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myocardial ischemic damage
相关语句
  心肌缺血损伤
     ② After the last gastric perfusion for 1 hour the rats in the isoproterenol group and fenofibrate group were given 5 mg/kg isoproterenol with intraperitoneal injection to replicate acute myocardial ischemic damage models.
     ②末次灌胃后1h异丙肾上腺素组和非诺贝特组给予异丙肾上腺素5m g/kg腹腔注射复制急性心肌缺血损伤模型;
短句来源
     Relationship between the activation of peroxisome proliferate-activated receptor alpha and the expression of tumor necrosis factor alpha in acute myocardial ischemic damage
     急性心肌缺血损伤中过氧化物酶体增殖物激活受体α活化与肿瘤坏死因子α表达的关系
短句来源
     34 healthy adult Zelanian rabbits are used in this experiment. Establish the models of myocardial ischemic damage established by ligating the left anterior descending branch of the coronary artery. The autologous skeletal muscle myoblast are cultured and amplified in vitro and are marked with BrdU, (3.5-5) ×107cells are transplanted into the area of the autologous acute myocardial infarct.
     本实验采用健康成年新西兰兔34只,结扎冠状动脉左前降支建立心肌缺血损伤模型,体外培养扩增自体骨骼肌成肌细胞并用BrdU标记,将(3.5~5)×10~7细胞移植到自体急性心肌梗死区域。
短句来源
     Objective To investigate the protective mechanisms from myocardial ischemic damage of antihypertensive factor (AHF).
     目的 探讨抗高血压因子 (AHF)对心肌缺血损伤保护的机理。
短句来源
     Experimental results showed that 10mg/kg×20 of above three anti-oxidative components extracted from Chinese herbal medicines could also significantly decrease the leakage of LDH from myocardium and content of myocardium MDA, suggesting protective effect ou myocardial ischemic damage.
     结果显示上述3种中药抗氧化组分可明显地减少缺血小鼠心肌 LDH 漏出,降低心肌 MDA 含量,对实验性心肌缺血损伤有很好的保护作用。
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  “myocardial ischemic damage”译为未确定词的双语例句
     13.1%~26.3% cases are complicated with pericarditis,uremic myocardiosis and myocardial ischemic damage after hemodialysis.
     13.1%~ 2 6 .3%并发透析后心包炎、尿毒症性心肌病和心肌缺血性损伤等心脏并发症
短句来源
     Conclusions Em、Em/Am、QEm、TEm and IVRTm measured with pulsed DTI are promising indices for the quantitative assessment of global left ventricular diastolic dysfunction in patients with coronary artery disease. Em and IVRTm may even indicate the severity of myocardial ischemic damage.
     结论脉冲DTI所测Em、Em/Am、QEm、TEm及IVRTm均可反映冠心病患者左室整体舒张功能异常,Em和IVRTm尚可反映心肌缺血损害的严重程度。
短句来源
     Methods AHF was used to protect the models (Wistar rats) of myocardial ischemic damage induced by isoproterenal(ISO, 80 mg/kg s. c). The influence of AHF on the contents of myocardial molondialdehyde(MDA), myocardial calcium and TXB 2 in plasma and 6-keto-PGF 1a was observed.
     方法 以异丙肾上腺素性心肌损伤大鼠为模型 ,观察AHF对其心肌丙二醛、钙含量、血栓素B2 和 6-酮 -前列腺素F1a( 6-keto -PGF1a)的影响。
短句来源
     Protective effect of CD59 on the myocardial ischemic damage mediated by complements
     CD59对补体介导的心肌缺血损伤的保护作用
短句来源
     AIM: To observe the activation of peroxisome proliferate-activated receptor α(PPARα) by fenofibrate of agonists of PPARα after pre-application, and the effects on tumor necrosis factor alpha (TNF-α) in acute myocardial ischemic damage rats.
     目的观察预先应用过氧化物酶体增殖物激活受体α的激动剂非诺贝特活化过氧化物酶体增殖物激活受体α,对急性心肌缺血性损伤大鼠血清肿瘤坏死因子α的影响。
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  相似匹配句对
     Ischemic postconditioning and myocardial preservation
     缺血后处理与心肌保护
短句来源
     Mitochondrion and myocardial ischemic-reperfusion injury
     线粒体与心肌缺血-再灌注损伤
短句来源
     Laboratory Progress in Ischemic Myocardial Damage Detection
     缺血性心脏病的实验室诊断进展
短句来源
     Myocardial ischemic damage after acute stroke
     卒中后的心肌缺血样损伤
短句来源
     Myocardial ischemic damage was found in 8 cases.
     3 10例 (2 6 .3% )符合尿毒症性心肌病 ,8例 (2 1.1% )疑有心肌缺血性损伤等。
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  myocardial ischemic damage
The hearts subjected to 25 min of ischemia followed by 30 min of post-ischemic reperfusion showed typical signs of severe myocardial ischemic damage.
      


The effects of sophoridine (Sop, 30 mg / kg iv) on ischemic ventricular tachyarrhythmias and electrophysiologic consequences in both normal and ischemic myocardium were studied in the open+chest dogs subject to programmed electrical stimulation (PES) oh 5-8 d after acute myocardial infarction. Its effects were compared with those of procainamide (PA 25 mg / kg iv). Both drugs distinctly lengthened the QTc interval and the effective refractory period (ERP) of the normal and infarct myocardium in both ventricles,...

