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   tumor necrosis factor family 在 肿瘤学 分类中 的翻译结果: 查询用时:0.055秒
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tumor necrosis factor family
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  肿瘤坏死因子家族
    Backround and Objective:CD40 ligand (CD40L) is a member of the tumor necrosis factor family, which is expression on activated T cells and binds to CD40 present on the membrane of antigen-presenting cells(APC).
    背景及目的:CD40配体(CD40L)是肿瘤坏死因子家族中的一员,它在激活的T细胞上表达,可与抗原提呈细胞(APC)上的CD40结合。
短句来源
    Fas ligand (FasL) belongs to the tumor necrosis factor family and has the ability to induce apoptosis in susceptible target cells by binding to its receptor, Fas.
    FasL属于肿瘤坏死因子家族,它通过与其受体Fas结合诱导敏感的靶细胞凋亡。
短句来源
    The T N F related apoptosis inducing ligand ,TRAIL is referred to as Apo2 ligand, Apo2L or death ligand, it belongs to tumor necrosis factor family and leads to tumor cell apoptosis selectively, but normal cell can run from TRAIL'S killing effect and it is hopeful that TRAIL becomes to a new anti - cancer drugs, so people are paying attention to it.
    肿瘤坏死因子相关的凋亡诱导配体(TNF related apoptosis inducingligand,TRAIL)又称凋亡素2配体(Apo2 ligand,Apo2L)或死亡配体,属于肿瘤坏死因子家族,能选择性的诱导肿瘤细胞凋亡,而正常细胞则可以逃逸他的杀伤作用,有望成为一种新的抗肿瘤制剂,尤为引人关注。
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  “tumor necrosis factor family”译为未确定词的双语例句
    AFP restrain DCs transform to matured antigen presenting cell through regulating the phenotype molecule express in the membrane of DCs, and AFP also influence the expression of tumor necrosis factor family and its receptor in lymphocytes or in hepatoma cells, AFP also could inhibit the activity of caspase in tumor cells, possesses these effects of AFP result in the hepatoma cells escape the immune surveillance.
    AFP可能通过调节DCs表型分子的表达,抑制DCs转变为成熟的抗原提呈细胞(APC)以及通过影响淋巴细胞或肝癌细胞的肿瘤坏死因子(TNF)家族及其受体的表达,抑制癌细胞内Caspase活性,导致肝癌细胞逃避机体免疫监视。
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  tumor necrosis factor family
Fas ligand is a member of the tumor necrosis factor family that can induce both apoptosis and activation of various cells.
      
Differentiation of osteoclasts is regulated by receptor activator of nuclear factor NF-kB ligand (RANKL), a member of tumor necrosis factor family.
      
Natural killer (NK) cells and cytotoxic T lymphocytes (CTLs) kill target cells by the granule-exocytosis pathway and by the engagement of molecules belonging to the tumor necrosis factor family.
      
APO2 ligand (APO2L) is a CD95 ligand (CD95L)-related cytokine of the tumor necrosis factor family that interacts with agonistic (DR4, DR5) and antagonistic (DcR1, DcR2) receptors.
      
The ectodysplasin 1 gene (ED1) encodes a signaling molecule of the tumor necrosis factor family that is involved in fetal development of ectodermal appendages.
      
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Alpha fetoprotein(AFP) plays multi-role in regulating the growth of hepatocellular carcinoma cells, because of AFP has a complex molecular structure. AFP not only can promote the proliferation of hepatoma cells, but also can inhibit the immune response in liver cancer patients. Recently, documents showed that AFP has a capability to impair the function of dentritic cells(DCs), and AFP could induce DCs to death. AFP restrain DCs transform to matured antigen presenting cell through regulating the phenotype molecule...

Alpha fetoprotein(AFP) plays multi-role in regulating the growth of hepatocellular carcinoma cells, because of AFP has a complex molecular structure. AFP not only can promote the proliferation of hepatoma cells, but also can inhibit the immune response in liver cancer patients. Recently, documents showed that AFP has a capability to impair the function of dentritic cells(DCs), and AFP could induce DCs to death. AFP restrain DCs transform to matured antigen presenting cell through regulating the phenotype molecule express in the membrane of DCs, and AFP also influence the expression of tumor necrosis factor family and its receptor in lymphocytes or in hepatoma cells, AFP also could inhibit the activity of caspase in tumor cells, possesses these effects of AFP result in the hepatoma cells escape the immune surveillance.

甲胎蛋白(AFP)复杂的结构决定其在肝癌细胞生长过程中扮演多功能的角色。AFP不仅具有促进肝癌细胞增生的作用,而且还具有抑制肿瘤患者的免疫应答作用。最近研究表明AFP能损伤肝癌患者树突状细胞(DCs)的功能和诱导DCs凋亡;AFP可能通过调节DCs表型分子的表达,抑制DCs转变为成熟的抗原提呈细胞(APC)以及通过影响淋巴细胞或肝癌细胞的肿瘤坏死因子(TNF)家族及其受体的表达,抑制癌细胞内Caspase活性,导致肝癌细胞逃避机体免疫监视。

 
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