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  pre-administration
     at 5th -6th hours,the energies of F3,F7,F8,C3and T4of α1, of F7, F4 of δ, and of F7、 T3、 T4、 T5、 T6 of θ were significantly higher than that of pre-dministratian (P<0. 05),and at 12th -24th hours basically recoverd to the levels of pre-administration;
     α_1频段F_3、F_7、F_8、C_3、T_4导联,δ频段F_7、T_4导联,θ频段F_7、T_3、T_4、T_5、T_6导联能量第5~6小时较用药前显著增高(P<0.05),第12~24小时基本恢复用药前水平;
短句来源
     Results : There was higher value that the UtA, UmA, PI, RI and the ratio of S/D post-administration is more than that of the pre-administration,RI,S/D of UtA and S/D of UmA were significantly higher after administration,there were obviously differences(P <0. 05).
     结果:用药后UtA和UmA的PI、RI和S/D值均较用药前升高,其中UtA的RI,S/D和UmA的S/D升高值较用药前的数值差异显著(P<0.05),胎盘血管内皮细胞呈损伤性改变。
短句来源
     Results :After the once oral administration of clozapine,the energies of O1 and O2leads of α3 frequence domain, of O2 of β at 5th -6th hours were significantly lower than that of pre-administra-tion,and at 24th hour basically recovered to the level of pre-administration;
     结果,服用一次量25mg氯氮平后,α_3频段O_1、O_2导联,β频段O_2导联能量在第5~6小时较服药前显著降低(P<0.05),第24小时基本恢复用药前水平;
短句来源
     Results: After the once oral administration, the energies of Fp2 and F8 leads of a2 frequence domain, of F4 nd F8 of a3, F8 and 01 of βat 5th -6th hours were significantly lower than that of pre-administration (P<0. 05), at 24th bour basically recovered to the level of preadministration;
     结果,服用一次量25mg氯氮平后,α_2频段F_(P2)、F_8导联,α_3频段F_4、F_8导联,β频段F_8、O_1导联脑电能量在第5~6小时较服药前显著降低(P<0.05),第24小时基本恢复用药前水平;
短句来源
     At dissecting nasal septum,HR,SBP,DBP,MAP of the four groups increased at some degree,as compared with those of pre-administration,HR of group A increased significantly,SBP,DBP,MAP of Groups A,B,C also increased,but the change was not significant,as compared with those of pre-administration and Group A,B,C. The HR,SBP,DBP,MAP of group D increased significantly (P<0.05).
     鼻中隔分离时,四组患者的HR、SBP、DBP、MAP均有升高,A、B、C组SBP、DBP、MAP与用药前相比无统计学差异,A组HR与用药前相比有统计学差异,D组的HR、SBP、DBP、MAP与用药前及A、B、C三组相比差异显著(P<0.05)。
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  pre-medication
     There was a highly positive correlation between SCL and SNRT(r = 0.8573 pre-medication and r = 0.8718 post-medication).
     SCL与SNRT呈高度正相关(用药前r=0.8573,用药后r=0.8718)。
短句来源
     After Ami iv, the refractory period was shortened by S1 - St (50 ms, 330 Hz, 6V) GP<0. 01) and minimal inhibitory voltage of Sa was higher than that of pre-medication in different S1 - S2 intervals (20, 25 ms).
     Ami iv后,在高电压S_t(50ms,330Hz,6V),可出现不应期缩短。 另外,使用S_1—S_s—S_2法,当S_s—S_2间期等于20,25ms时,S_s的最小抑制电压值高于用药前水平(P<0.05)。
短句来源
     The responders’ serum IL-11 level of pre-medication was (21.81±1.88)ng/L, lower than that of non-responders(P<0.05).
     显效组用药前平均IL11水平为(21.81±1.88)ng/L,低于无效组(35.75±2.10)ng/L(P<0.05);
短句来源
     Results Compared with pre-medication, both β blockers produced a sustained and significant improvement in New York Heart Association class,left ventricular(LV) diastolic end diameter and LVEF(all P<0.05) after the treatment of 6 months.
     结果治疗后,2组左室射血分数均比用药前明显增加,且左室收缩和舒张期末容量及心胸比例均明显减少,肺瘀血情况和NYHA分级亦明显改善。
短句来源
     The blood pressure began to rise in 15 minutes and restored to pre-medication level in 60 minutes.
     15min后血压开始回升,60min后恢复至用药前水平。
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  “用药前”译为未确定词的双语例句
     RESULT In the experimental group,the serum IgE and IL-4 concentration dropped markedly(P<0.01) while this concentration was much higher than that in the control group before the treatment.
     结果实验组变应性鼻炎患者在用药前、后比较,血清中IgE浓度和IL-4浓度明显下降(P<0.01),而实验组用药前与对照组比较,IgE浓度和IL-4浓度明显增高。
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     There was very significant difference between before and 1,4 hours(P<0.01) and 8 hours(P<(0.05)) after treatment with GSH,but no significant difference after 12 hours(P>0.05). Conclusions GSH can interfere the results of some biochemical items examination relating to oxydase reaction.
     用药后1和4 h各生化项目结果与用药前比较差异有统计学意义(P<0.01),其中以1 h差异最大,用药后8 h各生化项目结果与用药前比较差异有统计学意义(P<0.05),12 h后各生化项目的结果与用药前比较差异无统计学意义(P>0.05)。
短句来源
     Clinical symptoms, Schirmer test and BUT were observed before treatment and 20 days after treatment.
     观察用药前及用药20d后,患者的自觉症状及Schirmer试验及BUT结果。
短句来源
     Compared with pre-treatment, the level of hs-CRP in the chinese medicine group decreased markedly, there was a significant difference between them(P < 0.01).
     第24周中药组hs-CRP水平明显降低,与用药前相比差异显著(P<0.01)。
短句来源
     Results After combined treatment,there were significant decreases on the degree of pruritus and the maternal blood level of biochemical parameters such as TBA,DB/TB,ALT/AST(P<0.01);
     结果用药后观察组的TBA、TB、DB、ALT/AST明显下降,与用药前相比差异有统计学意义(P<0.01); 两组瘙痒评分相比差异有统计学意义(P<0.01)。
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  pre-administration
The results were as follows: (1) On the 7th day after the first administration of BIS, the cytostatic and cytotoxic effects of MC on TC showed obvious increase over pre-administration.
      
