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小儿all
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  childhood all
     The DNA aneuploidy rate in childhood ALL patients was41.8%. The DNA aneuploidy of ALL was found in9of17samples with deletion of p16protein and in4of14samples with positive expression of p16,respectively(P=0.2749).
     小儿ALL异倍体发生率为41.8 % ,p16蛋白表达阴性者17例中DNA异倍体9例 ,p16蛋白表达阳性者中异倍体4例 ,两组差异无显著性 (P=0.2749)。
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     CONCLUSION:There exists decreased expression of TAP in both childhood ALL and AML, which probably contributes to the escape of leukemia cells from immune surveillance.
     结论:小儿ALL和AML均存在TAP分子低表达,这可能促使白血病细胞逃避免疫监视;
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     In order to investigate the incidence of p16 gene deletion in childhood ALL and to determine the association between this gene deletion and the clinical characteristics of childhood ALL, the deletion of p16 gene was detected in 42 children with ALL by PCR analysis.
     作者用PCR方法检测了42例小儿急性淋巴细胞白血病(ALL)p16基因3个外显子缺失状况,初步探讨了pl6基因缺失与小儿ALL的发病、临床表现及预后的相关性。
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     Conclusion Low dose L-Asp has definite efficacy for childhood ALL, while avoids serious side effects from standard dose L-Asp.
     结论应用小剂量L-Asp治疗小儿ALL,即使对因用常规剂量L-Asp发生过不良反应的患儿也具有肯定的安全性,而且对小儿ALL具有较确切的药效性。
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     Conclusion:There were multiple IgH bi allelic rearrangements and co existing clones in childhood ALL.
     结论:从基因水平证实,小儿ALL多发生IgH双等位基因重排,且有多个克隆并存的现象。
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  “小儿all”译为未确定词的双语例句
     [Conclusion] Continuous intervenous infusion of HDMTX (each time 2 g/m2 or 3 g/m2) to treat children with ALL is safe and effective.
     结论大剂量MTX(每次2 g/m2,3 g/m2)连续24 h静滴治疗小儿ALL是安全有效的。
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     Clinical analysis of the therapeutic effect of 43 children with ALL
     43例1~7岁小儿ALL临床疗效分析
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     ObjectiveTo compare the clinical and MIC characteristics of childhood acute lymphoblastic leukemia between prednisone good responder (PGR) and poor responder (PPR).
     分析小儿ALL泼尼松试验敏感者(prednisone good responder,PGR)与不敏感者(prednisone poor responder,PPR)临床及MIC特点。
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     Conclusion The cure rate is high, when employing combined therapy to treat ALL complicating ARDS.
     结论采用综合疗法治疗小儿ALL并ARDS,治愈率高。
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     In order to explore the availability and its clinical significance for detecting the minimal leukemic cells of CSF in patients with acute lymphoblastic leukemia (ALL), 21 ALL cases were determined with nested PCR amplifying TCR Vδ2-Vδ3 rearranged fragment.
     为探索急性淋巴细胞白血病(ALL)患儿脑脊液(CSF)中微量白血病细胞的检测方法及其意义,本文采用巢式聚合酶链反应(PCR)扩增T细胞受体的Vδ_2-Dδ_3重排片段方法,对21侧小儿ALL进行检测。
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     Objective:To explore the curative effect of children with acute lymphoblastic leukemia(ALL).
     目的:探讨小儿急性淋巴细胞白血病(ALL)的治疗效果。
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     To investigate the clonal diversity in childhood acute lymphoblastic leukemia (ALL).
     目的探讨小儿急性淋巴细胞白血病(ALL)的克隆多样性。
短句来源
     All.
     建议All.
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     in adults and decreased appropriately in children.
     ,小儿酌减。
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     ( 2 ) As for the 2 groups of All.
     ( 2 ) All.
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  childhood all
Methods: The GCR level of venous blood lymphocytes was measured by receptor radioligand binding assay in 50 cases with childhood ALL and 41 normal children.
      
