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mean arterial
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  平均动脉
     In clinic, four parameters are used in the description to blood pressure, that is: systolic blood pressure (SBP), diastolic blood pressure (DBP), mean arterial pressure (MAP), pulse pressure (PP).
     在临床上常用的四个参数来描述血压:收缩压(systolic blood pressure,SBP)、舒张压(diastolic blood pressure,DBP)、平均动脉压(mean arterial pressure,MAP)、脉压(pulse pressure, PP)。
短句来源
     After injection,the cardiac output(CO),heart rate(HR),mean arterial pressure(MAP),central venous pressure(CVP),CSFP and urine volume were detected at the end of injection(T0),15min(T1),30min(T2),60min(T3),90min(T4) and 120min(T5),then the cerebral perfusion pressure(CPP) was calculated.
     输完后分别于输注完即刻(T0)、15min(T1)、30min(T2)、60min(T3)、90min(T4)、120min(T5)检测心排量(CO)、心率(HR)、平均动脉压(MAP)、中心静脉压(CVP)、CSFP、尿量,计算脑灌注压(CPP)。
短句来源
     the mean variation of mean arterial pressure was -2.4% ±2.5% , and that of the control group 10.7%±2.5%.
     艾司洛尔组的平均动脉压平均变化为 - 2 .4 %± 2 .5 % ,对照组为 10 .7%± 2 .5 %。
短句来源
     The heart rate(HR),mean arterial pressure(MAP),oxygen saturation by pulse oximetry(SPO2),time for tracheostomy,and postoperative complications were recorded at the time before operation(T1),during tracheostomy(T2),1 min(T3),3 min(T4),and 5 min(T5) after tracheostomy,respectively.
     比较气管造口前(T1)、气管造口即刻(T2)、气管造口后1 min(T3)、3 min(T4)、5 min(T5)时心率(heart rate,HR)、平均动脉压(mean arterial pressure,MAP)和脉搏血氧饱和度(saturation pulse oxygen,SPO2),以及气管造口完成时间和术后并发症。
短句来源
     ②The 24-hour mean systolic pr essure,mean diastolic p ressure and mean arterial pressure were ob-viously hig her in the 51to 60-year-o ld group than in the 21to 30,31to 40an d41to 50-year-old groups(119±4),(76±5),(90±4)mm Hg ;
     ②51~60岁组24h平均收缩压、平均舒张压及平均动脉压明显高于21~30岁组,31~40岁组,41~50岁组犤(119±4),(76±5),(90±4)mmHg;
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  平均动
     Mean arterial pressure and SpO_2 were 11.4±2.2 kPa,10.6±2.1 kPa and 98±1.0,96±1.5 respectively before and after ten minutes of the operation,with no significant difference.
     麻醉前后10 min平均动脉压分别为(11.4±2.2)kPa和(10.6±2.1)kPa,SaO2分别为(98±1.0)%和(96±1.5)%,均无显著性差异。
短句来源
     Results Nocturnal systemic blood pressure(18.90±0.91kPa)and mean arterial pressure(MAP)(12.94± 0.12 kPa)were significantly higher than that in the microalbuminuria group and in the control group( 16.30 ± 0.81 kPa,11.83±0.69kPa)respectively.
     结果 ①微量白蛋白尿组的夜间收缩压、平均动脉压分别为 18.90± 0 .91kPa和 12 .94± 0 .12kPa ,较正常组 16 .30± 0 .81kPa和 11.83± 0 .6 9kPa明显升高 (P <0 .0 5 )。
短句来源