The ratios on day 4 reached 4.7- and 5.3-fold the pre-administration values in the 10 and 20?mg/kg per day groups, respectively.
      
Pre-administration of proglumide significantly shifted the haloperidol dose response curve to the left.
      
Pre-administration of proglumide had no effect on this haloperidol dose response curve.
      
Results: In the group pre-treated with saline, the NA concentration increased to 13 times the pre-administration level, rectal temperature increased to more than 40°C, and all of the animals died 75?min later.
      
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  pre-medication
injection of 0.8 mg prindolol the AH intervals were prolonged compared with the pre-medication phase, whereas the HV times remained unchanged.
      
Again exercise induced a decrease of the AH intervals, but the exercise induced HV prolongation in the pre-medication phase could not be reproduced after administration of the betablocker.
      
infusion with standard pre-medication every third week) in patients with relapsing or progressing epithelial ovarian cancer following platinum.
      
The elimination constant k2 of aminopyrine and the excretion of glucaric acid in the pre-medication period were correlated (p>amp;lt;0.05).
      
Pre-medication test doses of 1 g ASA always inhibited platelet aggregation in vivo.
      
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The LD_(50) in mice was found to be 38.5g/kg for the infusion of Kwang-Ti-lung, given by intravenous injection. The infusion has been orally administered to rats (0.1g/kg/day) for 45 days without any toxic effect. The infusion and extract with 70% alcohol by intravenous injections to anaesthetized dogs (0.1g/kg) or by oral administrations to normal rats (10g/kg) produced a fall of blood pressure. This hypotensive effect might last for 2—3 hours. Ten renal hypertensive rats were treated with the extract at a...

The LD_(50) in mice was found to be 38.5g/kg for the infusion of Kwang-Ti-lung, given by intravenous injection. The infusion has been orally administered to rats (0.1g/kg/day) for 45 days without any toxic effect. The infusion and extract with 70% alcohol by intravenous injections to anaesthetized dogs (0.1g/kg) or by oral administrations to normal rats (10g/kg) produced a fall of blood pressure. This hypotensive effect might last for 2—3 hours. Ten renal hypertensive rats were treated with the extract at a dosage of 0.1 g/kg/day. The drug was given to all the rats for 2 weeks by oral administration. Hypotensire effect in most rats was observed in 3—7 days after medication, and became more remarkable in the second week of the therapeutic period or the first week after cessation of medication. The blood pressure in most rats returned in 2 weeks, and some rats in 4—5 weeks after the stopping of medication. The fall of blood pressure produced by Kwang-Ti-lung was neither due to the effect of the vagus, nor due to the direct dilatation of the blood vessels and the blocking of the ganglia of the vegetative nerves. The mechanism of its hypotensive effect is thought to be chiefly due to the dilatation of splanchnic vessels produced by the direct or reflex effect of Kwang-Ti-lung on the central nervous system above spinal cord.

