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in the cells    
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  组细胞
    The EGFP-VP3 granules in the nucleus became coarse and the cells became small and round, and finally into pieces. Apoptosis in the cells of HepG2/EGFP-VP3 was shown by Annexin V-PE dying.
    在HepG2/EGFP-VP3组细胞中,EGFP-VP3以荧光颗粒形式集中在细胞核,细胞核中EGFP-VP3颗粒逐渐变粗,细胞变得小而圆,最后成为碎片,Annexin V-PE染色显示细胞凋亡;
短句来源
    Twenty-four hours later, the expression of GFP in the cells was observed by fluorescence microscope, the numbers of positive expression cells in every high power field (×400) were counted.
    24h后用荧光显微镜观察各转染组细胞荧光蛋白表达情况,计录每高倍视野(400×)荧光蛋白表达阳性细胞数。
短句来源
    Results In the cells of Lovo/EGFP and 3T3/EGFP-VP3 group, EGFP was fairly well-distributed in whole cell. No change was found in cell morphology and apoptosis rate on transfected cells than natural ones.
    结果在Lovo/EGFP组和3T3/EGFP-VP3组细胞中,EGFP均匀分布于细胞中,细胞形态无明显变化,细胞凋亡率较非转染细胞组亦无明显增加。
短句来源
    In the cells of Lovo/EGFP-VP3 group, EGFP-VP3 was concentrated in nucleus as fluorescent granules, furthermore became coarser and the cells became small and round, and finally pieces. The rate of apoptosis in the cells of Lovo/EGFP-VP3 group was obviously higher than other control group cells (P < 0.01).
    而在Lovo/EGFP-VP3组细胞中,EGFP-VP3以荧光颗粒形式集中在细胞核,并逐渐变粗,最后细胞碎裂成片状,流式细胞技术检测Lovo/EGFP-VP3组的细胞凋亡率明显高于其他对照组(P<0.01)。
短句来源
    mito-MPG mRNA and protein were detected in the cells transfected with pCMV-Script/mito-MPG (MPG group), but not in the cells transfected with pCMV-Script (P group) and untransfected cells (C group).
    转染pCMV-Script/mito-MPG载体组(MPG组)检测到mito-MPGmRNA的表达,转染pCMV-Script载体组(P组)及未转染组(C组)细胞内则未检测到; MPG组细胞线粒体内检测到MPG,P组及C组则未检测到;
短句来源
  在细胞
    Role of Telomerase in the Cells Eternalization and Diagnosis of Tumor
    端粒酶在细胞永生化及肿瘤诊断中的作用
短句来源
    Recombination of TK gene in the cells was examined by blot hybridization and sensitivity of A549-TK cells to GCV was observed in vitro.
    斑点杂交检测外源基因在细胞中整合,并体外观察A549-TK对GCV的敏感性。
短句来源
    Moreover, a similar amount of PpIX was found in the cells incubated with 0.2 mmol/L He ALA or 2 mmol/L ALA and a similar level of cell survival was reached following light exposure.
    实验发现 ,0 .2mmol/L的He ALA药物浓度与 2mmol/LALA的药物浓度在细胞中生成的PpIX含量相当 ;
短句来源
    RARα ex-isted in the cells in single, but the PMI-RARa in com-plex form.
    结果显示:RARα在细胞中以单体形式存在,而PML-RARα则以复合体形式存在;
短句来源
    Results Similar to the E-cadherin expressed in normal epidermis,E-cadherin was strongly expressed in cell of samples BCC,SK,and KA,whereas marked decrease or lose of E-cadherin expression was found in the cells of SCC. Compared with the normal epidermal cells the expression of E-cadherin in AK and Bowen disease was normal or reduced,with complete absence of staining in dysplasis arease.
    结果 E cad在BCC ,SK ,KA表皮中的表达与正常表皮相似 ,为表皮全层细胞间较强的染色 ,而在SCC中表达显著减弱或完全无表达 ,在AK和Bowen病表皮正常区域表达正常或下调 ,但在细胞间变区域无染色。
短句来源
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  在细胞内
    Dox accumulation in the Har-resistant HL60 cells treated by Tet was increased. CONCLUSION: Dox resistance in the Har-resistant HL60 cells treated by Tet or Dau was reduced, due to the increase of Dox accumulation in the cells.
