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cns injury
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  cns损伤
     2. The protection of hypothalamus and medullatreatment of NPE after CNS injury.
     2、CNS损伤后保护下丘脑及延髓将成为治疗NPE的有力措施,及时用α-肾上腺素阻滞剂亦将是治疗NPE的关键。
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     Objective To screen the genes related to the development and injury of central nervous system (CNS) for providing some suitable target genes applied for CNS injury repair by rational genetic engineering.
     目的 探讨中枢神经系统 (CNS)发育、损伤相关基因 ,为基因工程治疗CNS损伤提供以一些可能的靶基因。
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     This experiment not only supported and advanced the ensheathing cell research but also of- fered ideal materials of in vivo transplantation for the repair of CNS injury.
     本实验支持和丰富了OECs发育的相关理论,为进一步体内移植修复CNS损伤提供了理想的材料。
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     Understanding the mechanisms of radiation-induced CNS injury may help to develop strategies to increase the radiation tolerance and treat CNS injury induced by radiation.
     了解中枢神经系统(centralnervoussystem,CNS)的辐射损伤机制,对增强CNS的辐射耐受程度和治疗辐射引起的CNS损伤具有重要意义。
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     Data indicated that nov gene might participate in the processes of regeneration and repair after CNS injury. In recent years, scientists have shown great interest in a kind of glial cell in the research of CNS injury.
     有资料表明nov基因与动物中枢神经系统的损伤和修复之间存在一定的关系,可能参与CNS损伤后的再生和修复过程。
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  中枢神经系统损伤
     DETECTION OF Nogo-A PROTEOLYTIC FRAGMENT DURING CNS INJURY
     中枢神经系统损伤过程中Nogo-A降解片段的检测
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     To elucidate the molecular mechanisms underlying the neural plasticity after CNS injury, the gene expression profile in the rat hippocampus following perforant path transections was explored by several methods including custom differential screening, custom cDNA array and cDNA microarray.
     为了阐明中枢神经系统损伤后神经可塑性的分子机制,本实验探索了多种方法,包括传统差异筛库、传统cDNA array和cDNA microarray,对穿通纤维切断后大鼠海马的基因表达图谱进行了研究。
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     Therefore, the importance of rehabilitation of cerebral stroke survivors to improve their living quality and help them go back to the society has aroused increasingly great attention and so has the investigation into the mechanism of functional rehabilitation after CNS injury.
     因此,为了提高患者生活质量及尽早重返社会,脑卒中的康复已日益受到重视,探讨中枢神经系统损伤后功能恢复的机制也得到广泛关注。
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     Objective: To understand if there are positive nestin cells in different part of human fetal brain and search for source of NSCs to treatment CNS injury.
     目的:观察人胎脑各部分有无Nestin阳性细胞存在,探索中枢神经系统损伤修复治疗中神经干细胞的来源问题。
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     To reinforce research on reparation of optic nerve is of great importance for treatment of optic nerve trauma and CNS injury.
     因此,加强视神经损伤后修复的研究,对于视神经损伤的治疗乃至中枢神经系统损伤后的治疗具有极其重要的意义。
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  中枢神经损伤
     The Mechanism of CNS Injury Induced by Fast Decompressing and the Therapeutic Effects of Hyperbaric Oxygenation
     快速减压致中枢神经损伤机制及高压氧效用的研究
短句来源
     Conclusion CNS injury by unsafe fast decompression activated the synthesis and secretion of TNF-α and increased the content of TNF-α in central nerve tissue.
     结论快速减压致中枢神经损伤激活了脑组织内小胶质细胞,增加了中枢神经组织内TNFα含量;
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     Research development on disinhibition of the regeneration after CNS injury
     中枢神经损伤再生修复去抑制作用的研究
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     ASTROCYTES NEEDS TO PERFORM MACROPINOCYTOSIS AND SYNTHESIZE NEW GFAP TO BECOME HYPERTROPHIC UNDER CNS INJURY
     中枢神经损伤中星型胶质细胞肥大的两大新特征:内吞现象和GFAP细胞骨架重排
     Conclusion: The responses of neuronal somas in PNS and CNS were different following injury, which might be associated with the difference in regeneration between PNS and CNS injury.
     结论 :外周和中枢神经损伤后再生过程中其胞体反应性不同 ,这一差异可能与外周和中枢神经损伤后再生差异有关
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  “cns injury”译为未确定词的双语例句
     Our results indicate that Tet-On-regulated NT-3 expression in OECs was efficient, offering novel material of in vivo transplantation for the repair of CNS injury.
     这些结果表明,Tet-On调控NT-3表达在OECs转染成功,为进一步体内移植修复损伤提供了理想的材料。
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     Advances in study of cellular and molecular mechanisms of radiation-induced CNS injury
     中枢神经系统辐射损伤的细胞与分子机制研究进展
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     Treatment of Ischemic CNS Injury in Mice with Mesenchymal Stem Cells from Murine Fetal Liver
     小鼠胎肝间充质干细胞治疗缺血性脑损伤的实验研究
短句来源
     These proteins,which mainly refer to Nogo-A,MAG ( myelin associated glycoprotein) and OMgp (oligodendrocyte myelin glycoprotein),are recognized as MAIFs (myelin associated inhibitory factors). MAIFs cause growth cone collapse and block axonal sprouting,and they have been found to transmit signals intracellularly through binding the same receptor-NgR (Nogo-66 receptor) . Therefore, as a potent target of treating CNS injury,NgR is getting more and more attention.
     人们从CNS髓鞘中提取纯化了多种抑制性蛋白质,称为髓鞘相关抑制因子(myelin associated inhibitory factors,MAIFs),主要包括Nogo-A,MAG(myelin associated glycoprotein)和OMgp(oligodendrocyte myelin glycoprotein);
短句来源
     From high risk to low, the factors which affected the mortality of severe asphyxia were, in order, severe CNS injury, ≥1 organ/system injury, respiratory failure, metabolic abnormality, electrolyte imbalance, blood-gas abnormality, pulmonary involvement, 10 min Apgar score≤3, gestational age<37 weeks, hepatic involvement, cardiac involvement, raised PCO_2, and hematologic involvement.
     与死亡有关的危险因素依次为中枢神经系统严重受累,1个以上脏器严重受损,呼吸衰竭,代谢紊乱,电解质紊乱,血气异常,肺损害,10分钟Apgar评分≤3,胎龄<37周,肝脏损害,心脏损害,PCO2增高和血液系统损伤。
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  cns injury
Neurobiology of the CNS injury and repair: New roles of amino acids, growth factors and neuropeptides - Introduction
      
