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暂时性缺血
相关语句
  temporarily ischemic
     Methods The middle cerebral artery of rats was made to be temporarily ischemic, and the contents of Ca 2+ , Na + and K + in their brain tissues were measured to compare the effects when they were punctured respectively by the proxinmal Baihui (GV 20) and Renzhong (GV 26), and the distal Huantiao(GB 30) and Kunlun (BL 60).
     方法 采用大鼠大脑中动脉暂时性缺血模型 ,以脑组织 Ca2 + 、Na+ 、K+ 含量为指标 ,比较电针近脑部的百会、人中穴和远脑部的环跳、昆仑穴对脑缺血的影响。
短句来源
  “暂时性缺血”译为未确定词的双语例句
     EXPRESSION OF c-fos PROTOONCOGENE PROTEIN (FOS) IN THE RAT BRAIN INDUCED BY TRANSIENT GLOBAL CEREBRAL ISCHEMIA
     全脑暂时性缺血诱导c-fos原癌基因蛋白(FOS)在大鼠脑内的表达
短句来源
     THE CHARACTERISTICS OF C-FOS PROTOONCOGENE PROTEIN (FOS) EXPRESSION IN RAT HIPPOCAMPUS INDUCED BY TRANSIENT GLOBAL CEREBRAL ISCHEMIA
     全脑暂时性缺血诱导原癌基因蛋白(Fos)在大鼠海马结构表达的特征
短句来源
     Expression of c—fos Protooncogene Protein(Fos)In The Rat Hypothalamus Induced By Transient Global Cerebral Ischemia
     全脑暂时性缺血诱导c—fos癌基因蛋白(FoS)在大鼠下丘脑的表达
短句来源
     Nimodipine (1 mg/kg) was injected intraperitoneally following brain ischemia, the calcium content in hippocampus had no significant difference between Nimodipine treated group and ischemic group.
     缺血-尼莫地平组的大鼠在夹闭颈总动脉造成鼠脑暂时性缺血的同时於腹腔内注射尼莫地平。 比较对照组及暂时性缺血再灌注72小时后海马组织钙含量,用原子吸收分光光度计测定发现缺血组海马组织钙含量增高,但缺血-尼莫地平组海马组织钙含量与缺血组差异无显著性。
短句来源
     The immunocytochemical method(ABC)has been used to detect the distribution and time course of c—fos proto—oncogene protein(Fos)expression in the rat hy- pothalamus induced by global cerebral ischemia and recirculation.
     本文用免疫组化(ABC)方法探查了大鼠全脑暂时性缺血诱导的c—fos癌基因蛋白(Fos)在下丘脑表达的分布以及时间过程。
短句来源
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  相似匹配句对
     Experimental Study of Stress Echocardiography Testing for Detecting Transient Myocardial Ischemia: Effects of Quantitative Dipyridamole Stress Test on the Heart of Dogs with Critical Coronary Stenosis
     负荷超声心动图检测暂时性心肌缺血的试验研究
短句来源
     Acute myocardial ischemia
     心肌缺血
短句来源
     (3)ischemia(I) ;
     (3)缺血组(I);
短句来源
     The Protection Effect of Temporary Portocaval Shunt for the Cold Ischemia Reperfusion Injury in Rats Liver
     暂时性门体分流对大鼠冷缺血再灌注肝脏保护作用的研究
短句来源
     MR imaging of transient osteoporosis
     暂时性骨质疏松的MRI表现
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  temporarily ischemic
Inflation of the balloon causes the overlying skin and dermis to blanch and become temporarily ischemic.
      


Angina pectoris is a clinical syndrome caused by transient myocardial ischemia which is due to an imbalance between myocardial oxygen supply and demand. Many drugs for the treatment of angina pectoris have been shown to increase myocardial tolerance of hypoxia, so it is of theoretical significance and practical value to search for compounds for improving the myocardial tolerance of hypoxia. In view of the fact that vitamin B_(15) can improve the tolerance of hypoxia and Lidocaine possesses antiarrythmic activity,...

Angina pectoris is a clinical syndrome caused by transient myocardial ischemia which is due to an imbalance between myocardial oxygen supply and demand. Many drugs for the treatment of angina pectoris have been shown to increase myocardial tolerance of hypoxia, so it is of theoretical significance and practical value to search for compounds for improving the myocardial tolerance of hypoxia. In view of the fact that vitamin B_(15) can improve the tolerance of hypoxia and Lidocaine possesses antiarrythmic activity, a series of ring compounds carrying substituted amino acetylamino groups were synthesized by combining these two structures in one molecule. These compounds were synthesized by condensation of suitable aniline or alcohol with chloroacetylchloride, followed by reacting with corresponding amines. Preliminary pharmacological examination revealed that some of the compounds especially ω-isopropylamino-2,4-dimethylacetanilide (15), the hydrochloride of ω-isopropylamino acetanilide (19) as well as the hydrocbloride of ω-diethylamino-3,4-dichloro-acetanilide (11) showed significant activity to improve the tolerance of hypoxia for heart, while, 4,6-Bis (isopropyl-amino acetylamino) 1,3-xylol (61), 4,6-Bis-(s-butylamino acetylamino) 1,3-xylol (62), 4,6-Bis (diisopropylaminoacetyl-amino) 1,3-xylol (60) did not exhibit such activity but possessed a strong antiarrythmic action.