    结论:Tet和Dau减低抗Har的HL60细胞对Dox的抗性,其机制是增加Dox在细胞内积聚。
短句来源
    After incubation in conventional medium for 24 h over 90% WSA and 70% of the 3H-Liposomes still remained in the cells, while only 35% of the 125I-Trastuzumab remained.
    细胞摄取125ITrastuzumabWSA3H脂质体后,在普通培养液中培养24和48h,分别有90%、67%的WSA滞留在细胞内
短句来源
    Dendritic cells (DC) are special antigen presenting cells (APC) in the body. They intake, process and treat antigens, then bind with MHC-Ⅱmolecules in the cells and present the antigen-MHC-Ⅱmolecules compounds to naive T cells, thus elicit specific immune response.
    树突状细胞(DC)是人体内的专职抗原提呈细胞,它摄取、加工、处理抗原,然后在细胞内与MHC-Ⅱ类分子结合,将MHC-Ⅱ类分子、抗原复合物提呈给初始T淋巴细胞,激发特异性免疫反应。
短句来源
    The results showed that EBV might make a great impact on the pathway of DNA synthesis regulation, gene transcription and so on by its gene expression products, and then altered protein expression pattern, consequently promoted cell growth and suppressed cell apoptosis, while OA could inhibit phosphatase to cause the over-phosphorylation of related proteins which affected pathways of the signal transduction in the cells.
    OA则可能是通过对蛋白磷酸酶的抑制,使细胞内相关蛋白过度磷酸化,影响细胞内的信号转导通路,改变基因表达; 同时,EB病毒与OA在细胞内可能会对相同的途径影响发挥作用,在某些通路中可能相互促进,而在某些通路中可能存在一定拮抗性的作用。
短句来源
    Results The plus and minus-strand of HCV RNA was intermittently detected in the cells and the supernatant of the culture medium over a period of more than three months after incubation. Various antigen against HCV were expressed in cells.
    结果 细胞内和培养上清中在孵育后的 3月余均可间断地检测出HCV正链和 /或负链RNA ,多种HCV抗原在细胞内能稳定表达 ;
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  后细胞
    Expression of Survivin protein in the cells treated with SB at 2.500 mmol/L was detected by immunohistochemistry SP method.
    采用免疫组织化学SP法检测2.500mmol/LSB作用24h、48h、72h后细胞抗凋亡蛋白Survivin表达的变化。
短句来源
    The cell VDRas3 which stably expressed VDR antisense mRNA was used to observe the effect of 1,25(OH) 2 D 3 on the proliferation of HOS-8603 cells and the induction of p21 mRNA, one of the VDR target genes, when the VDR in the cells was blocked.
    并进一步利用稳定表达VDR反义mRNA的细胞株VDRas3细胞研究VDR被阻断后细胞增殖以及基因转录的变化。
短句来源
    The arrangement of microfilament in the cells became regular and was similar to nonmalignant normal cells, but there was no effect on microtubule when the bel 7402 cells were treated with lidamycin 10 nmol/L for 8 h.
    10nmol/L力达霉素处理细胞8h后,细胞伸展,微丝排列整齐,向非恶性细胞转变,但对微管没有影响。
短句来源
    The fluorescence emission spectrum of the cells incubated with He ALA was identical to that of PpIX, indicating that He ALA could induce PpIX in the cells.
    细胞的荧光显微图象显示 ,经He ALA培育后 ,细胞中生成了PpIX。
短句来源
    RESULTS:The PGE 2 level in the cells cultured supernatant decreased by NIM in a concentration and time dependent manner.
    结果 :NIM处理A 5 49细胞后 ,细胞培养上清液PGE2 水平下降 ,呈浓度和时间依赖性 ;
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  in the cells
Enzyme activity was revealed in the cells grown on medium with elemental sulfur or in the presence of various sulfide minerals and concentrates of sulfide ores.
      
