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   grey values 在 呼吸系统疾病 分类中 的翻译结果: 查询用时:0.193秒
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呼吸系统疾病
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grey values
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  灰度值
    Results ①Immunohistochemistry results showed that there was no obvious difference for the mean grey values(0-black to 255-white) of MMP-2 positive products in the airway epithelia and the pulmonary inflammatory cells between normal saline control group(185.60±4.81,161.47±5.71) and simply sensitized group(184.80±9.64,160.33±4.03)(P>0.05).
    结果①免疫组织化学结果显示:豚鼠气道上皮和肺内炎性细胞的MMP-2阳性免疫反应产物的平均灰度值(0-深色,255-浅色),生理盐水组(185.60±4.81、161.47±5.71)与单纯致敏组(184.80±9.64、160.33±4.03)比较没有显著差异(P>0.05);
短句来源
    Immunohistochemistry results showed that [The intensity of staining reactivity, determined by means of an image analysis system, was expressed as grey value (0-black to 255-white)]: there was no obvious difference in the mean grey values of IL-1β positive products between normal saline control group and simply sensitized group (P>0.05).
    免疫组织化学结果显示(染色反应强度用灰度值表示):生理盐水组与单纯致敏组豚鼠IL-1β阳性免疫反应产物的平均灰度值没有显著差异(P>0.05);
短句来源
    The mean grey values of IL-1β positive products decreased significantly in the lower respiratory tract, nerve terminals in vascular smooth muscle, C_7-T_5 spinal ganglia and corresponding spinal dorsal horn of the asthmatic guinea pigs compared with the normal saline control group and the simply sensitized group (P<0.01).
    与这两个对照组相比,哮喘豚鼠气道上皮、肺内炎性细胞、血管平滑肌内的神经末梢、C7~T5段脊神经节及相应节段脊髓背角内IL-1β阳性反应产物的平均灰度值均明显降低(P<0.01);
短句来源
    However, the mean grey values of IL-1β positive products in the lower respiratory tract, nerve terminals in vascular smooth muscle, C_7-T_5 spinal ganglia and corresponding spinal dorsal horn was much higher in NGF antibody group (inhalation of NGF antibody into nasal cavity) than in asthmatic group (P<0.01).
    在哮喘+NGF抗体组(鼻腔吸入NGF抗体),上述部位内IL-1β阳性反应产物的平均灰度值均明显高于哮喘组(P<0.01)。
短句来源
    Results:The mean grey values of TrkA positive products decreased significantly in the lower respiratory tract,nerve terminals in vascular smooth muscle,C_7-T_5 spinal ganglia and corresponding spinal dorsal horn of the asthmatic guinea pigs compared with the normal saline control group and the simple sensitized group.
    结果:(1)与对照组相比,哮喘豚鼠气道上皮、肺内炎性细胞、血管平滑肌内的神经末梢、C_7~T_5段脊神经节及相应节段脊髓背角内 TrkA 阳性反应产物的平均灰度值均明显降低;
短句来源
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  “grey values”译为未确定词的双语例句
    The mean grey values of MMP-2 positive products decreased significantly in the lower respiratory tract(141.93±4.66,129.77±4.07)of the asthmatic guinea pigs compared with the normal saline control group and the simply sensitized group(P<0.01).
    哮喘组(141.93±4.66、129.77±4.07)明显低于生理盐水组和单纯致敏组(P<0.01);
短句来源
    However,the mean grey values were much higher in NGF antibody group(inhalation of NGF antibody into nasal cavity,179.73±6.11,150.23±8.13) than inasthmatic group(P<0.01).
    哮喘+NGF抗体组(鼻腔吸入NGF抗体,179.73±6.11、150.23±8.13)则明显高于哮喘组(P<0.01)。
短句来源
    The mean grey values of MMP-2 positive products decreased significantly in the C_(7)-T_(5) spinal ganglia(139.87±4.88)and the corresponding spinal dorsal horn(125.93±4.49)of the asthmatic guinea pigs compared with the normal saline control group and the simply sensitized group(P<0.01).
    哮喘组(139.87±4.88、125.93±4.49)明显低于生理盐水组和单纯致敏组(P<0.01);
短句来源
    However,the mean grey values were much higher in NGF antibody group(inhalation of NGF antibody into nasal cavity,169.73±6.24、152.37±8.67) than in asthmatic group(P<0.01).
    哮喘+NGF抗体组(鼻腔吸入NGF抗体,169.73±6.24、152.37±8.67)则明显高于哮喘组(P<0.01)。
短句来源
    However,the mean grey values were much higher in NGF antibody group than those in asthmatic group.
    哮喘+NGF 抗体组则明显高于哮喘组。
短句来源
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  grey values
There are often position dependent trends in the grey values of a region, that is; the image is piecewise smooth.
      