本文对广地龙毒性、降压作用和降压机制作了初步探討。广地龙热浸剂给小鼠靜脉注射时,LD_(50)=38.5克/公斤。热浸剂(0.1克/公斤)連續給大鼠灌胃45天,未发現毒性反应。 給麻醉狗按0.1克/公斤靜脉注射热浸剂或乙醇浸出液,在給药30—45分钟出現血压下降,一般可維持2—3小时。正常大鼠一次用大剂量(10克/公斤)灌胃或腎型高血压大鼠用个剂量(50毫克/公斤)长时間(2周)灌胃,均有明显的降血压作用。在后一种情况下,多数大鼠血压下降出現于給药的第3—7天,并于停药后第2周回升到用药前水平。广地龙降压机制可能是由于它直接作用于脊髓以上的中枢神經系統或通过某些內感受器反射地影响中枢神經系統,引起部分內脏血管的扩张而使血压下降。

The influences of 15-methyl-prostaglandin F_(2α)(15-methyl-PGF_(2α))and 13-dehydro-ω-ethyl-prostaglandin F_(2α)(13-dehydro-ω-ethyl-PGF_(2α))on plasma pro-gesterone(P)and their effects on the termination of early pregnancy were studiedin pregnant rats by the same doses of 0.5mg/rat i.m.b.i.d on Day 7 of gestation.Theplasma Plevels at 4,8,24 hours after 15-methyl-PGF_(2α)were lowered down to 56.6%,43.7% and 13.3% of the initial values respectively.Pregnancy was terminated72 hours after 15-methyl-PGF(2α)administration...

The influences of 15-methyl-prostaglandin F_(2α)(15-methyl-PGF_(2α))and 13-dehydro-ω-ethyl-prostaglandin F_(2α)(13-dehydro-ω-ethyl-PGF_(2α))on plasma pro-gesterone(P)and their effects on the termination of early pregnancy were studiedin pregnant rats by the same doses of 0.5mg/rat i.m.b.i.d on Day 7 of gestation.Theplasma Plevels at 4,8,24 hours after 15-methyl-PGF_(2α)were lowered down to 56.6%,43.7% and 13.3% of the initial values respectively.Pregnancy was terminated72 hours after 15-methyl-PGF(2α)administration in all the rats of this group.Theplasma P levels at 4,8,24 hours after 13-dehydro-ω-ethyl-PGF_(2α)were reducedto 63.5%,34.4% and 51.9% of the initial values respectively.It was found inthis group that foetuses still remained in the uteri in 5 out of 6 rats 72 hours afterdrug administration,but they were smaller and free from the uteri as comparedwith controls.Concomitant treatment with 20 i.u.of HCG prevented the decrease in P in-duced by 15-methyl-PGF_(2α).The plasma P levels at 4,8,24 hours after HCGand 15-methyl-PGF_(2α)were not significantly different from those of the controls,Pregnancy was not interrupted in all the rats of this group.The size and the numberof the foetuses were similar to those of the controls.In another group,15-methyl-PGF_(2α)(20μg) was given as intravenous infusionto anaethetized pregnant rats,30 minutes after the infusion,the amount of P inutero-ovarian venous blood decreased from 1.271±0.154μg/10 min to 0.279±0.83μg/10 min.No significant change was observed in the blood flow of utero-ovarian vein.When HCG(20 i.u.)was administered intravenously,it stimulated the corpusluteum and produced a rise in the amount of P in utero-ovarian venous blood frominitial value 1.123±0.162μg/10 min to 1.496±0.018μg/10 min.Concomitanttreatment with HCG brought the P content in the utero-ovarian blood back to1.179±0.042μg/10 rain,a value similar to the initial one.The result demonstrated that 15-methyl-PGF_(2α)has a longer suppressing effecton plasma P level of the pregnant rats than 13-dehydro-ω-ethyl-PGF_(2α).Theeffect in terminating early pregnancy in rats seems related to the duration of thereduction of plasma P and the plasma P level 24 hours after PGs administration.The mechanism of the luteolytic effect of 15-methyl-PGF_(2α)appears to be littlerelated to the utero-ovarian venous blood flow, but probably related to the inhibi-tion of the effect of intrinsic gonadotropic hormone at the ovarian level.

