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halothane hepatitis
相关语句
  氟烷性肝炎
     Antigen presenting action and MHC limitation of Kupffer cells in immune response of halothane hepatitis
     氟烷性肝炎免疫应答中Kupffer细胞的抗原递呈作用和MHC限制
短句来源
     It is concluded that an imbalance of TXA2/PGI2 may be partly responsible for halothane hepatitis.
     提示氟烷性肝炎与TXA_2/PGI_2平衡失调有一定的关系。
短句来源
     The proliferative effect on lymphocytes of specific antigen in immune response of Guinea pigs halothane hepatitis
     氟烷性肝炎免疫应答中特异性抗原的促淋巴细胞增殖作用
短句来源
     Objective To study the lymphocytes proliferation stimulated by TFA GSA antigen in immune response of Guinea pigs halothane hepatitis.
     目的 探讨豚鼠氟烷性肝炎免疫应答中特异性抗原 (TFA GSA抗原 )刺激淋巴细胞增殖的作用。
短句来源
     Conclusion:As the most important antigen presenting cells in immune response of halothane hepatitis, Kupffer cells can markedly promote immune response, the antigen presenting action of Kupffer cells is limited by major histocompatibility complex.
     结论 :Kupffer细胞是氟烷性肝炎免疫机制中重要的抗原递呈细胞 ,能明显促进机体免疫应答 ,其抗原递呈作用受 MHC限制。
短句来源
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  “halothane hepatitis”译为未确定词的双语例句
     The results suggest that trace ele-ments are released from the liver to blood during halothane-related liver injury and the imbalance of trace elementsmay enhance halothane hepatitis.
     提示氟烷肝损害过程中铜、铁、锌由损伤肝组织向血浆中释放,这种释放程度与肝损害程度相平行。 而且体内微量元素平衡的改变又会加重氟烷性肝损害的发生与发展。
短句来源
  相似匹配句对
     Die Hepatitis G
     庚型肝炎
短句来源
     HEPATITIS A
     A型肝炎
短句来源
     Stereological and cytochemical study on the pathogenesis of hepatitis induced by halothane in rats
     Stereological and cytochemical study on the pathogenesis of hepatitis induced by halothane in rats
短句来源
     A Stereological and Cytochemical Study on the Pathogenesis of Hepatitis Induced by Halothane in Rats
     应用体视学及细胞化学对大鼠氟烷性肝炎发生机理的研究
短句来源
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  halothane hepatitis
The patient reported here developed typical acute halothane hepatitis following the second exposure to the anesthetic; she remained ill and failed to show evidence of improvement during a five-week period.
      
Ultrastructural pathology of a case of halothane hepatitis
      
Halothane hepatitis with renal failure treated with hemodialysis and exchange transfusion
      
Lack of defect in oxidative hydroxylation of debrisoquine in a patient with halothane hepatitis
      
One animal out of 6 that were repeatedly exposed to the blend and sacrificed at 24?h showed liver lesions similar to halothane hepatitis.
      
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Male Sprague-Dawley rats were pretreated with phenobarbital and randomly divided into 6 groups arid wereexposed to O2/N2/1.2MAC anesthetics for 1 hr:NC,21%O2/79%N2;HC,14%O2/86%N2;NH,21%O2/79%N2/1.2MAC halothane;HH,14%O2/86%N2/1.2MAC halothane;NS,21%O2/79%N/1.2MAC sevonurane;HS,14%O2/86%N2/1.2MAC sevoflurane. Plasma amino-acid patterns were analysed using HPLC 24 Hrs after exposureconcluded.Plasma concentrations of serine,glutamine, arginine and tyresine were significantly raised in NH,HHgroups. Plasma concentrations...

Male Sprague-Dawley rats were pretreated with phenobarbital and randomly divided into 6 groups arid wereexposed to O2/N2/1.2MAC anesthetics for 1 hr:NC,21%O2/79%N2;HC,14%O2/86%N2;NH,21%O2/79%N2/1.2MAC halothane;HH,14%O2/86%N2/1.2MAC halothane;NS,21%O2/79%N/1.2MAC sevonurane;HS,14%O2/86%N2/1.2MAC sevoflurane. Plasma amino-acid patterns were analysed using HPLC 24 Hrs after exposureconcluded.Plasma concentrations of serine,glutamine, arginine and tyresine were significantly raised in NH,HHgroups. Plasma concentrations of the three branched chain amino-acid valine,isoleucine,and leucine were notmarkedly changed in all groups,These date suggest that the plasma amino-acid patterns of halothane hepatitis isthe same as that of fulminant hepatic failure, The imbalance of the aromatic and the branched chain amino-acidsmay also be a main cause of hepatic coma of halothane hepatitis.