     Fourteen patients (M11, F 3; age 54+s4a) with essential hypertension received nitrendipine po 2-3 h after the initial dose of 20 mg, results showed that 7B 3.8+1.4h, Tmax 4.4+1.5 h, total peripheral resistance and mean arterial pressure were reduced by 40%, 20% respectively, and were maintained for 12 h;
     原发性高血压14例(男11例,女3例;年龄54±s4a),首次口服尼群地平20mg2-3h后,消除T_(1/2)为3.8±1.4h,T_(max)4.4±1.5h,总外周阻力、平均动脉压分别下降40%,20%,降压作用持续12h。
短句来源
     The left atrial pressure fell from 2.53±0.28 to 1.11 ±0.21 kPa immediately after the dilatation (P<0.001). However, no remarkable changes in the right atrial pressure, mean arterial pressure and the heart rate were observed.
     结果表明,术后即刻左房压力显著下降(前后分别为2.53±0.28与1.11±0.21 kPa,P<0.001),而右房压、平均动脉压及心率均无显著变化;
短句来源
     the mean arterial pressure were (12.7±0.33)kPa and (13.6±0.33)kPa respectively, the difference between two groups was significant.
     平均动脉压分别为(12.7±0.33)kPa、(13.6±0.33)kPa。 两组对比有显著差异(P<0.05)。
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  “mean arterial”译为未确定词的双语例句
     60min after administion, the mean arterial pressure (MAP) of the group a was 10 4±2 1kPa, while those of two control (B、C) were 7 9±2 4 and 7 7±2 6kPa respectively ( P <0 01).
     给药60min后,A组MAP为10.4±2.1kPa,与B、C组7.9±2.4和7.7±2.6kPa间均有差异(P<0.01);
短句来源
     mean arterial pressure had positive correlation with levels of AngⅡ and CORT in all miners(r=0.412,P<0.01 and r=0.309,P<0.01).
     矿工平均动脉血压与血浆AngⅡ和CORT水平均呈显著正相关(r=0.412,P<0.01和r=0.309,P<0.01)。
短句来源
     Results:The mean AG was (22.8 ± 1.6) mmol/L and the mean UAG was (3.9 ± 0 .8) mmol/L. The mean arterial bind lactate concentration was (5 .8 ± 0.7) mmol/L.
     结果:AG=(22.8±1.6)mmol/L,UAG=(3.9±0.8)mmol/L,乳酸=(5.8±0.7)mmol/L。
短句来源
     HR, SBP, DBP and mean arterial pressure (MAP) of subjects undergoing AGSM on the ground were (87.29±9.23) beats/min, (186.00±11.11) mmHg, (147.86±9 96)mmHg and (160 57±8 54)mmHg respectively.
     在地面做L-1动作时心率、收缩压、舒张压及平均动脉血压分别为(87.29±9.23)min-1、(186.00±11.11)mmHg、(147.86±9.96)mmHg及(160.57±8.54)mmHg;
短句来源
     After reperfusion and administration of rhEDIL-8(250μg/kg),the plasma content of 6-keto-PGF_(1α) was remarkably increased and its levels significantly correlated with that of mean arterial blood pressure(r=0.746).
     给予rhEDIL-8(250μg/kg)静脉注射后血浆6~keto-PGF_(1α)含量明显升高,其血浆水平与MABP呈明显的正相关性(r=0.746);
短句来源
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  mean arterial
Mean arterial pressure (MAP) was registered on the day before surgery, before anesthetic induction, during surgery, and upon admission in the intensive care unit (ICU).
      
