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hypertonic glucose
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  高渗葡萄糖
     The Effect of Hypertonic Glucose on Erythrocyte Membrane Na~+-K~+-ATPbase
     高渗葡萄糖处理后脱水红细胞膜Na~+-K~+-ATP酶的变化
短句来源
     Treatment of 232 cases with epidemic hemorrhagic fever by hypertonic glucose solution
     高渗葡萄糖溶液佐治流行性出血热232例疗效探讨
短句来源
     Purpose To evaluate the effectiveness of injection of hypertonic glucose for the sclerosis therapy in aged huge hepatic cysts.
     目的评价高渗葡萄糖对老年人巨大肝囊肿囊腔内硬化治疗的效果。
短句来源
     Objective To investigate the clinical value of the 50% hypertonic glucose injection in the treatment of recurrent pneumothorax.
     目的 :探讨 5 0 %的高渗葡萄糖在治疗复发性气胸中的临床价值。
短句来源
     Methods Forty five patients with recurrent pneumothorax were injected with 60~80 ml of 50% hypertonic glucose into the pleural cavity after air discharge and then were instructed to turn their body to the right and the left quickly.
     方法 :对 4 5例复发性气胸患者在胸腔内气体基本排除的情况下注入 5 0 %的高渗葡萄糖 6 0~ 80ml,嘱患者迅速左右辗转身体。
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  高张葡萄糖
     Reasonable Use of Hypertonic Glucose
     合理使用高张葡萄糖
短句来源
     In clinical practice the authors found that the use of hypertonic glucose produced a certain hazard in some circumstance or made the condition be worse,even endangered the patient's life.
     临床护理工作中发现,高张葡萄糖的使用,在某些情况下存在一定的危险性,使用不当则加重病情的发展,甚至危及病人的生命。
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  “hypertonic glucose”译为未确定词的双语例句
     Intravenous infusion of hypertonic glucose solution(30—40%, 2—3 ml per minute for 40—45 minutes) caused great augmentation of the acid-induced parotid secretion, but infusion of 7% NaCl(iso-osmotic to 40% glucose solution)and of 5% glucose-saline was without effect on the volume of secretion.
     当由静脉内灌注与40%糖溶液渗透压相当的7%NaCl 溶液或同样量的5%葡萄糖生理盐水时,则酸反射量无大改变。
短句来源
     Objective To study the effect of hypertonic glucose on erythrocyte membrane Na+_K+_ATP base in vivo.
     方法对全血与50 %葡萄糖 (50 %GS)溶液混合前后红细胞膜Na +_K +_ATP酶的变化 ,以及脱水红细胞于等渗条件下其Na+ _K+ _ATP酶的恢复情况进行了测定。
短句来源
     Clinical Observation of Hypertonic Glucose in IVP
     高渗糖在静脉肾盂造影中的临床观察
短句来源
     (2)In acute experiments infusion of hypertonic glucose solution and of normal saline neither induced spontaneous secretion nor affected the volume of the nerve- stimulated secretion of the parotid or submaxillary glands.
     在急性实验,高血糖不能诱发唾液腺的分泌,亦不能使刺激鼓索神经、耳颞神经引起的唾液分泌量增加。
短句来源
     From January 1985 to September 1992, nutritional support in 57 patients with acute necrotizing pancreatitis was divided into three stages: total parenteral nutritional (TPN) was started in the early stages of postoperation, and their nutritional solution which was composed of hypertonic glucose, intralipid and amino acids was given by peripheral intravenous infusion;
     自1985年1月~1992年9月,我们对57例急性坏死性胰腺炎病人的营养支持治疗分三期进行,即术后早期经周围静脉输注高渗糖、脂肪乳剂和氨基酸等营养液行全胃肠外营养(TPN);
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  hypertonic glucose
Thermal enhancement of ACNU and potentiation of thermochemotherapy with ACNU by hypertonic glucose in the BT4An rat glioma
      
Isotonic and hypertonic glucose, beer, and fermented glucose significantly increased plasma levels of cholecystokinin (CCK), but the effect was significantly higher after hypertonic glucose than after isotonic glucose, beer, or fermented glucose.
      
Hypertonic glucose evoked a significantly smaller level of motor activity compared with both saline (at given osmolarities) and an elemental diet (at given energy loads).
      
Hypertonic glucose, mannitol, and saline infusions significantly increased plasma concentrations of enteroglucagon, whereas other solutions had no effect.
      
A similar degree of inhibition was obtained for pepsin secretion with hypertonic glucose and mannitol.
      
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(1)The cardiac disturbances of hypothermic dog under ether anaesthesia showed the following types:ventricular fibrillation,asystole or asystolic fibrillation.The mean terminal temperature of animals was 16.9±1.14℃.There was no correlation between the type of cardiac disturbance and the value of the terminal temperature. (2)The hypothermic ventricular fibrillation of dog could be completely prevented by systemic administration of hypertonic glucose solution,whereas hypertonic sucrose solution was...

(1)The cardiac disturbances of hypothermic dog under ether anaesthesia showed the following types:ventricular fibrillation,asystole or asystolic fibrillation.The mean terminal temperature of animals was 16.9±1.14℃.There was no correlation between the type of cardiac disturbance and the value of the terminal temperature. (2)The hypothermic ventricular fibrillation of dog could be completely prevented by systemic administration of hypertonic glucose solution,whereas hypertonic sucrose solution was ineffective in this regard.The mean terminal temperature of the experimental animal was not changed by the administration of either of these solutions.It was therefore suggested that glucose exerted a specific action on the hypothermic cardiac metabolism. (3)Yohimbine,quinidine and molar sodium lactate failed to prevent ventricular fibrillation under our experimental conditions. (4)Administration of procaine markedly reduced the incidence of hypothermic ventricular fibrillation,with a significant lowering of the mean terminal temperature to 12.3±1.13℃,per- haps owing to its trophic action on the cardiac muscle. (5)On the basis of afore-mentioned data,the concept that metabolic disturbances in cardiac muscle induced by hypothermia per se was the causative factor in the occurrence of ventricular fibril- lation might be advanced.

