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acute toxin
相关语句
  急性毒性
     Safety as food experiments: Acute toxin experiments, four dosage were selected: 10. Og/kg, 21. 5g/kg, 46. 4g/kg and lOOg/kg, giving them to mice within 24hours, 12hours of feeding prohibition of them before experiments.
     安全食效性试验:急性毒性试验用Horn法设计剂量,通过24h内多次给样的办法,使最高剂量达到100g/kg。
短句来源
     In the study the best formula of the compound, its hypoglycemic effects and acute toxin were investigated.
     本文主要就其配方的筛选,降血糖作用和急性毒性等进行了初步研究。
短句来源
     On the basis of having studied on the effect of high temperature on acute toxin of carbon mon9xide(CO),this paper deals with the effect of different temperature on subacute toxin of CO.
     本文在研究高温对CO急性毒性影响的基础上,进行了不同温度条件下大鼠动式吸入CO气体亚急性染毒实验研究。
短句来源
     There was no unusual sign in the white mouse acute toxin experiment of the pigment.
     急性毒性试验中白鼠无异常表现。
短句来源
  急性毒
     Acute toxin test showed that no acute toxicity was found on the tested mice when feeding 10.0, (15.0,) 22.5, (33.8,) 50.6 g·kg~(-1) of J9311 propolis capsule on the mice, giving us a clue that J9311 propolis capsule is safe product for human~′s consumption.
     急性毒理实验显示,10.0、15.0、22.5、33.8、50.6g·kg-1剂量的J9311蜂胶囊对小鼠无急性毒理作用.
短句来源
     In this study, the water solution of J9311 Propolis Capsule was used to test the anti-tumor effect on S180 and H22 tumor cells from mice. Also the phagocytosis variation of phagocyte, the components analysis as well as the acute toxin action of propolis capsule tests were carried out to prove its positive effect.
     本实验以J9311蜂胶囊水溶液为样品,对小鼠进行了抑制S_(180)实体瘤(小鼠肉瘤细胞系)和H_(22)实体瘤(小鼠肝癌细胞系)实验、对巨噬细胞吞噬能力影响实验、成分合理性分析实验、急性毒理实验。 结果如下:
短句来源
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On the basis of having studied on the effect of high temperature on acute toxin of carbon mon9xide(CO),this paper deals with the effect of different temperature on subacute toxin of CO.The results of the experiment showed that high temperature (35℃) could enhance the toxic effects of CO.The effects were mainly that the increase of carboxyhemoglobin(COHb) level in the blood of the rats is more obvious.On the other hand,CO could also reduce the heat tolerance of rats.The results showed an additive...

On the basis of having studied on the effect of high temperature on acute toxin of carbon mon9xide(CO),this paper deals with the effect of different temperature on subacute toxin of CO.The results of the experiment showed that high temperature (35℃) could enhance the toxic effects of CO.The effects were mainly that the increase of carboxyhemoglobin(COHb) level in the blood of the rats is more obvious.On the other hand,CO could also reduce the heat tolerance of rats.The results showed an additive effect in the combined exposure of high temperature and CO.

本文在研究高温对CO急性毒性影响的基础上,进行了不同温度条件下大鼠动式吸入CO气体亚急性染毒实验研究。结果表明高温可加强CO的毒作用,主要表现为血中碳氧血红蛋白含量明显升高。CO也可降低机体耐热力,表现为体温升高更明显,两者之间为协同作用。

Purpose: The present studies were first to determine the plasma levels of tissue factor path-way inhibitor (TFPI) in several type of hepatitis and decompenstion cirrhosis. Methods: TFPI antigen (TFPI:Ag) was determined with an enzyme linked immunity (ELISA). TFPI: active (TFPI:A) was de-termined with chromogenic peptide substrate method. Results: Acute toxin hepatitis (n=14), acute viral hepatitis (n=20), chronic B type hepatitis (n=48)and decompensation cirrhosis (n=20), TFPI:Ag/ TFPI: A was: 160.3±26.1...

Purpose: The present studies were first to determine the plasma levels of tissue factor path-way inhibitor (TFPI) in several type of hepatitis and decompenstion cirrhosis. Methods: TFPI antigen (TFPI:Ag) was determined with an enzyme linked immunity (ELISA). TFPI: active (TFPI:A) was de-termined with chromogenic peptide substrate method. Results: Acute toxin hepatitis (n=14), acute viral hepatitis (n=20), chronic B type hepatitis (n=48)and decompensation cirrhosis (n=20), TFPI:Ag/ TFPI: A was: 160.3±26.1 ng/ml/515. 8±50.2 u/ml, 197.7±18.6ng/ml/532.1±98.7u/ml, 162.5±28.3 ng/ml/636.7±?54.8u/ml, 81.7±16.7ng/ml/747.2±81.2u/ml respectively. Except for group of cirrhosis, the other groups TFPI: Ag was increased than control(103. 2±?11.5 ng/ml) and TFPI: A were increased than control(112. 5±23.6 u/ml) in all groups. Conclusion: TFPI can over showdown on inflammation due to active of mono-macrophages and endothelial cell of vessel irritated by inflammatory fac-tor. But TFPI: Ag decreased to low levels of normal in group of cirrhosis, be concerned with thrombosis of portal vein system, The TFPI:Ag would decreased in a consumption coagulation.

