斑点杂交法检测3组大鼠骨骼肌细胞内GLUT4mRNA含量。结果糖尿病大鼠骨骼肌细胞内GLUT4基因表达明显降低,GLUT4mRNA含量与正常对照组相比减少54.9%。糖尿病运动组大鼠经过6周运动训练,血糖由18.5±1.9mmol/L降至14.0±3.3mmol/L(t=4.64,P<0.01),骨骼肌细胞内GLUT4mRNA含量与糖尿病大鼠相比增加56%(t=15.56,P<0.01)。结论在糖尿病高血糖状态下,大鼠骨骼肌细胞内的GLUT4基因表达受到抑制,这可能是胰岛素抵抗受体后缺陷的重要原因。运动训练能够促进糖尿病大鼠受抑的GLUT4基因表达的部分恢复,这可能是运动增加外周组织对胰岛素的敏感性,降低血糖的机制之一。

 
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