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micronodular cirrhosis
相关语句
  小结节性肝硬化
     Seventy-five percent of animals exhibited features of steatohepatitis, 57 % were associated with hepatic fibrosis, and 11 % were complicated with micronodular cirrhosis.
     其中75 %伴发不同程度脂肪性肝炎 ,5 7%有不同程度纤维化 ,11%形成小结节性肝硬化
短句来源
     Results:4 weeks after modeling,typical micronodular cirrhosis formed,and 6 weeks after modeling,the hepatic injury in the model group had some remission as compared with that at 4 weeks.
     结果:造模4周后大鼠形成典型小结节性肝硬化,6周后模型组大鼠肝组织损伤较4周时有所缓解。
短句来源
  小结节肝硬变
     Abstract The nuclear DNA content of hepatocytes in 67 human cases was measured using an automated image analyser in normal liver, mild chronic active hepatitis (MCAH), severechronic active heptitis (SCAH), micronodular cirrhosis (MINC), macronodular cirrhosis(MANC), pericarcinoma cirrhosis (PC) and hepatocarcinoma (HCC).
     利用图像分析技术对轻度慢性活动性肝炎(MCAH)、重度慢性活动性肝炎(SCAH)、小结节肝硬变(MINC)、大结节肝硬变(MANC)、癌周肝硬变(PC)和肝细胞癌(HCC)及正常对照共67例样本的肝细胞或癌细胞DNA含量进行测定分析。
短句来源
     Methods The nuclear DNA contents and 13 morphometric parameters of hepatocytes in 67 human cases were measured by using an automated image analyser in normal liver, mild chronic hepatitis, severe chronic hepatitis, micronodular cirrhosis, macronodular cirrhosis, pericarcinoma cirrhosis (PC) and hepatocellular carcinoma (HCC). Variance (q-test) and stepwise discrimination analysis analyzed the data.
     方法 :利用图像分析技术对 67例正常肝、轻度慢性肝炎、重度慢性肝炎、小结节肝硬变、大结节肝硬变、癌旁肝硬变和肝细胞癌分别作了 13项参数的形态定量研究和肝细胞DNA含量测定分析 ;
短句来源
     The nuclear DNA contents and 13 morphometric parameters of hepatocytes in 67 humancases were measured by using an automated image analyser in normal liver, mild chronic hepatitis, severe chronic hepatitis, micronodular cirrhosis, macronodular cirrhosis, pericarcinoma cirrhosis and hepatocellular carcinoma.
     利用自动化图象分析技术对轻度慢性肝炎、重度慢性肝炎、小结节肝硬变、大结节肝硬变、癌周肝硬变和肝细胞癌等 6种慢性肝病及正常对照共 6 7例样本 ,分别作了 13项参数的形态定量研究和肝细胞DNA含量测定分析 .
短句来源
  “micronodular cirrhosis”译为未确定词的双语例句
     In the above results we have shown that type B hepatitis leads to cirrhosic mixed type and type non-A:non-B hepatitis leads to micronodular cirrhosis.
     结果表明,乙型肝炎是引起混合结节型肝炎的主要原因之一,非甲非乙型肝炎引起的多属小结节型肝硬变。
短句来源
     he nuclear DNA content of hepatocytes in 48 human cases was measured using anautomated image analyser in normal liver,severe chronic active heptitis(SCAH)micronodular cirrhosis,macronodular cirrhosis and pericarcinoma cirrhosis. The resultsshowed:(1)There was a decrease in tetraploid and binuclear cells in SCAH. Inmacronodular cirrhosis,the percentage of diploid cells was increased, but the S-phase frac-tion valus andhnuclear cells decreased.
     利用自动化图象分析仪对48例正常肝、重度慢性活动性肝炎、小结节性肝硬变、大结节及混合结节肝硬变、癌旁肝硬变的肝细胞DNA含量进行测定分析,结果:①重度慢性活动性肝炎4N细胞及双核细胞比率下降;
短句来源
     Results Liver histology revealed that there were massive or sub massive necrosis of hepatic cells on the basis of chronic hepatitis and micronodular cirrhosis in the early and the meta stages of the disease;
     结果 早、中期肝组织学主要表现为在慢性肝炎和小结节性肝硬变基础上出现大块或亚大块坏死 ;
短句来源
     intermediate or mixed patterns of macronodular and micronodular cirrhosis were common in most cases but some with appearance of heavy chronic hepatitis in the convalescence;
     恢复期肝脏以大小结节混合型肝硬变为主 ,少数为重度慢性肝炎的病理表现 ;
短句来源
     Methods:By laparoscopy or abdominal operation,the morphologic changes in the 31 cases were directly observed which were 4 types(micronodular cirrhosis [MicroNC],macronodular cirrhosis [MacroNC],mixed macronodular and micronodular cirrhosis [MixedNC] and incomplete septal cirrhosis ).
     方法:通过腹腔镜或开腹手术的方法,直接对肝脏进行肝硬化形态学诊断(小结节型、大结节型、大小结节混合型、橘皮样变);
短句来源
  相似匹配句对
     Bonedensity in cirrhosis
     肝硬化患者骨密度分析
短句来源
     objective cirrhosis.
     目的 探讨肝硬变状态下胆石发生的机理。
短句来源
     Results In CCL4-treated mice, the liver surface was micronodular, and the liver showed the histologically characteristic features of cirrhosis.
     结果 小鼠经CCL。 注射后肝脏表面出现细小结节,镜-下呈现肝硬化的组织学特征。
短句来源
     intermediate or mixed patterns of macronodular and micronodular cirrhosis were common in most cases but some with appearance of heavy chronic hepatitis in the convalescence;
     恢复期肝脏以大小结节混合型肝硬变为主 ,少数为重度慢性肝炎的病理表现 ;
短句来源
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  micronodular cirrhosis
The results demonstrated acharacteristic micronodular cirrhosis in all the liversstudied; the microcirculation displayed the presence ofnewly formed perinodular plexus.
      
