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necrosis foci
相关语句
  细胞坏死灶
     (1) 60.0% cases were still undergoing HBV active replication or reactivation in which HBcAg cytoplasmic and membreneous expression was often associated with the hepatic necrosis foci;
     发现:①60.0%(9/15)患者存在HBV活动性复制或HBV复制重新激活,观察到表达浆膜型HBcAg肝细胞多紧紧毗邻肝细胞坏死灶;
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  “necrosis foci”译为未确定词的双语例句
     There were clear necrosis foci in liver histopathology of 200 mg/kg and 130 mg/kg groups.
     13 0和 2 0 0mg/kg组均出现不同程度的肝脏炎症和坏死。
短句来源
     There were cle ar necrosis foci in liver histopathology of 200 and 130 mg·kg -1 groups.
     130和 2 0 0mg·kg- 1组均有不同程度的肝脏炎症和坏死。
短句来源
     High expression of tTG contributed around the necrosis foci and apoptotic cells in astrcytomas.
     在胶质母细胞瘤中,坏死和凋亡组织周围的肿瘤细胞tTG呈强阳性表达;
短句来源
     Furthermore, we found that hepatocytes containing HBV DNA or surface or core antigen expression were often close to hepatic necrosis foci, suggesting that HBV replication and its antigen(s) expression in hepatocytes could be an explanation for chronic, active and necrotic inflammation occurring in the liver of the patients in question.
     含HBV DNA的肝细胞及表达HBsAg,HBcAg的肝细胞大多毗邻灶状坏死和(或)碎屑样坏死灶,提示HBV复制及其产物表达与患者的慢性化、活动性坏死性炎症密切相关。
短句来源
     The viral antigens were expressed in cytoplasm and positive cells were located at the center of necrosis foci or surrounded them. Antigen expressions could also be seen at the sites of piecemeal necrosis.
     阳性信号位于胞浆中 ,阳性信号表达阳性细胞可位于坏死灶中或灶旁 ,碎屑样坏死处也可见该病毒抗原表达。
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  相似匹配句对
     Central necrosis of the tumor foci was seen in 44 cases.
     4 4例肿瘤内出现坏死。
短句来源
     The liver showed edema,vacuolar degeneration and foci necrosis.
     肝水肿,狄氏间隙增宽,肝细胞空泡变性及坏死;
短句来源
     And it was associated with necrosis and inflammation.
     同时与肿瘤组织内坏死及癌间质中炎症反应有关(P均<0.05)。
短句来源
     pancreas degeneration, necrosis;
     胰腺腺泡变性、坏死。
短句来源
     NATURAL PLAGUE FOCI IN YUNNAN
     云南的鼠疫自然疫源地
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  necrosis foci
The incidence and the size of coagulation necrosis foci of cardiomyocytes developed 6 h after isoproterenol injection significantly decreased with increasing in the interval between rubomycin injection and subsequent administration of isoproterenol.
      
Microthrombi could be found at small vessels in the peripheral part of necrosis foci.
      


To explore the role of HBV antigen expression and monocyte infiltrate in situ in chronic hepatitis,HBV markers and the composition and number of mo-nocytes in the liver of chronic hepatitis B were immunohistochemically located and identified,and correlated with hepatocyte necrosis It was found that hepatic necrosis frequently took place in the centre or boundary of membraneous HBsAg and/or HBcAg expression and the majority of monocytes accumulated jn the necrosis foci showed CD8+cell token in HBeAg-positive...

To explore the role of HBV antigen expression and monocyte infiltrate in situ in chronic hepatitis,HBV markers and the composition and number of mo-nocytes in the liver of chronic hepatitis B were immunohistochemically located and identified,and correlated with hepatocyte necrosis It was found that hepatic necrosis frequently took place in the centre or boundary of membraneous HBsAg and/or HBcAg expression and the majority of monocytes accumulated jn the necrosis foci showed CD8+cell token in HBeAg-positive stage of chronic active hepatitis(CAH).On the other hand,with HBeAg converted into anti-HBe positive,HBV antigen expression significantly declined,and CD4+ cells remarkably increased as compared with HBeAg positive status.These findings suggest that there may be distinct pa-thogenesis of hepatic necrosis in different stages of chronic HBV infection.

