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major subset
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  “major subset”译为未确定词的双语例句
     In islet of control group the major subset of the infiltrating T cells was CD 8 + T cells, but in study group in 90% mice appeared the normal pancreatic islet structure without T cell infiltration.
     对照组胰岛内浸润的T细胞以CD8+为主 ( 5 9% ) ;
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     Conclusion SpA is a common rheumatism,of which,the major subset was AS and uSpA.
     结论SpA是常见的风湿性疾病,其主要构成为AS和uSpA。
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     Human cervical cancer represent a major subset of female tumors throughout the world, especially in the developing countries.
     宫颈癌是常见的妇科肿瘤之一,特别是在发展中国家更多见。
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  相似匹配句对
     Choose the major
     选专业不要“雾里看花”
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     var. major N.
     var. major N.
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     Conclusion SpA is a common rheumatism,of which,the major subset was AS and uSpA.
     结论SpA是常见的风湿性疾病,其主要构成为AS和uSpA。
短句来源
     Drinking water is the major therapy for this subset of chronic hypernatremia.
     此类慢性高钠血症的治疗以单纯补水为主。
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     The Construction of Mobile Subset
     移动子集的构造方法
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  major subset
The reciprocals of these lattices are used in lattice integration rules, of which number theoretic rules form a major subset.
      
Thus, a major subset of LIL is phenotypically similar to mouse NK1.1+ "intermediate" T cells.
      
With about 20% of all lung cancers small cell lung cancer (SCLC) represents a major subset of this entity.
      
We show that if s2cf(α)>amp;lt;s2p(α), then noα-recursively enumerable set (α-r.e.) with complement of order type less than ts2p(α) can have a major subset.
      
Background: Myxoid liposarcomas (ML) make up the major subset of liposarcomas, which in most series represent the second or third most common type of soft tissue sarcoma.
      
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Objective To study the effect of chemotactic activity of mouse medullary type epithelial cell line MTEC1 secreted chemokines on thymocyte subsets. Methods Based on the distinct expression of CD4 and /or CD8 molecules,four major subsets of CD4 +CD8 +(DP),CD4 -CD8 -(DN),CD4 +CD8 -(SP) and CD4 -CD8 +(SP) mouse thymocytes were isolated by the combination of antibody plus complement killing,immunobeads and panning separation.The chemotaxis of MTEC1 secreted chemokines(in SN)to four thymocyte...

Objective To study the effect of chemotactic activity of mouse medullary type epithelial cell line MTEC1 secreted chemokines on thymocyte subsets. Methods Based on the distinct expression of CD4 and /or CD8 molecules,four major subsets of CD4 +CD8 +(DP),CD4 -CD8 -(DN),CD4 +CD8 -(SP) and CD4 -CD8 +(SP) mouse thymocytes were isolated by the combination of antibody plus complement killing,immunobeads and panning separation.The chemotaxis of MTEC1 secreted chemokines(in SN)to four thymocyte subsets was assessed in Boyden Chamber and the chemotactic activity was quantitatively determined. Results MTEC1 SN was a strong chemoattractant to DP and CD4SP with CI=6.6±1.0 and 6.1±1.8, repectively,and showed an intermidiate activity to CD8SP(CI=3.2±1.0), by contrast,MTEC1 SN had very weak chemotactic activity to DN thymocytes(CI=1.3±0.6). To confirm those results,purified MCP 1, a major chemokine that MTEC1 SN contains,exhibited strong chemotactic activity to CD4SP thymocytes(CI=5.6), but no detectable activity to DN thymocytes. Conclusions Mouse thymic medullary type epithelial cell line(MTEC1) could secret chemokines,which were chemoattractant to DP, CD4SP and CD8SP thymocytes,but not to the DN thymocytes.It implies that MTEC1 secreted chemokines may chemotactically attract the intermidiate (DP) and late(SP) stages of developing thymocytes to migrate directly and retain in thymic medulla.