The noncarcinomatous area revealed typical micronodular cirrhosis due to excessive alcohol consumption.
      
We report the second case of a patient with intrahepatic cholestasis that developed after treatment with thiabendazole and progressed to severe micronodular cirrhosis.
      
The patients, all over 50 years old, comprised two females and one male with steatohepatitis and three females with micronodular cirrhosis.
      
We found evidence of micronodular cirrhosis of the liver and an apparent Roux-en-Y anastomosis.
      
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Liver cirrhosis may be caused by type B hepatitis and type non-A:non-B hepatitis,but there may be difficult to distinguish from the lesions of two causes.The study was made by methods of morphologic analysis,histochcmistry,immunohistochemistry.In the above results we have shown that type B hepatitis leads to cirrhosic mixed type and type non-A:non-B hepatitis leads to micronodular cirrhosis.

乙型肝炎与非甲非乙型肝炎均可引起肝硬变,但这两型肝炎引起的肝硬变间有无差别,本文应用显微图象计量方法,免疫组化,组化方法进行研究.结果表明,乙型肝炎是引起混合结节型肝炎的主要原因之一,非甲非乙型肝炎引起的多属小结节型肝硬变。

he nuclear DNA content of hepatocytes in 48 human cases was measured using anautomated image analyser in normal liver,severe chronic active heptitis(SCAH)micronodular cirrhosis,macronodular cirrhosis and pericarcinoma cirrhosis.The resultsshowed:(1)There was a decrease in tetraploid and binuclear cells in SCAH. Inmacronodular cirrhosis,the percentage of diploid cells was increased, but the S-phase frac-tion valus andhnuclear cells decreased.These changes suggested that the degree of cellulardifferentiation...

he nuclear DNA content of hepatocytes in 48 human cases was measured using anautomated image analyser in normal liver,severe chronic active heptitis(SCAH)micronodular cirrhosis,macronodular cirrhosis and pericarcinoma cirrhosis.The resultsshowed:(1)There was a decrease in tetraploid and binuclear cells in SCAH. Inmacronodular cirrhosis,the percentage of diploid cells was increased, but the S-phase frac-tion valus andhnuclear cells decreased.These changes suggested that the degree of cellulardifferentiation was inhibited.It mav be associated with the carcinogenesis ofhepatocarcinoma.(2) Although the ration of 2N:4N remaind unchanged,an increased frac-tion of≥5N cells and a decreased percentage of binuclear cells was indicated inpericarcinoma cirrhosis.The distribution of DNA contents in these cases was also much widerthan other cirrhosis.It is suggested that the hepatocytes had a tengency to develop theaneuploid cell populations with a reduced degree of differentiation。This cirrhosis pattern maybe a precursor of hepatocarcinoma。3)The micronodular cirrhosis contained a signiflcantlyhigher fraction of S-phase valus and binuclear cells,and a lower diploid eells than other threecirrhosis.It mav be attained increasing degrees of differentiation during liver regeneration after injury,which may be not associated with carcinogenesis So we suggest that the DNA con-tent of liver cirrhosis might objectively reflect the condition of liver proliferation. It may beuseful in the diagnosis and prognosis of cirrhosis.

