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   myc 的翻译结果: 查询用时:0.163秒
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myc     
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  myc基因
     Results\ The positive rate of bcl 2 and C MYC gene expression in colorectal carcinoma and adenoma were significantly higher than that in normal colorectal mucosa ( χ 2= 4.20 - 20.66 , P <0.05). The positive rate of bcl 2 and C MYC gene expression in colorectal carcinoma was also significantly higher than that in colorectal adenoma ( χ 2 = 9.40 , 8.75 , P <0.05).
     ③结果大肠癌和大肠腺瘤bcl-2和C-MYC基因阳性表达率均显著高于正常大肠黏膜(χ2=4.20~20.66,P<0.05),大肠癌bcl-2和C-MYC基因阳性表达率显著高于大肠腺瘤(χ2=9.40,8.75,P<0.05);
短句来源
     Positive expression of c myc in grade 1 to 2 was 68.2% (15/22), and 27.3% (3/11) in grade 1 (P<0.05).
     ⅡⅢ度睑板腺癌c myc基因阳性表达率为 68.2 % ( 15 / 2 2 ) ,Ⅰ度为 2 7.3 % ( 3 / 11) ,P <0 .0 5 ;
短句来源
     The expression intensity of c myc gene was markedly higher in GC+DENA group that in DENA group.
     cmyc基因表达强度GC+DENA组明显高于DENA组。
短句来源
     Results The rates of positive staining for p16, p53 and c myc were 34.8%, 95.7% and 100% for malignant GST,and 45.5%, 77.3% and 90.9% for borderline GST,and 65.4%, 73.1% and 65.4% for benign GST respectively.
     结果  71例GST中p1 6、p53、c myc基因蛋白阳性表达率分别为 :恶性 34 .8%、95 .7%、1 0 0 % ; 交界性 45 .5 %、77.3 %、90 .9% ;
短句来源
     In colorectal carcinoma, the rate of bcl 2 and C MYC gene expression had no correlation with the age and sex of patients, tumor histologic grade, lymph node metastasis, the depth of invasion and Dukes' stages of tumors ( χ 2=0- 1.56 ,P > 0.05 ).
     bcl-2和C-MYC基因在大肠癌中的表达率与病人的性别、年龄、组织类型、浸润深度、淋巴结转移及Dukes分期均无明显关系(χ2=0~1.56,P>0.05)。
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  myc蛋白
     Relationship between apoptosis and cell proliferation and expression of Rb, bcl 2 and c myc proteins in human breast cancer
     乳腺癌中凋亡与细胞增殖及Rb、bcl-2、c-myc蛋白表达的关系
短句来源
     P53,Cmyc expressions of 47 mice with leukemia were significantly higher than those of control group(P<0.05).
     47只白血病小鼠P53、C myc蛋白阳性表达率均比正常对照组高(P<0.05);
短句来源
     Results:The rates of positive immunostaining of cyclin D1,P53 and c myc in gastric carcinoma were 32.75% ,25.40% and 52.38%,respectively.
     结果 :63例胃癌中cyclinD1、P53及c myc蛋白染色阳性率分别为 32 .75 %、2 5 .40 %及 52 .38% ;
短句来源
     Methods After treatment of HepG 2 cells with 0~1.5 g/L matrine for 3 d, the protein expressions of AFP, PCNA, wp53, Rb, N ras and c myc were detected with immunocytochemical method, mRNA expressions of wp53, c myc with in situ hybridization;
     方法  0~ 1 5g/L苦参碱作用HepG2 细胞 3d ,免疫组化检测AFP、PCNA、wp5 3、Rb、N ras、c myc蛋白的表达 ,原位杂交法检测wp5 3、c mycmRNA的表达 ,真彩色图像定量分析检测结果 ,Microsoft-Excel软件统计学处理结果 ;
短句来源
     P53,Cmyc expressions of 23 mice without leukemia didn't have difference with control group(P>0.05).
     23只未形成白血病的实验组小鼠P53、C myc蛋白阳性表达率均与正常对照组无显著差异(P>0.05).
短句来源
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  myc
     The expression intensity of c myc gene was markedly higher in GC+DENA group that in DENA group.
     cmyc基因表达强度GC+DENA组明显高于DENA组。
短句来源
     Conclusion:As2O3 can induce GLC 82 cell apoptosis mainly with regulation of c myc,p53 and p16 gene expression.
     结论:As2 O3 能显著抑制GLC82 细胞生长、诱导细胞凋亡,并主要通过调节cmyc,p16 和p53 等基因的表达来实现。
短句来源
     Detection of P53 and c myc with immunohistochemical technique were carried out for 34 surgical specimens of malignant tumor of adrenal gland,25 adrenal benign tumors and in 12 adrenal tissues free of tumor.
     为探讨肾上腺肿瘤发病机理,为鉴别肿瘤良恶性及其分级分期提供依据,收集34例肾上腺恶性肿瘤应用SP法作P53及cmyc免疫组化检测,并与25例肾上腺良性瘤及12例非瘤肾上腺组织对比。
短句来源
     Methods The pathological features of EPS were observed in 17 cases, and the expressions of HCG, HPL, EMA, PRL, PLAP, Vim, actin, c erbB 2 and c myc were analyzed immunohistochemically.
     方法:对17 例经病理检查诊断为EPS的病例进行病理学形态观察,采用免疫组化方法标记滋养细胞HCG、HPL、EMA、PRL、PLAP、Vim 、actin、cerbB2 和cmyc
短句来源
     Expression of c_fos and c_myc were inhibited and expression of p53 was enhanced in the exercise group.
     运动组cfos和cmyc明显被控制,p53表达增强。
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  myc表达
     Results Compared with control, aM2 remarkably reduced Myc expression (by 37.6%), while aM1 only slightly reduced the expression down to 98.8%, and in contrast, aM3 elevated Myc expression by 21.7%.
     结果 与对照组相比,aM2使c-myc表达量减少了37.6%,aM1为对照组的98.8%,而aM3却使c-myc表达增高21.7%;
短句来源
     Results 1.5 3.0 g/L Matrine could induce HepG 2 apoptosis and up regulate the expression of apoptotic genes wp53, bax, Fas and down regulate the expression of anti apoptotic genes bcl 2, c myc.
     结果  1.5~ 3 .0g/L苦参碱可诱导HepG2 凋亡 ,并且使凋亡相关基因wp5 3 ,bax ,Fas表达上调 ,而抗凋亡基因bcl 2 ,c myc表达下调。
短句来源
     Results The results showed that the positivity rates of p53, pzlras, c myc and PCNA were 78 8%, 51 5%,81 8% and 97% respectively.
     结果:p53表达为78.8%,p21ras表达为51.5%,c-myc表达为81.8%,PCNA表达为97%。
短句来源
     Objetive To study the relationship between apoptosis induced by etoposide in PC 3 cells and the expression of bcl 2 and c myc oncogene.
     目的 研究 VP- 1 6诱导 PC- 3细胞凋亡和癌基因 bcl- 2及 C- myc表达的关系。
短句来源
     The expression of survivin was significantly correlated with the expression of c myc in DTC ( P <0.01).
     DTC中Survivin和c myc表达呈明显正相关 (P <0 .0 1)。
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      myc
    Such are the suggested effects on telomerase of Myc, p53, Waf1, protein kinases B and C, Wnt5A, TGFβ, WT1, and estrogens.
          