Gene expression arrays and comparative genomic hybridization have further extended our appreciation of the known immunophenotypic and genetic diversity of childhood ALL.
      
Future studies of neurobehavioral outcome are briefly elaborated in the context of current chemotherapy approaches used in the treatment of childhood ALL.
      
It was suggested that GSTM1 null genotype might be a risk genotype of childhood ALL.
      
While there as no correlation between GSTT1 null genotype and childhood ALL.
      
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Objective To search for the relationship between glucocorticoid receptor (GCR) quantity and therapeutic effect and prognosis in the Children with acute lymphatic leukemia (ALL).Methods The levels of GCR of lymphocyte in venous blood in 50 children with ALL and 41 healthy children were determined by radioligand reccptor assay (RRA) respectively, and observing the relationship between GCR and the therapeutic effect and prognosis.Results The value of lymphocyte GCR in the venous blood in healthy children is 4651±1617...

Objective To search for the relationship between glucocorticoid receptor (GCR) quantity and therapeutic effect and prognosis in the Children with acute lymphatic leukemia (ALL).Methods The levels of GCR of lymphocyte in venous blood in 50 children with ALL and 41 healthy children were determined by radioligand reccptor assay (RRA) respectively, and observing the relationship between GCR and the therapeutic effect and prognosis.Results The value of lymphocyte GCR in the venous blood in healthy children is 4651±1617 combining site/cell, the range for 95 percent normal reference value is froxn 1482 to 7800 colnbining site/cell. The value of lymphoeyte GCR (6695±5256 combining site/cell) of the chilbren with ALL is obviously higher than that of the healthy children (t = 2, 50, P<0.05). The value of GCR in good prognosis group is obviously higner than that in bad prognosis group in the children with ALL.Conclusion The GCR value of lymphocyte in venous blood in the children with ALL could be regarded as a biochemical index for judging prognosis and therapeutic effect.

研究目的探讨ALL患儿LGCR数量与化疗效果及预后的关系。研究方法以受体放射配基结合分析法检测50例ALL患儿及41例正常小儿静脉血淋巴细胞GCR水平,并观察其与化疗效果与预后的关系。研究结果正常小儿静脉血淋巴细胞GCR值为4651±1617结合位点/细胞,其95%正常参考值范围为1482~7800结合位点/细胞。ALL患儿淋巴细胞GCR值(6695±5256结合位点/细胞)明显高于正常小儿(t=2.50,P<0.05)。小儿ALL预后好组GCR值明显高于预后差组(t=4.39,P<0.001)。结论ALL患儿外周静脉血淋巴细胞GCR值可以作为一项生化指标而应用于判断预后及指导联合化疗。

AbstractThe cytolvtic effect of normal adult donors’LAKcells on 11 cases of ALL-blasts of children at the tirneof diagnosis or relapse before treatnient wasinvestigated. The cytolytic activrty of LAK cells induced by rhIL- 2 ( 1000U/ml) a alone or combinationof rhIL-2 (250U/ml) with rhTNFa (5000U/ml)onchildren’s ALL-blasts was  ̄35.8%±9.4%,42.5%± 7. 3% respectively. The result showed that the cy-tolytic activity was similar when the dose of rhlL-2decreased 3/4 in combination wkh a low dose ofrhTNFa. The productive...

