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physical hypothermia
相关语句
  物理降温
     Application of Improved Physical Hypothermia in Management of 37 Cases of Severe Craniocerebral Injury
     改良物理降温在37例重型颅脑损伤抢救中的应用
短句来源
     mild hypothermia-treated group (H), 15 minutes after brain injury, maintained the body temperature between 32~34℃(anal temperature) for 3 hours by physical hypothermia method;
     H组,创伤15分钟后,采用物理降温法,大鼠体温控制在32~34℃(肛温),维持3小时;
短句来源
     mild hypothermia-treated group (H), 15min after brain injury, maintained the body temperature between 32-34℃(anal temperature) for 3h by physical hypothermia method;
     浅低温组(H),创伤15min后,采用物理降温法,大鼠体温控制在32~34℃(肛温),维持3h;
短句来源
     The physical hypothermia in III and V was induced by padding one slight ice-bag under the rabbits and setting another six ice-bags at the axilla,groin and the sides of abdomen 30min before occlusion and constituted 30min.
     Ⅲ的物理降温采用冰袋降温法:自制一薄冰袋垫于动物身下,同时将6只冰袋分别置于前肢根部、腹股沟及腹部两侧,维持30min后撤去;
短句来源
  物理低温
     Ⅱ(control group, n=7),Ⅲ(physical hypothermia group, n=6);
     Ⅱ对照组(n=7); Ⅲ物理低温组(n=6);
短句来源
     Ⅳ(chlorpromazine group, n=6) andⅤ(chlorpromazine + physical hypothermia group, n=7).
     Ⅳ氯丙嗪组(n=6); Ⅴ氯丙嗪+物理低温组(n=7)。
短句来源
     II :control group(ischemia reperfusion,I/R),n=7; III :physical hypothermia group(PH),n=6;
     Ⅱ:对照组(n=7):Ⅲ:物理低温组(n=6);
短句来源
  “physical hypothermia”译为未确定词的双语例句
     To investigate the hypothermic methods for central hyperthermia, 30 cases of central hyperthermia with the body temperature over 39℃ following physical hypothermia or drug hypothermia received the intravenous drip of cold solution (solution temperature: 0~10℃, drip speed: 40~60 gtt/min).
     为了探讨中枢性高热的降温方法,对30例在接受物理或药物降温后体温仍高于39℃的中枢性高热患者,静脉滴注低温液体,液温0~10℃,40~60gt/min进行降温。
短句来源
     Clinical research progress on physical hypothermia for patients with central high fever
     中枢性高热物理降温的临床研究进展
短句来源
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To investigate the hypothermic methods for central hyperthermia, 30 cases of central hyperthermia with the body temperature over 39℃ following physical hypothermia or drug hypothermia received the intravenous drip of cold solution (solution temperature: 0~10℃, drip speed: 40~60 gtt/min). The results showed that the effective rate of hypothermia was up to 100% with the statistical difference being significant before and after the hypothermia ( P< 0 05). Intravenous infusion of cold...

To investigate the hypothermic methods for central hyperthermia, 30 cases of central hyperthermia with the body temperature over 39℃ following physical hypothermia or drug hypothermia received the intravenous drip of cold solution (solution temperature: 0~10℃, drip speed: 40~60 gtt/min). The results showed that the effective rate of hypothermia was up to 100% with the statistical difference being significant before and after the hypothermia ( P< 0 05). Intravenous infusion of cold solution had a significant effect on the central hyperthermia. The main points for nursing care included mastering indications, monitoring vital signs, determining body temperature and volume of solution, and expectant nursing care.

为了探讨中枢性高热的降温方法,对30例在接受物理或药物降温后体温仍高于39℃的中枢性高热患者,静脉滴注低温液体,液温0~10℃,40~60gt/min进行降温。结果:降温有效率达100%,降温前后体温差经统计学检验,P<005。提示静脉输注低温液体对中枢性高热有显著疗效。护理要点:掌握适应证,严密监护生命体征,确定液温、液量,适时对症护理。

Objective To investigate the effect of mild hypothermia on ischemia-reperfusion myocardium of anesthetized rabbits. Methods Acute ischemia-reperfusion model was made by ligation/de-ligation of anterior descending branch in coronary artery on anesthetized rabbits. 32 rabbits were randomly divided into 5 groups,Ⅰ(shame operated group, n=6);Ⅱ(control group, n=7),Ⅲ(physical hypothermia group, n=6);Ⅳ(chlorpromazine group, n=6) andⅤ(chlorpromazine + physical hypothermia group, n=7). The rabbits in Ⅱ、Ⅲ、Ⅳ...