The mutants varied in Cr(VI) resistance, the degree of chromium accumulation in the cells (from 0.1 to 11.6 mg/g dry cells), and the degree of Cr(VI) reduction (from 50% to complete disappearance of bichromate from the culture liquid).
      
Treatment with ambiol enhanced the differentiation of the internal membrane system of plastids in the cells of original and transgenic plants, especially the tubular membrane systems.
      
The concentration of eight kinds of catechins in solution decreased by 29.6-47.6%, respectively; some catechins were absorbed and accumulated by yeast cells, but the amount in the cells was very low during the fermentation process.
      
Specific attention is given to regulation of enzymes of antioxidative defense, operating in the cells of strict anaerobes under the conditions of oxidative stress caused by oxygen, superoxide anion, or hydrogen peroxide.
      
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(1) A comparative study has been made on the induction of tryptophan pyrrolase by substrate and corticoids in liver tumor and the precancerous liver induced by 3'-MeDAB.(2) The liver tumor, contrary to normal liver, was inactive in response to both tryptophan and hydrocortisone for the induction of tryptophan pyrrolase, while the tissue adjacent to the tumor was active with respect to both substrate and hormonal induction.(3) A decrease in both substrate and hormonal induction was observed in the precancerous...

(1) A comparative study has been made on the induction of tryptophan pyrrolase by substrate and corticoids in liver tumor and the precancerous liver induced by 3'-MeDAB.(2) The liver tumor, contrary to normal liver, was inactive in response to both tryptophan and hydrocortisone for the induction of tryptophan pyrrolase, while the tissue adjacent to the tumor was active with respect to both substrate and hormonal induction.(3) A decrease in both substrate and hormonal induction was observed in the precancerous liver developed by feeding 3'-MeDAB for different time intervals. In experiments by injecting intraperitoneally the carcinogen into animals,for 25 hours, similar results were obtained as in the feeding experiments.(4) 2-MeDAB, a non-carcinogenic substance, caused the same effect on the induction of tryptophan pyrrolase in every case as did 3'-MeDAB. It thus appears that the effect of 3'-MeDAB on the enzyme induction may not be specific.(5) No inhibitors of tryptophan pyrrolase or activators of kynureninase were found in the cell sap of liver tumor and the liver of rats fed 3'-MeDAB or 2-MeDAB in the course of induction.(6) The cell sap from liver tumor (non-induced) contained only a small amount of enzyme protein, as shown by the fact that the enzyme activity being only slightly increased by the addition of either normal microsome or hematin, of which the level has not elevated by the administration of tryptophan or hydrocortisone.(7) Similar experiments have shown that the cell sap from the substrate and hormonal induced liver in the precancerous stage contained a decreased amount of enzyme protein as compared to that of the control. The same was true of the rats fed 2-MeDAB.(8) Microsomes from liver tumor have lost almost completely the ability of activating tryptophan pyrrolase in the cell sap. The ability of activation due to microsomes from the precancerous liver was remarkably reduced, though not yet completely lost, while the microsomes from the liver of rats fed 2-MeDAB were normal. It was in this respect that the effect of non-carcinogen (2-MeDAB) was found to be different from that of the carcinogen (3'-MeDAB).(9) From the results presented, it was concluded that the default of tryptophan pyrrolase induction observed in the liver tumor and the precancerous liver was mainly due to an inadequate amount of apoenzyme, rather than a deficiency of co-factor (hematin) or an increment of protein other than the enzyme. The possible cause of these effects was briefly discussed.

