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liver injury     
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  肝损伤
     EXPERIMENTAL STUDY OF CHAIZHI MISTURA IN TREATMENT OF CHRONIC ALCOHOLIC LIVER INJURY
     柴枳合剂治疗慢性酒精性肝损伤大鼠的实验研究
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     The Role of CXCL16 in Murine Immunological Liver Injury
     CXCL16在免疫性肝损伤中的作用
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     Study on the Mechanism of the Action of Galectin-3 in Liver Injury Caused by CCl_4
     galectin-3在CCl_4肝损伤中作用机制的研究
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     Pharmacological Study on the Protective Action of Bicyclol Against Experimental Liver Injury and Fibrosis in Animals
     双环醇对实验性肝损伤、肝纤维化的保护作用和作用机制的研究
短句来源
     STUDIES ON THE EFFECT OF OLEANOLIC ACID ON EXPERIMENTAL LIVER INJURY
     齐墩果酸防治实验性肝损伤作用的研究
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  肝损害
     EXPERIMENTAL STUDY OF CYTOPROTECTIVE EFFECT OF PROSTAGLANDIN E_2 (PGE_2) ON CARBON TETRACHLORIDE (CCI_2)-INDUCED LIVER INJURY IN RAT
     前列腺素E_2(PGE_2)对CCI_4诱导急性肝损害细胞保护作用的实验研究
短句来源
     Significance and changes of cytokines TNF-α,IL-6 in alcoholic liver injury of rats
     大鼠酒精性肝损害时血浆细胞因子TNF-α、IL-6水平的变化及其意义
短句来源
     Experimental study on the early liver injury and expression of TNF-α mRNA after burns complicated by endotoxemia
     烧伤并发内毒素血症早期肝损害与TNF-α mRNA表达的实验研究
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     Genetic polymorphism of cytochrome CYPⅡE1 related to the development of drug-induced liver injury
     CYPⅡE1基因多态性与药物性肝损害
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     The preliminary study of autoimmunity mechanism of TNT or CCl_4 induced toxic liver injury.
     TNT及CCl_4中毒性肝损害的自身免疫机制初步探讨
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  肝损伤的
     To observe the protective effect of selenium enriched yeast(SEY)with different ig dose(30,60,90 mg/kg)for 7d on chemical liver injury produced by CCl 4 and D-Gal-N.
     观察不同剂量(30,60,90mg/kg)载硒酵母(selminm-enrichedyeast,SEY)ig7天对CCl4和D-Gal-N引起的化学性肝损伤的保护作用。
短句来源
     Experimental Study on Liver Injury Induced by HBsAg specific CTL in HBV Transgenic Mice
     HBsAg特异性CTL诱发HBV转基因小鼠肝损伤的实验研究
短句来源
     Effects of 2-ethyl-3-hydroxy-6-phenylthio-4-(1H)-pyridinone on experimental liver injury in mice and rats
     2-乙基-3-羟基-6-苯硫基-4(1H)-吡啶酮对动物实验性急性肝损伤的保护作用
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     The projective effects of J - 911 on the acute liver injury in mice and the chronic liver in jury in rats were observed and studied by establishing animal models.
     通过建立小鼠急性肝损伤及大鼠慢性肝损伤模型,观察并研究海洋药物J- 911对小鼠急性肝损伤及大鼠慢性肝损伤的保护作用。
短句来源
     Aim:To study the protective effect of 2-ethyl-3-hydroxy-6-phenylthio-4-(1H)-pyridinone (HPP) on acute liver injury induced by CCl 4、TAA and D-GlaN.
     目的 :研究 2 -乙基 - 3-羟基 - 6-苯硫基 - 4 ( 1H) -吡啶酮 (HPP)对四氯化碳 (CCl4 )、硫代乙酰胺 (TAA)及D -氨基半乳糖 (D -GalN)引发的小鼠急性肝损伤的影响 .
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  肝脏损伤
     Hepatic SOD and GSH decreased, MDA and TG increased as liver injury and fibrosis developed.
     随着肝脏损伤与肝纤维化的发展,肝脏SOD与GSH水平下降,MDA与TG水平上升。
     The expression of CD19+CD25+ cells in peripheral blood in patients would reflect degree of liver injury and status of humoral immunity, that may be helpful to instruct clinical therapy and judge the prognosis.
     检测肝硬化患者外周血CD19+CD25+细胞表达水平能反映患者的肝脏损伤程度和体液免疫状况,有助于指导临床治疗和预后判断。
短句来源
     The Role of Serum NO and sIL-2R in Pathogenesis of Liver Injury
     血清NO和sIL-2R在肝脏损伤发病机制中作用的探讨
短句来源
     Conclusions Apoptosis,abnormal expression of TGFβ 1 and elevation of lipid peroxidation involved liver injury mechanisms induced by ADR. [
     结论 阿霉素诱导肝细胞凋亡可能是其肝脏损伤机制之一,并且细胞凋亡与转化生长因子 β1的高表达和血清中脂质过氧化水平升高有密切关系。
短句来源
     Conclusions Ischemia preconditioning relieves the liver injury after reduced-size liver transplantation during early period,and facilitates the expression of Ref-1 protein in hepatic tissue after transplantation, implying the protection mechanism is at least partially relevant to the promotion of Ref-1 protein expression.
     结论缺血预处理可以减轻减体积肝移植后早期肝脏损伤,促进肝组织Ref-1蛋白表达,提示缺血预处理保护减体积肝移植物早期损伤的机制可能与促进Ref-1蛋白的表达有关。
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  liver injury
Changes in sphingomyelinase activity, tumor necrosis factor α expression, and level of lipid peroxidation products in the course of development of cholestatic liver injury have been studied.
      
