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hepatocyte injury
相关语句
  肝细胞损伤
     (2) EGF of different doses (50, 100, 500, and 1000ng/ml) added to the medium prior to acetaminophen could reduce hepatocyte injury in a dose-dependent manner.
     (2)提前1h加入不同剂量(50,100,500,1000ng/ml)的EGF可减轻醋氨酚引起的肝细胞损伤
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     (4) hepatocyte injury was ameliorated.
     (4)肝细胞损伤减轻。
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     The hepatocyte injury and detection of serum cytokines after hepatopathy rats attacked by Vibrio vulnificus
     肝病大鼠创伤弧菌攻击后肝细胞损伤和血清细胞因子检测
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     Conclusion: The fragments of C 3 and C 5 in ZAHS can stimulate KC to secrete NO, by which they can mediate hepatocyte injury, which maybe the potential mechanism of liver injury mediated by complements in some pathological condition.
     结论 :ZAHS中的C3、C5片断可激发KC生成NO ,介导肝细胞损伤 ,这可能是某些病理情况下补体介导肝损伤的机制之一
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     Cadmium (Cd)-induced cytotoxicity and intracellular Ca2+ alteration ([Ca2+]i) in hepatocytes and intervention with selenium (Se) were studied to discuss mechanism of Cd-induced hepatocyte injury and protective effect by Se.
     本文通过研究镉诱发鼠肝细胞毒性和胞内游离Ca2+变化及硒的干预效应,探讨镉致肝细胞损伤机制及硒的保护作用。
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  肝细胞损害
     Conclusions: TBA is a sensitive test to reflect hepatocyte injury in patients with liver cirrhosis.
     结论:TBA是反映肝硬化患者肝细胞损害的敏感指标之一。
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     Relationship between sinusoidal endothelial cell apoptosis and hepatocyte injury after their transplantation into rats
     肝移植大鼠肝窦内皮细胞细胞凋亡与移植肝肝细胞损害的关系
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     Effect of bcl-2 gene in hepatocyte injury caused by obstructive jaundice in rats
     bcl-2基因在阻塞性黄疸大鼠肝细胞损害中的作用
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     Background: Bile acid is synthesized and excreted in liver, therefore it can reflect the hepatocyte injury.
     背景:胆汁酸在肝内合成和分泌,因此可以作为反映肝细胞损害的指标之一。
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     There had significant difference between the contents of HBV-DNA copies and the severe degree of hepatocyte injury(P<0.05); The contents of HBV-DNA copies had no correlation with the severe degree of hepatocyte injury(P>0.05).
     两组肝细胞损害指标ALT、AST、TBA比较,差异有显著性(P<0.05),HBV-DNA含量与肝细胞损害各指标均无相关性。
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  肝细胞损伤的
     Experimental Study of Hepatocyte Injury after Cardiopulmonary Resuscitation in Rats
     心肺复苏后大鼠肝细胞损伤的实验研究
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     hepatocyte injury.
     以CCl4 体外损伤的原代培养大鼠肝细胞为实验模型 ,通过测定培养液中ALT、AST、MDA水平和MTT法检测细胞增殖活性 ,观察EMD对急性肝细胞损伤的影响。
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     AIM To investigate the effects of polydatin on mouse hepatocyte injury induced by hydrogen peroxide(H 2O 2).
     目的 观察中药虎杖的活性成分白藜芦醇苷对过氧化氢 (H2 O2 )所致肝细胞损伤的影响。
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     Expression of HBV Markers of Chronic Hepatitis B and Its Relation with Hepatocyte Injury
     慢性乙型肝炎HBV标志的表达与肝细胞损伤的关系
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     Aim: To evaluate the effect of rhubarb on hepatocyte injury after intestinal ischemia reperfusion in rats.
     目的:评价大黄对大鼠肠缺血所致肝细胞损伤的防治作用。
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  “hepatocyte injury”译为未确定词的双语例句
     Study one The establishment of the hepatocyte injury model made on BRL-3 A rat hepatocyte induced by CCl4Methods: rat hepatocytes are divided into control group, 5mmol/L CCl4 group, 10mmol/L CCl4 group, 15mmol/L CCl4 group, 20mmol/L CCl4 group, the culture medium without serum is added into the control group cells, the CCl4 with corresponding concentration is added into cells.
     方法:将大鼠肝细胞分为正常组、5mmol/L CCl_4组、10mmol/L CCl_4组、15mmol/LCCl_4组、20mmol/L CCl_4组,加入相应浓度的CCl_4。 再分别培养3h、6h、12h、24h、48h后,吸出上清液,测ALT。
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     Methods:40 male rats were divided into 4 groups, including group A、B、C and D. Acute hepatocyte injury was induced in all male rats by CCL4 administration about 4 weeks.
     方法:40只Wistar雄性大鼠随机分为A、B、C和D共4组,均采用CCL4诱导肝细胞急性损伤4周。
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     H2O2 positive precipitation on outer plasma membrane of hepatocytes was obviously increased at 3 h,and disappeared at 24 h but hepatocyte injury was very severe at this time,at 48 h hepatocyte recovered through regeneration.
     肝细胞膜上H2O2的阳性沉积物3小时最严重,24小时已消失,但此时细胞损伤最严重,48小时肝细胞再生、修复。
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     Effects of XCHT and bupleurum root-scutellaria root on hepatocyte injury model induced by CCl_4 in vitro
     小柴胡汤和柴胡—黄芩含药血清对CCl_4损伤肝细胞的影响
短句来源
     Conclusion: The detection of serum glutamate dehydrogenase is of significance in early clinical diagnosis and prediction of hepatic disease, especially in hepatocyte injury and necrotic hepatopathy.
     结论 :检测血清中谷氨酸脱氢酶的活性对肝病早期诊断、掌握病情、判断预后有着独特的临床意义 ,是诊断肝细胞病变 ,特别是坏死性肝病的重要指征。
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  hepatocyte injury
It was shown that PGI2 could circumvent the augmented catabolic rate of 4,5-phosphatidyl inositol-diphosphate (PIP2) in CCl4 induced hepatocyte injury.
      