本文对大鼠肝癌及癌前期肝内TP的底物诱导和激素诱导作了比较研究。在3′-MeDAB诱发的肝癌中,TP活性很低,且不因注射色氨酸或氢可地松而升高,而癌周组织则仍保留对底物和激素诱导的能力。喂3′-MeDAB 13天、28天、90天大鼠肝内TP的底物诱导效应都较对照组为低。急性注射3′-MeDAB25小时,以100毫克/100克体重的L-色氨酸进行诱导,TP的诱导效应亦较对照组为低。氢可地松诱导的结果与底物诱导的相似,无论在喂或急性注射3′-MeDAB的情况下,TP的诱导效应都受到抑制。但在相同条件下,非致癌物,2-MeDAB,对TP的底物诱导和激素诱导(慢性的或急性的实验)也有相似的作用。诱导后肝癌组织或喂偶氮染料的肝组织中都未发现有TP的抑制物或狗尿酸酶的激活物。微粒体及正铁血红素与上清液的加合实验表明:(1)肝癌微粒体几乎完全不具有激活TP的活力;癌前期(3′-MeDAB组)肝微粒体已部分失去此种生化功能,但2-MeDAB组微粒体则否。3′-MeDAB对肝微粒体中辅助因子(正铁血红素)的结构并无破坏,而可能使辅助因子的含量减少。(2)微粒体对激活上清液TP的效果较自由的正铁血红素差,即使加入过量微粒体亦不能...

本文对大鼠肝癌及癌前期肝内TP的底物诱导和激素诱导作了比较研究。在3′-MeDAB诱发的肝癌中,TP活性很低,且不因注射色氨酸或氢可地松而升高,而癌周组织则仍保留对底物和激素诱导的能力。喂3′-MeDAB 13天、28天、90天大鼠肝内TP的底物诱导效应都较对照组为低。急性注射3′-MeDAB25小时,以100毫克/100克体重的L-色氨酸进行诱导,TP的诱导效应亦较对照组为低。氢可地松诱导的结果与底物诱导的相似,无论在喂或急性注射3′-MeDAB的情况下,TP的诱导效应都受到抑制。但在相同条件下,非致癌物,2-MeDAB,对TP的底物诱导和激素诱导(慢性的或急性的实验)也有相似的作用。诱导后肝癌组织或喂偶氮染料的肝组织中都未发现有TP的抑制物或狗尿酸酶的激活物。微粒体及正铁血红素与上清液的加合实验表明:(1)肝癌微粒体几乎完全不具有激活TP的活力;癌前期(3′-MeDAB组)肝微粒体已部分失去此种生化功能,但2-MeDAB组微粒体则否。3′-MeDAB对肝微粒体中辅助因子(正铁血红素)的结构并无破坏,而可能使辅助因子的含量减少。(2)微粒体对激活上清液TP的效果较自由的正铁血红素差,即使加入过量微粒体亦不能使TP活性增高到加入正铁血红素的水平;微粒体对3′-MeDAB组上清液的激活不如对2-MeDAB组及对照组上清液(底物或激素诱导)的激活显著,而正铁血红素对三组上清液都有显著激活。(3)肝癌细胞上清液只合有极少量的TP蛋白,且不因注射色氨酸或氢可地松而增加;癌前期肝细胞上清液的TP蛋白因底物或激素诱导而增加的量都较对照粗低。2-MeDAB组也有相似现象。以上结果表明,肝癌及癌前期肝内TP诱导的受损,主要是由于诱导后TP蛋白的缺少,而不是由于辅助因子(如正铁血红素)的不足,或非酶蛋白的增多。

Observations on the localization of AFP in rat liver tissue in correlation to serum AFP dynamics during 3'-MeDAB carcinogenesis were carried out by the immunoenzyme technique and the radio-rocket-electrophoresis autography.The following results were obtained:(1) During the precancerous stage, AFP was found in a few liver cells in the pseudolobules, in the cells of the basophilio anaplastic regenerative nodules and in some of the "survival" liver cells and "transitional" cells. Most of these...

