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cirrhosis patients
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  肝硬化患者
     Detection of platelet Ca~(2+i), CD_(62)P, CD_(63) and plasma CD_(62)P in cirrhosis patients
     肝硬化患者血小板胞内Ca~(2+)浓度、CD_(62)P、CD_(63)及血浆CD_(62)P的检测
短句来源
     Results:The contents of plasma LPO were (4 61±0 69)μmol/L and (5 34± 1 33)μmol/L in primary hepatocarcinoma and liver cirrhosis patients respectively,they were raised markedly as compared with control group (2 48±0 35)μmol/L (all P <0 001);
     结果:原发性肝癌、肝硬化患者血浆LPO含量分别为(4.61±0.69)μmol/L和(5.34±1.33)μmol/L,较正常对照组〔(2.48±0.35)μmol/L〕明显升高(P均<0.001);
短句来源
     Results:SS was (39.5 ± 9.3)μg/L, (24.1 ± 4.4)μg/L,and (20.3 ± 7.6)μg/L in healthy persons,duodenal ulcer and hepatic cirrhosis patients respectively.
     结果:血浆SS正常人为(39.5±9.3)μg/L,Du组为(24.1±4.4)μg/L,肝硬化患者为(20.3±7.6)μg/L。
短句来源
     CLINICAL SIGNIFICANCE OF TNFα、IL-6、HA、C_(Ⅳ)OF LIVER CIRRHOSIS PATIENTS
     肝硬化患者TNFα、IL-6、HA、C_(Ⅳ)水平测定的临床意义
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     In the liver cirrhosis patients, the levels of plasma insulin before (fasting) and 2 h after the OGTT were 12.23±5.4 mU/L and 105.16±40.70 mU/L, respectively, which were significantly higher than those in the healthy controls (P <0.05).
     而胰岛素测定结果可见肝硬化患者空腹及OGTT2h后胰岛素水平分别为:12.23±5.4mU/L、105.16±40.70mU/L,均明显高于对照组;
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  肝硬化
     Detection of platelet Ca~(2+i), CD_(62)P, CD_(63) and plasma CD_(62)P in cirrhosis patients
     肝硬化患者血小板胞内Ca~(2+)浓度、CD_(62)P、CD_(63)及血浆CD_(62)P的检测
短句来源
     Results1 Ghrelin was significantly elevated in liver cirrhosis patients compared with healthy controls (3.4±3.3 ng/ml vs 2.1±1.4 ng/ml,P<0.05).
     结果肝硬化组血浆ghrelin水平显著高于正常对照组(3.4±3.3 ng/ml vs 2.1±1.4 ng/ml;P<0.05)。
短句来源
     3 Leptin was significantly elevated in liver cirrhosis patients compared with healthy controls (6.3±8.0 ng/ml vs 1.2±1.6 ng/ml,P<0.001).
     肝硬化组血清瘦素水平显著性高于正常对照组(6.3±8.0 ng/ml vs 1.2±1.6 ng/ml,P<0.001);
短句来源
     Results:The contents of plasma LPO were (4 61±0 69)μmol/L and (5 34± 1 33)μmol/L in primary hepatocarcinoma and liver cirrhosis patients respectively,they were raised markedly as compared with control group (2 48±0 35)μmol/L (all P <0 001);
     结果:原发性肝癌、肝硬化患者血浆LPO含量分别为(4.61±0.69)μmol/L和(5.34±1.33)μmol/L,较正常对照组〔(2.48±0.35)μmol/L〕明显升高(P均<0.001);
短句来源
     The value of ISI in liver cirrhosis patients was significantly lower than that in the healthy controls (1.47±0.54 vs 2.73±0.62, P <0.05).
     肝硬化组ISI显著低于对照组(1.47±0.54vs2.73±0.62,P<0.05).
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  “cirrhosis patients”译为未确定词的双语例句
     the erythrocyte SOD activity was (1 695 5±229 7)U/g and (2 651 5±445 6)U/g in primary hepatocarcinoma and liver cirrhosis patients respectively,they were decreased obviously as compared with control group (3 330 2±552 2)U/g (all P <0 001).
     2组红细胞SOD活性分别为(1695.5±229.7)U/g和(2651.5±445.6)U/g,较正常对照组〔(3330.2±552.2)U/g〕明显降低,P均<0.001;
短句来源
     Results The abnormal rate of intelligent mental test was 58.6% in cirrhosis patients ,which was much higher than that of EP test, and the positive rate was 68.9%(80/116).
     结果心理智能测试异常率为58.6%,显著高于EP检测异常率25.9%,两者总异常率达68.0%(80/116例)。
短句来源
     5. AKP,γ—GT increased continually and AFP was higher than nomal in hepatitis cirrhosis patients.
     5)肝炎肝硬变患者AKP、γ—GT持续增高,AFP持续高于正常值。
短句来源
     The prevalence of SHE in cirrhosis patients was 51.3%. Highly statistical significance was found ( x2 = 23.910, P < 0.01) among the Child-pugh A, B, C groups (39.9%, 55.2% and 71.8%).
     Child—pugh A、B、C级患者的患病率分别为39.9%(75/188)、55.2%(79/143)、71.8%(56/78),三组间差异有显著性,x2=23.910,P<0.01。
短句来源
     Objective To analyze the actions of transforming growth factor β 1 (TGF-β 1) and tumor necrosis factor α(TNF-α) in the liver fibrosis formation in chronic viral hepatitis patients and liver cirrhosis patients.
     目的 分别测定慢性病毒性肝炎、肝炎肝硬变患者血清转化生长因子 β1(TGF - β1)、肿瘤坏死因子(TNF -α) ,探讨两者在肝纤维化中的作用。
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  cirrhosis patients
A total of 125 patients were classified into three groups: inactive HBsAg carriers, inactive liver cirrhosis patients, and resolved chronic HBV-infected patients with loss of HBsAg.
      
