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et levels
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  et levels
Although hypoxia had no effect on the frequency of rotations induced by APO, it significantly attenuated a decrease in the ET levels in the circulation of rats with the most extensive lesions.
      
In normoxia, the rotation sessions were associated with significant decreases in circulating ET levels, from 21.32±4.06 to 9.32±1.08 pg/ml.
      
Also NEM, even et levels 2-3 times greater than those required to inhibit oxidative phosphorylation in intact mitochondria, did not inhibit coupled processes in submitochondrial particles.
      
ET levels in the plasma did not change from the respective controls over the same 24 h period.
      
From 5 min to 24 h after the Arthus reaction (AR), endothelin (ir-ET) levels in bronchoalveolar lavage fluid (BALF) and in plasma were measured by radioimmunoassay.
      
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Concentrations of plasma endothelin(ET) were measured by radioimmuno-assay in 10 cases of primary hypertension and 19 oases of uremia resulting from various kidney diseases and compared with those in 87 normal humans, The concentrations of plasma ET in the normal controls were 45.5±17.6pg/ml. No significant difference in ET concentrations could be found among various age groups. Compared with the controls, the values of ET in patients with primary hypertension and uremia were slightly...

Concentrations of plasma endothelin(ET) were measured by radioimmuno-assay in 10 cases of primary hypertension and 19 oases of uremia resulting from various kidney diseases and compared with those in 87 normal humans, The concentrations of plasma ET in the normal controls were 45.5±17.6pg/ml. No significant difference in ET concentrations could be found among various age groups. Compared with the controls, the values of ET in patients with primary hypertension and uremia were slightly and markedly increased respectively (P<0.01). These results suggest that the plasma ET level is constant in normal children and adults. The pathegenesis of primary hypertension may be related to ET. High plasma ET levels may result in further damage to renal function in patients with uremia.

放射免疫方法检测10例原发性高血压和19例各种肾脏病所致尿毒症患者血浆内皮素(ET)浓度,与87例正常人值相比。正常对照组血浆ET浓度为45.5:k17.6Pg/ml;不同年龄正常人血浆ET值无差别。与对照组相比,原发性高血压与尿毒症患者血浆ET值分别呈中度和明显升高(P均<0.01)。结果表明正常儿童至成人血浆ET水平较为恒定;提示血浆ET值与原发性高血压发病有一定关系,尿毒症患者血浆ET值升高可加重肾功能损害。

In order to investigate the pathoge'netic role of endothelin in septic shock, the effect of endothelin-antiserum on septic shock was evaluated on late sepsis model ( 20h ) produced by cecal ligature and puncture ( CLP ) in rats. The results showed that ET level elevated significantly in septic rats (7.7±1.4 vs sham 3. 1±0.2, pg/ml p<0.01)and ET-antiserum administration ( 0.5ml ) dramatically reduced plasma ET level and alleviated tissue injury. It suggested that ET is an important humoral...

In order to investigate the pathoge'netic role of endothelin in septic shock, the effect of endothelin-antiserum on septic shock was evaluated on late sepsis model ( 20h ) produced by cecal ligature and puncture ( CLP ) in rats. The results showed that ET level elevated significantly in septic rats (7.7±1.4 vs sham 3. 1±0.2, pg/ml p<0.01)and ET-antiserum administration ( 0.5ml ) dramatically reduced plasma ET level and alleviated tissue injury. It suggested that ET is an important humoral factor participating in the pathogenesis of septic shock.

为探讨内皮素(Endothelin,ET)在败血症休克中均发病学意义,本工作在结扎大鼠盲肠并穿孔的晚期败血症模型上观察内皮素抗血清(ET-Ab)列败血症休克大鼠的治疗作用。结果发现,败血症休克大鼠血浆ET含量(放免测定)显著增加(77±1.4 vs假手术动物3.1±0.2pg/ml,p<0.01)。用特异的内皮素抗血清治疗明显降低血浆内皮素水平,改善休克动物的低血压、低血糖状态和组织细胞的损伤。结果证实,内皮素是败血症休克发病重要体液因素之一。

This work observed on isolated perfused rat hearts, that 10~(-9)mol/L endothelin (ET) induced myocardial contracture, decrease of heart function and coronary flow, intramyocardial Ca~(2+) accumulation and Mg~(2+) loss and etc. These cardiac action of ET were obviously more prominent when Mg~(2+) in the perfusion fluid was lowered to 0.12 mmol/L, and were significantly alIeviated when Mg~(2+) content was increased to 4.8mmol/L (normal plasma Mg~(2+) content is about 1.2 mmol/L). The mechanism of the...

This work observed on isolated perfused rat hearts, that 10~(-9)mol/L endothelin (ET) induced myocardial contracture, decrease of heart function and coronary flow, intramyocardial Ca~(2+) accumulation and Mg~(2+) loss and etc. These cardiac action of ET were obviously more prominent when Mg~(2+) in the perfusion fluid was lowered to 0.12 mmol/L, and were significantly alIeviated when Mg~(2+) content was increased to 4.8mmol/L (normal plasma Mg~(2+) content is about 1.2 mmol/L). The mechanism of the Mg~(2+) effect on the cardiac action of ET may be related to its inhibition of Ca~(2+) influx into the myocytes. These results suggested that without magnesium defficiency, suitable replenishment of Mg~(2+) probably have practical clinical significance in the prevention and therapy of myocardial injury occurred during diseases with elevated circulatory ET level.

本实验在离体大鼠心脏灌流模型上,观察到10~(-9)mol/L内皮素灌流引起心肌挛缩,心功能降低,冠脉灌流量减少,以及心肌Ca~(2+)聚积,Mg~(2+)丧失等表现。灌流液含低镁(0.12mmol/L)时内皮素对心脏的上述作用显著增强,而在含高镁(4.8mmol/L)时明显减弱。Mg~(2+)影响内皮素对心脏作用的机制可能与其抑制心肌Ca~(2+)内流有关。实验结果提示避免缺镁,适当补充Mg~(2+)对于防治伴有循环内皮素水平增加的疾病或病理过程中的心脏损伤可能具有临床意义。

 
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