The effects of sophoridine (Sop, 30 mg / kg iv) on ischemic ventricular tachyarrhythmias and electrophysiologic consequences in both normal and ischemic myocardium were studied in the open+chest dogs subject to programmed electrical stimulation (PES) oh 5-8 d after acute myocardial infarction. Its effects were compared with those of procainamide (PA 25 mg / kg iv). Both drugs distinctly lengthened the QTc interval and the effective refractory period (ERP) of the normal and infarct myocardium in both ventricles, decreased the dispersion of ERP in infarct myocardium (IDR) as well as the dispersion of ERP in left ventricle (VDR) and increased the diastolic excitability threshold(DET) of the normal and infarct myocardium in both ventricles remarkably. The PES-induced ventricular tachycardia (VT) or ventricular fibrillation (VF) was prevented in the 5 out of the 6 Sop and PA treated dogs respectively. Sop prevented spontaneous VF in 5 dogs (n = 6); PA prevented spontaneous VF in 4 dogs (n = 6); normal saline (NS) did not prevented PES-induced VT or VF and spontaneous VF. The results suggested that Sop might be effective in preventing the onset of reentrant ventricular tachyarrhythmias and sudden coronary death after myocardial ischemic damage.

用心脏程控刺激技术(PES),研究了ⅳ槐定对慢性心肌梗塞犬的正常心肌和梗塞心肌的电生理及快速室性心律失常的影响,并与普鲁卡因胺对比。两药均可显著延长OTc间期,RERP,NERP及IERP,缩小IDR和VDR,提高DET,抑制PES诱发的VT或VF,槐定尚可预防犬心肌梗塞后由急性心肌缺血所致的自发性室颤,证实了槐定抗缺血性快速室性心律失常的作用。

A canine model of ischemic ventricular tachyarrhythmias was established in open-chest dogs subjected to programmed electrical stimulation (PES) for 5 ̄8 days after acute myocardial infarction. The electrophysiologic effects of sophoridine (Sop) and procainamide (PA) were observed in this canine model. With routine methods of PES, ventricular tachycardia (VT) and ventricular fibrilation (VF) could be reproducibly initiated in this model. Both drugs distinctly lengthened the QTc interval ( P <0.01) and the effective...

A canine model of ischemic ventricular tachyarrhythmias was established in open-chest dogs subjected to programmed electrical stimulation (PES) for 5 ̄8 days after acute myocardial infarction. The electrophysiologic effects of sophoridine (Sop) and procainamide (PA) were observed in this canine model. With routine methods of PES, ventricular tachycardia (VT) and ventricular fibrilation (VF) could be reproducibly initiated in this model. Both drugs distinctly lengthened the QTc interval ( P <0.01) and the effective refractory period (ERP) in normal and ischemic ventricular myocardium ( P <0.01), decreased the dispersion of ERP in ischemic myocardium and the dispersion of ERP in left ventricle (P <0.05), and increased the diastolic excitability threshold of normal and ischemic ventricular myocardium remarkably ( P <0.01). Both drugs effectively prevented the PES-induced VT or VF and ischemia-induced VF ( P <0.05). The results indicated that this canine model is a good and reliable one, sophoridine and procainamide may be effective in preventing the onset of reentrant ventricular tachyarrhythmias after myocardial ischemic damage.

用心脏程控电刺激技术(PES),研究静脉注射槐定对慢性心肌梗塞犬的正常心肌和梗塞心肌的电生理及快速室性心律失常的影响,并与普鲁卡因胺(PA)对比。研究结果表明,两药均可显著延长QTC间期,RERP、NERP及IERP.缩小IDR和VDR,提高DET,抑制PES诱发的持续性室速和室颤,并能有效地预防心肌梗塞后缺血所致的自发性室颤,证实了槐定有抗缺血性快速室性心律失常的心电生理作用。

Myocardial ischemia in NIH mice was modeled by using pituitrin.Detecting serum lactic dehydrogenase(LDH) and myocardium LDH content and plasma malondialdehyde(MDA) and myocardium MDA content,the protective effect of Crataegus flavone,Cynomorium tannin and green tea polyphenol on experimental myocardial ischemic damage was stud- ied.Experimental results showed that 10mg/kg×20 of above three anti-oxidative components extracted from Chinese herbal medicines could also significantly decrease the leakage of...

Myocardial ischemia in NIH mice was modeled by using pituitrin.Detecting serum lactic dehydrogenase(LDH) and myocardium LDH content and plasma malondialdehyde(MDA) and myocardium MDA content,the protective effect of Crataegus flavone,Cynomorium tannin and green tea polyphenol on experimental myocardial ischemic damage was stud- ied.Experimental results showed that 10mg/kg×20 of above three anti-oxidative components extracted from Chinese herbal medicines could also significantly decrease the leakage of LDH from myocardium and content of myocardium MDA, suggesting protective effect ou myocardial ischemic damage.The mechanism of the protective effect was discussed from anti-free radicals damage of reperfusion.

用垂体后叶素造成 NIH 小鼠急性心肌缺血,从血清、心肌乳酸脱氢酶(LDH)和血浆、心肌丙二醛(MDA)含量变化研究了山楂黄酮、锁阳单宁和绿茶多酚对心肌缺血的保护作用。结果显示上述3种中药抗氧化组分可明显地减少缺血小鼠心肌 LDH 漏出,降低心肌 MDA 含量,对实验性心肌缺血损伤有很好的保护作用。对3种中药抗氧化组分的心肌缺血损伤保护作用从抗再灌注自由基损伤角度进行了讨论。

 
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