分析胸腺髓质上皮样细胞系MTEC1分泌的化学趋化因子对胸腺细胞亚群的趋化作用。方法以抗体加补体杀伤结合免疫磁珠及panning法,将小鼠胸腺细胞分离纯化,获得CD4+CD8+(DP),CD4-CD8-(DN),CD4+CD8-(CD4SP)及CD4-CD8+(CD8SP)四亚群细胞,用Boyden小室分析MTEC1┐SN对四群胸腺细胞的趋化作用。结果MTEC1┐SN对DP及CD4SP胸腺细胞有趋化活性(CI=6.6±1.0及6.1±1.8);对CD8SP细胞有中度趋化活性(CI=3.2±1.0);对DN趋化活性微弱(CI=1.3±0.6)。化学趋化因子MCP┐1纯品对CD4SP胸腺细胞显示强趋化活性(CI=5.6),对DN胸腺细胞则无可测出趋化活性。结论MTEC1分泌的化学趋化因子对DP,CD4SP及CD8SP胸腺细胞有显著趋化作用,对DN胸腺细胞几乎无趋化作用。提示此类化学趋化因子有趋使胸腺发育中后期阶段的细胞向胸腺髓质区迁移和定位的作用。

Objective To investigate the activation and expression of nuclear factor κB (NF κB) and effects of anti inflammatory treatment on NF κB in the intestinal mucosa of patients with ulcerative colitis (UC). Methods Ten pieces of colon mucosal biopsy specimens were obtained from 31 cases with UC, 17 of which received sulphasalazine (SASP)or SASP plus glucocorticoid and 14 of which received no medication. Samples of normal mucosa around the lesion taken from 11 patients with colon cancer were used as controls....

Objective To investigate the activation and expression of nuclear factor κB (NF κB) and effects of anti inflammatory treatment on NF κB in the intestinal mucosa of patients with ulcerative colitis (UC). Methods Ten pieces of colon mucosal biopsy specimens were obtained from 31 cases with UC, 17 of which received sulphasalazine (SASP)or SASP plus glucocorticoid and 14 of which received no medication. Samples of normal mucosa around the lesion taken from 11 patients with colon cancer were used as controls. NF κB DNA binding activaty was evaluated by electrophoretic mobility shift assay. NF κB p65 expression was determined by Western blot analysis and immunohistochemical staining with a NF κB p65 antibody. The type of cells containing activited NF κBp65 was identified by double immunofluorescence confocal laser scanning microscopy. Results The expression of NF κB p65 and NF κB DNA binding activity were significantly higher in patients with UC than in the control ( P <0.05), and were correlated with the degree of inflammation. The NF κB expression was significantly stronger in the nuclei than in the cytoplasm in patients with UC without pharmacotherapy. The NF κB espression in nuclei was significantly stronger in the group without pharmacotherapy than in the group with pharmacotherapy ( P <0.05). Only a few NF κB p65 positive cells were seen in the controls. NF κBp65 expression was found in all major subsets of mononucler cells, including macrophages, B lymphocytes, T lymphocytes, and cryptal epithelial cells. Conclusion The increased activation of NF κB and increased expression of NF κB may be involved in the pathogenesis of UC. Glucocorticoids and SASP strongly inhibited NF κB activation and expression. The inhibition of NF κB activation may be a central part of the anti inflammatory action of glucocorticoids and SASP, which might represent an important pharmacological mechanism in treatment of patients with UC. NF κB will be an important target for cytokine based therapy of UC.

目的 探讨溃疡性结肠炎患者肠粘膜κ基因结合核因子 (NF κB)的活化以及柳氮磺胺吡啶 (SASP)和糖皮质激素对其的影响。方法  31例溃疡性结肠炎患者中 17例使用过药物 (SASP或SASP +糖皮质激素 )治疗 ,14例未用过任何与溃疡性结肠炎治疗相关的药物 ,11例同期结肠癌患者(取其癌旁正常组织 )作为对照。采用凝胶电泳迁移率改变分析 (EMSA)检测NF κBDNA结合活性 ;Western蛋白印迹分析检测NF κBp6 5蛋白表达情况 ;免疫组化方法原位检测肠粘膜组织中NF κBp6 5蛋白的表达情况 ;荧光双标激光共聚焦显微镜确定表达NF κBp6 5的细胞类型。结果 溃疡性结肠炎患者肠粘膜NF κBp6 5的表达和NF κBDNA结合活性与对照组比较明显升高 (P <0 .0 5 ) ,且与炎症程度有关 ;溃疡性结肠炎患者肠粘膜巨噬细胞、T淋巴细胞、B淋巴细胞以及隐窝上皮细胞均有NF κBp6 5的活化 ;糖皮质激素和SASP明显抑制NF κB的活性及表达。结论 NF κB活性和表达的增加可能与溃疡性结肠炎的发生发展有关 ;糖皮质激素和SASP的抗炎作用可能是通过抑制NF κB的活性实现的...