心绞痛是由于心肌对氧的供需失去平衡而引起暂时性缺血的临床症状。很多治疗心绞痛的药物可以增加心肌缺氧的耐受力。因此寻找能耐缺氧的化合物对冠心病的治疗可能具有实用价值与理论意义。鉴于维生素B_(15)能改善缺氧的能力,以及利多卡因有抗心律不齐的作用。作者将两类化合物进行拼合,合成了一系列带有取代胺基乙酰胺基的环状化合物。合成方法是以取代的苯胺或醇与氯乙酰氯缩合,再与各种胺类反应,即形成所需产物。药理试验结果显示,其中许多化合物具有良好的耐缺氧作用,尤以异丙胺乙酰(2,4-二甲基苯胺)(15)、异丙胺乙酰苯胺盐酸盐(19)及二乙胺乙酰(3,4-二氯苯胺)盐酸盐(11)的作用更为显著,而4,6-双(异丙胺乙酰胺基)1,3-二甲苯(61)、4,6-双(仲丁胺乙酰胺基)1,3-二甲苯(62)及4,6-双(二异丙胺乙酰胺基)1,3-二甲苯(60)虽无耐缺氧作用,但却具有较强的抗心律不齐作用。

Data on stroke risk in patients with carotid stenosis is inadequate. Using carotid Doppler, we compared 491 patients with asymptomatic neck bruits, 646 with transient ischemic attack and on neck bruits, 255 with transient ischemic attack and neck bruits, 195 with stroke and no neck bruits,66 with stroke and bruits, and 500 controls.Four percent of the arteries of the control group had abnormal Dopplers. Forty percent of the asymptomatic neck bruit group were abnormal with a bimodal distribution and ischemic...

Data on stroke risk in patients with carotid stenosis is inadequate. Using carotid Doppler, we compared 491 patients with asymptomatic neck bruits, 646 with transient ischemic attack and on neck bruits, 255 with transient ischemic attack and neck bruits, 195 with stroke and no neck bruits,66 with stroke and bruits, and 500 controls.Four percent of the arteries of the control group had abnormal Dopplers. Forty percent of the asymptomatic neck bruit group were abnormal with a bimodal distribution and ischemic ceremic cerebral events predominating in the severe stenosis group. Severity of internal carotid stenosis increased prior to the events. In the transient ischemic attack and stroke groups, the bimodal. junction was at 85%(15 kilohartz)stenosis(shif to the right) increased in all symptomatic groups when neck bruits were present. This distribution was not explained by age and sex using analysis of variance. The consistent bi-modal distribution in these patients indicates that 85% carotid stenosis is critical to outcome.

关于颈动脉狭窄患者发生中风的危险度的资料尚少。我们应用颈动脉多普勒观察五组患者。1.无症状、有颈部血管杂音491例; 2.暂时性缺血发作、无颈部血管杂音646例;3.暂时性缺血发作、有颈部血管杂音255例;4.中风、无颈部血管杂音195例;5.中风、有颈部血管杂音66例和对照组(500例)作了比较。对照组中,4%的多普勒是异常的。无症状、有颈部血管杂音组中,40%有异常而具有双峰基分布,在严重的狭窄组中,缺血性脑中风居多。在发作之前,颈内动脉狭窄的严重程度有所增加。在暂时性缺血发作组和中风组内,85%的狭窄患者可出现双峰基连接。在全部有症状组中,当出现颈部血管杂音时,颈内动脉狭窄的范围和严重程度均增加。应用方差分析,用年龄和性别不能解释这种分布。在这些(颈动脉狭窄)患者中,出现持续性的双峰基分布,表明有85%预后是危急的。

The immunocytochemical method(ABC)has been used to detect the distribution and time course of c—fos proto—oncogene protein(Fos)expression in the rat hy- pothalamus induced by global cerebral ischemia and recirculation.The results indicated that Fos expression was rapidly and transiently induced in the broad area and nuclei of the hypothalamus from 15 minutes to 12 hours after global ischemia—recirculat- tion.Fos immunoreactivity products were located in the nucleus of the neurons.Fos induction appeared at 15min,peaked...

The immunocytochemical method(ABC)has been used to detect the distribution and time course of c—fos proto—oncogene protein(Fos)expression in the rat hy- pothalamus induced by global cerebral ischemia and recirculation.The results indicated that Fos expression was rapidly and transiently induced in the broad area and nuclei of the hypothalamus from 15 minutes to 12 hours after global ischemia—recirculat- tion.Fos immunoreactivity products were located in the nucleus of the neurons.Fos induction appeared at 15min,peaked at 3 hr and subsided at 12 hr after ischemia— recirculation.The earliest and strongest expression occurred in the periventricle zone, particularly in the supraoptic and paraventricle nuclei,followed by the medial and later- al zones of the hypothalamus.According to these results,we postulate that the rapid and transient induction of c—los protooncogene protein in the hypcthalamus are involved in mediating the endocrine,autonomic and behavioral responses and maintaining the in- dividual homeostasis to the global ischemia.

本文用免疫组化(ABC)方法探查了大鼠全脑暂时性缺血诱导的c—fos癌基因蛋白(Fos)在下丘脑表达的分布以及时间过程。结果如下:四管阻塞全脑缺血后15分钟至12小时内,Fos在下丘脑广泛区域和核团出现短暂而快速的表达。Fos免疫阳性(Fos—IR)产物定位于神经元的胞核内。缺血再循环后15分钟开始表达,3小时达到高峰,12小时消退。最早和最强的表达出现在室周区(特别是视上核和室旁核),其次是内侧区和外侧区。结果提示全脑缺血诱导的Fos在下丘脑短暂而快速的表达与调节缺血引起的内分泌、自主功能、情感和行为反应和维持机体内环境的稳定有关。

 
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