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ischemic brain tissue
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  缺血脑组织
     CONCLUSION:VEGF165 gene can transform into the ischemic brain tissue and express VEGF mRNA and VEGF,and VEGF protein protects nerve cells by restraining the expression of Bax and impelling the expression of Bcl 2.
     结论:VEGF165基因可以转化到缺血脑组织中并表达VEGFmRNA和VEGF,后者可能通过抑制Bax和增强Bcl-2的表达而保护神经细胞。
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     5. PD can reduce NOS activity in the ischemic brain tissue, which has not significance compared with ischemic model group(P>0.05).
     5. 与缺血模型组相比较,0.6%PD(12mg/kg)能降低缺血脑组织NOS活性,但无显著性差异(P>0.05)。
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     Results:The expressions of IL-6 and TNF-α mRNA in ischemic brain tissue increased,reached the peak after 6 hours,and returned to the baseline after 48 hours.
     结果:缺血脑组织中IL-6的mRNA的表达水平升高,6h达到高峰,48h恢复到基础水平。
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     Protective role of vascular endothelial growth factor and its gene in ischemic brain tissue and its correlation with the expression of Bax and Bcl-2
     血管内皮生长因子及其基因对缺血脑组织保护作用与Bax和Bcl-2表达的相关性
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     ConclusionThe results indicate the rAAV-VEGF165 gene application by CSF or intravenous route can transfect the ischemic brain tissue and express VEGF protein.
     在rAAV为载体介导下,VEGF_(165)基因可以通过脑脊液及静脉导入转染到大鼠缺血脑组织中并表达VEGF,促进脑缺血边缘区新生血管的形成,进而保护神经细胞,治疗脑梗死。
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  缺血区脑组织
     MAIN OUTCOME MEASURES:The content of Ca2+, Mg2+, EAA and ET in ischemic brain tissue in different time period.
     主要观察指标:检测各个时段大鼠缺血区脑组织内Ca2+,Mg2+,EAA及血浆内皮素含量。
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     Influence of well acupoints blood-letting therapy on apoptosis associated protein of ischemic brain tissue in rats with acute cerebral ischemia
     井穴放血法对急性脑缺血大鼠缺血区脑组织凋亡相关蛋白的影响
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     Dynamic observation of the influence of blood-letting puncture of hand twelve well points on partial pressure of oxygen in ischemic brain tissue in rats with experimental cerebral ischemia
     手十二井穴刺络放血对实验性脑缺血大鼠缺血区脑组织氧分压影响的动态观察
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     Another 50 healthy SD rats(weighing 250-300 g,provided by Animal Experime ntal Center of the Chinese PLA General Hospital) were selected to establish foca l cerebral ischemia models by using the bypass technique described by Carys M Ba nnister,and then divided into 10 groups with 5 in each group to measure lactic a cid content in ischemic brain tissue before ischemia,60 minutes after ischemia,2 0,60,120 minutes after reperfusion, respectively.
     另选体质量250~300g健康SD大鼠50只(解放军总医院实验动物中心提供),以CarysMBannister转流法制作局灶缺血模型。 转流法模型50只大鼠5只一组分为10组,各用于测定盐酸法舒地尔和生理盐水治疗动物缺血前,缺血60min,再灌注20,60,120min缺血区脑组织乳酸含量。
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     Results In MCAO group,SOD activity in the ischemic brain tissue was significantly decreased while MDA content was increased markedly. In MCAO+CLB group,alterations of above parameters were more obvious.
     结果 在手术后各时间点 ,MCAO大鼠缺血区脑组织 SOD活力显著降低而 MDA含量明显增多 ,在 MCAO+CLB大鼠上述指标的变化更加严重。
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  缺血侧脑组织
     Results: Compared with those in shamoperated group,the(TUNEL)(positive) neuron and the expressions of Bcl2 and FasL protein increased in ischemic brain tissue of rats in the model group(all P<0.01).
     结果:与假手术组相比,模型组大鼠缺血侧脑组织TUNEL阳性神经细胞及B cl 2、F as L阳性细胞表达增多(P均<0.01)。
短句来源
     【Results】Baicalin reduced the scores of injured nerve function,increased SOD,GSH-Px and CAT contents and decreased MDA content in ischemic brain tissue,the difference being significant as compared with those in the model group (P<0.05 or P<0.01).
     【结果】黄芩苷能降低MCAO模型大鼠的神经功能缺损评分,升高缺血侧脑组织中SOD、GSH-Px、CAT的含量,降低MDA的含量,与模型组比较具有显著性差异(均P<0.05或P<0.01)。
短句来源
     2. In MG, the NF-κB positive cells in the ischemic brain tissue(IBT)reached the peak at 1st day,began to decrease slightly at 2nd day and 3rd day, decreased significantly at 5th day, there was a remarkable difference compared with each time point of SG (P<0.05);
     2. 模型组缺血侧脑组织 NF-κB 阳性细胞在 1d 即达高峰,2d、3d稍下降,5d 明显下降,与假手术组比较各时间点均显著增高(P<0.05)。
短句来源
     Results (1) No HSP70 was found in non ischemic brain tissue but HSP70 was observed in the parietal cortices 30 minutes after brain ischemia.
     结果(1)非缺血侧脑组织无HSP70表达,缺血30分钟缺血侧大脑皮层可见HSP70表达;
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     Nitrite(NO2-) ,nitrate(NO3-) and myeloperoxidase activity of ischemic brain tissue were detected, and the expression of E-selectin was measured by immunofluoresecent staining and RT-PCR.
     测定缺血侧脑组织NO代谢物含量、MPO活性、间接免疫荧光染色和RT-PCR方法检测E-选择素表达。
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  “ischemic brain tissue”译为未确定词的双语例句
     On day 6 after MCAO,the level of DC expressed MHC Ⅱmolecules(OX62+and OX6+) in the ischemic brain tissue also significantly increased as compared with the sham operative group(t=2.975,P< 0.05).
     脑缺血第6天,缺血组与假手术组进行比较,DC表达MHC-Ⅱ类分子(OX62+OX6+)显著升高(t=2.975,P<0.05)。
短句来源
     3. PD can reduce MDA level and increase SOD, GSH-Px, CAT activity in the ischemic brain tissue, which has significance compared with ischemic model group(P<0.01).
     3. 缺血前PD处理组脑组织MDA含量明显降低、SOD、GSH-Px、CAT活性显著升高,与缺血模型相比较有显著性差异(P<0.01)。
短句来源
     Conclusion The increased expression of CSB/ERCC6 mRNA after cerebral ischemia-reperfusion is significantly correlated with the degree of neuronal DNA damage in focal ischemic brain tissue. CSB/ERCC6 mRNA,a transcription-coupled repair factor,may contribute to neuronal DNA repair.
     结论 脑缺血再灌注后表达增高的CSB/ERCC6mRNA与局部缺血区域神经元DNA损伤密切相关 ,CSB/ERCC6mRNA作为转录修复偶联因子 ,可能调节了神经元DNA损伤后的自身修复能力。
短句来源
     And the contents of cAMP in ischemic brain regions were remarkably decreased,the Na~+-K~+ATPase activity of ischemic brain tissue was significantly increased after ICV of V_2 and V_1/V_2 receptor antagonist as compared with that of ischemic control.
     且中枢应用 V_2,V_1/V_2受体拮抗剂后,各脑区 cAMP 含量均显著低于缺血对照组,脑组织的 Na+-K+ATP 酶活力显著升高;
短句来源
     Conclusions\ Reperfusion following forebrain ischemia lead to upregulation of the expression of P 53 and P 21 proteins in ischemic brain tissue, and cycloheximide could reduce their expression, also protect neurons from DND, suggesting P 53 and P 21 proteins participated the apoptosis process of DND.
     结论 全脑缺血再灌流损伤后,脑组织P53、P21蛋白表达增加,放线菌酮可抑制其表达,并对DND起保护作用,提示P53、P21蛋白参与了全脑缺血后DND的凋亡机制,并对其起促进作用
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  ischemic brain tissue
They also suggest that alkaline pH shifts that occur after blood reperfusion of ischemic brain tissue might be critical for the survival of cells.
      