Thus, our study demonstrates that the serum IL-6 level is a possible marker for identifying the clinical status in acute hepatitis and that this cytokine may have some roles in hepatocyte injury.
      
Furthermore, we found that a significant correlation between serum IL-6 level and prothrombin time existed in these patients and that the elevated serum IL-6 returned to a normal range after recovery from their hepatocyte injury.
      
To study the mechanisms of hepatocyte injury, we examined serum interleukin-6 (IL-6) level in acute hepatitis patients.
      
Oxidative stress may initiate significant hepatocyte injury in subjects with fatty liver.
      
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To understand the relationship between the expression of HBV-markers andinjury of hepatocytes,we have examined HBsAg,HBcAg in liver tissue with PAPmethod and HBV-markers in serum with ELISA method of 70 patients with CH-B.The results showed that the expression patterns of HBsAg could be divided into 4types,those of HBcAg into 3 types.The positive percentages of HBsAg and HBcAgin CPH were higher than in CAH under which the expression patterns were notconsidered.When considering the expression patterns,positive...

To understand the relationship between the expression of HBV-markers andinjury of hepatocytes,we have examined HBsAg,HBcAg in liver tissue with PAPmethod and HBV-markers in serum with ELISA method of 70 patients with CH-B.The results showed that the expression patterns of HBsAg could be divided into 4types,those of HBcAg into 3 types.The positive percentages of HBsAg and HBcAgin CPH were higher than in CAH under which the expression patterns were notconsidered.When considering the expression patterns,positive percentages of mem-brane type of HBsAg and cytoplasmic membrane type of HBcAg in CAH were higherthan in CPH.It was suggested that the expressions of HBV-Ag of these types wereconsistent with the degree of hepatocytic injury.The combination positive percen-tages of HBsAg+HBeAg+Anti-HBc,and HBsAg+Anti-HBc in serum in CAH,which indicated HBV replication,were higher than in CPH.The positive percen-tage of HBeAg increased along with the increase of SGPT.It revealed that HBV-markers in serum which indicated HBV replication could also reflect the conditionof hepatocyte injury.Target antigen and relation between the expression of HBV-markers in liver tissue and in serum were discussed.

为了探讨HBV标志的表达与肝细胞损伤的关系,本文应用 PAP 法和 ELISA 法检测了70例 CH-B 患者肝组织 HBsAg、HBcAg 和五项血清 HBV 标志。结果发现 HBsAg 在肝内有4种表达型式,HBcAg 有3种表达型式。HBsAg 和 HBcAg 在不分表达型式的情况下,CPH 的检出率高于 CAH,但按不同表达型式进行比较,则膜型 HBsAg 和浆膜型 HBcAg 的检出率反以CAH 为高,提示这二型抗原的表达与肝细胞损伤程度具有一致性。表示病毒复制的血清 HBsAg+HBeAg+抗-HBc 与 HBsAg+抗-HBc 联合检出率在 CAH 组高于 CPH 组,HBeAg 的检出率随着 SGPT 值的增高而增高。表明作为病毒复制指标的血清 HBV 标志也能反映肝细胞损伤情况。本文对靶抗原和肝组织与血清 HBV 标志表达的关系进行了讨论。

We studied the acetaminophen (AAP)-induced primary cultured rat hepatocyte injury and its mecha-nism. The results showed that in monolayer-culture condition, AAP-induced lactic dehydrogenase (LDH)escape from the hepotocytes happened only when intracellular GSH was significantly reduced. This sug-gested the intracellular GSH depletion might be the causc of AAP-induced hepatocyte injury. By directdetermination of GSH and [Ca~(2+)f]c synchronously, we demonstrated for the first time that AAP couldcause...

