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intraventricular hemorrhage
相关语句
  脑室内出血
    Ultrastructure study of cerebral cells in intraventricular hemorrhage in dog models
    犬脑室内出血动物模型脑细胞超微结构的研究
短句来源
    INTRAVENTRICULAR HEMORRHAGE IN PRETERM NEONATES AND THE RELATIONSHIP WITH EXTRACELLULAR MATRIX BOTH IN CHOROID PLEXUS AND THE WALL OF LATERAL VENTRICLES
    早产儿室管膜下和脉络丛微血管基膜与脑室内出血的相关性研究(英文)
短句来源
    Specimens of total 20 cases which were taken from lateral ventricular wall and choroid plexus in 10 preterm neonates died of intraventricular hemorrhage (IVH) and other 10 preterm neonates died of non intraventricular hemorrhage,were marked with antibodies against type Ⅳ collagen,laminin,and fibronectin.
    10 例死于脑室内出血(Intraventricular hem orrhage,IVH)的早产儿和10 例死于非脑室内出血的早产儿,每例取侧脑室壁和脉络丛两份标本。 用抗Ⅳ型胶原、lam inin 和fibronectin 抗体标记。
短句来源
    Methods:Experimental intraventricular hemorrhage were produced in 18 mongrel dogs by injection blood into the ventricles,and ultrastructural pathological changes were observed by electron microscopy.
    方法:对18 只杂种家犬采用脑室内注血法建立脑室内出血动物模型,电镜观察其病理改变。
短句来源
    Conclusions:Canine models of intraventricular hemorrhage by injecting blood into the ventricles appear to be a simple and reliable method.
    结论:脑室内注血法是建立犬脑室内出血模型的最简单、最可靠的方法。
短句来源
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  intraventricular hemorrhage
Spontaneous primary intraventricular hemorrhage: clinical data, etiology and outcome
      
The clinical features, etiology, and neurological outcome in patients with primary intraventricular hemorrhage (PIVH) have rarely been reported.
      
Severe intraventricular hemorrhage caused by extension from subarachnoid hemorrhage or intracerebral hemorrhage leads to hydrocephalus and often to poor outcome.
      
Patients with primary intraventricular hemorrhage were excluded.
      
Intraventricular hemorrhage (IVH) in adults usually occurs in the setting of aneurysmal subarachnoid hemorrhage or hypertension-related intracerebral hemorrhage.
      
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Abstract Objective To discuss the clinical criteria for diagnosing diffuse axonal injury (DAI). Methods Clinical and computed tomographic features of 117 patients with severe closed head injury were analyzed. The authors preliminarily put forward CT diagnostic criteria of DAI, that is, 1) single or multiple small intraparenchymal hemorrhages in the cerebral hemispheres (<2 cm in diameter); 2) intraventricular hemorrhage; 3) hemorrhage in the corpus callosum; 4) small focal areas of hemorrhage...

Abstract Objective To discuss the clinical criteria for diagnosing diffuse axonal injury (DAI). Methods Clinical and computed tomographic features of 117 patients with severe closed head injury were analyzed. The authors preliminarily put forward CT diagnostic criteria of DAI, that is, 1) single or multiple small intraparenchymal hemorrhages in the cerebral hemispheres (<2 cm in diameter); 2) intraventricular hemorrhage; 3) hemorrhage in the corpus callosum; 4) small focal areas of hemorrhage adjacent to the third ventricle (<2 cm in diameter); and 5) brain stem hemorrhage. All patients were divided into two groups, DAI and non DAI group, according to the criteria. There were 42 patients in the DAI group and 75 patients in the non DAI group. The injury causes, Glasgow coma scale (GCS) scores on admission, coexisting injuries and outcomes were compared between the two groups. The relationship between DAI and the outcomes in the patients with severe head injury was analyzed. Results Traffic accident was the main injury cause in the cases of DAI. GCS scores on admission in patients with DAI were significantly lower than those in patients without DAI. The incidence of diffuse brain swelling (DBS) in the DAI group was significantly higher than that in the non DAI group, whereas the incidences of both skull fracture and epidural hematoma (EDH) in the DAI group were significantly lower than those in non DAI group. There was no significant difference between the incidence of subarachnoid hemorrhage (SAH) and subdural hematoma (SDH) in the two groups. The incidence of poor outcome in the DAI group was significantly higher than that in the non DAI group, although there was no significant difference between the mortalities in the two groups. Conclusions Because DAI is a very important factor worsening the outcome of patients with head injury, it is essential to make a diagnosis as soon as possible. The clinical manifestations of DAI, however, are not specific and DAI does not show directly on CT scans, so it is difficult to make a definite diagnosis. The CT diagnostic criteria of DAI put forward in this article are practicable, though they are by no means perfect.