In conclusion, induction of HO-1 provides protection against liver injury during cold ischemia preservation and improves the preservation of liver graft.
      
Effectiveness of pectin extracted from the eelgrass Zostera marina for alleviating lead-induced liver injury
      
The effects of pectin from the eelgrass Zostera marina on toxic liver injury induced by enteral administration of lead acetate are examined in experiments on rats.
      
The hepatoprotective potential of earthworm extract (EE) (Lampito mauritii, Kinberg) was evaluated against paracetamol-induced liver injury in Wistar albino rat, in comparison with silymarin, the standard hepatoprotective drug.
      
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Toxic hepatitis was induced with carbon tetrachloride in mice. L-4-oxalysine (I-677) l00mg/kg injected daily has been proved to be effective in lowering the serum glutamic-pyruvate transaminase (SGPT) level in both aoute and subacute liver injuries. Pathological examinations also showed that the degeneration and necrosis of liver cells were protected by the administration of I-677. In the course of inducing experimental liver oirrhosis with CCl_4, I-677 administered for 8 weeks affected neither...

Toxic hepatitis was induced with carbon tetrachloride in mice. L-4-oxalysine (I-677) l00mg/kg injected daily has been proved to be effective in lowering the serum glutamic-pyruvate transaminase (SGPT) level in both aoute and subacute liver injuries. Pathological examinations also showed that the degeneration and necrosis of liver cells were protected by the administration of I-677. In the course of inducing experimental liver oirrhosis with CCl_4, I-677 administered for 8 weeks affected neither liver function besides a lowering of serum bilirubin, nor liver collagen content and histological appearance. The mechanism of serum GPT-lowering was found to be a lowering of hepatic enzyme activity. No direct inhibition on the activity of both liver and serum GPT was observed in vitro. The transaminase-lowering effect of I-677 could not be antagonized by simultaneous administrations of L-lysine. Amino-oxyacetic acid, a GPT inhibitor, showed potent inhibition on the GPT activity in vitro, but not in vivo. Large doses of I-677 (8-32 times the clinical dose) for 11 weeks in rats induced a lowering of serum albumin, a decrease of incorporation of ~3H-lysine into serum protein, and an increase of the lipid of liver. This revealed that albumin synthesis and lipid transport function of liver were inhibited by I-677, but this effect could be antagonized by L-lysine.

用四氯化碳引起小白鼠中毒性肝炎,同时每天注射I-677 100毫克/公斤,对一次中毒的急性肝损伤或反复中毒5次疗程15天的亚急性肝损伤都有明显的降低血清谷丙转氨酶(GPT)作用,病理切片证明肝细胞变性和坏死程度都较对照组为轻。在四氯化碳中毒连续8周引起肝硬化的过程中,每日给予I-677,除血清胆红素降低外,对其他肝功能、肝胶元含量和肝组织切片检查,均无明显影响。I-677作用机理的研究发见,血清GPT的降低与肝组织GPT的降低有关,同时给予L-赖氨酸,不能对抗其降酶作用。I-677与肝匀浆或高GPT血清在体外培养,未观察到对GPT的直接抑制作用。与GPT抑制剂氨氧醋酸比较,后者在体外有明显抑制GPT作用而体内则无降酶效果。大白鼠每日灌胃I-677 80~320毫克/公斤(临床剂量的8~32倍),连续11周,可引起血清白蛋白的降低和丙球蛋白的升高。用氚标记赖氨酸对蛋白的掺入试验也证明,大剂量I-677对肝脏的蛋白质合成和脂肪转运有一定影响,但这种作用可被赖氨酸所对抗。

Schizandrin B, one of the components isolated from Fructus Schizandrae, was found to have protective action on liver injury and to increase the weight of liver in mice. In order to elucidate the cause and implications of the liver enlargement by Schizandrirn B, some studies were carried out in mice. The results indicate that the content of water, protein, RNA, glycogen and total lipid per gram liver of the group given Schizandrin B was almost the same as those of the control group, but...

