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intra-nuclear
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  核内
     The theoretical frames of MEND are spherical optical model,intra-nuclear cascade model,pre-equilibrium emission theory based on exciton model,evaporation model and Hauser-Feshbach statistical theory with width fluctuation correction.
     MEND程序的理论框架基于球形光学模型、核子的核内级联发射模型、以激子模型为基础的预平衡发射理论、蒸发模型和带宽度涨落修正的Hauser-Feshbach统计理论。
短句来源
     Results Compared to that in control group,the nuclear translocation of monocytic p50 and p65 took place 30 min after the PBMCs were stimulated by burn serum,peaking at 30 to 60 min,but it gradually recovered to pre-stimulation state at 2 hrs with decreased intra-nuclear collection.
     结果 与对照组比较 ,刺激 30min后烧伤血清组PBMC中p5 0、p6 5即发生核移位 ,30~ 6 0min达高峰 ,12 0min后核内聚集减少 ,回复至刺激前状态。
短句来源
     The nuclear reaction is calculated using intra-nuclear cascade model.
     核反应过程采用核内级联模型。
短句来源
     In this study, RAPD marker and mitochondria DNA sequencing techniques were used to study the intra-nuclear and extra-nuclear genetic material variation, and to analyze the phylogeny of the deers.
     本研究试图通过RAPD标记和线粒体DNA测序,对存在于核内及核外的遗传物质变异情况进行研究,同时对鹿类动物的系统发育进行分析。
短句来源
     After comparison with the result published and experimental data,it showed that it is a very good choice using intra-nuclear cascade model to simulate protons' nuclear reaction.
     经比较和分析发现,用核内级联模型模拟质子的核反应,在入射质子的能量很高(>50MeV)时与文献和实验结果吻合得非常好;
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  “intra-nuclear”译为未确定词的双语例句
     On the first day of AngⅡ and RY stimulation, NFAT3 was shown mainly as intra-nuclear expression rather than cytoplasmic expression as seen in the control group.
     AngⅡ和雷尼丁刺激第 1天 ,心肌细胞NFAT3表达由胞质转入胞核表达为主。
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  相似匹配句对
     Intra-Abundant semigroups
     内禀富足半群(英文)
短句来源
     (5) intra-alveolar hemorrhage;
     (5)肺泡内出血;
短句来源
     The Enhanced H.264 Intra Prediction
     H.264帧内方向预测增强模式
短句来源
     Treatment of intra articular fractures
     重视关节内骨折的治疗
短句来源
     NUCLEAR MULTIFRAGMENTATION
     原子核的多重碎裂
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  intra-nuclear
Intra-nuclear localization of two envelope proteins, gB and gD, of herpes simplex virus
      
Studies with the monkey-intra-nuclear-inclusion-agent (MINIA) and foamy-agent
      
Studies with the monkey-intra-nuclear-inclusion-agent (MINIA) and foamy-agent derived from spontaneously degenerating monkey kid
      
This case of interstitial pneumonia was caused by the measles virus, with intracytoplasmic inclusion bodies in giant cells and type II alveolar cells in alveolar and bronchiolar spaces, with rare intra-nuclear inclusion bodies.
      
The intra-nuclear rod which extends from the middle piece to the base of the acrosome is derived from the proximal centrosome.
      
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Developing nuclear data needs towards to sustainable development on fission reactor design and many nuclear applications out the field of fission reactor technology that are growing economic significance and that have substantial data requirements are introduced. International standard codes used in nuclear data evaluation and calculation are presented and compared with each other. Generally speaking, three rather wide groups of nuclear reaction models used usually are also described. They are the formal theories...

Developing nuclear data needs towards to sustainable development on fission reactor design and many nuclear applications out the field of fission reactor technology that are growing economic significance and that have substantial data requirements are introduced. International standard codes used in nuclear data evaluation and calculation are presented and compared with each other. Generally speaking, three rather wide groups of nuclear reaction models used usually are also described. They are the formal theories of resonance reactions, generalized statistical models including fission, pre equilibrium and direct processes and combined with the optical model, and the intra nuclear cascade models. Recent progress of these nuclear reaction theories are given briefly, specially the recent progress of multi step compound and direct and state density are introduced in more detail.

简要介绍了国际上发展中的核科学和技术及其应用对微观核数据的需求和核数据理论计算中现有标准程序的基本情况 ,并做了相应的分析和比对 .同时也简要介绍了国际上核数据理论计算中模型方法研究的新进展

ObjectiveTo investigate the role of [Ca 2+ ]i from different origins in the course of myocardial hypertrophy mediated by CaN-NFAT3 signal transduction. MethodsThe primarily cultured cardiomyocyte were irritated with angiotensin (Ang) Ⅱand ryanodine (RY) which cause Ca 2+ inflow and release respectively. Then to observe the changes of CaN-NFAT3 pathway were then observed. Western blotting was employed to semi-quantify CaN, NFAT3 and GATA4. The distribution of NFAT3 was shown with immunocytochemistry,...