We studied the acetaminophen (AAP)-induced primary cultured rat hepatocyte injury and its mecha-nism. The results showed that in monolayer-culture condition, AAP-induced lactic dehydrogenase (LDH)escape from the hepotocytes happened only when intracellular GSH was significantly reduced. This sug-gested the intracellular GSH depletion might be the causc of AAP-induced hepatocyte injury. By directdetermination of GSH and [Ca~(2+)f]c synchronously, we demonstrated for the first time that AAP couldcause GSH depletion and [Ca~(2+)f]c elevation in suspensionn-cultured hepatocytes. Dithiothreitol (DTT)could prevent the GSH depletion induced by AAP and completely antagonize the AAP-induced elevat-ion of [Ca~(2+)]c. It indicated that [Ca~(2+)f]c elevation could be the direct cause of AAP-induced rathepatocyte injury and that the elevation of [Ca~(2+)f]c might be linked with GSH depletion.

醋氨酚(AAP)引起大鼠肝细胞膜损伤发生在肝细胞还原型谷胱甘肽(GSH)明显下降之后,提示GSH下降是AAP损伤肝细胞的原因之一。同步测定培养大鼠肝细胞GSH和胞浆自由钙离子浓度([Ca~(2+)f]c)的结果证明:加10mmol/L AAP培养2.5小时可引起GSH显著降低,[Ca~(2+)f]c明显升高。二巯基苏糖醇(DTT)5mmol/L可使加AAP后,细胞内GSH仍维持较高水平,[Ca~(2+)f]c升高几乎被完全拮抗。首次直接证明AAP所致的[Ca~(2+)f]c升高与GSH等巯基物质被消耗有关。

We have studied pathologically 36 cases of carcinomas of extrahepatic bile ducts,ofwhich 27 were surgically removed,9 from a autopsy series.Of all 36 cases 24 were carcinomas aris-ing in the proximal portions of extrahepatic bile ducts.The gross appearence could be classified as inrta-luminal papillary and wall infiltrating carcinomas respectiverly.Histoloigcally,it is classified as 4types:(1)papillary adnocarcinoma with intraluminal growth;(2)well differentiated adnocarcino-ma;(3)poorly differentiated adenocarcinoma;(4)carcinoma...

We have studied pathologically 36 cases of carcinomas of extrahepatic bile ducts,ofwhich 27 were surgically removed,9 from a autopsy series.Of all 36 cases 24 were carcinomas aris-ing in the proximal portions of extrahepatic bile ducts.The gross appearence could be classified as inrta-luminal papillary and wall infiltrating carcinomas respectiverly.Histoloigcally,it is classified as 4types:(1)papillary adnocarcinoma with intraluminal growth;(2)well differentiated adnocarcino-ma;(3)poorly differentiated adenocarcinoma;(4)carcinoma simplex.The latter 3 all showed infil-trating growth into the duct wall.Invasion of nerves were observed in 24 cases.Carcinomas arising inthe proximal portion of the bile ducts may extend into the liver.In 9 cases intrahepatic bile stasis wasfound in partially removed liver tissue which also showed various degrees of hepatocyte injury(atro-phy,degeneration and necrosis).The pathological features of the carcinomas of extrahepatic bileducts,their biological behavior and clinico-pathological correlation was discussed.

对我院近几年手术切除的27例肝外胆管癌及9例尸检肝外胆管癌进行了病理学研究。36例中以高位胆管癌为多(24例),占66.7%。在大体形态上,分为腔内乳头状型6例、管壁浸润型26例及结节型4例。组织学上分为4型:(1)乳头状腺癌(6例),(2)高分化腺癌(25例);(3)低分化腺癌(3例);(4)单纯癌(2例)。后3型均在胆管壁内浸润生长,其中24例见癌组织侵犯神经现象。高位胆管癌易侵及肝组织。在9例尸检及切除部分肝组织的9例中,均见有肝内淤胆现象,肝细胞有萎缩、变性甚至坏死。本文对肝外胆管癌的病理学特点、生物学行为及其与临床联系进行了讨论。

 
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