Preface - Special Issue: Molecular mechanisms of CNS injury and neuroprotection: new roles of amino acids, heme oxygenase system
      
These results indicate that the CNS injury in AIDS is entirely dependent on events involving the peripheral immune system mediated by trafficking of SIV-infected monocytes into the brain.
      
This review presents the current understanding of the biology of microglia during normal CNS function as well as in response to CNS injury or neurodegenerative disease.
      
The increased expression of fibronectin, the only known ligand for the α5β1 integrin receptor, known to occur around the site of a CNS lesion suggests a possible role for the α5β1 receptor in the response of neurons in the vicinity of a CNS injury.
      
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Nitric oxide (No) is a gaseous neural active substance acting as both second rnessenger and cellular toxicant. Ex-cessive NO is one of the major factors leading to neuronal death. The present study aimed at dynamically observing the be-havioral, electrophysiological and histological changes induced by spinal cord injury (SCI) and the effect of NO synthase (NOS) inhibitor, N-Nitro-L-Arginine Methyl Ester (L-NAME), on the repairation of the injury during 8 weeks. one week after injury, behavioral...

Nitric oxide (No) is a gaseous neural active substance acting as both second rnessenger and cellular toxicant. Ex-cessive NO is one of the major factors leading to neuronal death. The present study aimed at dynamically observing the be-havioral, electrophysiological and histological changes induced by spinal cord injury (SCI) and the effect of NO synthase (NOS) inhibitor, N-Nitro-L-Arginine Methyl Ester (L-NAME), on the repairation of the injury during 8 weeks. one week after injury, behavioral abilities of the rats;hindlimbs recovered gradually. In grorp L-NAME, the hindrance of hindlimbs was lighter and the recovery was more rapid and better than those in group NS l descending spinal field potential were recov-ered and recorded in group L-NAME. Morphologically, the degree of cavity formation and SC atrophy in group NS was markedly more serious than those in group L-NAME;and the number of NOS-positive neurons was increased gradually after SCI and reached a higher level in about 4 weeks and then was decreased but the Iesion area enlarged; Nissl body in SC neu-rons became translocated, decreased in amount or vanished, whereas all these changes were much le8s in group L-NAME.These rsults indicate that CNS injury induces the expression of NOS, the NOS inhibitor L-NAME improves the repair of SCI, and its mechanism seems to be related to the bIockade of NO's neurotoxicity.

