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小管间质     
相关语句
  tubulointerstitial
    TGF-β/Smad Signaling Pathway and Renal Tubulointerstitial Fibrosis
    TGF-β/Smad信号通路在肾小管间质纤维化中的作用及机制的实验研究
短句来源
    The Protective Effects and Mechanisms of PPARγ and TZDs on Renal Tubulointerstitial Inflammation and Fibrosis
    PPARγ和TZDs对肾小管间质炎症和纤维化的保护作用及其机制
短句来源
    Clinical Study on the Relationship between Transforming Growth Factor Beta-1 and Tubulointerstitial Fibrosis in Obstructive Nephropathy
    转化生长因子β_1与梗阻性肾病肾小管间质纤维化关系的研究
短句来源
    Clinical Tubulointerstitial Nephritis——Clinical Analysis of 18 Cases
    慢性小管间质性肾炎——18例临床分析
短句来源
    Renal Tubulointerstitial Lesions in 820 Cases of Glomerular Diseases
    820例肾小球疾病的肾小管间质病变
短句来源
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  tubular interstitial
    The study of Tubular Interstitial Damages in Lupus Nephritis
    狼疮性肾炎小管间质病变的研究(附49例临床及病理分析)
短句来源
    Method Group A(without renal tubular interstitial damage,n=12) and group B(with renal tubular interstitial damage,n=19) were compared and analysed.
    方法 将患者分A组 (无合并肾小管间质损害 ,n =12 )和B组 (合并肾小管间质损害 ,n =19)进行对比分析。
短句来源
    Results The occurrence rate of the renal tubular interstitial damage in nephrotic syndrome was very high(19/31,61.29%). The urinary protein in 24 hours of group B was higher than that of group A( P <0.05),and the difference of creatinine in blood between group A and B was not singificant( P >0.05).
    结果 NS时肾小管间质病变的发病率相当高 ( 19/31;6 1.2 9% ) ,B组 2 4小时尿蛋白排出量明显高于A组 (P <0 .0 5 ) ,两组血肌酐值差别无显著性意义 (P >0 .0 5 )。
短句来源
    All 24 renal specimens showed tubular interstitial lesions,70.8% was acute tubular necrosis.
    肾活检 2 4例 ,均为肾小管间质病变 ,70 .8%为肾小管坏死。
短句来源
    Methods Group A(without renal tubular interstitial damage,n=33) and group B(with renal tubular interstitial damage,n=49) were compared and analysed.
    方法 将患者分A组 (无合并肾小管间质损害 ,n =33)和B组 (合并肾小管间质损害 ,n =4 9)进行对比分析。
短句来源
更多       
  tubule interstitial
    Relationship between urine α1-MG, NAG and renal tubule interstitial pathological change
    尿α_1-MG和尿NAG与肾小管间质病理变化的关系
短句来源
    Roles of transforming growth factor β1 in renal tubule interstitial fibrosis in obstructive nephropathy rats
    转化生长因子-β1在梗阻性肾病大鼠肾小管间质纤维化中的作用
短句来源
    Objective To investigate the relationship between urine alpha 1-microglobulin (α1-MG), N-acetyl-beta-D-glucosamidase (NAG) and renal tubule interstitial pathological change.
    目的 探讨尿α1-微球蛋白(α1-MG)、尿N-乙酰-β-D-葡萄糖苷酶(NAG)与肾小管间质病理变化的关系。
短句来源
    there was a correlation between urine α1-MG, NAG and renal tubule interstitial pathological change (P <0. 01). Conclusion Renal tubule interstitial pathological change could be reflected by both urine α1-MG and NAG.
    结果 尿α1-MG、尿NAG水平与肾小管间质病变程度呈显著正相关(P<0.01)。
短句来源
    Urine α1-MG and NAG could be considered as an important reference index for the prognosis of the renal tubule interstitial pathological change.
    结论 尿α1-MG和NAG可动态观察肾小管间质病变和临床治疗效果,以判断肾脏疾病的预后。
短句来源
更多       
  renal tubulointerstitial
    TGF-β/Smad Signaling Pathway and Renal Tubulointerstitial Fibrosis
    TGF-β/Smad信号通路在肾小管间质纤维化中的作用及机制的实验研究
短句来源
    The Protective Effects and Mechanisms of PPARγ and TZDs on Renal Tubulointerstitial Inflammation and Fibrosis
    PPARγ和TZDs对肾小管间质炎症和纤维化的保护作用及其机制
短句来源
    Renal Tubulointerstitial Lesions in 820 Cases of Glomerular Diseases
    820例肾小球疾病的肾小管间质病变
短句来源
    Role of type I plasminogen activator inhibitor in mediating renal tubulointerstitial injury of patients with IgA nephropathy
    Ⅰ型纤溶酶原激活物抑制物在IgA肾病肾小管间质损伤中的作用研究
短句来源
    Relationship between expression of Smad6 and Smad7 and renal tubulointerstitial injury following unilateral ureteral obstruction in rats
    Smad6、7蛋白表达与梗阻性肾病小管间质损伤关系的实验研究
短句来源
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      tubulointerstitial
    Expression of renal cubilin and its potential role in tubulointerstitial inflammation induced by albumin overload
          
