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acute alcoholic
相关语句
  急性酒精中毒
     Observation of the curative effect of naloxone in the treatment of 46 cases of acute alcoholic poisoning
     纳洛酮治疗急性酒精中毒46例疗效观察
短句来源
     Objective:To summarize the effect and experience of naloxone in the treatment of acute alcoholic poisoning.
     目的:总结纳洛酮(naloxone)治疗急性酒精中毒的疗效和经验。
短句来源
     The hepatocyte growth factor in the blood serum of acute alcoholic intoxication patients with severe hepatic injury was higher than that of chronic alcoholic intoxication patients with gentle hepatic injury (P<0.05),and it was significantly higher than that of control groups (P<0.01).
     急性酒精中毒患者伴有肝功能异常者血清中肝细胞再生因子水平高于慢性酒精中毒 (P <0 0 5 ) ,并显著高于正常对照组 (P <0 0 1)。
短句来源
     Method:A total of 165 acute alcoholic toxicosis cases were studied by using randomized control trial from Jan,2003 to Dec.,2005.Of them,81 cases were set in study group and the other 84 cases were in control group at random.
     方法:急性酒精中毒患者共165例随机分为治疗组81例,对照组84例,在一般治疗基础上,治疗组用醒脑静,对照组用纳洛酮。
短句来源
     Methods:The clinical data in 46 patients with acute alcoholic poisoning treated with naloxone were analyzed.
     方法:对用纳洛酮治疗的46急性酒精中毒患者的临床资料进行分析。
短句来源
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  “acute alcoholic”译为未确定词的双语例句
     Objective To study the protective effect of heme oxygenase-1 induced by hemin on acute alcoholic liver injury in rats.
     目的观察氯化血红素(hemin)对大鼠肝脏血红素加氧酶-1(HO-1)的诱导表达作用, 并探讨HO-1对大鼠急性乙醇性肝损害的防治作用。
     Objective To establish the acute alcoholic liver injury model and apply in evaluation for health care foods.
     目的 建立小鼠急性酒精性肝损伤模型 ,并将该模型应用于保护酒精性肝损伤功能的保健食品的筛选与评价。
短句来源
     Objective:To investigate the liver function damage in patients with acute alcoholic pancreatitis(AAP).
     目的:探讨酒精性急性胰腺炎(alcoholic acute pancreatitis,AAP)并发肝损害的临床特点。
短句来源
     Study on Models of Acute Alcoholic Liver Injury in vivo and in vitro
     体内外急性酒精性肝损伤模型的研究
短句来源
     OBJECTIVE: To study the effects of radix bupleuri and radix scutellariae in compatibility with the ratio of different dosage (2∶1, 1∶1, 1∶2) on acute alcoholic liver injury by regulating the system of endocrine-immunity network in rats.
     目的:从整体观念出发探讨柴胡、黄芩以不同剂量比例配伍(2∶1,1∶1,1∶2)对大鼠急性酒精性肝损伤的防治作用。
短句来源
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  相似匹配句对
     RAT MODEL OF ACUTE ALCOHOLIC LIVER INJURY
     大鼠急性酒精性肝损伤模型分析
短句来源
     Objective To study the impact of hyperlipidemia on the alcoholic acute pancreatitis.
     目的探讨高脂血症对急性酒精性胰腺炎发病及病情的影响。
短句来源
     Acute pericarditis
     急性心包炎
短句来源
     Acute Appendictics
     急性阑尾炎
短句来源
     On the Liabilities of Alcoholic Crime
     论醉酒人犯罪的刑事责任
短句来源
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  acute alcoholic
A comparison is made between patients treated by other methods, and with chlorpromazine, who were suffering from severe acute alcoholic intoxication, post-intoxication withdrawal, delirium tremens and postdelirious agitation.
      
It was found that some, especially those with an acute alcoholic hallucinosis, carried their auditory hallucinations directly into their dreams with similar content as during the waking state.
      
This combination of factors could increase susceptibility to acute alcoholic pancreatitis through a hyperstimulation mechanism.
      
Group I consisted of 11 patients who had presumed acute alcoholic pancreatitis.
      
Serum lipase and amylase ratio in acute alcoholic and nonalcoholic pancreatitis by using Dupont ACA discrete clinical analyzer
      
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AIM and METHOD:The effects of alcohol on lipopolysaccharide (LPS) eliminating actions of liver Kupffer cells in rats with chronic alcohol-induced liver injury and in patients with severe acute alcoholic hepatitis were observed.RESULTS:Ten minutes after the injection of LPS(0.2 mg/kg body weight )via portal vein, Kupffer cells in the liver of normal rats showed strong FITC fluorescent reactions to LPS, but only slight increase of FITC fluorescent reactions to LPS was found in Kupffer cells in the liver...

AIM and METHOD:The effects of alcohol on lipopolysaccharide (LPS) eliminating actions of liver Kupffer cells in rats with chronic alcohol-induced liver injury and in patients with severe acute alcoholic hepatitis were observed.RESULTS:Ten minutes after the injection of LPS(0.2 mg/kg body weight )via portal vein, Kupffer cells in the liver of normal rats showed strong FITC fluorescent reactions to LPS, but only slight increase of FITC fluorescent reactions to LPS was found in Kupffer cells in the liver tissues of chronic alcohol induced-liver injury rats. Under electron microscope, few positive reactions to LPS was found in Kupffer cells in the liver of alcohol injury rats, but positive reactions to LPS increased in sinusoidal epithelial cells and hepatocytes. FITC fluorescent intensity in Kupffer cells in the liver samples of patients with acute severe alcoholic hepatitis was significantly lower than of normal controls. CONCLUSION:Alcohol diminisk the LPS-eliminating actions of Kupffer cells in the liver, and LPS might play a role in the mechanism of liver injury induced by alcohol. 

