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propylene glycol alginate
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propylene glycol alginate
Propylene glycol alginate forms strong, covalently bonded gel when mixed with certain proteins in alkaline conditions.
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Additionally, the propylene glycol alginate (PGA) vehicle and the 24% ethylenediaminetetra-acetate (EDTA) gel were tested.
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Egg albumen foams (8%) with and without added propylene glycol alginate or xanthan gum (both at 0.2% concentration on wet weight basis) formed after 1-5?min whipping were tested for their consistency using squeezing flow viscometry and stability.
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Propylene glycol alginate is regulated as a food additive in 21 CFR 172.858.
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The viscosity properties of food thickening agent propylene glycol alginate (PGA) are studied. Experiment indicate: there is semi- logarithmic linear relation between the viscosity and concerntration of PGA (R 2=0.974 0); In range of pH 2～10, the viscosity of PGA almost is constant, when pH≥11, the viscosity of PGA reduces. There is good viscosity synergism between PGA and CMC, the synergism between PGA and sodium alginate is more complex. 研究了食品增稠剂藻酸丙二醇酯（ＰＧＡ）的粘度特性。实验表明：ＰＧＡ的粘度与其浓度之间存在着半对数线性关系（Ｒ２＝０．９７４０）；在ｐＨ２～１０范围内，ＰＧＡ的粘度几乎无变化，但当ｐＨ≥１１时，ＰＧＡ的粘度下降；ＰＧＡ与羧甲基纤维素（ＣＭＣ）具有良好增粘效应，但与海藻酸钠之间的效应较复杂。 文摘来源 AIM and METHODS: The effect of propylene glycol alginate sodium sulphate (PSS), eight cytokines including thrombospondin (TSP), PF 4, TGFβ 1, IL-3,IL-6, EPO, GM-CSF, bFGF and aplastic pig serum (APS) known to be rich in TPO, alone or in combination, on megakaryocytopoiesis and the mechanism of PSS action were studied by plasma clot system (PCS), serum free agar system (SFAS) and [ 125 I]-bFGF binding test. RESULTS: PSS stimulated the growth of CFU -MK in PCS, but no... 目的和方法：采用血浆凝块（ＰＣ）、无血清琼脂（ＳＦＡ）培养及［１２５Ｉ］－ｂＦＧＦ结合试验等方法研究ＰＳＳ及ＴＳＰ等８种细胞因子对巨核细胞生成的作用与机制。结果：在ＰＣ系统，ＰＳＳ刺激ＣＦＵ－ＭＫ形成；在ＳＦＡ系统，ＰＳＳ无此作用。ＰＳＳ加强ｂＦＧＦ或再障血清对ＣＦＵ－ＭＫ形成的刺激作用，但ＰＳＳ不增加ｂＦＧＦ与其高亲和力受体的结合。ＴＳＰ及具有肝素结合位点（ＨＢＤ）的ＴＳＰ多肽片段明显抑制ＣＦＵ－ＭＫ的形成，无ＨＢＤ的ＴＳＰ片段则无此作用。ＴＳＰ、ＰＦ４及ＴＧＦβ１对巨核细胞的抑制作用可被ＰＳＳ所逆转。结论：ＰＳＳ对巨核细胞生成起正调控作用，其机制与增强巨核细胞生成刺激因子活性及中和抑制因子的活性有关；ＴＳＰ是巨核细胞生成负调控因子，ＨＢＤ是其重要功能基团。 文摘来源 
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