Improvement of organ perfusion and oxygenation is traditionally thought to rely on stabilization of mean arterial blood pressure to levels ≥70 mmHg.
      
A few hours later the patient developed profound vasodilatatory shock (mean arterial pressure >amp;lt;40mmHg) due to generalized ischemia-reperfusion injury.
      
We achieved a hemodynamic improvement (mean arterial pressure 50 to 60mmHg), which persisted for 12 hours.
      
The rate of administration of resuscitation fluids should maintain urine outputs between 0.5 and 1 ml/kg/h and mean arterial pressures of >amp;gt;70 mmHg.
      
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The intoxication from tartar emetic is presumed to be concerned with certain disorders of the central nervous system, but the study of the cerebral circulation in this aspect is still lacking. We used unanesthetized male rabbits, ligated the vertebral arteries and all the branches of the common carotid arteries except the internal carotid. Both common carotid arteries were connected to the bubble flow meter for measuring the intracranial blood flow. The averages and the standard deviations of 84 normal rabbits:...

The intoxication from tartar emetic is presumed to be concerned with certain disorders of the central nervous system, but the study of the cerebral circulation in this aspect is still lacking. We used unanesthetized male rabbits, ligated the vertebral arteries and all the branches of the common carotid arteries except the internal carotid. Both common carotid arteries were connected to the bubble flow meter for measuring the intracranial blood flow. The averages and the standard deviations of 84 normal rabbits: the mean arterial blood pressure (BP) was 107±17 mm Hg, the cerebral blood flow (CBF) 142±35 ml/100 g brain/minute, and the cerebral vascular resistance (CVR) 0.80±0.24 mm Hg/ml blood/100 g brain/minute. Intravenous injection of a dose of tartar emetic 4-6 mg/kg or three-day's intensive treatment (4 mg/kg b. i. d.) had no measurable influence on the cerebral circulation. Intravenous drip of 15-20 mg/kg produced a slowing of CBF, but no action on CVR. Intraperitoneal administration of 20 mg/kg gave only slight alterations; with increasing doses up to 80 mg/kg, the decrease of CBF and the increase of CVR became remarkable. So the therapeutic doses of tartar emetic rendered little change in the cerebral circalation, while the toxic doses yielded a reduction of CBF and a constriction of the intracranial vessels. Before the cardiac arrhythmias occurred, the CBF had been diminishing and the CVR enhancing. At the time that the cerebral circulation was in a state of very low BP, very slow CBF and very high CVR, the electro cardiogram also revealed arrhythmias. Intracarotid injections of small doses (below 4μg) of tartar emetic did not modify the cerebral circulation, whereas a large dose (0:8 mg) induced a striking rise of CBF and a precipitous drop of CVR, lasting a few minutes. Intravenous administration of sodium dimercaptosuccinate 0.25 g/kg conferred no marked action, but a dose twice the amount brought about cardiac arrhythmia together with a dwindling CBF and an augmenting CVR. Intravenous injection of atropine sulfate 1 mg/kg yielded a tendency to decreasing CBF and increasing CVR, while 2 mg/kg produced an evidently high CBF accompanied by a low CVR. Intracarotid injections of either sodium dimercaptosuccinate or atropine elicited a prompt rise of CBF and a fall Of CVR.

(一)用气泡流量計测定84只不麻醉雄兔的脑血流,頸动脉平均血压(BP)是107±17毫米汞柱,脑血流量(CBF)是142±35毫升/100克脑/分鐘,脑血管阻力(CVR)为0.80±0.24毫米汞柱/毫升血/100克脑/分鐘。在持續試驗2小时过程中比較稳定。 (二)吐酒石的作用:治疗量和三日疗法对兔脑血流无明显影响,靜脉和腹腔用藥40毫克/千克以上时,均呈現不同程度的CBF减慢和CVR增大。CVR主要反映脑血管口徑的变化,所以中毒剂量的吐酒石有收縮脑血管的作用。發生心律紊乱前,CBF已在逐漸减慢,而CVR正行加大。再繼續观察,当处于BP显著低、CBF显著慢、OVR显著大的状态时,心电圖也發現心律紊乱。 頸动脉注射吐酒石4微克以下对脑血流无影响,只有在極高的濃度(0.8毫克)时,發生CBF驟增,CVR頓时銳减,数分鐘后复原。 (三)靜脉注射二巯基丁二酸鈉0.25克/千克时,未發現对CBF和CVR的明显作用。而剂量加倍时則引起心律紊乱、CBF减慢和CVR加大。靜脉注射硫酸阿託品1毫克/千克时,有CBF减少和CVR增大的趋势。2毫克/千克时,CBF明显增多,CVR同时变小。 頸动脉注射二巯基丁二酸鈉4.8毫克或硫酸...

(一)用气泡流量計测定84只不麻醉雄兔的脑血流,頸动脉平均血压(BP)是107±17毫米汞柱,脑血流量(CBF)是142±35毫升/100克脑/分鐘,脑血管阻力(CVR)为0.80±0.24毫米汞柱/毫升血/100克脑/分鐘。在持續試驗2小时过程中比較稳定。 (二)吐酒石的作用:治疗量和三日疗法对兔脑血流无明显影响,靜脉和腹腔用藥40毫克/千克以上时,均呈現不同程度的CBF减慢和CVR增大。CVR主要反映脑血管口徑的变化,所以中毒剂量的吐酒石有收縮脑血管的作用。發生心律紊乱前,CBF已在逐漸减慢,而CVR正行加大。再繼續观察,当处于BP显著低、CBF显著慢、OVR显著大的状态时,心电圖也發現心律紊乱。 頸动脉注射吐酒石4微克以下对脑血流无影响,只有在極高的濃度(0.8毫克)时,發生CBF驟增,CVR頓时銳减,数分鐘后复原。 (三)靜脉注射二巯基丁二酸鈉0.25克/千克时,未發現对CBF和CVR的明显作用。而剂量加倍时則引起心律紊乱、CBF减慢和CVR加大。靜脉注射硫酸阿託品1毫克/千克时,有CBF减少和CVR增大的趋势。2毫克/千克时,CBF明显增多,CVR同时变小。 頸动脉注射二巯基丁二酸鈉4.8毫克或硫酸阿託品20微克后,均立卽出現CBF加快,CVR减小。

The purpose of this study was to evaluate the effects of gallbladder stretchingon cardiac performance in anesthetized dogs.We used the ratio of the pre-ejec-tion period to the left ventricular ejection time(PEP/ET)as the index of the cardiacpump function.Following control measurements,the gallbladder was stretchedfor 20 min and the PEP/ET ratio was estimated at each 5 rain intervals.Theheart rate and mean arterial pressure were calculated simultaneously.In thefirst series of experiments,it was shown that...