Inhibition of excitatory neurotransmitters should therefore result in cytoprotection of ischemic brain tissue.
      
The release of the neurotransmitter, glutamate, and the activation of receptor operated calcium channels, may increase the degree of damage in ischemic brain tissue.
      
The results from our study suggest that this relation may also exist in vivo in ischemic brain tissue.
      
Cerebral blood vessels, atherosclerotic endothelium, and ischemic brain tissue are good targets of gene transfer.
      
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Light and electron microscopic evaluations of the dog brain after middle cerebral artery(MCA)occlulsion and omental pedicle graft was carried out. The animals were killed 4 weeks after operation. The ischemic brain tissue and the tissue near the junction of graft omentumwere examined pathologically. Light and electron microscopy revealed irreversible brain damag in the territory which supplied by the occludede MCA, Three basic types of neuronal injury were represented within neurons around...

Light and electron microscopic evaluations of the dog brain after middle cerebral artery(MCA)occlulsion and omental pedicle graft was carried out. The animals were killed 4 weeks after operation. The ischemic brain tissue and the tissue near the junction of graft omentumwere examined pathologically. Light and electron microscopy revealed irreversible brain damag in the territory which supplied by the occludede MCA, Three basic types of neuronal injury were represented within neurons around cerebral infarction. Ischemic changes of neurons and swollen perivascular astrocytic processes showed mild than control, but there was no obvious difference between the early and delayed operative cases Area of vascularization and omental degeneration were found. These findings showed that the ischemic area, may got more blood supply. Reduction of perivascular glial swelling may improve the delayed postischemic hypoperfusion. It may preventing neurological deficit and benefit the functional recover.