利用自动化图象分析仪对48例正常肝、重度慢性活动性肝炎、小结节性肝硬变、大结节及混合结节肝硬变、癌旁肝硬变的肝细胞DNA含量进行测定分析,结果:①重度慢性活动性肝炎4N细胞及双核细胞比率下降;大结节及混合结节肝硬变2N比率增高;S期及双核细胞下降,显示细胞分化受抑制,可能与肝癌发生相关,②癌旁肝硬变2N/4N比率无明显改变,但≥5N细胞增高,双核细胞下降,DNA直方图增宽,表明肝细胞分化受抑制且有向异倍体发展趋势,具有癌前病变性质,③小结节性肝硬变2N细胞比率显著下降,S期及双核细胞比率增高,为分化性代偿修复病变而与肝癌发生无关,肝细胞DNA含量的定量分析对于了解肝硬变肝细胞的再生修复状态及患者预后评估将有重要的意义。

Abstract The nuclear DNA content of hepatocytes in 67 human cases was measured using an automated image analyser in normal liver, mild chronic active hepatitis (MCAH), severechronic active heptitis (SCAH), micronodular cirrhosis (MINC), macronodular cirrhosis(MANC), pericarcinoma cirrhosis (PC) and hepatocarcinoma (HCC). The results showed:(1) The polyploidization process was inhibited in MCAH, SCAH and MANC. Thesechanges suggested that the differentiative ability was reduced, which might be...

Abstract The nuclear DNA content of hepatocytes in 67 human cases was measured using an automated image analyser in normal liver, mild chronic active hepatitis (MCAH), severechronic active heptitis (SCAH), micronodular cirrhosis (MINC), macronodular cirrhosis(MANC), pericarcinoma cirrhosis (PC) and hepatocarcinoma (HCC). The results showed:(1) The polyploidization process was inhibited in MCAH, SCAH and MANC. Thesechanges suggested that the differentiative ability was reduced, which might be associatedwith the hepotocarcinogenesis. (2) All HCCs were aneuploid tumors, whose cells possesedan increasing proliferative ability and a reduced degree of differentiation. (3) Ploidy distributions and the percent of binucleated cells in PC were intermediate betweem HCCs and othernon-cancinous lesions. It suggested that the hepatocytes had a tendency to develop the aneuploid cell populations with a reduced degree of differentiation. This cirrhosis pattern mightbe a precursor of hepatocarcinoma. (4) This predominant polyploidization could be seen inMINC. It was attained in the increasing degrees of differentiation during liver regenerationafter injury, which was not associated with carcinogenesis. So we suggest that the plodidypatterns of hepatocytes can objectively reflect the condition of liver proliferation. It may beuseful in the diagnosis and prognosis of these liver diseases.Keyworks: Liver-Neoplasms, Lirrhosis, Hepatitis, DNA-Analysis, Ploidies.

利用图像分析技术对轻度慢性活动性肝炎(MCAH)、重度慢性活动性肝炎(SCAH)、小结节肝硬变(MINC)、大结节肝硬变(MANC)、癌周肝硬变(PC)和肝细胞癌(HCC)及正常对照共67例样本的肝细胞或癌细胞DNA含量进行测定分析。结果(1)MCAH、SCAH和MANC多倍体化受抑制,显示细胞分化能力下降,可能与肝癌的发生相关;(2)HCC均为异倍体肿瘤,细胞具有较高的增殖能力并处于较低的分化状态;(3)PC倍体分布和双核细胞率介于其他非癌病变和HCC之间,细胞分化受抑制且有向异倍体发展趋势,具有癌前病变性质;(4)MINC明显多倍体化,为分化性的再生修复病变而与肝癌发生无关。因此肝细胞DNA含量的定量分析对于了解肝细胞的增生状态,以及患者的预后评估有重要意义。

 
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