目的 探讨溃疡性结肠炎患者肠粘膜κ基因结合核因子 (NF κB)的活化以及柳氮磺胺吡啶 (SASP)和糖皮质激素对其的影响。方法  31例溃疡性结肠炎患者中 17例使用过药物 (SASP或SASP +糖皮质激素 )治疗 ,14例未用过任何与溃疡性结肠炎治疗相关的药物 ,11例同期结肠癌患者(取其癌旁正常组织 )作为对照。采用凝胶电泳迁移率改变分析 (EMSA)检测NF κBDNA结合活性 ;Western蛋白印迹分析检测NF κBp6 5蛋白表达情况 ;免疫组化方法原位检测肠粘膜组织中NF κBp6 5蛋白的表达情况 ;荧光双标激光共聚焦显微镜确定表达NF κBp6 5的细胞类型。结果 溃疡性结肠炎患者肠粘膜NF κBp6 5的表达和NF κBDNA结合活性与对照组比较明显升高 (P <0 .0 5 ) ,且与炎症程度有关 ;溃疡性结肠炎患者肠粘膜巨噬细胞、T淋巴细胞、B淋巴细胞以及隐窝上皮细胞均有NF κBp6 5的活化 ;糖皮质激素和SASP明显抑制NF κB的活性及表达。结论 NF κB活性和表达的增加可能与溃疡性结肠炎的发生发展有关 ;糖皮质激素和SASP的抗炎作用可能是通过抑制NF κB的活性实现的。NF κB可能为溃疡性结肠炎新药的研究与开发提供一个有价值的靶标。

Objective To evaluate the prevalence of spondyloarthropathies in Beijing area.Methods Samples of 1982 workers and their adults relatives of a factory in Beijing were studied.The revised questionnaire of LIAR China study was administered in a house to house survey.Physical examination was done in all who gave positive answers.For those who gave positive response to certain set of questions,rheumatoid factor(RF),HLA B 27 ,and radiographs of pelvis and lumbar spine were done.The X ray films were read blindly...

Objective To evaluate the prevalence of spondyloarthropathies in Beijing area.Methods Samples of 1982 workers and their adults relatives of a factory in Beijing were studied.The revised questionnaire of LIAR China study was administered in a house to house survey.Physical examination was done in all who gave positive answers.For those who gave positive response to certain set of questions,rheumatoid factor(RF),HLA B 27 ,and radiographs of pelvis and lumbar spine were done.The X ray films were read blindly and separately by 2 radiologists.The 1984 Revised New York criteria for AS,and Amor Criteria for SpA were used.Results The prevalence of SpA was 16.1‰(32/1982),in which the prevalence of AS was 3.0‰(6/1982),and uSpA was 12.1‰(24/1982),excepted 2 cases of psoriatic arthritis,no other subset of SpA was found.Sex ratio of male to female was 5∶1 in AS and 1.2∶1 in uSpA.Of the uSpA cases,the main clinical symptoms were inflammatory low back pain(83.3%),asymmetric oligoarthritis in the low limbs(62.5%).RF was negative in all patients.25.0%(6/24) cases were HLA B 27 positive.Conclusion SpA is common in Beijing area,of which,the major subset was AS and uSpA.Clinical follow up of uSpA is helpful in the early diagnosis of AS and the other SpA.

目的 了解北京地区血清阴性脊柱关节病 (SpA)的流行情况。 方法 用修订的中国 国际抗风湿联盟风湿病调查表 ,对北京市区某工厂职工及 16岁以上家属 1982名挨户逐人询问 ,阳性者进行体检 ,疑似者行类风湿因子 (RF)、HLA B2 7和X线检查。结果 脊柱关节病患病率 16 1‰ ,其中强直性脊柱炎 (AS)3 0‰ ,未分化脊柱关节病 (uSpA) 12 1‰ ,除 2例银屑病性关节炎外 ,未发现其他SpA。uSpA主要症状为炎症性腰痛 (83 3% )、非对称性下肢大关节痛 (6 2 5 % ) ,RF均 (- ) ,2 5 0 %HLA B2 7(+) ,男女比例 1 2∶1。结论 SpA在北京地区常见 ,其主要构成为AS和uSpA。对uSpA的随访有助于AS和其他SpA的早期诊断

 
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