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brain tissue injury
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  脑组织损伤
     Conclusion ET-1 played a crucial role in brain tissue injury postresuscitation,one of the possible mechanism was associated with inhibition of Na +-K + ATPase activity. [
     结论 心脏停跳及复苏后脑组织ET - 1含量升高引起脑组织损伤 ,其可能的作用机制之一是抑制了Na+ -K+ ATP酶的活性。
短句来源
     DATA EXTRACTION:A total of 50 papers about the bionomics of AT2 receptors and the renovation of damaged tissue especially on heart,kidney as well as renovation of brain tissue injury were collected.
     资料提炼:共收集到50余篇关于2型受体生物学特性及其与组织损伤修复,特别是与心、肾、脑组织损伤修复相关的文章。
短句来源
     It displayed an obvious brain tissue injury after reperfusion.
     结果表明 :老龄大鼠脑缺血再灌注脑组织损伤明显 ,大黄具有明显保护作用。
短句来源
     raise t PA activity of blood, abate brain tissue injury and improve behavior disorder.
     可升高血浆中t PA的活性、降低PAI的活性 ; 减轻脑组织损伤 ,明显改善行为障碍。
短句来源
     These findings indicated that brain tissue injury persists,and may become even severe in the early stage of reperfusion.
     本文实验结果表明再灌流早期脑组织损伤继续存在,甚至更为严重。
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  脑组织损伤的
     The expression of the advanced glycation end product receptor mRNA in the brain tissue of mice was detected with reverse transcription-polymerase chain reaction (RT-PCR), its level in the diabetic tissue after oxidation stress and its role in mediating brain tissue injury were evaluated.
     应用反转录-聚合酶链反应法测定小鼠大脑组织糖基化终产物受体mRNA的表达,评估其在糖尿病发生氧化应激组织中的水平和其介采脑组织损伤的作用。
短句来源
     Radix et Rhizoma Rhei′s Protective Action on Brain Tissue Injury of Cerebral Ischemia with Reperfusion in Aged Rats
     大黄对老龄大鼠脑缺血再灌注脑组织损伤的保护作用
短句来源
     Objective To explore the molecular mechanism of brain tissue injury induced by endotoxin through observation of LPO content, SOD activity, and ⅠκBα mRNA expression of cortex in endotoxic shock rats.
     目的 :通过观察内毒素休克大鼠脑皮质中 L PO含量、 SOD活力和 κBα m RNA表达及人参二醇组皂苷 (PDS)对其的影响 ,探讨内毒素引起脑组织损伤的分子机制。
短句来源
     AIM: To explore the molecular mechanism of brain tissue injury induced by endotoxin.
     目的:通过观察内毒素休克大鼠脑皮质中NOS活性、NO含量和TLR4 MRNA的表达及人参二醇组皂苷(PDS)对其的影响,探讨内毒素引起脑组织损伤的分子机制。
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  “brain tissue injury”译为未确定词的双语例句
     ② Huoxue huayu tang reduces remarkably the level of complement C3 and C4 in brain homogenate and serum and decreases serum IgG content explaining that the medicine alleviates brain tissue injury by startup complement system. The remarkable decrease of C-reactive protein further suggests the inhibition of such medicine on inflammatory reaction.
     ②给予活血化瘀中药可使大鼠脑匀浆和血清中补体C3和C4水平明显降低,血清IgG 含量降低,说明该药可通过启动补体系统而减轻对脑组织的损伤,C 反应蛋白也明显降低,更进一步提示该药可抑制炎症反应。
短句来源
     Conclusion; The intake of 3H - TdR in brain tissue injury was maximum from the 3rd to 7th day after surgery.
     结论:伤后脑组织摄取~3H-TdR以3天至1周后最多。
短句来源
     Conclusion:Acupuncture of"Shuigou"(DU 26) and "Fengfu"(DU 16) can reduce the release of EAA(Glu,ASP) and balance EAA/IAA, that may relieve ICH induced brain tissue injury.
     结论 :针水组、针风组能抑制不同兴奋性氨基酸(Glu、ASP)的释放 ,纠正兴奋性氨基酸 (EAA) /抑制性氨基酸 (IAA)失衡 ,从而达到减轻脑出血后脑组织损害的作用。
短句来源
     lt has become to be hot spot to study mechanism and protective method of reperfusion injury since 1985.In our study we investigated the influence of ginseng total saponin on brain tissue injury after ischemia-reperfusion by the model of brain ischemia-reperfusion with rat models.
     本课题利用大鼠脑缺血再灌注模型,观察人参总皂甙对缺血再灌注后脑组织损伤的影响,目的是探讨人参总皂甙对大鼠缺血再灌注脑组织的保护作用机制。
短句来源
     C-fos are involved in brain tissue injury following reperfusion of ischemia retina.
     (2)在外侧膝状体中c-fos的变化在中枢调节作用中起着中介作用,c-fos可能参与引起脑组织改变。
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  brain tissue injury
The second opening, recognizable after a delay, is associated with a severe ischemic brain tissue injury.
      
