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   grey values 在 内分泌腺及全身性疾病 分类中 的翻译结果: 查询用时:0.067秒
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内分泌腺及全身性疾病
呼吸系统疾病
口腔科学
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grey values
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  灰度值
    Results The average grey values(0~255,black~white) of p38mRNA positive cells in synovium and lung of the arthritis rats were much lower than those in the normal control rats(P<0.01),p38 mRNA positive cells were the macrophage cells,fibroblast cells and lymphocytes in the synovium.
    结果AA大鼠滑膜和肺组织p38mRNA阳性细胞的平均灰度值(0~255,深~浅)明显低于正常大鼠(P<0.01)。 AA大鼠滑膜的巨噬细胞,成纤维细胞和淋巴细胞的胞浆内均有p38mRNA的表达。
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    At 15 d,the grey values of GAP 43 were increased in hypothyroidism (P<0.01),there were no statistical difference between experimental pups and controls (P>0.05) at 30 d.
    图像分析显示 :生后 15 d甲减鼠大脑生长相关蛋白灰度值较正常对照组明显升高 (P <0 .0 1) ; 生后 30 d,其灰度值虽低于正常对照组 ,但经统计学分析差异无显著意义 (P >0 .0 5 )。
短句来源
    The grey values of GAP-43 were decreased in hypothyroidism( P <0.01),there were no statistical difference between excessive iodine pups and control( P >0.05) of CA1 hippocampal formation during critical period of brain development.
    图像分析显示 :甲减鼠海马CA1区GAP - 4 3灰度值与对照组比明显升高 (P <0 0 1) ; 高碘鼠同一脑区其灰度值虽低于正常对照组 ,但经统计学分析无显著性差异 (P >0 0 5 )。
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  grey values
There are often position dependent trends in the grey values of a region, that is; the image is piecewise smooth.
      
Multibeam sidescan data were recorded along with depth measurements and show reflectivity variations from light grey values at the centre of the H?kon Mosby Mud Volcano to dark grey values (less reflective) at the surrounding moat.
      
These are characterised by different colours or grey values.
      
However, in many confocal microscopy volumetric images of neurons, the grey values of the object and/or background are usually uneven.
      
The objective of this paper is to develop a novel digital subtraction technique for serial intra-oral radiography, which would allow the detection of subtle variations in grey values.
      
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In order to study the aging changes of somatostation(SS)neurons in aged rat hippocampus,24 SpragueDawley rats were divided into zinc deficient group(2~3 months old,n=8),normal zinc group((2~3 month old,n=8)and aged group (24~26 month old,n=8).()n the experimental 15th day,we stained the hippocampal SS neurons by immunocytochemic method,and used the quantitive analytical method to observe the changes of the hippocampal SS neurons. The results showed that the number of SS neuron reduced in both groups of aged...

In order to study the aging changes of somatostation(SS)neurons in aged rat hippocampus,24 SpragueDawley rats were divided into zinc deficient group(2~3 months old,n=8),normal zinc group((2~3 month old,n=8)and aged group (24~26 month old,n=8).()n the experimental 15th day,we stained the hippocampal SS neurons by immunocytochemic method,and used the quantitive analytical method to observe the changes of the hippocampal SS neurons. The results showed that the number of SS neuron reduced in both groups of aged rats and zinc deficient rats as compared with the normal zinc group(P<0. 05).The grey values (ABS)of SS neurons decreased also,but the distributive patterns of SS neurons in hippocampus of these groups had no difference. These results suggest that zinc-deficiency in the aged rats may be one of the important reasons causing aged-dependent deterioration of the SS neurons in hippocampus.

为分析衰老大鼠脑内生长抑素(SS)神经元变化的可能原因,实验采用免疫组化结合图像分析技术比较了常锌(2~3月龄)、缺锌(2~3月龄)和老龄(24~26月龄)3组大鼠海马SS神经元的变化。结果表明:缺锌15d时,缺锌组与老年组大鼠海马SS神经元数目减少,免疫反应强度明显减低,但SS神经元的分而模式及胞体形态没有明显改变。说明衰老过程中海马SS神经元的变化可能与体内Zn的增龄性减少有密切关系

Objective To study the expression of GAP 43 in developing cerebral of hypothyroid rats. Methods The model hypothyroidism was established by pre treating with Thiamazole and immunocytochemical method.Image analysis was used to observe changes of GAP 43 in cerebral of both normal and hypothyroid neonatal rats.Results The distribution tendency of GAP 43 in parietal neocortex and hippocampal formation was similar in experimental pups as compared to age matched controls.At 15 d,the grey values of GAP...