These results suggest that the combination of EVL plus PSE can prevent the recurrence of varices, progression to variceal bleeding, and death in cirrhosis patients with esophageal varices and thrombocytopenia.
      
In a prospective study, 84 cirrhosis patients with esophageal varices and thrombocytopenia (platelet count >amp;lt; 50,000/mm3) underwent EVL plus PSE (N = 42) or EVL alone (N = 42).
      
The aim of this study was to assess the efficacy of the combination of endoscopic variceal ligation (EVL) and partial splenic embolization (PSE) compared with EVL alone in cirrhosis patients with thrombocytopenia.
      
Improved Prognosis of Cirrhosis Patients with Esophageal Varices and Thrombocytopenia Treated by Endoscopic Variceal Ligation Pl
      
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Yeast cells were used for examining phagocytosis and intraccllular bacteriocidal function of neutrophils in 44 cases of cirrhosis, 10 chronic persistent hepatitis and 38 normal subjects as controls. The mean value of phagocytosis and killing rates of ncutrophils were 23 ± 7 and 39 ± 3 respectively in controls, and 15±5 and 30±7 respectively in the cirrhosis The values were significanlly lower in the cirrhosis and much higher in chronic hepatitis than those in controls, but there was no marked...

Yeast cells were used for examining phagocytosis and intraccllular bacteriocidal function of neutrophils in 44 cases of cirrhosis, 10 chronic persistent hepatitis and 38 normal subjects as controls. The mean value of phagocytosis and killing rates of ncutrophils were 23 ± 7 and 39 ± 3 respectively in controls, and 15±5 and 30±7 respectively in the cirrhosis The values were significanlly lower in the cirrhosis and much higher in chronic hepatitis than those in controls, but there was no marked difference among all kinds of cirrhosis. The effect of liver cirrhosis on the function of ncutrophils is still not clear. According to the detection of TgA (15 cases) and C3 ( 10 cases), it was found that the level of IgA was significanlly higher and C3 was much lower than those in controls. Tin's indicates that the susceptibility of cirrhosis patient is due to the abnormality in phagocytosis and killing ability of ncutrophils, which may be correlated with the increase in IgA and/or decrease in C3.

本文用普通酵母菌对44例门脉性肝硬化患者、10例慢性迁延型肝炎患者以及38例正常人进行中性粒细胞吞噬及细胞内杀菌功能试验,结果,38例正常人中性粒细胞吞噬率及杀伤率均值分别为23±7及39±3,而44例肝硬化患者即为15±5及30±7,明显低于正常对照组(P<0.001).而24例失代偿期肝硬化与12例代偿期肝硬化患者,8例肝硬化合并肝癌者与36例未合并肝癌肝硬化者吞噬及杀伤功能无差异.慢迁肝患者吞噬及杀菌能力高于正常对照组(P<0.05),但高于肝硬化组(P<0.001).肝硬化对中性粒细胞功能的影响机理尚不清楚.本组44例肝硬化患者中15例检测了IgA及10例检测了C_3含量,结果表明,15例患者IgA高于正常值,10例C_3含量低于正常值具有统计学意义(P<0.001).由此推测,本组肝硬化患者吞噬及杀菌功能异常似与IgA升高和(或)C_3降低有关.IgA升高抑制了中性粒细胞吞噬作用抑或与抗原形成抗原抗体复合物,后者封闭了中性粒细胞Fc和C_3受体,致使被调理的微生物不能被中性粒细胞所吞噬或杀伤.C_3b由C_3裂解而来,C_3降低势必导致C_3b减少,结果微生物因C_3b减少而不能被调理,不能被中性粒...