    Infiltration of CD-3 and ED-1-positive cells was predominant in tubulointerstitial areas displaying signs of increases of cubilin expression and albumin accumulation.
          
    Cubilin was required for handling a greater amount of protein in nephrotic status and albumin-induced production of MCP-1 and RANTES by renal tubular cells, which further initiated tubulointerstitial inflammation in proteinuric disease.
          
    Key factors involved in progression of renal disease include accumulation of extracellular matrix in the glomerular and tubulointerstitial compartments, epithelial to mesenchymal transformation, and vascular changes.
          
    Expression of connective tissue growth factor in renal tubulointerstitial fibrosis in rats and its pathogenic role
          
    更多          
      tubular interstitial
    A patient with myoglobininduced acute tubular interstitial nephritis was found to have initially an asymmetric renal affection as evidenced by renography.
          
    Tubular interstitial alterations in these disorders other than cast nephropathy were firmly documented when careful ultrastructural studies were conducted experimentally and using clinical material.
          
      renal tubulointerstitial
    Expression of connective tissue growth factor in renal tubulointerstitial fibrosis in rats and its pathogenic role
          
    In order to explore the role of connective tissue growth factor (CTGF) in the pathogenesis of renal tubulointerstitial fibrosis, 48 Wistar rats were randomly divided into sham-operated and unilateral ureteral obstruction (UUO) group.
          
    The renal tubulointerstitial injury index was evaluated according to the MASSON staining.
          
    On the post-UUO day 7, the protein level of CTGF was positively related to the renal tubulointerstitial injury index (r=0.62,P>amp;lt;0.01), the expression of TGF-β1 (r=0.85,P>amp;lt;0.01), col I (r=0.78,P>amp;lt;0.01), and PAI-1 (r=0.76,P>amp;lt;0.01).
          
    The role of protease activated receptor-2 (PAR-2) in the renal tubulointerstitial lesion induced by unilateral ureteral obstruction (UUO) was explored.
          
    更多          


    Eighteen patients of chronic tubulointerstitial nephritis were reported with theclinical and experimental data analized.The results showed that infection and drugdamage might be the main causes of CTIN.The prominent clinical features of thesecases were due to disturbance of renal concentration and acidification ability usuallymanifestating polyuria,polydipsia and renal tubular acidosis accompanied by azote-mia,osteodystrophy and anemia.The pathogenesis and clinical diagnostic criteria ofCTIN were discussed.It...

    Eighteen patients of chronic tubulointerstitial nephritis were reported with theclinical and experimental data analized.The results showed that infection and drugdamage might be the main causes of CTIN.The prominent clinical features of thesecases were due to disturbance of renal concentration and acidification ability usuallymanifestating polyuria,polydipsia and renal tubular acidosis accompanied by azote-mia,osteodystrophy and anemia.The pathogenesis and clinical diagnostic criteria ofCTIN were discussed.It was suggested that diagnosis could be established by meansof clinical data though in lack of histopathogical evidences.