NO是兼有细胞间第二信使和细胞毒性作用的气体性神经活性物质。过量NO是导致神经细胞死亡的主要因素之一、应用大鼠脊髓磁控过半夹损模型,在施加NOS抑制剂L-NAME和生理盐水作为对照条件下,手术后8周内动态观察行为学、电生理学和形态学变化.结果表明;损伤后1周,后肢行为能力逐渐增强,其中抑制剂组恢复速度较快、程度也较好,可记录到恢复的下行性脊髓诱发电位,在空洞形成和脊髓萎缩程度方面抑制剂组也均明显轻于对照组,抑制剂组未见NOS表达;但对照组伤后NOS阳性神经元渐增,至4周时达较高水平.后渐降并伴有损伤区不断扩大,神经元尼氏体移位、减少或消失。结果提示;神经损伤可诱导NOS表达,NOS抑制剂L-NAME对脊髓损伤的修复有促进作用,其机制可能与NO介导的神经毒性受阻有关。

This paper reviewed Trauma Regisrry Data of all motor vehicle crash(MVC) death occurring in 16 hospitals over 30 months to determine the epidemiologic features of MVC deaths in hospitals: 4390 patients were admitted. Location of death was main ly at emergency room (ER) (30. 9%) and ICU (42% ). Of the nonsurvivers, 52% died within 24 hours ;44. 8% died after 24 hours,The two peak age group of death were 21 40 years old group and 61 - 80 one respec tively. CNS injury was responsible for 52% of death. The...

This paper reviewed Trauma Regisrry Data of all motor vehicle crash(MVC) death occurring in 16 hospitals over 30 months to determine the epidemiologic features of MVC deaths in hospitals: 4390 patients were admitted. Location of death was main ly at emergency room (ER) (30. 9%) and ICU (42% ). Of the nonsurvivers, 52% died within 24 hours ;44. 8% died after 24 hours,The two peak age group of death were 21 40 years old group and 61 - 80 one respec tively. CNS injury was responsible for 52% of death. The mean complications of the died was 1. 453 which differed significatly from that (0. 2554) of the servivors. Among complications, cardiovascular, ARDS and sepsis were 39 (21. 5%), 34 (18. 8%) and 18 (9. 9% ) respectively. The study also did comparison of deaths in different phases of hospital and found that patients who died in some different phases differed significantly in revised trauma score (RTS),injury severi ty score and probility of surval (PS) at the 0. 05 level.

本文评价了十六家医院连续三个月,因车祸意外(MVC:MotoVehicleCrash)死亡的创伤登记资料,获得以下车祸意外医院内死亡的流行病学特征:4390病人入院,181例死亡,死亡地点主要在急诊科(30.9%)和ICU(42%)。死亡病人的55.2%死于创伤后24小时内,44.8%死于24小时后,有2个死亡年龄高峰组,分别是21-40岁和61-80岁,中枢神经系统损伤占各类损伤的52%.井发症占整个死亡原因的5%,死亡病例平均并发症发生均数为1.453,死亡病人并发症发生率与存活病人有明显差异.在所有并发症中,心血管、ARDS、败血症分别各占39(21.5%)、34(18.8%)和18(9.9%).本文也比较院内各阶段死亡病例,其RTS*、ISS*、PS*、GCS*有统计学差异.

The most Cytokines are found to be produced and functional in both of immune systemsand nervous system, which become a foundation of interaction between nervous and immune system.Macrophage and microglia may play an important role in the study of CNS injury and regeneration.

一些相同的受体和配体共同存在于免疫系统和神经系统内,成为免疫物质在脑内产生效应和神经介质在免疫系统中发挥作用的基础。巨噬细胞和小胶质细胞在脑损伤及脑可塑性中起重要作用。它们的迁移、活性及其释放的细胞素,直接影响了各种脑损伤局部的环境,决定了是否有利于神经元的再生。

 
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