本文用普通酵母菌对44例门脉性肝硬化患者、10例慢性迁延型肝炎患者以及38例正常人进行中性粒细胞吞噬及细胞内杀菌功能试验,结果,38例正常人中性粒细胞吞噬率及杀伤率均值分别为23±7及39±3,而44例肝硬化患者即为15±5及30±7,明显低于正常对照组(P<0.001).而24例失代偿期肝硬化与12例代偿期肝硬化患者,8例肝硬化合并肝癌者与36例未合并肝癌肝硬化者吞噬及杀伤功能无差异.慢迁肝患者吞噬及杀菌能力高于正常对照组(P<0.05),但高于肝硬化组(P<0.001).肝硬化对中性粒细胞功能的影响机理尚不清楚.本组44例肝硬化患者中15例检测了IgA及10例检测了C_3含量,结果表明,15例患者IgA高于正常值,10例C_3含量低于正常值具有统计学意义(P<0.001).由此推测,本组肝硬化患者吞噬及杀菌功能异常似与IgA升高和(或)C_3降低有关.IgA升高抑制了中性粒细胞吞噬作用抑或与抗原形成抗原抗体复合物,后者封闭了中性粒细胞Fc和C_3受体,致使被调理的微生物不能被中性粒细胞所吞噬或杀伤.C_3b由C_3裂解而来,C_3降低势必导致C_3b减少,结果微生物因C_3b减少而不能被调理,不能被中性粒细胞吞噬及杀伤.因此本试验也提示了,肝硬化患者易感染性与他们中性粒细胞吞噬及杀菌功能减退有一定的联系.

In this work, C-13-methacetin contents in serum were measured in several normal adults and hepatic patients by isotope reverse dilution analysis using GC-MS technique. The result showed that the serum C-13-methacetin content of hepatic cirrhosis patients (1.050±SEM 0.227 μg/ml) was singnificantly higher than normal (0.209± SEM 0.038μg/ml). Therefore it is clear that the delay of 13CO2 elimination in hepatic patients is mainly caused by the malfunction of liver in oxidizing mathacetin, and the influence...

In this work, C-13-methacetin contents in serum were measured in several normal adults and hepatic patients by isotope reverse dilution analysis using GC-MS technique. The result showed that the serum C-13-methacetin content of hepatic cirrhosis patients (1.050±SEM 0.227 μg/ml) was singnificantly higher than normal (0.209± SEM 0.038μg/ml). Therefore it is clear that the delay of 13CO2 elimination in hepatic patients is mainly caused by the malfunction of liver in oxidizing mathacetin, and the influence of absorption rate is negligible.

我们过去的工作表明,口服~(13)C-美沙西汀后,呼出气中~(13)CO_2的排出因肝功能障碍而延迟。这种延迟有两个可能的原因,一是肝细胞氧化美沙西汀的能力降低,二是肝功能障碍导致的肠吸收美沙西汀变慢。为此,用GC-MS技术以同位素反稀释法测定了口服~(13)C-美沙西汀15分钟后,血清中该标记物的浓度。测定结果。肝硬化患者血清~(13)C-美沙西汀含量显著高于正常人。证明肠吸收不良对~(13)C-美沙西汀呼气试验的结果无明显干扰,~(13)CO_2的排出延缓主要反映肝功能障碍。此外,血清含量的测定本身也有反映肝功能障碍的作用。

It was observed that in 81.5% of 140 cases with liver cirrhosis, the serum was HBVM positive, and the liver biopsy showed characteristic features of CAH. HBsAg could be detected with VB stains in 77.6% of HBVM seropositive cases, 66.7% of HBsAg seronegative cases, and 60.0% of the cases whose serum HBVM had not been examined. There was no significant difference among them statistically. It was our belief that negative serum HBsAg did not rule out the posibility of HBV infection in liver cirrhosis patients....

It was observed that in 81.5% of 140 cases with liver cirrhosis, the serum was HBVM positive, and the liver biopsy showed characteristic features of CAH. HBsAg could be detected with VB stains in 77.6% of HBVM seropositive cases, 66.7% of HBsAg seronegative cases, and 60.0% of the cases whose serum HBVM had not been examined. There was no significant difference among them statistically. It was our belief that negative serum HBsAg did not rule out the posibility of HBV infection in liver cirrhosis patients. Our observations suggested that there might be a very close relationship between HBV infection and liver cirrhosis in our country, and liver cirrhosis was the outcome of HBV infection. Therefore, in the prevention of liver cirrhosis it should be emphasized that HBV infection should be prevented and CAH well treated.

报告经病理确诊为肝硬变140例,证明HBVM阳性占81.5%,HE染色均见慢活肝病理特点,Victoria blue染色在血清HBVM阳性、HBsAg阴性和未查HBVM病例中,肝组织内能检出HBsAg者分别为77.6%、66.7%和60.0%,三者无明显差异。说明肝硬变患者血清HBsAg阴性并不能除外HBV感染可能。因此,认为我国肝硬变与HBV感染间存在密切因果关系,是HBV感染后慢活肝的一种结局,预防肝硬变主要应在预防HBV感染和慢活肝的治疗。

 
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