Multibeam sidescan data were recorded along with depth measurements and show reflectivity variations from light grey values at the centre of the H?kon Mosby Mud Volcano to dark grey values (less reflective) at the surrounding moat.
      
These are characterised by different colours or grey values.
      
However, in many confocal microscopy volumetric images of neurons, the grey values of the object and/or background are usually uneven.
      
The objective of this paper is to develop a novel digital subtraction technique for serial intra-oral radiography, which would allow the detection of subtle variations in grey values.
      
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Objective To understand the interaction between surfactant proteins and pneumocystis carinii pneumonia (PCP), and the impact of corticosteriods on surfactant proteins、 Methods We established rat models of PCP and bacterial pneumonia induced by subcutaneous injection of 25mg cortisone acetate、 At 8-12?wk, the bronchoalveolar lavage fluid (BALF) of rats was collected、 Total nucleated cells of BALF were counted and differentiated, and the concentrations of surfactant protein A (SP-A) and surfactant protein D...

Objective To understand the interaction between surfactant proteins and pneumocystis carinii pneumonia (PCP), and the impact of corticosteriods on surfactant proteins、 Methods We established rat models of PCP and bacterial pneumonia induced by subcutaneous injection of 25mg cortisone acetate、 At 8-12?wk, the bronchoalveolar lavage fluid (BALF) of rats was collected、 Total nucleated cells of BALF were counted and differentiated, and the concentrations of surfactant protein A (SP-A) and surfactant protein D (SP-D) were measured by immunoblotting assay、 The rats were divided into three immunosuppressive groups and a normal control group、 Group Ⅰ, normal control (n=6), consisted of healthy SD rats; group Ⅱ, negative control (n=6), consisted of rats with cortisone acetate injection for over 8 wk without lung infection; group Ⅲ, bacterial pneumonia (n=11), rats were injected with cortisone acetate over 8 wk that resulted in bacterial pneumonia without other pathogens isolated; and group Ⅳ, PCP (n=14), rats with injected cortisone acetate for 8-12?wk and developed PCP without other pathogens isolated、 Results Our results indicated that the total cell count in BALF in the negative control group was lower than that in the normal control group (P<0、001)、 During PCP infection, the total cell count and the percentage of polymorphonuclearcytes (PMNs) in BALF were significantly increased (P<0、01), but were lower than those in the bacterial pneumonia group、 The concentration of SP-A of BALF in PCP (45、1±22、1?μg/ml) was significantly increased in comparison with that in the negative control (16、2±9、9?μg/ml, P<0、05) and bacterial pneumonia groups (6、2±5、6?μg/ml, P<0、001)、 We also found that the relative content of SP?-?D was significantly higher in PCP (24?249±4780 grey values) than that in the negative control (13?384±2887 grey values, P<0、001) and that in bacterial pneumonia (11?989 grey values, P<0、001)、 SP-A and SP-D were also higher in the moderate to heavy group of PCP than those seen in the mild group (P<0、01, P<0、001)、 SP-A and SP-D were higher in the negative control group than those in the normal control group, but there was no significant difference between the 2 groups、 Conclusion These results suggest that the concentrations of SP-A and SP-D in BALF are increased by pneumocystis carinii specific stimulation, but the alteration is not related to the corticosteriod usage、

目的 为进一步明确表面活性蛋白与卡氏肺孢子虫肺炎 (PCP)的关系及肾上腺皮质激素对表面活性蛋白的影响。方法 通过每周两次皮下注射 2 5mg醋酸可的松 8- 12周建立PCP及细菌性肺炎大模型。收集支气管肺泡灌洗液 (BALF) ,计数BALF中有核细胞数并分类 ,用免疫印迹法检测表面活性蛋白A(SP A)及表面活性蛋白D(SP D)。将大分为免疫抑制组 (共三组 )及正常对照组。Ⅰ组 :正常对照 (n =6 ) ,为健康SD大 ;Ⅱ组 :阴性对照 (n =6 ) ,为注射醋酸可的松 8周以上无肺部感染的大 ;Ⅲ组 :细菌性肺炎 (n =11) ,为注射醋酸可的松 8周以上出现细菌性肺炎而未分离到其它病原体的大 ;Ⅳ组 :PCP(n =14 ) ,为注射醋酸可的松 8- 12周出现PCP而未分离到其它病原体的大。结果 阴性对照组BALF中细胞总数低于正常对照组 (P <0 0 0 1)。PCP组BALF中细胞总数及多形核细胞(PMNs)百分数高于阴性对照组 (P <0 0 1) ,但低于细菌性肺炎组。PCP组BALF中SP A浓度 (45 1± 2 2 1μg/ml)显著高于阴性对照组 (16 2...