    本文报告18例慢性小管间质性肾炎,并分析其临床与实验室资料,结果表明感染与药物损伤为本病的主要原因,本病的突出临床表现是由于肾脏的浓缩与酸化功能障碍,常表现为多尿多饮及肾小管性酸中毒并伴有氮质血症、骨营养不良及贫血。文中讨论了慢性 TIN 的发病机制及临床诊断指标,认为虽无病理组织学资料,而凭临床资料亦能作出诊断。

    This paper reports and analyses the immunofluorescence features of 20 children with IgA nephropathy, and correlation between immunofluorescence features, clinical manifest and pathological changes in kidney. According to the children without GBM deposition of IgA or with GBM deposition of IgA, twenty patients were grouped in to two groups, namely group 1 (8 cases) and 2 (12 cases). The patients who had the symptoms of acute glomerulonephritis or persistent hematuria at onset of the disease were in group 2. The...

    This paper reports and analyses the immunofluorescence features of 20 children with IgA nephropathy, and correlation between immunofluorescence features, clinical manifest and pathological changes in kidney. According to the children without GBM deposition of IgA or with GBM deposition of IgA, twenty patients were grouped in to two groups, namely group 1 (8 cases) and 2 (12 cases). The patients who had the symptoms of acute glomerulonephritis or persistent hematuria at onset of the disease were in group 2. The severity of glomerular damages of both groups had significant difference. The children with more severe glomerular damages (≥2) were significantly more in group 2. The children with more severe tubular atrophy and/or interstitial fibrosis (≥++) in group 2 were also more than in group 1. We failed to demostrated correlation between the deposition of IgM in glomeruli and glomerulosclerosis. Although the patients with IgA immunofluorescence ≥ + + were significant more in group 2. but the patients with IgA≥+++ in group 9 were not significant more than in group 1 .

    本文报告和分析了20例小儿IgA肾病的免疫荧光表现及其与临床表现和病理改变之间的关系。8例IgA单独沉积于系膜区(组1);12例同时有毛细血管壁沉积(组2)。临床上以急性肾炎和持续血尿起病者均在组2。两组虽各有1例肾病综合征,但组1病例对皮质激素治疗呈完全效应,组2病例则为部分效应。组2肾小球病变≥2度,肾小管间质病变≥++者显著多于组1(P <0.05)。肾小球有无IgM沉积和小球有无硬化改变无关。虽然IgA荧光强度≥++在组2显著多于组1(P<0.05),但≥+++两组无显著差异。

    Analysis and comparisons were made between the clinical and pathological data obtained from 42 cases of type I and 17 cases of type Ⅲ mesangiocapillary glomerulonephritis (MCGN). It was shown that the type Ⅰ MCGN distincted from type Ⅲ marked by following point; (1)Nephrosis was a salient feature of type Ⅲ while hypertension, impairment of renal function and hematuria were more common in type I. (2) Pathological glomerular sclerosis, tubular interstitial changes and crescents formation occured frequently in...

    Analysis and comparisons were made between the clinical and pathological data obtained from 42 cases of type I and 17 cases of type Ⅲ mesangiocapillary glomerulonephritis (MCGN). It was shown that the type Ⅰ MCGN distincted from type Ⅲ marked by following point; (1)Nephrosis was a salient feature of type Ⅲ while hypertension, impairment of renal function and hematuria were more common in type I. (2) Pathological glomerular sclerosis, tubular interstitial changes and crescents formation occured frequently in type I but not type Ⅲ. (3) The long term prognosis for type Ⅲ was much better than type Ⅰ. In conclusion the type Ⅲ MCGN is a distinct clinical entity which differs from the classic type I MCGN in many aspects.

    对比分析42例Ⅰ型系膜毛细血管性GN(MCGN)和17例Ⅲ型MCGN的临床和病理资料。结果表明:Ⅰ型MCGN与Ⅲ型McGN有以下几点明显不同。1.Ⅲ型MCGN多表现为肾病,而高血压、肾功能损害和血尿则以Ⅰ型MCGN常见。2.在病理方面,肾小球硬化、小管-间质损害和新月体形成,Ⅰ型较Ⅲ型常见。3.Ⅲ型患者的长期预后较Ⅰ型患者好。总之,代们认为Ⅲ型MCGN是独立的疾病,它在许多方面与典型的Ⅰ型MCGN不同。

     
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