Objective To study the expression of GAP 43 in developing cerebral of hypothyroid rats. Methods The model hypothyroidism was established by pre treating with Thiamazole and immunocytochemical method.Image analysis was used to observe changes of GAP 43 in cerebral of both normal and hypothyroid neonatal rats.Results The distribution tendency of GAP 43 in parietal neocortex and hippocampal formation was similar in experimental pups as compared to age matched controls.At 15 d,the grey values of GAP 43 were increased in hypothyroidism (P<0.01),there were no statistical difference between experimental pups and controls (P>0.05) at 30 d.Conclusions Thyroid hormones affects expression of GAP 43 directly or indirectly.By regulating GAP 43,thyroid hormones have an effect on G 0 protein signal transduction system and may lead to brain developmental retardation.

目的 研究甲状腺激素缺乏对新生鼠大脑神经生长相关蛋白表达的影响。方法 选用他巴唑复制的甲状腺功能减退 (甲减 )大鼠动物模型 ,用免疫细胞化学方法结合图像分析对正常及甲减新生鼠大脑内神经生长相关蛋白的动态变化进行研究。结果 甲减新生鼠顶叶皮质及海马结构内生长相关蛋白的分布趋势与正常对照组相似。顶叶皮质表现为分子层染色最深 ,海马结构表现为腔隙层、放射层及始层梯度染色 ,神经元胞体一般不染色。图像分析显示 :生后 15 d甲减鼠大脑生长相关蛋白灰度值较正常对照组明显升高 (P <0 .0 1) ;生后 30 d,其灰度值虽低于正常对照组 ,但经统计学分析差异无显著意义 (P >0 .0 5 )。结论 甲状腺激素直接或间接影响神经生长相关蛋白的表达。甲状腺激素可能通过调控神经生长相关蛋白 ,进而影响 G0 蛋白信号转导系统及第二信号系统 ,调控鼠脑的发育。

ObjectiveTo study the expression of GAP-43 hippocampal formation during brain developmental critical period of both hypothyroid and excessive iodine rats.MethodsThe model of hypothyroid and excessive iodine rats were established by pre-treatment with Thiamazole and Injection of iodine respectively,analysis of thyroid hormones,immunocytochemical method combining with image analysis were used to observe changes of GAP-43 in hippocampal formation of both normal and hypothroid as well as excessive iodine neonatal...

ObjectiveTo study the expression of GAP-43 hippocampal formation during brain developmental critical period of both hypothyroid and excessive iodine rats.MethodsThe model of hypothyroid and excessive iodine rats were established by pre-treatment with Thiamazole and Injection of iodine respectively,analysis of thyroid hormones,immunocytochemical method combining with image analysis were used to observe changes of GAP-43 in hippocampal formation of both normal and hypothroid as well as excessive iodine neonatal rats.ResultsThe distribution tendency of GAP-43 in hippocampal formation was similarity in experimental pups as compared to age-matched controls.The grey values of GAP-43 were decreased in hypothyroidism( P <0.01),there were no statistical difference between excessive iodine pups and control( P >0.05) of CA1 hippocampal formation during critical period of brain development.Conclusion(1)Excessive iodine could lead to thyroid hormones imbalance;(2)GAP-43 may be a molecular that thyroid hormones regulated directly or indirectly.By regulating GAP-43,thyroid hormones had an effect on Go protein signal transduction system,as occured in the wake of the second signal process and may affect the abilities of learning and memory.

目的 研究高碘对甲状腺激素以及甲减、高碘对脑发育临界期大鼠海马组织神经生长相关蛋白表达的影响。方法 分别选用他巴唑及碘化油诱导建立的甲减及高碘大鼠动物模型 ,用免疫细胞化学方法结合图像分析对正常、高碘及甲减大鼠脑发育临界期海马组织神经生长相关蛋白的变化进行定性、定量研究。结果  (1)甲减及高碘脑发育临界期大鼠海马结构内神经生长相关蛋白的分布趋势与正常对照组相似。表现为腔隙层、放射层及始层梯度染色 ,神经元胞体一般不染色。图像分析显示 :甲减鼠海马CA1区GAP - 4 3灰度值与对照组比明显升高 (P <0 0 1) ;高碘鼠同一脑区其灰度值虽低于正常对照组 ,但经统计学分析无显著性差异 (P >0 0 5 )。 (2 )高碘可引起血清甲状腺激素紊乱(T3 ↓、T4↑、T3 /T4↓ )。结论  (1)高碘可引起甲状腺机能紊乱。 (2 )高碘及甲减在脑发育临界期可直接或间接影响神经生长相关蛋白的表达 ,进而影响Go蛋白信号转导系统及第二信号系统 ,导致学习、记忆障碍。

 
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