MRI, including fluid-attenuated inversion recovery, gradient echo T2* and diffusion-weighted sequences, is useful in determining the severity of acute brain tissue injury and may help to predict outcome.
      


In this study,the cerebral ischemia model of the animal was established in 22 Wistar rats by temporarily occluding bilateral common carotid arteries with clip.Three minutes after occlusion,the amplitudes of P waves of somatosensory evoked potentials,extracellular calcium ionic activities and tissue oxygen pressures in neocortex decreased by 52.7%(n=7),45.8%(n=10) and 20.5%(n=5) as.compared with the control value of 96.5μV,0.55mmol/L and 1.66±0.45kPa,respectively.The difference in extracellular calcium ionic...

In this study,the cerebral ischemia model of the animal was established in 22 Wistar rats by temporarily occluding bilateral common carotid arteries with clip.Three minutes after occlusion,the amplitudes of P waves of somatosensory evoked potentials,extracellular calcium ionic activities and tissue oxygen pressures in neocortex decreased by 52.7%(n=7),45.8%(n=10) and 20.5%(n=5) as.compared with the control value of 96.5μV,0.55mmol/L and 1.66±0.45kPa,respectively.The difference in extracellular calcium ionic activities between pre-and post-ischemia was statistically significant(P<0.01).During the early phase(1-3 minutes) of reperfusion,the three parameters declined further compared with occlusion phase,then restored gradually to normal level after reperfusion for 10-15 minutes.These findings indicated that brain tissue injury persists,and may become even severe in the early stage of reperfusion.

实验在22只Wistar大鼠上进行,夹闭双侧颈总动脉造成脑缺血,夹闭3min时,皮层体感区诱发电位P波幅值、皮层细胞外钙离子活度及皮层内氧分压分别由正常对照值96.5μV、0.55mmol/L和1.66±0.45kPa降低了52.7%(n=7)、45.8%(n=10)和20.5%(n=5),其中钙离子活度与对照值比较具有非常显著的差异(P<0.01)。放夹后再灌流的早期(1~3min)上述三项指标较夹闭时有更进一步下降,此后逐步恢复,至再灌流10~15min才达到正常水平。本文实验结果表明再灌流早期脑组织损伤继续存在,甚至更为严重。

We reported the ultrastructural pathology and changes of calcium cytochemistary in neurotrauma.Brain tissue injury following CNS primary trauma results in cerebral delayed or secondary damage or slow cell death by endogenous factors.These factors include products of phospholipid hydrolysis such as free fatty acids,free radicals and excitatory amino acids as well as calcium influx. Ultrastructural demonstred cell edema and organals degeneration such as mitochondria swollen,microfilament dissolve.Microcirculate...