    However, Myc, p53, WT1, estrogens, protein kinases B and C, and TGFβ can also directly influence telomerase independently of the G1-S checkpoint mechanism.
          
    To study some additional factors necessary for such transformation, c-myc and N-rasAsp12 were consecutively introduced into REF52 cells by retroviral infection, and the cell cultures obtained were analyzed.
          
    The proliferating-cell gene cluster included MET, VIM, MYC, TOP2A, PCNA.
          
    In pCI.ori-neo, chromosomal ori was from the human c-myc locus.
          
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    In apple growing region of Liaoning Province,Glomerella cingulata,the causal fungus of apple bitter rot,overwintered as dormant myc-elium in small mummified fruits, dead fruiting spurs,and rough-skinedt wigs on trees.Conidia that had remained on infected fruit mummiesfailed to germinate in the next spring.Infected fallen fruits on ground-under fruit trees played little role as source of primary infection.Und-er the climatic conditions of southern Liaoning,conidia production anddispersion started from mid-June,and...

    In apple growing region of Liaoning Province,Glomerella cingulata,the causal fungus of apple bitter rot,overwintered as dormant myc-elium in small mummified fruits, dead fruiting spurs,and rough-skinedt wigs on trees.Conidia that had remained on infected fruit mummiesfailed to germinate in the next spring.Infected fallen fruits on ground-under fruit trees played little role as source of primary infection.Und-er the climatic conditions of southern Liaoning,conidia production anddispersion started from mid-June,and young fruits were infected short-tlg after petal fall, The causal fungus penetrated into the fruits thro- ugh intact epidermis. The incubation period lasted about one month du-ring the early fruit stage.Disease incidence and development covered aperiod from mid-July to mid-September. Based on the results obtainedfrom field experiments, the following spray schedule was recommerdedas practicle: spraying Bordeaux Mixture (1:2:200) or Zn-Cu Lime Mixt-ure (Zn sulfate 0.5 kg,Cu sulfate 0.5 kg, Lime 2 kg,Water 200) thricefrom mid-June at an intervals of about 20 days. The first spray shouldbe performed immediately after conidia dispersion as detected with sporetrapping method.