ObjectiveTo investigate the role of [Ca 2+ ]i from different origins in the course of myocardial hypertrophy mediated by CaN-NFAT3 signal transduction. MethodsThe primarily cultured cardiomyocyte were irritated with angiotensin (Ang) Ⅱand ryanodine (RY) which cause Ca 2+ inflow and release respectively. Then to observe the changes of CaN-NFAT3 pathway were then observed. Western blotting was employed to semi-quantify CaN, NFAT3 and GATA4. The distribution of NFAT3 was shown with immunocytochemistry, 3H-Leu incorporation was used as an index of myocyte hypertrophy. Cyclosporin A (CsA) was applied to restrain CaN-NFAT3 pathway as a kind of CaN-selective antagonist. ResultsCaN, NFAT3, GATA4 expression significantly increased 1 and 3 days after the stimulation of cardiomyocytes with AngⅡ and RY(10 -7 mol/L) as compared with that of a control group (P<0.05) and 3H-Leu incorporation distinctly increased after AngⅡ and RY (10 -7 mol/L) stimulation (P<0.05 versus control group). On the first day of AngⅡ and RY stimulation, NFAT3 was shown mainly as intra-nuclear expression rather than cytoplasmic expression as seen in the control group. All of the above effects were suppressed by CsA administration, but they were rarely suppressed if CsA was not administered (P<0.05). ConclusionsIt si shown that both Ca 2+ inflow and release may activate CaN-NFAT3 signal pathway, which responds to increase of [Ca 2+ ] i and is independent of its origin, indicating the augment of [Ca 2+ ] i may trigger CaN-NFAT3 signal transduction and consequently induce myocyte hypertrophy. Moreover, CsA may restrain the expression and activation of CaN-NFAT3 and protein synthesis of myocytes in response to AngⅡ and RY stimulation.

目的 探讨不同来源的细胞内Ca2 + ([Ca2 + ]i)在钙调神经磷酸酶 (CaN) 活化T细胞核因子 3 (NFAT3 )介导的心肌肥大中的作用。方法 分别用血管紧张素Ⅱ (AngⅡ )或雷尼丁刺激培养的大鼠心肌细胞外Ca2 + 跨膜内流或细胞内Ca2 + 释放 ,检测CaN、NFAT3、锌指转录因子 (GATA4)蛋白量、NFAT3定位以及氚 亮氨酸 (3H Leu)掺入量 ,环孢素A作为CaN特异抑制剂。结果 AngⅡ、雷尼丁刺激 1、3d ,心肌细胞CaN、NFAT3、GATA4蛋白表达及3H Leu掺入量较对照组明显增高(P值 <0 0 5或 <0 0 1)。AngⅡ和雷尼丁刺激第 1天 ,心肌细胞NFAT3表达由胞质转入胞核表达为主。环孢素A可抑制上述作用 ,与刺激组相比差异有显著性 (P <0 0 5或 <0 0 1)。结论 刺激心肌细胞Ca2 + 内流及Ca2 + 释放 ,均可激活CaN NFAT3信号通路。CaN NFAT3信号通路的激活与[Ca2 + ]i增加有关 ,而与 [Ca2 + ]i的来源无关。环孢素A能够抑制AngⅡ和雷尼丁介导的CaN NFAT 3 GATA ...

目的 探讨不同来源的细胞内Ca2 + ([Ca2 + ]i)在钙调神经磷酸酶 (CaN) 活化T细胞核因子 3 (NFAT3 )介导的心肌肥大中的作用。方法 分别用血管紧张素Ⅱ (AngⅡ )或雷尼丁刺激培养的大鼠心肌细胞外Ca2 + 跨膜内流或细胞内Ca2 + 释放 ,检测CaN、NFAT3、锌指转录因子 (GATA4)蛋白量、NFAT3定位以及氚 亮氨酸 (3H Leu)掺入量 ,环孢素A作为CaN特异抑制剂。结果 AngⅡ、雷尼丁刺激 1、3d ,心肌细胞CaN、NFAT3、GATA4蛋白表达及3H Leu掺入量较对照组明显增高(P值 <0 0 5或 <0 0 1)。AngⅡ和雷尼丁刺激第 1天 ,心肌细胞NFAT3表达由胞质转入胞核表达为主。环孢素A可抑制上述作用 ,与刺激组相比差异有显著性 (P <0 0 5或 <0 0 1)。结论 刺激心肌细胞Ca2 + 内流及Ca2 + 释放 ,均可激活CaN NFAT3信号通路。CaN NFAT3信号通路的激活与[Ca2 + ]i增加有关 ,而与 [Ca2 + ]i的来源无关。环孢素A能够抑制AngⅡ和雷尼丁介导的CaN NFAT 3 GATA 4表达的增加和蛋白质合成

Objective To investigate the effects of burn serum on nuclear translocation of monocytic NF-κB heterodimers p50/p65 and the degradation of inhibiting κB(IκBα),so as to further explore the role of burn serum on the activation of monocytes. Methods Peripheral blood monocytes (PBMCs) isolated from healthy volunteers were employed as the target cells.The cells were stimulated by the serum from healthy volunteers and burn patients,and by burn serum together with pyrrolidine dithioncarbamate(PDTC).Sera from normal...