目的 为进一步明确表面活性蛋白与卡氏肺孢子虫肺炎 (PCP)的关系及肾上腺皮质激素对表面活性蛋白的影响。方法 通过每周两次皮下注射 2 5mg醋酸可的松 8- 12周建立PCP及细菌性肺炎大模型。收集支气管肺泡灌洗液 (BALF) ,计数BALF中有核细胞数并分类 ,用免疫印迹法检测表面活性蛋白A(SP A)及表面活性蛋白D(SP D)。将大分为免疫抑制组 (共三组 )及正常对照组。Ⅰ组 :正常对照 (n =6 ) ,为健康SD大 ;Ⅱ组 :阴性对照 (n =6 ) ,为注射醋酸可的松 8周以上无肺部感染的大 ;Ⅲ组 :细菌性肺炎 (n =11) ,为注射醋酸可的松 8周以上出现细菌性肺炎而未分离到其它病原体的大 ;Ⅳ组 :PCP(n =14 ) ,为注射醋酸可的松 8- 12周出现PCP而未分离到其它病原体的大。结果 阴性对照组BALF中细胞总数低于正常对照组 (P <0 0 0 1)。PCP组BALF中细胞总数及多形核细胞(PMNs)百分数高于阴性对照组 (P <0 0 1) ,但低于细菌性肺炎组。PCP组BALF中SP A浓度 (45 1± 2 2 1μg/ml)显著高于阴性对照组 (16 2± 9 9μg/ml,P <0 0 5 )及细菌性肺炎组 (6 2± 5 6 μg/ml,P <0 0 0 1)。我们也发现PCP组中SP D相对含量 (2 4 2 4 9± 4 780灰度值 )显著高于阴性对照组 (13384± 2 887,P <0 0 0 1)及细菌性肺炎组(11989± 2 75 0灰?

Objective To explore the expression of matrix metalloproteinase-2(MMP-2) and the regulatory effect of nerve growth factor(NGF) in the lower respiratory tract of the asthmatic guinea pig.Methods The changes of MMP-2 immunoreactivity were investigated by means of immunohistochemistry in lower respiratory tract of the guinea pigs in different groups.The expressions of NGF and MMP-2 were observed by Western blot in the lung of all the groups.The results were analyzed by the Luzex-F real time image analysis system...

Objective To explore the expression of matrix metalloproteinase-2(MMP-2) and the regulatory effect of nerve growth factor(NGF) in the lower respiratory tract of the asthmatic guinea pig.Methods The changes of MMP-2 immunoreactivity were investigated by means of immunohistochemistry in lower respiratory tract of the guinea pigs in different groups.The expressions of NGF and MMP-2 were observed by Western blot in the lung of all the groups.The results were analyzed by the Luzex-F real time image analysis system and the gel imaging analysis system respectively.Results ①Immunohistochemistry results showed that there was no obvious difference for the mean grey values(0-black to 255-white) of MMP-2 positive products in the airway epithelia and the pulmonary inflammatory cells between normal saline control group(185.60±4.81,161.47±5.71) and simply sensitized group(184.80±9.64,160.33±4.03)(P>0.05).The mean grey values of MMP-2 positive products decreased significantly in the lower respiratory tract(141.93±4.66,129.77±4.07)of the asthmatic guinea pigs compared with the normal saline control group and the simply sensitized group(P<0.01).However,the mean grey values were much higher in NGF antibody group(inhalation of NGF antibody into nasal cavity,179.73±6.11,150.23±8.13) than inasthmatic group(P<0.01).②Western blot results showed that: compared with normal saline control group andsimply sensitized group,the ratios between Integrated Density Value(IDV) for MMP-2 or NGF protein band and IDVforβ-actin protein band increased markedly in the lung of asthmatic group(P<0.01),however,the ratios of NGFantibody group reduced markedly in comparison with asthmatic group(P<0.01),indicating that NGF can upregu-late the expression of MMP-2.Conclusions MMP-2 might play a role in airway remodeling in asthma,the overex-pression of MMP-2 induced by NGF might be one of the mechanisms for the involvement of NGF in the pathogenesisof asthma,and inhibitory expression of MMP-2 by NGF antibody in the lower respiratory tract might inhibit airway re-modeling and be a new route for the prevention and treatment of the bronchial asthma.

