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hyperglycemia exacerbates
相关语句
  高血糖明显加重
     Conclusion Diabetic hyperglycemia exacerbates postischemic brain damage and results frequently in postischemic seizures.
     结论 糖尿病高血糖明显加重缺血后脑损伤并常引起脑缺血后癫痫发作。
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  相似匹配句对
     Conclusion Diabetic hyperglycemia exacerbates postischemic brain damage and results frequently in postischemic seizures.
     结论 糖尿病高血糖明显加重缺血后脑损伤并常引起脑缺血后癫痫发作。
短句来源
     Urethane-induced hyperglycemia
     乌拉坦诱导的高血糖反应(英文)
短句来源
     Critical illness and hyperglycemia
     危重病人与高血糖(综述)
短句来源
     Disconnection exacerbates PHG and shunt ameliorate it.
     分流术后凋亡改善 ,PHG病变程度缓解。
短句来源
     This exacerbates the situation induced by ischemia.
     而这又使缺血引发的状况更为恶化。
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Objective: To compare the effects of long term experimental stressors on blood glucose level and insulin secretion in the aged and young mice with diabetes prone and to explore the mechanism. Methods: Diabetes prone was induced by streptozotocin (STZ) in 40 1.5 month old mice and 40 15 month old mice. The young and the aged STZ mice were divided randomly into the stress group and the control group, respectively. Each group contained 20 mice. Both the aged and the young stress mice were exposed to multiple...

Objective: To compare the effects of long term experimental stressors on blood glucose level and insulin secretion in the aged and young mice with diabetes prone and to explore the mechanism. Methods: Diabetes prone was induced by streptozotocin (STZ) in 40 1.5 month old mice and 40 15 month old mice. The young and the aged STZ mice were divided randomly into the stress group and the control group, respectively. Each group contained 20 mice. Both the aged and the young stress mice were exposed to multiple stressors (restrain, rotation, crowding) for 6 weeks. Fasting blood glucose (FBG), fasting plasma insulin (FINS), pancreas lipid peroxidate (LPO) and the metabolic products of nitric oxide (NO - 2, NO - 3) levels and superoxide dismutase (SOD) activity were measured. The grade of insulitis in STZ mice was also evaluated. Results: After 6 week stress, FBG level, pancreastissue LPO and NO - 2, NO - 3 contents were significantly higher while FINS level and SOD activity lower in the aged stress mice than those in the youngs. The insulitis malignization in the aged stress group was severer than that in the young stress group. There were correlativities among the above variables. Conclusion: The effects of long term stress in promoting hyperglycemia exacerbating insulitis, and inhibiting insulin secretion are more significant in the aged STZ mice than in the young ones. This difference may result from the excessive NO and the peroxide injury to the pancreas.

目的:比较长期实验性应激源对老龄、青龄糖尿病倾向鼠血糖和胰岛素分泌的不同影响并探讨其机制。方法:取1-5 月龄( 青龄) 和15 月龄( 老龄) 昆明小鼠各40 只,用链脲佐菌素诱导糖尿病倾向后(STZ 鼠) ,各分为应激组与对照组。两组应激STZ 鼠均给予6 周实验性应激源刺激( 限制、旋转、拥挤) ,检测其空腹血糖(FBG) 、空腹血浆胰岛素(FINS) 、胰腺组织脂质过氧化物(LPO) 和一氧化氮代谢产物(NO-2 ,NO-3 ) 水平,观察胰腺组织超氧化物歧化酶(SOD) 活性的变化,并对STZ 鼠胰岛炎的严重程度进行评分。结果:经长期实验性应激的老龄STZ 鼠FBG 水平增高较青龄者显著,且FINS 水平明显下降;胰岛炎恶化较青龄STZ鼠严重;胰腺组织LPO 和NO-2 ,NO-3 含量增高及SOD 活性降低均较青龄STZ鼠明显;上述各指标间存在相关性。结论:长期实验性应激促使老龄STZ 鼠血糖增高、胰岛炎加重和胰岛素分泌机能耗竭的效应较青龄STZ 鼠明显;胰腺NO 过量及过氧化损伤可能参与这一差异的产生机制

Objective To explore the effect of streptozotocin induced hyperglycemia (HG) on ischemic brain damage in rats. Methods Streptozotocin induced diabetic rats were subjected to 10 min of forebrain ischemia in 4 weeks after the induction of diabetes. Normoglycemic (NG) rats exposed to the same duration of ischemia,and sham operated diabetic rats (Sham)served as controls.The animals underwent evaluation of histopathological analysis of brain damage and clinical seizures. Results In the CA 1 and in the subcular...

Objective To explore the effect of streptozotocin induced hyperglycemia (HG) on ischemic brain damage in rats. Methods Streptozotocin induced diabetic rats were subjected to 10 min of forebrain ischemia in 4 weeks after the induction of diabetes. Normoglycemic (NG) rats exposed to the same duration of ischemia,and sham operated diabetic rats (Sham)served as controls.The animals underwent evaluation of histopathological analysis of brain damage and clinical seizures. Results In the CA 1 and in the subcular regions of hippocampus, the severity and distribution of damage were similar between HG and NG animals. However, HG rats had more severe neuronal necrosis in the parietal cortex and the neuronal damage involved“additional brain structures”compared with NG animals. Postischemic seizures developed in 42.1% of HG rats. None of the diabetic rats with plasma glucose concentrations below 12 mmol/L exhibited postischemic seizure activity. Conclusion Diabetic hyperglycemia exacerbates postischemic brain damage and results frequently in postischemic seizures.

目的 研究链脲佐菌素所致糖尿病性高血糖 (HG)对大鼠缺血性脑损伤的影响。方法 链脲佐菌素引起糖尿病 4周的大鼠造成 10 min前脑缺血 ,观察缺血后脑组织的病理改变和癫痫发作情况。结果 在海马 CA1区和下脚区 ,HG组与正常血糖 (NG)组动物脑神经元损伤程度相似。然而 ,HG组动物有严重的顶叶皮质神经元坏死和“额外脑结构损伤”。 HG组中 42 .1%的动物脑缺血后出现癫痫发作 ,血浆葡萄糖浓度低于 12 mm ol/ L者未见癫痫发作。结论 糖尿病高血糖明显加重缺血后脑损伤并常引起脑缺血后癫痫发作。

 
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