Objective To investigate the effects of burn serum on nuclear translocation of monocytic NF-κB heterodimers p50/p65 and the degradation of inhibiting κB(IκBα),so as to further explore the role of burn serum on the activation of monocytes. Methods Peripheral blood monocytes (PBMCs) isolated from healthy volunteers were employed as the target cells.The cells were stimulated by the serum from healthy volunteers and burn patients,and by burn serum together with pyrrolidine dithioncarbamate(PDTC).Sera from normal healthy volunteers were taken as control.The nuclear translocation of monocytic p50 and p65 at 30th,60th,120th and 480th post stimulation minutes (PSM) was observed with laser confocal microscopy.The degradation of monocytic IκBα protein at 30th,60th,90th and 120th PSM was determined by Western blot. Results Compared to that in control group,the nuclear translocation of monocytic p50 and p65 took place 30 min after the PBMCs were stimulated by burn serum,peaking at 30 to 60 min,but it gradually recovered to pre-stimulation state at 2 hrs with decreased intra-nuclear collection.Meanwhile, the IκBα degradation occurred within 30 min after PBMCs being stimulated by burn serum,and it peaked at 60 mins. However,IκBα gradually reappeared in the cytoplasm after 2 hrs of stimulation.PDTC (an antioxidants) could effectively inhibit monocytic IκBα degradation and nuclear translocation of NF-κB induced by burn serum. ConclusionBurn serum could induce nuclear translocation of p50 and p65 components of NF-κB in monocytes into the nucleus and degradation of IκBα,leading ultimately to the secretion of cytokines from the PBMCs.

目的 观察烧伤患者血清 (以下称烧伤血清 )对单核细胞核因子κB(NF κB)异二聚体p5 0、p6 5核移位及核抑制因子κBα(IκBα)降解的影响 ,进一步探讨烧伤血清对单核细胞活化的作用。 方法 收集体外培养的人外周血单核细胞 (PBMC),分别用正常人血清、烧伤血清、烧伤血清 +吡咯烷二硫代氨基甲酸盐 (PDTC)刺激PBMC(依次分为对照组、烧伤血清组、PDTC组 ) ,应用激光共聚焦显微镜观察血清刺激 30、6 0、12 0、4 80min后PBMC的p5 0、p6 5核移位 ;采用Western印迹法检测刺激 30、6 0、90、12 0min时PBMC的IκBα蛋白降解情况。 结果 与对照组比较 ,刺激 30min后烧伤血清组PBMC中p5 0、p6 5即发生核移位 ,30~ 6 0min达高峰 ,12 0min后核内聚集减少 ,回复至刺激前状态。刺激 30min后烧伤血清组PBMCIκBα发生降解 ,刺激 6 0min后含量几乎为零 ,与对照组比较差异有非常显著性意义 (P <0.0 1),12 0min后表达水平逐渐恢复。PDTC组PBMCIκBα降解 [刺激 6 0min后含量为 (11...

目的 观察烧伤患者血清 (以下称烧伤血清 )对单核细胞核因子κB(NF κB)异二聚体p5 0、p6 5核移位及核抑制因子κBα(IκBα)降解的影响 ,进一步探讨烧伤血清对单核细胞活化的作用。 方法 收集体外培养的人外周血单核细胞 (PBMC),分别用正常人血清、烧伤血清、烧伤血清 +吡咯烷二硫代氨基甲酸盐 (PDTC)刺激PBMC(依次分为对照组、烧伤血清组、PDTC组 ) ,应用激光共聚焦显微镜观察血清刺激 30、6 0、12 0、4 80min后PBMC的p5 0、p6 5核移位 ;采用Western印迹法检测刺激 30、6 0、90、12 0min时PBMC的IκBα蛋白降解情况。 结果 与对照组比较 ,刺激 30min后烧伤血清组PBMC中p5 0、p6 5即发生核移位 ,30~ 6 0min达高峰 ,12 0min后核内聚集减少 ,回复至刺激前状态。刺激 30min后烧伤血清组PBMCIκBα发生降解 ,刺激 6 0min后含量几乎为零 ,与对照组比较差异有非常显著性意义 (P <0.0 1),12 0min后表达水平逐渐恢复。PDTC组PBMCIκBα降解 [刺激 6 0min后含量为 (11 5 7± 1.98)× 10 4积分灰度值 ]及p5 0、p6 5核移位程度比烧伤血清组轻 (P <0 0 1)。  结论 烧伤血清可导致PBMCIκBα降解和 p5 0、p6 5核移位 ,进而活化NF κB,诱导PBMC分泌细胞因子。PDTC对此变化有抑制作用

 
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