目的探讨基质金属蛋白酶-2(MMP-2)在哮喘豚鼠下呼吸道的表达及神经生长因子(NGF)对MMP-2的调节作用。方法应用免疫组织化学方法检测各组豚鼠下呼吸道MMP-2免疫反应的变化。用W estern b lot蛋白印迹方法检测各组豚鼠肺组织NGF和MMP-2的表达。用Luzex-F实时图像分析系统和凝胶成像分析系统分别对结果进行图像分析。结果①免疫组织化学结果显示:豚鼠气道上皮和肺内炎性细胞的MMP-2阳性免疫反应产物的平均灰度值(0-深色,255-浅色),生理盐水组(185.60±4.81、161.47±5.71)与单纯致敏组(184.80±9.64、160.33±4.03)比较没有显著差异(P>0.05);哮喘组(141.93±4.66、129.77±4.07)明显低于生理盐水组和单纯致敏组(P<0.01);哮喘+NGF抗体组(鼻腔吸入NGF抗体,179.73±6.11、150.23±8.13)则明显高于哮喘组(P<0.01)。②W estern b lot结果显示:与生理盐水组和单纯致敏组比较,哮喘组豚鼠肺组织MMP-2和NGF阳性产物的IDV(Integrated Density Value)与β-...

目的探讨基质金属蛋白酶-2(MMP-2)在哮喘豚鼠下呼吸道的表达及神经生长因子(NGF)对MMP-2的调节作用。方法应用免疫组织化学方法检测各组豚鼠下呼吸道MMP-2免疫反应的变化。用W estern b lot蛋白印迹方法检测各组豚鼠肺组织NGF和MMP-2的表达。用Luzex-F实时图像分析系统和凝胶成像分析系统分别对结果进行图像分析。结果①免疫组织化学结果显示:豚鼠气道上皮和肺内炎性细胞的MMP-2阳性免疫反应产物的平均灰度值(0-深色,255-浅色),生理盐水组(185.60±4.81、161.47±5.71)与单纯致敏组(184.80±9.64、160.33±4.03)比较没有显著差异(P>0.05);哮喘组(141.93±4.66、129.77±4.07)明显低于生理盐水组和单纯致敏组(P<0.01);哮喘+NGF抗体组(鼻腔吸入NGF抗体,179.73±6.11、150.23±8.13)则明显高于哮喘组(P<0.01)。②W estern b lot结果显示:与生理盐水组和单纯致敏组比较,哮喘组豚鼠肺组织MMP-2和NGF阳性产物的IDV(Integrated Density Value)与β-actin IDV的比值均明显升高(P<0.01),而哮喘+NGF抗体组则明显低于哮喘组(P<0.01),表明NGF可上调MMP-2的表达。结论MMP-2可能是哮喘气道重塑的一种调控因素,NGF诱发MMP-2的表达可能是NGF参与哮喘发病的机制之一,NGF抗体抑制哮喘时下呼吸道MMP-2的表达有可能延缓气道重塑的发生,可能是治疗哮喘的新途径。

To explore the role and neuroimmune mechanism of nerve growth factor (NGF) in the pathogenesis of asthma. The changes of interleukin-1β (IL-1β) immunoreactivity were investigated by means of immunohistochemistry in lower respiratory tract and viscerosensory afferent sites of the guinea pigs among different groups. The expression of NGF and IL-1β were observed respectively by Western blot in the lung, C_7-T_5 spinal ganglia and the corresponding spinal dorsal horn of all the groups. The results were analyzed...