(一)在乙醚麻醉下,低温所致狗心脏活动障碍有三种型式,即心室颤动、心跳停止和心跳停止式心室颤动。动物的最终死亡温度为16.9±1.14℃。在最终温度与心脏活动障碍的型式之间,并无任何依从关系。(二)高渗葡萄糖溶液能防止低温下心脏发生颤动,但不能使动物死亡的最终温度降低。高渗蔗糖溶液并无此类作用,这说明葡萄糖对低温心肌代谢有特异作用。(三)育亨宾、奎尼丁和乳酸钠并不能防止低温性心室颤动的发生,由此表明交感-肾上腺系统的机能亢进、酸中毒等并非低温性心室颤动的重要诱因。(四)应用普鲁卡因溶液能显著减少低温性心室颤动的发生,并使动物的最终温度明显降至12.3±1.13℃(P<0.01)。普鲁卡因的这种效应似应归之于它对心肌组织的良性营养作用。(五)基于葡萄糖和普鲁卡因对低温所致心室颤动有良好的防止效果,因而有理由认为低温性心室颤动是低温直接引起心肌内代谢失调的结果。(六)本工作的结果可能为临床上防止低温性心室颤动提供有效措施。

The effects of hyperglycemia upon dog's salivary secretion were observed in chronic and acute experiments The main results were as follows: (1)Chronic experiments were conducted on 6 dogs. Intravenous infusion of hypertonic glucose solution(30—40%, 2—3 ml per minute for 40—45 minutes) caused great augmentation of the acid-induced parotid secretion, but infusion of 7% NaCl(iso-osmotic to 40% glucose solution)and of 5% glucose-saline was without effect on the volume of secretion. (2)In acute...

The effects of hyperglycemia upon dog's salivary secretion were observed in chronic and acute experiments The main results were as follows: (1)Chronic experiments were conducted on 6 dogs. Intravenous infusion of hypertonic glucose solution(30—40%, 2—3 ml per minute for 40—45 minutes) caused great augmentation of the acid-induced parotid secretion, but infusion of 7% NaCl(iso-osmotic to 40% glucose solution)and of 5% glucose-saline was without effect on the volume of secretion. (2)In acute experiments infusion of hypertonic glucose solution and of normal saline neither induced spontaneous secretion nor affected the volume of the nerve- stimulated secretion of the parotid or submaxillary glands. (3)It is suggested that in the chronic experiments the effect of hyperglycemia was most probably produced via the parasympathetic center in the central nervous system.

本工作系用具有一侧腮腺瘘的清醒狗及麻醉狗,观察了高血糖对唾液分泌的潜伏期、分泌量等的影响,发现在慢性实验中,人工地提高血糖浓度(静脉灌注30—40%糖溶液),可使唾液分泌酸反射量有显著的增加。当由静脉内灌注与40%糖溶液渗透压相当的7%NaCl 溶液或同样量的5%葡萄糖生理盐水时,则酸反射量无大改变。在急性实验,高血糖不能诱发唾液腺的分泌,亦不能使刺激鼓索神经、耳颞神经引起的唾液分泌量增加。推测高血糖对清醒动物的上述作用可能是兴奋副交感神经中枢的结果。

Total parenteral nutrition(TPN)was used in 14 cases of digestive tract fistula,severeinfection or persistent postoperative gastrointestinal stasis for an average of 22.9 days.The superior vena cava was catheterized for infusion of hypertonic glucose and crystal-line amino acids solution. The infusion fluids contained 0.2-0.3 gm nitrogen/kg/dayand 35-50 cal/kg/day.The patients without severe infection could be maintained inpositive nitrogen balance.Two patients were complicated with catheter sepsis.Glucoseintolerance...

Total parenteral nutrition(TPN)was used in 14 cases of digestive tract fistula,severeinfection or persistent postoperative gastrointestinal stasis for an average of 22.9 days.The superior vena cava was catheterized for infusion of hypertonic glucose and crystal-line amino acids solution. The infusion fluids contained 0.2-0.3 gm nitrogen/kg/dayand 35-50 cal/kg/day.The patients without severe infection could be maintained inpositive nitrogen balance.Two patients were complicated with catheter sepsis.Glucoseintolerance was the chief hindrance to TPN and led to hyperosmolar hyperglycemic non-ketotic coma and fatty liver in 2 cases.There were 7 deaths and one of which died ofTPN complications.We stress that it is essential to adhere strictly to the indications ofTPN and aseptic technic,and avoid dextrose overload.Resumption of oral feeding shouldbe instituted as soon as digestive functions recovered.

本文报道用全静脉营养治疗消化道瘘、术后持续胃肠道潴留和严重感染14例,共320天。所用营养液为结晶氨基酸及高渗葡萄糖。每日每公斤体重给予热量35~50卡,氮0.2~0.3克。除严重感染者外,病人可获正氮平衡。发生导管滑脱10次、堵塞5次;导管静脉端培养25次,有菌生长15次(60%)。发生导管性败血症2例,高血糖8例,低血糖2例,高渗综合征2例,脂肪肝1例。作者认为应严格掌握全静脉营养的适应证。

 
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