We reported the ultrastructural pathology and changes of calcium cytochemistary in neurotrauma.Brain tissue injury following CNS primary trauma results in cerebral delayed or secondary damage or slow cell death by endogenous factors.These factors include products of phospholipid hydrolysis such as free fatty acids,free radicals and excitatory amino acids as well as calcium influx. Ultrastructural demonstred cell edema and organals degeneration such as mitochondria swollen,microfilament dissolve.Microcirculate and brain blood barrier are disordes.We have found calcium deposit in cytoplasm and synaptic vesicles.A hypothesis has been proposed that,in response to primary injury,cells can produce so called “killer proteins”,which cause delayed cell death.Identification of these endogenous factors and their regulation will help to clarify mechanisms of secondary tissue damage and may lead to novel therapies.We suggested that the traumatic injury results in vasospasm and brain ischemic,alterations in mitochondrial function and reduce ATP synthesis,leading to gate openning of calcium channels,Ca  influx accumulate as well as excitatory neurotransmitter release. These biocative metabolites overstimulate the phospholipase A2 and proteinase results in cytoskeleton broken,cytotoxin edema and cell membrane destory.Therefore the traumatic theaputic should be include reduce calcium ion influx and inhibited bioactive metabolites producting decreased glutamate release and block NMDA receptor as well.

对17例人脑挫裂伤的神经组织进行了超微病理及钙细胞化学的形态学观察,研究结果显示星形细胞高度肿胀、细胞器崩解,神经细胞内脂褐素增多,血脑屏障破坏,微循环障碍,兴奋性神经递质明显增多,小胶质细胞增生并迁移至神经元周围;损伤的神经组织内有多量钙离子沉积。文中结合外伤脑组织的超微形态学变化,讨论了脑损伤机制并提出了药物治疗的可能途径。

Six intracranial deep tumors were treated using the SLT contact laser produced by the Medi-Therm company of America.There were 4 basilar meningiomas,1 thalamic tumor and 1 ependymoma of occipital incisura tentorii. Total resections were done in 3 cases of meningiomas,including 1 sphenoid ridge meningioma and 2 olfactory groove meningioma,and subtotal in 2 cases,including 1 ependymoma and 1 recurrent petro-clivus meningioma.The other 1 case had gross partial removal of thalamic tumor.The operative effect was...

Six intracranial deep tumors were treated using the SLT contact laser produced by the Medi-Therm company of America.There were 4 basilar meningiomas,1 thalamic tumor and 1 ependymoma of occipital incisura tentorii. Total resections were done in 3 cases of meningiomas,including 1 sphenoid ridge meningioma and 2 olfactory groove meningioma,and subtotal in 2 cases,including 1 ependymoma and 1 recurrent petro-clivus meningioma.The other 1 case had gross partial removal of thalamic tumor.The operative effect was satisfactory.The SLT contact laser has the following advantages:having the ability to precisely dissect and vaporize the tumor,without peritumoral normal brain tissue injury very mild postoperative brain edema,and excellent hemostatic effect.Sum up,using SLT contact laser in the treatment of deep brain tumors is technically feasible,effective and safe.The manipulative skills of this device also have been discussed.

应用SLT接触式激光显微手术治疗脑深部肿瘤6例,即颅底脑膜瘤4例,丘脑肿瘤1例,枕部小脑幕切迹室管膜瘤1例。其中1例岩—斜坡复发性脑膜瘤次全切除,其余3例脑膜瘤中1例蝶骨嵴内侧脑膜瘤及2例嗅沟脑膜瘤全切。1例丘脑肿瘤大部切除,接触式激光直接接触肿瘤组织,局部温度可达400℃,可迅速汽化、切割肿瘤,避免了激光的散射及侧射,减少对周围组织的损害,术后脑水肿反应轻,止血效果好,又可控制穿透深度,对部位深在,或富于血管的颅底脑膜瘤切除效果满意

 
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