雄性SD大鼠饮用含苯巴比妥(1mg/ml)的饮水一周后,随机分为6组,每组6例:NC,21%O2/79%N2;HC,14%O2/86%N2;NS,21%O2/79%N2/1.2MAC七氟醚;HS,14%O2/86%N2/1.2MAC七氟醚;NH,21%O2/79%N2/1.2MAC氟烷;HH,14%O2/86%N2/1.2MAC氟烷。吸入时间1h、24h后用高效液相色谱法测定血浆中11种游离氨基酸的含量。结果NH与HH组酪、精、丝、甘、丙、苏、谷氨酰胺含量升高,而支链氨基酸无明显变化。提示氟烷性肝炎的氨基酸谱的变化类似于急性暴发性肝衰竭。

Male Sprague-Dawley rats were pretreated with phenobarbital and randomly divided into 4 groups, The ratswere exposed to O2/N2/1.2 MAC anesthetics for 1 hr and anesthetized by 14%O2/86%N2/(C), 14%O2/86%N2/1. 2MAC enflurane(E),14%O2/86%N2/1. 2MAC sevoflurane(S)and 14%O2/86%/N2/1. 2 MAC halothane(H)respectively. The livers of all rats given halothane had extensive centrilobular necrosis and denaturation. Accom-panying the morphologic damage was an increase in serum glutamic pyruvic transminase(ALT).The concentra-tions...

Male Sprague-Dawley rats were pretreated with phenobarbital and randomly divided into 4 groups, The ratswere exposed to O2/N2/1.2 MAC anesthetics for 1 hr and anesthetized by 14%O2/86%N2/(C), 14%O2/86%N2/1. 2MAC enflurane(E),14%O2/86%N2/1. 2MAC sevoflurane(S)and 14%O2/86%/N2/1. 2 MAC halothane(H)respectively. The livers of all rats given halothane had extensive centrilobular necrosis and denaturation. Accom-panying the morphologic damage was an increase in serum glutamic pyruvic transminase(ALT).The concentra-tions of TXB2 in plasma and the liver were significantly raised in group H(P< 0.01).With regard to 6-keto-PGF1a, no statistically significant difference was found in all groups.It is concluded that an imbalance of TXA2/PGI2 may be partly responsible for halothane hepatitis.

雄性SD大鼠饮用含苯巴比妥钠(1mg/ml)的饮水1周后,随机分为四组,每组8例,分别吸入:C,14%O_2/86%N_2;E,14%O_2/86%N_2/1.2MAC安氟醚;S,14%O_2/86%N_2/1.2MAC七氟醚;H,14%O_2/86%N_2/1.2MAC氟烷1h。24h后发现H组血浆ALT活性显著高于其它各组,并有明显的小叶中心性肝细胞坏死及汇管区炎细胞浸润,血窦重度充血。E组可见部分肝小叶内有小叶中心性坏死及空泡变性。H组肝匀浆及血浆中TXB_2含量显著高于其它各组。6-keto-PGF1a各组间均无显著差异。提示氟烷性肝炎与TXA_2/PGI_2平衡失调有一定的关系。

ale Sprague-Dawley rats were pretreated with phenobaraital and randomly divided into 4 groups. The ratswere exposed to O2/N2/1.2 MAC anesthetics for 1 hr : 21%O2/79%N2(NC); 14%O2/86%N2(HC); 14%O2/86%N2/1. 2 MAC sevoflurane(HS) and 14%O2/86%N2/1. 2MAC halothane(HH)respectively. The rats givenhalothane had significantiy lower hepatic Cu2+, Zn2+ values and higher hepatic Ca2+ values(P<0. 01 ). Plasmacontents of Fe2+”,Cu”, Zn”were significantly raised in group HH (P<0.05). The results suggest that trace ele-ments...

ale Sprague-Dawley rats were pretreated with phenobaraital and randomly divided into 4 groups. The ratswere exposed to O2/N2/1.2 MAC anesthetics for 1 hr : 21%O2/79%N2(NC); 14%O2/86%N2(HC); 14%O2/86%N2/1. 2 MAC sevoflurane(HS) and 14%O2/86%N2/1. 2MAC halothane(HH)respectively. The rats givenhalothane had significantiy lower hepatic Cu2+, Zn2+ values and higher hepatic Ca2+ values(P<0. 01 ). Plasmacontents of Fe2+”,Cu”, Zn”were significantly raised in group HH (P<0.05). The results suggest that trace ele-ments are released from the liver to blood during halothane-related liver injury and the imbalance of trace elementsmay enhance halothane hepatitis.

雄性SD大鼠饮用含苯巴比妥钠(1mg/ml)的饮水1周后,随机分为四组,每组8例:NC,21%O_2/79%N_2;HC,14%O_2/86%N_2;HS,14%O_2/86%N_2/1.2MAC七氟醚;HH,14%O_2/86%N_2/1.2MAC氟烷。吸入时间为1h。24h后用原子吸收分光光度法测定血浆及肝匀浆中锌、铜、铁、钙的含量。结果HH组肝匀浆中铜、锌的含量显著低于对照组(P<0.01),钙离子显著高于对照组(P<0.01),HH组血浆中铁、铜、锌含量均显著高于对照组(P<0.05)。提示氟烷肝损害过程中铜、铁、锌由损伤肝组织向血浆中释放,这种释放程度与肝损害程度相平行。而且体内微量元素平衡的改变又会加重氟烷性肝损害的发生与发展。

 
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