目的:测定各类型肝炎及失代偿期肝硬化患者血浆中的组织因子途径抑制物(Tissue factor pathway in-hibitor,TFPI),并探索其临床意义。方法:TFPI抗原(TFPI:Ag)测定采用双夹心ELISA抗原测定法,TFPI活性(TFPI:A)测定采用发色底物法。结果:急性中毒性肝炎(n=14)TFPI:Ag为160.3±26.1ng/ml,TFPI:A为515.8±50.2 u/ml;急性病毒性肝炎(n=20)各为197.7±18.6 ng/ml与532.1±98.7 u/ml;慢性乙型肝炎(n=48)各为162.5±28.3ng/ml与636.7±54.8 u/ml;失代偿期肝硬化(n=20)各为81.7±16.7 ng/ml与747.2±81.2u/ml。与对照组相比,TF-PI:Ag(正常值103.2±11.5 ng/ml)除肝硬化组接近正常低值外,其它各组均有增高(P<0.05);TFPI:A(正常值112.5±23.6 u/ml)各组均增高(P<0.01)。结论:炎症时因单核-巨噬细胞活跃及炎症因子刺激血管内皮细胞,故使TFPI过度表达,但在肝硬化时TFPI:Ag已趋向正常低值...

目的:测定各类型肝炎及失代偿期肝硬化患者血浆中的组织因子途径抑制物(Tissue factor pathway in-hibitor,TFPI),并探索其临床意义。方法:TFPI抗原(TFPI:Ag)测定采用双夹心ELISA抗原测定法,TFPI活性(TFPI:A)测定采用发色底物法。结果:急性中毒性肝炎(n=14)TFPI:Ag为160.3±26.1ng/ml,TFPI:A为515.8±50.2 u/ml;急性病毒性肝炎(n=20)各为197.7±18.6 ng/ml与532.1±98.7 u/ml;慢性乙型肝炎(n=48)各为162.5±28.3ng/ml与636.7±54.8 u/ml;失代偿期肝硬化(n=20)各为81.7±16.7 ng/ml与747.2±81.2u/ml。与对照组相比,TF-PI:Ag(正常值103.2±11.5 ng/ml)除肝硬化组接近正常低值外,其它各组均有增高(P<0.05);TFPI:A(正常值112.5±23.6 u/ml)各组均增高(P<0.01)。结论:炎症时因单核-巨噬细胞活跃及炎症因子刺激血管内皮细胞,故使TFPI过度表达,但在肝硬化时TFPI:Ag已趋向正常低值,可能与门静脉系血栓形成过程中TFPI不断大量消耗有关。

Objective To investigate the effect of Xue-Mai-Tong-Jiao-Nang (XMTJN) on acute toxin and chronic toxin in mice and rats.Methods Mice were given XMTJN (20、22 and 25g/kg ) by gavage, twice one day, the mice were observed 7 days after gavage XMTJN. Rats were received XMTJN (0.39、0.98 and 1.95g/kg) for 12 weeks by gavage. Observe the action changes and death of the rats. Serum and main organs were examined after 12 weeks.Results Mice weren’t obtained the maximum lethal dose. The maximum tolerance dose...

Objective To investigate the effect of Xue-Mai-Tong-Jiao-Nang (XMTJN) on acute toxin and chronic toxin in mice and rats.Methods Mice were given XMTJN (20、22 and 25g/kg ) by gavage, twice one day, the mice were observed 7 days after gavage XMTJN. Rats were received XMTJN (0.39、0.98 and 1.95g/kg) for 12 weeks by gavage. Observe the action changes and death of the rats. Serum and main organs were examined after 12 weeks.Results Mice weren’t obtained the maximum lethal dose. The maximum tolerance dose of the mice was 50g/kg (1282 times of the dose for adult). The general condition of mice and rats were normal during experiment, without death and abnormal reaction. It was shown that blood routine serum biochemistry and pathology were normal range.Conclusion XMTJN is a lower toxin and safe medicine.

目的观察连续给予血脉通胶囊后由于积蓄可能对机体产生的毒性反应 ,为拟定人用安全剂量提供参考。方法给小鼠按 2 0g/kg、2 2g/kg和 2 5g/kg剂量一天灌胃给药二次 ,观察药物的急性毒性。按 0 39g/kg、0 975g/kg、1 95g/kg(相当于临床用量的 10、2 5、5 0倍 )给大鼠连续灌胃 90天 ,观察大鼠的一般状况 ,试验结束时进行血液学、血液生化及病理检查。结果未见动物异常反应及死亡 ,小鼠最大耐受量达 5 0g/kg(相当于人用量的 12 82倍 )。大鼠连续给药 90天及停药后 15天 ,体重增长基本一致 ,主要组织的脏器系数、血常规及血液生化指标均在正常范围内 ,与对照组比较差异无显著意义 (P >0 0 5 )。主要脏器的肉眼尸检及病理组织学镜检也未见明显病理改变。结论血脉通胶囊是一种低毒和安全的药物。

 
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