Objective To investigate the effect of mild hypothermia on ischemia-reperfusion myocardium of anesthetized rabbits. Methods Acute ischemia-reperfusion model was made by ligation/de-ligation of anterior descending branch in coronary artery on anesthetized rabbits. 32 rabbits were randomly divided into 5 groups,Ⅰ(shame operated group, n=6);Ⅱ(control group, n=7),Ⅲ(physical hypothermia group, n=6);Ⅳ(chlorpromazine group, n=6) andⅤ(chlorpromazine + physical hypothermia group, n=7). The rabbits in Ⅱ、Ⅲ、Ⅳ and Ⅴ were ligated for 30 min and reperfused for 2 h. Body temperature and hemodynamic parameters were dynamically monitored, myocardial infarction size was determined by means of double-dyed (Evens and TTC) and the change of myocardial ultralstructure was examined in ischemia infarct region at end of reperfusion. Results Baseline level of body temperature, HR, LVSP and ±dp/dt_~max in Ⅲ and Ⅴ were lower than those in the other groups (P<0.05), but without differences between these two groups (P>0.05). The infact size of Ⅲ、Ⅳ and Ⅴ separately less than that of Ⅱ by 35.0%, 17.4% and 45.9% (P<0.05), also that of Ⅴ was smaller than that of Ⅲ and Ⅳ(P<0.05). The myocardial ultrastructural damage found under electron microscope in Ⅲ and Ⅴ were less than that of Ⅱ. Conclusions ①Mild hypothermia could protect acute ischmia-reperfusion myocardium in anesthetized rabbits. ②The protective effect inⅤgroup better than that of Ⅲ group may be due to the action of chlorpromazine itself.

目的探讨低温对麻醉兔急性缺血再灌注心肌的作用。方法采用麻醉开胸结扎/松开家兔左冠状动脉前降支制作急性缺血再灌注模型,32只动物随机分成5组Ⅰ空白组(n=6);Ⅱ对照组(n=7);Ⅲ物理低温组(n=6);Ⅳ氯丙嗪组(n=6);Ⅴ氯丙嗪+物理低温组(n=7)。其中Ⅱ~Ⅴ均结扎30min,再灌注2h。动态监测体温及血流动力学参数,Evens蓝和TTC双重染色计算心肌梗死范围(MI/RISK),再灌注末留缺血梗死区心肌观察超微结构变化。结果在降温后的各时间点,Ⅲ和Ⅴ的体温、HR、LVSP及±dp/dtmax均低于其他组(P<0.05),但它们之间无显著差异(P>0.05),Ⅲ、Ⅳ、Ⅴ的MI/RISK分别比Ⅱ减少35.0%、17.4%和45.9%(P<0.05),且Ⅴ低于Ⅲ和Ⅳ(P<0.05)。Ⅲ和Ⅴ电镜下心肌超微结构损伤轻于Ⅱ组。结论①低温对麻醉兔急性缺血再灌注心肌有一定保护作用;②氯丙嗪+物理低温组的保护效果优于物理低温组可能与药物本身的作用有关。

Objective To explore the effect of propofol-mild hypothermia on the apoptosis of neural cells in rat′s brain after traumatic brain injury by investigating the expression of Bax in this experiment.Methods By applying Feeney′s brain injury model, health male SD rats were divided randomly into five groups, eight rats for each group: sham-injured group (S), only treated with opening the skull without brain injury; model group (M), treated as Feeney′s brain injury model; mild hypothermia-treated group (H), 15min...