本文对实验性大脑中动脉缺血行早期、晚期带蒂大网膜脑移植的标本进行了光镜及电镜研究。取10只家犬。行单则MCA起始部持久性钳夹共8只,其中3只于MCA夹闭后3小时(早期组),3只于3~4周(晚期组)分别行同侧带蒂大网膜脑移植;2只为对照组,余2只不钳夹MCA,为模拟手术组。4周后杀死动物,取缺血区脑组织和网膜与脑表面交界区组织进行视察。结果表明,钳夹后MCA分布区有不可逆性脑损害;缺血区神经元分三种类型;神经元缺血性改变和血管周围胶质水肿手术组较对照组轻,但早期与晚期组无明显差别。新生血管明显增多。因而我们认为,新生血管的供血对缺血区神经元的有益作用和血管周围胶质水肿的减轻可能会改善慢性缺血后低灌流综合征,从而有助于防止神经功能和结构的损害。

This paper reported the changes of lactate, glucose, LPO and TXB_2, 6-keto-PGF_1 after administration of glucose, insulin and anisodamine on the ischemic brain tissue following recirculation with modified Pulsinelli's rat model. The results' showed that after administration of glucose, the lac-tate, LPO and TXB_2 were increased, and 6-keto-PGF_(13) was decreased mark-dly. While after administration of insulin, the lactate, LPO were decreased and 6-keto-PGF_(1a), was increased significantly, and after administration...

This paper reported the changes of lactate, glucose, LPO and TXB_2, 6-keto-PGF_1 after administration of glucose, insulin and anisodamine on the ischemic brain tissue following recirculation with modified Pulsinelli's rat model. The results' showed that after administration of glucose, the lac-tate, LPO and TXB_2 were increased, and 6-keto-PGF_(13) was decreased mark-dly. While after administration of insulin, the lactate, LPO were decreased and 6-keto-PGF_(1a), was increased significantly, and after administration of anisodamine, the LPO, TXB_2 were decreased markdly. Electron microscopi-tally the ischemic brain tissue showed serious damage only in the group of glucose. but slight damage in other groups. These data probably indicated that the glucose can aggravate the ischemic brain damage, but the insulin and anisodamine have protective effects for the isehemic brain tissue. The mech-nism on the effects of these drugs were also discussed.

本实验利用改良的Pulsinelli“大鼠模型,研究葡萄糖、胰岛素及山莨菪碱对血流再通后,缺血脑组织中的乳酸、葡萄糖、LPO、TXB_2及6-酮-PGF_(1α)含量的影响。结果:投用葡萄糖后,乳酸、LPO及TXB_2均明显增加,6-酮-PGF_(1α)明显减少;而投用胰岛素后,乳酸、LPO则明显减少,6-酮-PG_(1α)。明显增加;投用山茛菪碱后LPO及TXB_2明显减少。电镜检查发现,投用葡萄糖组的缺血性脑损害最为严重,而在其它组损害较轻。上述资料提示,葡萄糖可加重缺血性脑损害,而胰岛素及山莨菪碱对缺血脑组织具有保护作用。本文也讨论了这些药物的作用机制。

The effect of bilateral carotid occlusion for 60 min and reperfusion for 30 min on levels of water and lipid peroxides (LPO) and activities of Na+, K+ -ATPase and superoxide dismutase (SOD) in the forebrain was studied in gerbils (Meriones unguiculatus). Luring occlusion, increased water content and decreased activities of Na+, K+-ATPase and SOD were observed. After reperfusion, the activities of Na+, K+ -ATPase and SOD continued decreasing, but LPO elevated apparently. These results support a hypothesis that...

The effect of bilateral carotid occlusion for 60 min and reperfusion for 30 min on levels of water and lipid peroxides (LPO) and activities of Na+, K+ -ATPase and superoxide dismutase (SOD) in the forebrain was studied in gerbils (Meriones unguiculatus). Luring occlusion, increased water content and decreased activities of Na+, K+-ATPase and SOD were observed. After reperfusion, the activities of Na+, K+ -ATPase and SOD continued decreasing, but LPO elevated apparently. These results support a hypothesis that the free radicals may contribute to the damage of ischemic brain tissue. Pretreatment of gerbils with allopurinol offered protection to cerebral Na+, K+ -ATPase and SOD subsequent to secondary ischemia.

本实验利用结扎沙土鼠双侧颈总动脉制成的急性脑缺血模型,观察了缺血60min,重灌流30min后脑组织含水量、脂质过氧化物含量以及Na~+,K~+-ATP酶、SOD的活力变化.实验中发现,缺血60min以内,组织含水量增加,Na~+,K~+-ATP酶和SOD的活力降低。在继以30min的重灌流之后,Na~+,K~+-ATP酶与SOD的活力继续下降,而脂质过氧化物含量急剧升高。实验结果提示缺血重灌流以后产生自由基对缺血后的脑组织产生损害作用。提前给动物使用别嘌呤醇,能够对缺血后Na~+,K~+-ATP酶、SOD起到部分保护作用。

 
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