To explore the role and neuroimmune mechanism of nerve growth factor (NGF) in the pathogenesis of asthma. The changes of interleukin-1β (IL-1β) immunoreactivity were investigated by means of immunohistochemistry in lower respiratory tract and viscerosensory afferent sites of the guinea pigs among different groups. The expression of NGF and IL-1β were observed respectively by Western blot in the lung, C_7-T_5 spinal ganglia and the corresponding spinal dorsal horn of all the groups. The results were analyzed by the Luzex-F real time image analysis system and the gel imaging analysis system respectively. Immunohistochemistry results showed that [The intensity of staining reactivity, determined by means of an image analysis system, was expressed as grey value (0-black to 255-white)]: there was no obvious difference in the mean grey values of IL-1β positive products between normal saline control group and simply sensitized group (P>0.05). The mean grey values of IL-1β positive products decreased significantly in the lower respiratory tract, nerve terminals in vascular smooth muscle, C_7-T_5 spinal ganglia and corresponding spinal dorsal horn of the asthmatic guinea pigs compared with the normal saline control group and the simply sensitized group (P<0.01). However, the mean grey values of IL-1β positive products in the lower respiratory tract, nerve terminals in vascular smooth muscle, C_7-T_5 spinal ganglia and corresponding spinal dorsal horn was much higher in NGF antibody group (inhalation of NGF antibody into nasal cavity) than in asthmatic group (P<0.01). Western blot results showed that: compared with normal saline control group and simply sensitized group, the ratios between integrated density value (IDV) for IL-1β or NGF protein band and IDV for β-actin protein band increased markedly in the lung, C_7-T_5 spinal ganglia and the corresponding spinal dorsal horn of asthmatic group (P<0.01), however, the ratios between IDV for IL-1β or NGF protein band and IDV for β-actin protein band of NGF antibody group reduced markedly in comparison with asthmatic group (P<0.01), indicating that NGF can upregulate the expression of IL-1β. The present results indicate that the overexpression of IL-1β induced by NGF might be one of the mechanisms for the involvement of NGF in the pathogenesis of asthma, and inhibitory expression of IL-1β by NGF antibody in the lower respiratory tract and viscerosensory afferent sites might be a new route for the prevention and treatment of the bronchial asthma.

为探讨神经生长因子(NGF)在哮喘发病中的作用及其神经免疫调节机制,本研究应用免疫组织化学方法检测各组豚鼠下呼吸道和内脏感觉传入部位白介素-1β(IL-1β)免疫反应的变化。用Western blot方法分别检测各组豚鼠肺、C7~T5段脊神经节和相应节段脊髓背角NGF和IL-1β的表达。用Luzex-F实时图像分析系统和凝胶成像分析系统分别对结果进行图像分析。免疫组织化学结果显示(染色反应强度用灰度值表示):生理盐水组与单纯致敏组豚鼠IL-1β阳性免疫反应产物的平均灰度值没有显著差异(P>0.05);与这两个对照组相比,哮喘豚鼠气道上皮、肺内炎性细胞、血管平滑肌内的神经末梢、C7~T5段脊神经节及相应节段脊髓背角内IL-1β阳性反应产物的平均灰度值均明显降低(P<0.01);在哮喘+NGF抗体组(鼻腔吸入NGF抗体),上述部位内IL-1β阳性反应产物的平均灰度值均明显高于哮喘组(P<0.01)。Western blot结果显示:与生理盐水组和单纯致敏组比较,哮喘组豚鼠肺、C7~T5段脊神经节及相应节段脊髓样品中IL-1β或NGF目标带的IDV(integrated density value)与内参照IDV的...

为探讨神经生长因子(NGF)在哮喘发病中的作用及其神经免疫调节机制,本研究应用免疫组织化学方法检测各组豚鼠下呼吸道和内脏感觉传入部位白介素-1β(IL-1β)免疫反应的变化。用Western blot方法分别检测各组豚鼠肺、C7~T5段脊神经节和相应节段脊髓背角NGF和IL-1β的表达。用Luzex-F实时图像分析系统和凝胶成像分析系统分别对结果进行图像分析。免疫组织化学结果显示(染色反应强度用灰度值表示):生理盐水组与单纯致敏组豚鼠IL-1β阳性免疫反应产物的平均灰度值没有显著差异(P>0.05);与这两个对照组相比,哮喘豚鼠气道上皮、肺内炎性细胞、血管平滑肌内的神经末梢、C7~T5段脊神经节及相应节段脊髓背角内IL-1β阳性反应产物的平均灰度值均明显降低(P<0.01);在哮喘+NGF抗体组(鼻腔吸入NGF抗体),上述部位内IL-1β阳性反应产物的平均灰度值均明显高于哮喘组(P<0.01)。Western blot结果显示:与生理盐水组和单纯致敏组比较,哮喘组豚鼠肺、C7~T5段脊神经节及相应节段脊髓样品中IL-1β或NGF目标带的IDV(integrated density value)与内参照IDV的比值均明显升高(P<0.01),而哮喘+NGF抗体组上述部位样品中IL-1β或NGF目标带的IDV与内参照IDV的比值均明显低于哮喘组(P<0.01),表明NGF可上调IL-1β的表达。这些结果提示NGF诱发IL-1β的表达可能是NGF参与哮喘发病的机制之一,NGF抗体抑制哮喘时下呼吸道和内脏感觉传入部位IL-1β的表达可能是治疗哮喘的新途径。

 
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