Objective To explore the effect of propofol-mild hypothermia on the apoptosis of neural cells in rat′s brain after traumatic brain injury by investigating the expression of Bax in this experiment.Methods By applying Feeney′s brain injury model, health male SD rats were divided randomly into five groups, eight rats for each group: sham-injured group (S), only treated with opening the skull without brain injury; model group (M), treated as Feeney′s brain injury model; mild hypothermia-treated group (H), 15min after brain injury, maintained the body temperature between 32-34℃(anal temperature) for 3h by physical hypothermia method;propofol-treated group (P), propofol (50mg/kg) injected intraperitoneally immediately after brain injury, then the same dose was administered after 1h;propofol-mild hypothermia-treated group (PH), propofol injected combined with mild-hypothermia. The rats were killed 1days after brain injury. The number of apoptotic neuron around the injured cerebral cortex was quantified by TUNEL method, the expression of Bax was observed by immunohistochemical staining.Results The quantity of TUNEL (+) neural cell was the highest in Group M, and Group H and P was higher, and in Group PH was the lowest in four groups. Compared with Group M, the expression of Bax protein in Group H and P was decreased markedly (P<0.05, P<0.01). The expression of Bax protein in Group PH was decreased significantly, compared with Group M (P<0.01), and also with Group H and P (P<0.05, P<0.01).Conclusion After brain injury, the apoptosis of neural cells around the injured tissue increased significantly. Propofol-mild hypothermia could reduce the apoptosis of neural cells in the injured rat brain tissue, thus could protect the injured brain tissue through its anti-apoptic function by adjusting the expression of Bax.

目的本实验通过观察异丙酚复合浅低温治疗后Bax蛋白表达的变化,探讨异丙酚复合浅低温对大鼠脑创伤后神经细胞凋亡的影响。方法采用Feeney′s脑创伤模型,健康雄性SD大鼠随机分为5组,每组8只:假手术组(S),只颅骨开窗不致伤;模型组(M),制作大鼠脑创伤模型;浅低温组(H),创伤15min后,采用物理降温法,大鼠体温控制在32~34℃(肛温),维持3h;异丙酚组(P),创伤后立即按50mg/kg腹腔注射异丙酚,1h时后追加全量。异丙酚复合浅低温组(PH),既控制性降温,又腹腔注射异丙酚。脑创伤1d后处死大鼠。采用原位缺口末端标记(TUNEL)法检测创伤皮层周围神经细胞凋亡情况,采用免疫组化技术来检测Bax蛋白表达的变化。结果4组中,M组细胞凋亡数目最多,H组和P组次之,PH组最少。与M组比较,H组、P组Bax表达明显减少(P<0.05、P<0.01);与M组、H组和P组分别比较,PH组Bax表达明显减少(P<0.01、P<0.01、P<0.05)。结论脑创伤后创伤灶周围凋亡神经细胞显著增加,异丙酚复合浅低温能显著减少脑创伤后神经细胞凋亡的数量,对脑创伤具有保护作用,这可能是通过调节Bax蛋白的表达来抑制细胞凋...

目的本实验通过观察异丙酚复合浅低温治疗后Bax蛋白表达的变化,探讨异丙酚复合浅低温对大鼠脑创伤后神经细胞凋亡的影响。方法采用Feeney′s脑创伤模型,健康雄性SD大鼠随机分为5组,每组8只:假手术组(S),只颅骨开窗不致伤;模型组(M),制作大鼠脑创伤模型;浅低温组(H),创伤15min后,采用物理降温法,大鼠体温控制在32~34℃(肛温),维持3h;异丙酚组(P),创伤后立即按50mg/kg腹腔注射异丙酚,1h时后追加全量。异丙酚复合浅低温组(PH),既控制性降温,又腹腔注射异丙酚。脑创伤1d后处死大鼠。采用原位缺口末端标记(TUNEL)法检测创伤皮层周围神经细胞凋亡情况,采用免疫组化技术来检测Bax蛋白表达的变化。结果4组中,M组细胞凋亡数目最多,H组和P组次之,PH组最少。与M组比较,H组、P组Bax表达明显减少(P<0.05、P<0.01);与M组、H组和P组分别比较,PH组Bax表达明显减少(P<0.01、P<0.01、P<0.05)。结论脑创伤后创伤灶周围凋亡神经细胞显著增加,异丙酚复合浅低温能显著减少脑创伤后神经细胞凋亡的数量,对脑创伤具有保护作用,这可能是通过调节Bax蛋白的表达来抑制细胞凋亡的。

 
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