ClinicalstudiesondifuseaxonalinjuryinpatientswithsevereclosedheadinjuryWangHandong王汉东,DuanGuosheng段国升,ZhangJi张纪andZhouDingbia...

Extracellular matrix,such as type Ⅳ collagen,laminin and fibronectin are the significant compositions of basement membrane of microvessel.Specimens of total 20 cases which were taken from lateral ventricular wall and choroid plexus in 10 preterm neonates died of intraventricular hemorrhage (IVH) and other 10 preterm neonates died of non intraventricular hemorrhage,were marked with antibodies against type Ⅳ collagen,laminin,and fibronectin. Average areas of their positive staining around microvessels...

Extracellular matrix,such as type Ⅳ collagen,laminin and fibronectin are the significant compositions of basement membrane of microvessel.Specimens of total 20 cases which were taken from lateral ventricular wall and choroid plexus in 10 preterm neonates died of intraventricular hemorrhage (IVH) and other 10 preterm neonates died of non intraventricular hemorrhage,were marked with antibodies against type Ⅳ collagen,laminin,and fibronectin. Average areas of their positive staining around microvessels in IVH group showed less than that in non IVH group ( P <0 05),and microvessel count of discontinuous positive staining in IVH group was more than that in non IVH group( P <0 05) It was concluded that choroid plexus and subependymal microvessels of preterm neonates were short of type Ⅳ collagen,laminin,and fibronectin and this may be accounted for premature basement membrane of microvessels which is responsible for the occurrence of IVH.

细胞外基质成分Ⅳ型胶原、lam inin 和fibronectin 是微血管基膜的主要成分。10 例死于脑室内出血(Intraventricular hem orrhage,IVH)的早产儿和10 例死于非脑室内出血的早产儿,每例取侧脑室壁和脉络丛两份标本。用抗Ⅳ型胶原、lam inin 和fibronectin 抗体标记。侧脑室周围抗体的阳性产物多呈不连续线状。在IVH组中三种细胞外基质成分阳性产物的平均单位面积比非IVH组小(P< 001);IVH组的三种细胞外基质成分阳性产物呈不连续线状的微血管总数比非IVH 组多(P< 005),而阳性产物呈连续线状的微血管总数比非IVH组少(P< 005)。这表明IVH 组中三种细胞外基质成分比IVH 组少,这也是早产儿室管膜下和脉络丛微血管未成熟的特征,是易患IVH 早产儿的形态学基础之一

Objective:To study the ultrastructural pathological changes and their clinical significance.Methods:Experimental intraventricular hemorrhage were produced in 18 mongrel dogs by injection blood into the ventricles,and ultrastructural pathological changes were observed by electron microscopy.Results:Mitochondria swelling,endoplasmic reticulum swelling and chromatin margination were the main pathological manifestations in neurons,neurogliocytes and nerve fiber cells in the cerebrum,cerebellum and brain stem.Conclusions:Canine...

Objective:To study the ultrastructural pathological changes and their clinical significance.Methods:Experimental intraventricular hemorrhage were produced in 18 mongrel dogs by injection blood into the ventricles,and ultrastructural pathological changes were observed by electron microscopy.Results:Mitochondria swelling,endoplasmic reticulum swelling and chromatin margination were the main pathological manifestations in neurons,neurogliocytes and nerve fiber cells in the cerebrum,cerebellum and brain stem.Conclusions:Canine models of intraventricular hemorrhage by injecting blood into the ventricles appear to be a simple and reliable method.Mitochondria swelling and chromatin margination were main ultrastructural changes of neurocyte damage.Mitochondria swelling would inhibit ATP production,and as a result,the cerebral cells were short of energy,which manifested clinically as neurofunction disturbance.Death of cerebral cells caused by chromatin margination might lead to irreversible nerve cells damage.

目的:研究脑室内出血后脑细胞超微结构的病理变化及其临床意义。方法:对18 只杂种家犬采用脑室内注血法建立脑室内出血动物模型,电镜观察其病理改变。结果:大脑、小脑及脑干神经细胞、胶质细胞及神经纤维细胞内线粒体肿胀及染色质边集、内质网肿胀是其主要病理变化。结论:脑室内注血法是建立犬脑室内出血模型的最简单、最可靠的方法。脑室内出血后脑细胞受损的超微病理改变主要是线粒体肿胀及染色质边集;线粒体肿胀导致ATP产生受抑,脑细胞能量不足,从而表现为临床神经功能障碍;染色质边集导致脑细胞死亡,引起不可逆的神经功能损害

 
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