    在辽宁地区,苹果炭疽病菌主要以菌丝在苹果树上的小僵果、死果台、粗皮、爆皮枝等部位越冬。翌年在适宜温湿度条件下产生分生孢子,进行传播侵染。越冬后的分生孢子已失去萌发力,落地病果在初侵染中不起什么作用。病菌可以直接穿透表皮侵入果实。苹果在幼果期即感病,此时不抗侵入但抗扩展,潜育期长达一个月左右。七月中旬以后病害大量发生,直至9月中旬持续为害。根据果园中病菌孢子出现期,约在6月15至25日喷施第一次药,以后每隔20天左右连续喷施160~200倍波尔多液或锌铜石灰液三次,可以有效地控制炭疽病的为害。1964年和1965年,在辽宁地区进行大面积防治试验表明效果良好,可以大面积推广。

    To determine the association of fragile sites with leukemias and lymphomas,we examined 22 patients with leukemia or lymphoma (6 Hodgkin lymphomas,6 non-Hodgkin lymphomas,5 acute lymphocytic leukemia,3 acute nonlymphocytic leukemia,2 lymphosarcoleukemia) and 15 normal controls ranging in age from 4 to 56 years.The results were: (1) The average chromosome structural aberration rate of patients(12.98%) was much higher than that of the control group(0.73%).Fifteen cancer patients carried fragile sites,12 of them...

    To determine the association of fragile sites with leukemias and lymphomas,we examined 22 patients with leukemia or lymphoma (6 Hodgkin lymphomas,6 non-Hodgkin lymphomas,5 acute lymphocytic leukemia,3 acute nonlymphocytic leukemia,2 lymphosarcoleukemia) and 15 normal controls ranging in age from 4 to 56 years.The results were: (1) The average chromosome structural aberration rate of patients(12.98%) was much higher than that of the control group(0.73%).Fifteen cancer patients carried fragile sites,12 of them carried multiple fragile sites,but none of the controls carried any.There was a statistically significant difference between the two groups (P<0.005).(2) These patients carried 21 autosomal fragile sites (including 14 constitutive fragile sites and 7 heritable fragile sites): 1q44,2q11,2q23,2q37,3p14,4q31,5q31,6q26,8q22,8q24,9q13,10q22,10q23,10q25,11q13,12q13,13q34,14q13,14q24,16p12 and 16q22.(3) Eight breakpoints were located at the bands where oncogenes exist: 1p31 and Blym-1,2p23 and Nmyc,6q23 and myb,8q22 and mos.8q24 and myc,11q13 and bcl-1,15q26 and fes,17q21 and erbA.(4) Four breakpoints were near the fragile sites: 1p32 near fra(1p31),2p23 near fra(2p24),7p15 near fra(7p14),7q22 near fra(7q21).One breakpoint(1p12) was near the Nras(lp13).Five of the fragile sites (8q22,11q13,12q13,16p12 and 16q22) correspond with cancer breakpoints.The role of fragile sites in carcinogenesis is not yet known,but according to our results the association of fragile sites with leukemias and lymphomas is evident which deserves further investigation.

    对22例白血病、淋巴瘤病人和15例正常人进行了外周血淋巴细胞染色体脆性部位检测。结果表明,病人组的染色体畸变率、脆性部位检出率显著高于正常对照。通过G显带能准确定位的94个断点中包括了21种常染色体脆性部位,8个与癌基因在同一区带的断点。以上结果提示脆性部位同白血病、淋巴瘤之间有一定的相关。

    A recently devised protocol was used to obtain macrophage (M) lines with a variety of derived DNA to see if different oncogenes would lead to generation of M lines with different phenotypes. SV 40 DNA, cellular myc genes, U 937 DNA and Insulin gene have been used and 7 cell lines obtained. All these lines show heterogeneity in the cell phenotype and in each case there are Macrophage-like cells. They all secrete collagenase and lysozyme. 30-40% of these lines have Fc receptor and Fc receptor mediated phagocytosis...

    A recently devised protocol was used to obtain macrophage (M) lines with a variety of derived DNA to see if different oncogenes would lead to generation of M lines with different phenotypes. SV 40 DNA, cellular myc genes, U 937 DNA and Insulin gene have been used and 7 cell lines obtained. All these lines show heterogeneity in the cell phenotype and in each case there are Macrophage-like cells. They all secrete collagenase and lysozyme. 30-40% of these lines have Fc receptor and Fc receptor mediated phagocytosis and also complement receptor and phagocytosis. Heterogeneity within the lines is probably due to the fact that the lines are not cloned yet, It is supposed that this protocol can be used not only to derive M lines but also T and B cell lines.

    本文报告应用转染技术建立巨噬细胞传代株,探讨应用不同来源的DNA获得具有不同功能特性的巨噬细胞株,应用SV40DNA、C-myc基因,U937DNA和胰岛素基因已获得7株细胞,经鉴定它们均有巨噬细胞的特性,即可分泌胶元酶、溶菌酶,其中有30—40%的细胞带有Fc受体,并有经Fc受体介导的吞噬功能。细胞株内细胞功能各异是因为细胞株尚未克隆化。从本试验结果看不仅经转染方法可获得巨噬细胞株,而且有可能经此法获得B或T淋巴细胞传代株。

     
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