Schizandrin B, one of the components isolated from Fructus Schizandrae, was found to have protective action on liver injury and to increase the weight of liver in mice. In order to elucidate the cause and implications of the liver enlargement by Schizandrirn B, some studies were carried out in mice. The results indicate that the content of water, protein, RNA, glycogen and total lipid per gram liver of the group given Schizandrin B was almost the same as those of the control group, but the amount of all these biochemical constituents markedly increased. The DNA content per gram liver decreased, although the total amount of DNA in whole liver was the same as that of the control.In partially hepatectomized mice, Schizandrin B was also shown to increase the protein, RNA and DNA contents as well as, mitosis of liver cells. In addition, Schizandrin B Was found to enhance-the incorporation of ~(14)G-phenylalanine into liver protein, and to increase hepatic microsomal cytochrome P-450 and protein contents significantly. From these results, it may be concluded that Schizandrin B is'an inducing agent of drug metabolizing enzyme, and the enlargement of the liver caused by Schizandrin B is mainly, due to hypertrophy and to a lesser degree, hyperplasia. However, Schizandrin B is different from phenobarbital in the respectof inducing drug metabolizing enzyme. The difference is that phenobarbital also induced an increase of liver cell microsomal RNA and phospholipid contents. Besides, phenobarbital wasshown to be able to shorten the survival time of CC14-intoxicated mice, while Schizandrin B did not.

五味子乙素系从中药五味子分离出的成分之一,有抗肝损伤和解毒作用,同时亦引起小鼠肝脏重量增大。经进一步研究,发现五味子乙素对正常小鼠肝脏每克组织中水分、蛋白质、RNA、糖原及总脂含量均无明显影响,但在整个肝脏中上述成分含量均显著增加。每克肝组织中DNA含量稍降低,整个肝脏中DNA含量则无明显变化。对部分切除肝脏的小鼠再生肝,五味子乙素能明显引起整个肝脏中蛋白质,PLNA和DNA含量以及细胞核分裂数增加。此外,五味子乙素能显著促进~(14)C-苯丙氨酸掺入肝脏蛋白质,并使肝细胞微粒体细胞色素P-450及蛋白质含量显著增加。以上结果表明,五味子乙素对药酶有诱导作用,其引起的肝脏增大主要系肝实质细胞体积增大,同时伴有肝细胞增生。五味子乙素引起的肝脏增大似非病理性损害的表现,而是由于诱导药酶同时出现肝细胞肥大的表现。

The isolation, purification and identification of liver-specific membrane lipo-protein (LSP) is briefly presented. Human fetal liver was homogenized and submitted to 105,000 g for 1 hour. The supernatent was detected with presence of LSP. It was then fractioned by chromatography through Sephadex G-100 and Sephadex G-200. LSP revealed in the first peak.Nine rabbits of both sexes, weighing about 3 kg, were immunized with LSP to prepare antiserum. 80 sera of various liver disease patients were detected. Evidence...

The isolation, purification and identification of liver-specific membrane lipo-protein (LSP) is briefly presented. Human fetal liver was homogenized and submitted to 105,000 g for 1 hour. The supernatent was detected with presence of LSP. It was then fractioned by chromatography through Sephadex G-100 and Sephadex G-200. LSP revealed in the first peak.Nine rabbits of both sexes, weighing about 3 kg, were immunized with LSP to prepare antiserum. 80 sera of various liver disease patients were detected. Evidence of an immune response to the LSP was present in 18 of 23 cases (78.2%) of chronic active hepatitis. This observation suggests that the LSP may play an important role in the pathogenesis of liver injury.

本文报道了肝特异性膜脂蛋白(LSP)的分离提纯和鉴定。将人胎肝制成匀浆液,经105,000g/1小时离心后,上清液中存有肝特异性抗原。上液在Sephadex G-100及Sephadex G200凝胶层析后,LSP存在于第一峰中。用LSP免疫9只家兔(体重约3公斤),制备抗血清,应用抗-LSP检测了80例不同类型肝病患者的血清中LSP滴度,显示在18/23(78.2%)的慢性活动性肝炎患者中存在对LSP免疫反应,这一观察提示了在肝细胞损害的发病机理中,LSP起着重要的作用,

 
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