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gamma
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    Cytogenetic dosimetry for a victim accidentally exposed to 192 Ir gamma rays.
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  gamma
It is also shown that on the nilmanifold $\Gamma\backslash (H^3\times H^3)$ the balanced condition is not stable under small deformations.
      
For $\gamma\in\mathbb{R}$ let $C(\gamma)$ be the set of all $f\in{\mathcal S}^\prime$ for which$\sum_{n=0}^{\infty}\,|(f,h_n)|\,(n+1)^{\gamma}>amp;lt;\infty$, where $(h_n)_{n=0,1,\dots}$ is the orthonormal base of Hermite functions.
      
We show that $C(\gamma)\subset S_0$ if and only if $\gamma\geq\frac{1}{4}$ and that $S_0\subset C(\gamma)$ if and only if $\gamma\leq{-}\frac{1}{4}$.
      
Using these results we give an explicit solution of the problem of optimal reconstruction of functions from Sobolev's classes $W^{\gamma}_{p}(M^{d})$ in $L_{q}(M^{d}), 1 \leq q \leq p \leq \infty$.
      
This result generalizes the characterization of Fourier series of distributions with a distributional point value given in [5] by $\lim_{x\rightarrow\infty}\sum_{-x\leq n\leq ax}a_{n}e^{inx_{0}}=\gamma\ (\mathrm{C},k)\,$.
      
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This paper describes the dynamic observation of effect ofcontinuous low dose rate gamma irradiation during ninety days on T-lym-phocyte transformation rate in dogs.The certain effect of restraint on cellularimmunity function at accumulation dose of 8.9 rad was found.Althoughthe effect had some reversal after thirty days of stop irradiation,it did notreach the level of preirradiation.

本实验通过连续90天低剂量率γ照射对狗外周血淋转率的动态观察,发现8.9rad 的累积剂量即对狗的细胞免疫功能有一定的抑制作用。停止照射后一个月,虽有一定恢复,但尚未达到照前水平。

On September 1, 1930, a 25-year old worker was accidently exposed by a high-intensity 60Co source of approximate 206X1013Bq. An acute bone marrow-type radiation sickness was observed after an accidental nonuniform gamma-ray irradiation. The estimated equivalent whole-body uniform irradiation dose was 5.22 Gy.The patient survived and was followed up for a period of 3 years from 1981 to 1983. The patient has been recalled at intervals of one year for routine examinations and hematological, biochemical, cytogenetic...

On September 1, 1930, a 25-year old worker was accidently exposed by a high-intensity 60Co source of approximate 206X1013Bq. An acute bone marrow-type radiation sickness was observed after an accidental nonuniform gamma-ray irradiation. The estimated equivalent whole-body uniform irradiation dose was 5.22 Gy.The patient survived and was followed up for a period of 3 years from 1981 to 1983. The patient has been recalled at intervals of one year for routine examinations and hematological, biochemical, cytogenetic and ophthalmological examinations and special tests if indicated. The patient was examined most recently in July 1983.

国内大剂量~(60)Coγ线事故导致急性外照射放射病患者共有三起。本文报告一例男性患者于1980年9月1日受到206.1×10~(13)Bq~(60)Coγ线全身不均匀照射。全身等效均匀照射剂量为5.22Gy。初期临床表现和估算剂量符合急性重度骨髓型放射病。在外院经过胚胎肝输注及抗放药物治疗成功。患者在恢复期的最初三年,在我院进行系统随访观察,发现染色体畸变,精子的微观结构及脑电图尚未完全恢复正常,晶体混浊出现在照射后26个月,进展快。采用一般药物对症治疗。

Two inhibitory substances were isolated from the calf spleens and the porcine blood, which were respectively designated to as Granulopoiesis Inhibitory Factor (GIF) and Plasma Nonspecific Immunosuppressive Protein (PNIP).The biological activity of GIF and PNIP was examined.GIF could remarkably inhibit GM-CFU of the BaBL/C mouse bone marrow cells but had no effect on the 3H-TdR incorporation into the mouse spleen cells and mouse lymphoma L5178Y cells.PNIP could significantly suppress the proliferation of the...

Two inhibitory substances were isolated from the calf spleens and the porcine blood, which were respectively designated to as Granulopoiesis Inhibitory Factor (GIF) and Plasma Nonspecific Immunosuppressive Protein (PNIP).The biological activity of GIF and PNIP was examined.GIF could remarkably inhibit GM-CFU of the BaBL/C mouse bone marrow cells but had no effect on the 3H-TdR incorporation into the mouse spleen cells and mouse lymphoma L5178Y cells.PNIP could significantly suppress the proliferation of the lymphocytes from human peripheral blood and C57BL/6J mouse spleen and the malignant lymphoid cells,L5178Y cells and Raji cells, but had no effect on Hela cells and Vero-E6 cells which are non?lymphoid cells.GIF and PNIP could also result in the decline of the radiosensi-tivity of their responsive target cells to e(1Co Gamma rays .When the GIF-treated mouse bone marrow cells were irradiated,the number of their GM-CFU was much more than that of the control.The survival rate and the 3H-TdR incorporational rate of the L5178Y cells which were exposure to PNIP and then were irradiated was higher than that of the control.The radioprotective activity of the tissue inhibitory factors was closely related to the time of acting on the target cells, the concentration of them and the doses of irradiation.

作者分别从小牛脾脏和猪血浆中提取了粒细胞生成抑制因子(GIF)和血浆非特异性免疫抑制蛋白(PNIP),并对它们的生物学活性进行检测。GIF能够明显抑制BaBL/C小鼠骨髓粒-巨噬细胞集落形成单位(GM-CFU),但对小鼠脾脏细胞及小鼠淋巴瘤L5178Y细胞的~3H-TdR掺入无明显作用;PNIP能够显著地抑制C57BL/6J小鼠脾脏和人外周血淋巴细胞的增殖反应,同时也能够抑制小鼠淋巴瘤L5178Y细胞和人淋巴瘤Raji细胞的生长,但对非淋巴细胞系列的Hela细胞和Vero-E6细胞则无明显作用。GIF和PNIP均可使相应靶细胞的辐射敏感性降低。经GIF处理的骨髓细胞,受照射后其GM-CFU较对照组明显增多;经PNIP处理的L5178Y细胞受照射后其细胞生存率和~3H-TdR掺入率亦均较对照组为高。组织抑制因子的辐射防护作用与它们作用于靶细胞的时间和浓度以及照射剂量的大小密切相关。

 
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