The purpose of this study was to evaluate the effects of gallbladder stretchingon cardiac performance in anesthetized dogs.We used the ratio of the pre-ejec-tion period to the left ventricular ejection time(PEP/ET)as the index of the cardiacpump function.Following control measurements,the gallbladder was stretchedfor 20 min and the PEP/ET ratio was estimated at each 5 rain intervals.Theheart rate and mean arterial pressure were calculated simultaneously.In thefirst series of experiments,it was shown that the heart rate and mean arterial pres-sure were increased significantly while the PEP/ET ratio did not deviate substan-tially from the control.A possible explanation for this response pattern is thatthe cardiac performance revealed by PEP/ET ratio might be influenced by someconflicting factors arising from the different origins.This suspicion was con-firmed in subsequent experiments by means of the pretreatment of animals withspecific alpha and beta adrenergic blocking agents.These results indicated thatthe afferent input arising from gallbladder stretching elicited two distinct res-ponses:(1)a positive inotropic effects resulting from beta adrenergic stimulation;(2)a pressor effect resulting from alpha adrenergic vasoconstriction.It is empha-sized that these two conflicting influences on cardiac performance could canceleach other out in many conditions and thus lead to a misunderstanding as if therewould be no close correlation between heart performance and gallbladder stret-ching.The clinical implications of this correlation were discussed.

本工作观察了胆囊牵拉(或充气)对麻醉狗的心脏泵血功能的影响,并对与此有关的肾上腺素能受体的活动作了分析。实验表明,胆囊牵拉不影响用来反映左室射血分数的 PEP/ET 值,但引起有显著意义的心率加快和平均动脉压的升高;如预先给动物注射心得安,则牵拉不再能引起心率改变,但仍引起血压的升高,同时出现泵血功能的下降;如预先给动物注射酚妥拉明,牵拉不再引起血压改变,但出现心率的加快和泵血功能的增强。实验结果说明,胆囊牵拉可能通过心脏的β-肾上腺素能受体和动脉血管的α-肾上腺素能受体影响心脏的泵血功能,而这两种作用在正常情况下有可能相互抵消,造成胆囊激惹不显著影响泵血功能的假象。对这一现象在临床上的意义作了初步讨论。

Arterial blood pressure is mainly determined by the two variables, cardiac output (CO) and total peripheral resistance (TPR). The impedance cardiography as a non-invasive method was used to investigate the hemodynamic changes in different stages of essential hypertension. 86 essential hypertensives and 35 normatensives were studied. It was revealed that with the exception of CO in stage 1 hypertension, all the hemodyna-mic parameters of three stages showed significant difference from those of the normo-tensives....

Arterial blood pressure is mainly determined by the two variables, cardiac output (CO) and total peripheral resistance (TPR). The impedance cardiography as a non-invasive method was used to investigate the hemodynamic changes in different stages of essential hypertension. 86 essential hypertensives and 35 normatensives were studied. It was revealed that with the exception of CO in stage 1 hypertension, all the hemodyna-mic parameters of three stages showed significant difference from those of the normo-tensives. Mean TPR already higher than normal in stage Ⅰ steadily increased and the highest value being fonnd in stage Ⅲ. On the contrary, mean CO was reduced as the disease progressed to the late stage. Mean arterial pressure (MAP) was positively corre-lated with TPR, and CO was inversely correlated with TPR. No correlation was found between MAP and CO. It is concluded that the increased TPR is probably the major hemodynamic change in essential hypertension.

本文报告应用非侵入性阻抗心动图,测定86例原发性高血压患者和35名正常人的心输出量和总周围阻力.结果,高血压患者与正常人的各项指标大多数有显著性差异,而且随高血压病期的进展,心输出量逐渐降低而总周围阻力显著增高.作者认为,总周围血管阻力的增高,可能是原发性高血压血液动力学变化的—个主要特征.

 
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