Observations on the localization of AFP in rat liver tissue in correlation to serum AFP dynamics during 3'-MeDAB carcinogenesis were carried out by the immunoenzyme technique and the radio-rocket-electrophoresis autography.The following results were obtained:(1) During the precancerous stage, AFP was found in a few liver cells in the pseudolobules, in the cells of the basophilio anaplastic regenerative nodules and in some of the "survival" liver cells and "transitional" cells. Most of these cells showed basophilio cytoplasm and appeared dedifferentiated.(2) AFP was not detected in the bile duct carcinomas nor in most of the liver cell carcinomas of low grade malignancy, but was demonstrated in the poorly differentiated liver cell carcinomas. The intensity of AFP synthesis in liver cell carcinomas increased in coincidence with their activity in cell growth and cell proliferation but decreased with cell differentiation. AFP synthesis was not directly related to whether or not the cell was in the mitotic stage.(3) In the liver cell carcinomas, the extent of AFP positive reaction as visualized by the immunoenzyme technique basically paralleled the serum AFP level.The possible mechanism and the histologic basis of the serum AFP "saddle shaped" curve during 3'-MeDAB carcinogenesis are discussed, and a preliminary postulation on the histogenesis of liver cancer so induced is suggested.

本工作采用放射火箭电泳自显术和免疫酶标记定位技术,对大鼠3'-MeDAB诱癌过程血清和肝组织甲胎蛋白(AFP)的动态变化和定位情况进行了观察。发现(1)肝硬化假小叶内的少数肝细胞、嗜碱性间变再生结节细胞、少数“幸存肝细胞”及少数“过渡性细胞”,具有合成AFP的能力。未见肝内其他类型细胞合成AFP。合成AFP的细胞大多具有胞浆嗜碱性、生长活跃和形态上去分化的特点。(2)未见胆管癌细胞合成AFP。肝细胞癌分化好的癌细胞绝大多数也未见合成AFP,分化差的癌细胞合成AFP的能力,基本上与其生长活跃程度呈正相关,与分化程度呈负相关,而与癌细胞是否处于核分裂阶段关系不大。(3)肝癌组织AFP酶标的强度和范围与血清AFP水平之间基本上有平行关系。此外,本文还就什么细胞合成AFP、血清AFP“马鞍型”变化的成因及病理组织学基础等进行了讨论,并根据实验结果,对大鼠3'-MeDAB肝癌的组织发生提出了初步的设想。

In the present paper some factors affecting the superinduction of interferon in diploid cells were studied. The results showed that the titer of interferon superinduced in fetal skin muscle cell cultures was significantly higher than those in fetal human lung cell cultures. Cells treated with Astragalus membranaceus produced more interferon. Poly I:C and cycloheximide could be present simulstaneouly in the cell cultures during the whole course of superinduction. However, the duration...

In the present paper some factors affecting the superinduction of interferon in diploid cells were studied. The results showed that the titer of interferon superinduced in fetal skin muscle cell cultures was significantly higher than those in fetal human lung cell cultures. Cells treated with Astragalus membranaceus produced more interferon. Poly I:C and cycloheximide could be present simulstaneouly in the cell cultures during the whole course of superinduction. However, the duration of treatment with inhibitors of nucleic acid synthesis ( mithramycin and actinomy-cin D ) should not exceed one hour before the termination of superinduction. The. behaviour of superinduction by mithramycin was similar to that by actinomycin D The optimal time for combined poly I : C and cycloheximide treatment was about 9 hours. Under our experimental conditions, the interferon yield in the superinduction system was increased by 170-248 fold as compared with similar system without superinduction. The present paper describes a procedure for the large scale production of fibroblast interferon.

本文在人二倍体细胞培养上就影响干扰素超诱导的某些因素进行了研究。结果表明:人肌皮二倍体细胞株的干扰素超诱导能力高于人肺二倍体细胞株。黄芪处理细胞在超诱导系统中可以生产较高量的干扰素。聚I:C和环已亚胺能在超诱导的整个过程持续存在,但放线菌素D和光辉霉素则在超诱导终止前作用1小时最适宜。光辉霉素与放线菌素D有相似的干扰素超诱导作用。聚I:C与环已亚胺的适宜作用时间为7~9小时。在适宜条件下,干扰素的产量在超诱导系统中比单诱导增加170~248倍。本研究可为大量生产纤维细胞干扰素及提取干扰素mRNA提供依据,并为阐明干扰素超诱导机理提供基础。

 
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