目的和方法:应用免疫荧光及免疫电镜技术,对慢性酒精投与之实验大鼠及重症酒精性肝炎患者肝脏枯否细胞对LPS的清除功能进行了观察。结果:经门静脉注射LPS10min后,Lieber’s酒精饮食投与6周之大鼠肝脏枯否细胞内抗LPS荧光显色低于正常大鼠,电镜下枯否细胞内抗LPS阳性颗粒少见,而肝窦壁内皮细胞及肝细胞内抗LPS阳性物增多。重症酒精性肝炎患者肝穿组织枯否细胞内抗LPS荧光显色亦显著低于正常对照。结论:酒精刺激后肝脏枯否细胞对LPS的清除功能降低,且内毒素可能参与酒精致肝损伤的发生机制

To observe the effect of NX for treatment of acute alcoholic poisoning, 67 AAP patients were divided into two group. 30 patients in the treatment group were treated with NX plus furosemide and insulin until them consciousness. 37 patients in the controlgroup were treated with energymist plus insulin and furosemide. Result shown: for mild intoxication, the average time length of patients returning to consciousness was 31 minutes in NX group and 150 minutes in control-group ; for the severe intoxication,...

To observe the effect of NX for treatment of acute alcoholic poisoning, 67 AAP patients were divided into two group. 30 patients in the treatment group were treated with NX plus furosemide and insulin until them consciousness. 37 patients in the controlgroup were treated with energymist plus insulin and furosemide. Result shown: for mild intoxication, the average time length of patients returning to consciousness was 31 minutes in NX group and 150 minutes in control-group ; for the severe intoxication, the time length was respectively 7.5 hours and 24.8 hours. This proved that NX is effective and quick for treatment of AAP patients.

观察纳洛酮(NX)对急性酒精中毒的疗效。把67例酒精中毒者随机分为两组,30例治疗组在与对照组同样治疗的基础上加用NX入小壶治疗,直至病人清醒;37例对照组用能量合剂加胰岛素配合速尿治疗。结果,治疗组与对照组的平均清醒时间分别为:轻度中毒31min与150min;重度中毒7.5h与24.8h。证明:NX对急性酒精中毒治疗有效,且疗效快。

Objective To observe and follow-up the ocular changes of acute or chronic alcoholic with visual electrophysiologic test. Methods Scotopic ERG,P-or F-VEP and perimetry, fluorescine fundus angiography examination were performed for 18 eyes (9 cases) with alcoholic ocular changes. Results 6 eyes (3 cases) presented normal ERG and amplitudes reduce of F-or P-VEP P100 in acute alcoholic,4 eyes(2 eyes) of them returned to normal after treatment(66. 7% ) and 2 eyes(l cases) showed permanent changes. Visual field...

Objective To observe and follow-up the ocular changes of acute or chronic alcoholic with visual electrophysiologic test. Methods Scotopic ERG,P-or F-VEP and perimetry, fluorescine fundus angiography examination were performed for 18 eyes (9 cases) with alcoholic ocular changes. Results 6 eyes (3 cases) presented normal ERG and amplitudes reduce of F-or P-VEP P100 in acute alcoholic,4 eyes(2 eyes) of them returned to normal after treatment(66. 7% ) and 2 eyes(l cases) showed permanent changes. Visual field deficiency, irreversible abnormalitis of a,b wave amplitudes and P-VEP P100 latency were found in chronic alcoholics. Conclusion Acute or chronic alcoholism can damage retinal pigment layer, ERG and VEP can provide useful basis.

目的 对急慢性酒精中毒眼部损害者进行视觉电生理观察,了解酒精中毒性视力损害的机制。方法  对急性酒精中毒眼部损害者3例6眼和慢性中毒者6例12眼行暗适应视网膜电图(ERG),视觉诱发电位(VEP)和视野,眼底荧光造影等检查并进行 1月~2年的随访。结果 显示急性中毒3例6眼中暗适应 ERG正常,F-VEP或 P-VEP P100波严重下降,2例4眼治疗后完全恢复(66.7%),l例2眼成为永久性损害。经证实未恢复的1例2眼呈弥漫性色素上皮损害,眼底荧光血管造影显示全部眼底的透见荧光和色素游离的遮蔽荧光相间造影像,黄斑区视网膜尤甚。经2年随访无明显改善。慢性中毒6例 12眼有不同程度的视野缺损,暗适应 ERGa,b波、P-VEP P100波潜伏值均出现永久性异常,眼底荧光血管造影证实视网膜后极部数簇遮蔽荧光和透见荧光像。结论  认为急慢性酒精中毒可首先造成视网膜色素上皮的弥漫性或黄斑区的损害,而视神经损伤可能发生于上述改变之后;视觉电生理检测可对其损害提供可靠的诊断和跟踪随访依据。

 
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