AbstractThe cytolvtic effect of normal adult donors’LAKcells on 11 cases of ALL-blasts of children at the tirneof diagnosis or relapse before treatnient wasinvestigated. The cytolytic activrty of LAK cells induced by rhIL- 2 ( 1000U/ml) a alone or combinationof rhIL-2 (250U/ml) with rhTNFa (5000U/ml)onchildren’s ALL-blasts was  ̄35.8%±9.4%,42.5%± 7. 3% respectively. The result showed that the cy-tolytic activity was similar when the dose of rhlL-2decreased 3/4 in combination wkh a low dose ofrhTNFa. The productive ability of IL-2 from pa-tients’PBMNCs stimulated by PHA was evaluated.The mean value of ALL children (115.4±21.7U/ml)was higher than that of control group (78.3±12.2U/ml ). Analysis of linear correlation revealed that therewas posrtive correlation between the productive abilityof IL-2 from Patients’PBMNCs stimulated by PHAand the susceptibilrty of ALL-blasts to LAK cells

用1000U/ml重组人白细胞介素2(rhlL-2)诱导的正常人淋巴因子激活的杀伤细胞(LAK)对11例初诊或复发未治的小儿急性淋巴细胞性白血病(ALL)细胞进行了37次体外杀伤检测,结果全部表现出LAK细胞杀伤活性(35.8%±9.40%);另一组rhlL-2250U/ml+重组人肿瘤坏死因子。a(rhTNFa)500U/ml共同诱导的正常人LAK细胞对相同11例小儿ALL细胞进行了33次体外杀伤检测,也全部表现出杀伤活性(42.5%±7.3%)。两组间t检验差异无显著意义(P>0.05)。实验结果表明:1000U/mlrhlL-2诱导的正常人LAK细胞对小儿ALL。细胞具有明显的杀伤活性;当rhlL-2量减少3/4,加用中小剂量的rhTNFα可获得相近的杀伤活性。11例ALL患儿外周血单个核细胞(PBMNCs)经植物凝血素(PHA)刺激后产生白细胞介素2(IL-2)的能力为115.4±21.7U/ml,高于正常儿童组78.3±12.2U/ml(P<:0.01)。ALL患儿PBMNCs产生LL-2的能力与相应机体ALL细胞对LAK细胞杀伤敏感性呈正相关。提示了机体自身免疫特别是T细胞...

用1000U/ml重组人白细胞介素2(rhlL-2)诱导的正常人淋巴因子激活的杀伤细胞(LAK)对11例初诊或复发未治的小儿急性淋巴细胞性白血病(ALL)细胞进行了37次体外杀伤检测,结果全部表现出LAK细胞杀伤活性(35.8%±9.40%);另一组rhlL-2250U/ml+重组人肿瘤坏死因子。a(rhTNFa)500U/ml共同诱导的正常人LAK细胞对相同11例小儿ALL细胞进行了33次体外杀伤检测,也全部表现出杀伤活性(42.5%±7.3%)。两组间t检验差异无显著意义(P>0.05)。实验结果表明:1000U/mlrhlL-2诱导的正常人LAK细胞对小儿ALL。细胞具有明显的杀伤活性;当rhlL-2量减少3/4,加用中小剂量的rhTNFα可获得相近的杀伤活性。11例ALL患儿外周血单个核细胞(PBMNCs)经植物凝血素(PHA)刺激后产生白细胞介素2(IL-2)的能力为115.4±21.7U/ml,高于正常儿童组78.3±12.2U/ml(P<:0.01)。ALL患儿PBMNCs产生LL-2的能力与相应机体ALL细胞对LAK细胞杀伤敏感性呈正相关。提示了机体自身免疫特别是T细胞免疫状况对LAK细胞杀?

Seventeen children with acute

超大剂量程序化疗方案治疗小儿ALL17例,取得良好效果。该方案是根据肿瘤细胞动力学和抗肿瘤药物作用动力学理论制定的。强调药物配伍的治疗顺序和治疗时间的程序性。用VALP诱导治疗;用大剂量MTX作冲击治疗;用CPA或EMA作早期强化;用6-MP和MTX作维持治疗。强调治疗开始21周时,应用VALD作再次诱导治疗。强化治疗加用大剂量Ara-c冲击治疗。对普通型ALL重视晚期反复强化治疗,对高危型ALL重视早期强化治疗。为保证该方案顺利进行,必须预先克服超大剂量程序化疗产生的血液学毒性和临床学毒性。

 
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