本文应用兔疫组化方法研究慢性肝炎肝细胞坏死与乙型肝炎病毒(HBV)标志及T细胞亚群的关系,结果发现:e抗原阳性(eAg+)患者肝内HBsAg与HBcAg检出率明显高于e抗原阴性(eAg-)患者;在大多数膜HBsAg或浆膜型HBcAg表达部位中间或周围伴有肝细胞坏死,聚集在eAg+慢性活动型肝炎肝细胞坏死部位的单个核细胞大多为OKT~+_8细胞,OKT~+_4细胞少见,提示:eAg+的肝细胞破坏与HBV标志膜表达及OKT~+_8细胞浸润有一定关系。

To explore etiology of acute exacerbation in severe chronic active hepatitis,HBVDNA in situ hybridization was carried out in a group of patients,combined with detection of HBV markers in serum and liver as well as intrahepatic HDAg.Four subgroups were identified based on the etiological evidence.(1) 60.0% cases were still undergoing HBV active replication or reactivation in which HBcAg cytoplasmic and membreneous expression was often associated with the hepatic necrosis foci; (2) in 20% cases with HBsAg...

To explore etiology of acute exacerbation in severe chronic active hepatitis,HBVDNA in situ hybridization was carried out in a group of patients,combined with detection of HBV markers in serum and liver as well as intrahepatic HDAg.Four subgroups were identified based on the etiological evidence.(1) 60.0% cases were still undergoing HBV active replication or reactivation in which HBcAg cytoplasmic and membreneous expression was often associated with the hepatic necrosis foci; (2) in 20% cases with HBsAg or HBVDNA,despite absence of HBcAg expression,membraneous and homogeneous HBsAg expression was also closely related to hepatic necrosis; (3) 13.3% cases were with intrahepatic HDAg; (4) the remaining single case had no HBV infection evidence.All these findings suggest that HBV active replication or reactivation was the major cause of these exacerbation in severe chronic active hepatitis.In addition,HDV superinfection accounted for over 10% exacerbation and hepatitis V and C virus superinfection may contribute to some episodes of exacerbation.

为探讨慢性活动性肝炎患者急性加重的病因,对15例患者进行肝细胞内HBV DNA(乙肝病毒脱氧核糖核酸)原位杂交研究,同时检查血清与肝内HBV标志及肝内HDAg。发现:①60.0%(9/15)患者存在HBV活动性复制或HBV复制重新激活,观察到表达浆膜型HBcAg肝细胞多紧紧毗邻肝细胞坏死灶;②20.0%(3/15)患者肝内HBsAg或(和)HBVDNA阳性,虽无HBcAg表达,但见到浆膜型HBsAg表达或含HBV DNA肝细胞与肝细胞坏死灶关系密切的表现:③13.3%(2/15)患者有HDV二重感染;④1例患者缺乏HBV感染标志。表明慢性活动型肝炎急性加重主要与HBV活动性复制、HBV复制重新激活或HBV感染持续存在有关,其次是HDV二重感染,少数病例可能存在HAV或HCV的重叠感染。

To explore HBV infection status in a group of serologic HBV markers or HBsAg negative patients with chronic hepatitis, in situ hybridization to liver HBV DNA was carried out, in conjunction with detection of intrahepatic HBsAg and HBcAg. It was found that incidence of intrahepatocellular HBV DNA, HBsAg and HBcAg was 43.48% ,38.89% and 16.67% respectively, and 65.22% among the patients had HBV markers expression in the liver.These findings indicate that over half of patients with chronic hepatitis were still...

To explore HBV infection status in a group of serologic HBV markers or HBsAg negative patients with chronic hepatitis, in situ hybridization to liver HBV DNA was carried out, in conjunction with detection of intrahepatic HBsAg and HBcAg. It was found that incidence of intrahepatocellular HBV DNA, HBsAg and HBcAg was 43.48% ,38.89% and 16.67% respectively, and 65.22% among the patients had HBV markers expression in the liver.These findings indicate that over half of patients with chronic hepatitis were still undergoing HBV infection, despite serologic HBV marker or HBsAg negative. Furthermore, we found that hepatocytes containing HBV DNA or surface or core antigen expression were often close to hepatic necrosis foci, suggesting that HBV replication and its antigen(s) expression in hepatocytes could be an explanation for chronic, active and necrotic inflammation occurring in the liver of the patients in question.

本文采用原位杂交及免疫组化方法对23例血清HBV标志或HBsAg阴性慢性肝炎患者的HBV感染状态进行了研究。发现肝内乙肝病毒脱氧核糖核酸(HBV DNA),HBsAg,HBcAg的检出率分别为43.48%、38.89%.16.67%;肝内有HBV感染依据者占65.22%,表明半数以上的血清HBV标志或HBsAg阴性患者肝内仍然存在HBV复制或基因表达。含HBV DNA的肝细胞及表达HBsAg,HBcAg的肝细胞大多毗邻灶状坏死和(或)碎屑样坏死灶,提示HBV复制及其产物表达与患者的慢性化、活动性坏死性炎症密切相关。

 
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