15-甲-PGF_(2α)和13-去氢-ω 乙-PGF_(2α)在相同剂量时均能使妊娠7天的大鼠血浆孕酮浓度下降。15-甲-PGF_(α2)组于用药后4、8和24小时血浆孕酮浓度下降,分别为用药前的56.6%、43.7%和13.3%。在给药后72小时所有动物子宫中胚胎已被吸收。13-去氢ω-乙-PGF_(2α)组于用药后4、8和24小时血浆孕酮浓度分别为用药前的63.5%、34.4%和51.9%,给药后72小时大多数动物子宫中仍有胚胎,但胚胎比对照组显著为小,且多游离于子宫中。在给15-甲-PGF_(2α)前30分钟和第二次给15-甲-PGF_(2α)的同时,肌注 HOG 20国际单位,能完全对抗15-甲-PGF_(2α)的降低妊娠大鼠血浆孕酮浓度和抗早孕作用,给药后24小时内血浆孕酮浓度与对照组相似,全部动物维持妊娠,胚胎大小和数目也与对照组相似。恒速静注15-甲-PGF_(2α)(20微克)于麻醉妊娠大鼠,30分钟后已使子宫卵巢静脉血中孕酮含量由用药前1.271±0.154微克/10分钟下降到0.279±0.083微克/10分钟,给药后60分钟仍维持于低水平。如预先静注...

15-甲-PGF_(2α)和13-去氢-ω 乙-PGF_(2α)在相同剂量时均能使妊娠7天的大鼠血浆孕酮浓度下降。15-甲-PGF_(α2)组于用药后4、8和24小时血浆孕酮浓度下降,分别为用药前的56.6%、43.7%和13.3%。在给药后72小时所有动物子宫中胚胎已被吸收。13-去氢ω-乙-PGF_(2α)组于用药后4、8和24小时血浆孕酮浓度分别为用药前的63.5%、34.4%和51.9%,给药后72小时大多数动物子宫中仍有胚胎,但胚胎比对照组显著为小,且多游离于子宫中。在给15-甲-PGF_(2α)前30分钟和第二次给15-甲-PGF_(2α)的同时,肌注 HOG 20国际单位,能完全对抗15-甲-PGF_(2α)的降低妊娠大鼠血浆孕酮浓度和抗早孕作用,给药后24小时内血浆孕酮浓度与对照组相似,全部动物维持妊娠,胚胎大小和数目也与对照组相似。恒速静注15-甲-PGF_(2α)(20微克)于麻醉妊娠大鼠,30分钟后已使子宫卵巢静脉血中孕酮含量由用药前1.271±0.154微克/10分钟下降到0.279±0.083微克/10分钟,给药后60分钟仍维持于低水平。如预先静注 HOG 20国际单位,可使子宫卵巢静脉血中孕酮含量由用药前1.123±0.162微克/10分钟升高到1.496±0.018微克/10分钟,在 HCG作用的基础上再静脉恒速注入15-甲-PGF_(2α),虽可使子宫卵巢静脉血中孕酮含量下降到1.179±0.042微克/10分钟,但不能降低到

Amidarone is one of the effective anti-arrhythmic agents in pediatric tachyarrhythmias, especially showing its highly significant therapeutic effect in early stage. So, it is worth rationally using amidarone to be able to get a rapid control of the situation of arrhythmic patients. In our department 103 children with arrhythmias were treated with amidarone to observe its effectiveness in clinical practice. It was found that if there appeared no effective results in a maximum dose for 2 weeks or a maintenance...

Amidarone is one of the effective anti-arrhythmic agents in pediatric tachyarrhythmias, especially showing its highly significant therapeutic effect in early stage. So, it is worth rationally using amidarone to be able to get a rapid control of the situation of arrhythmic patients. In our department 103 children with arrhythmias were treated with amidarone to observe its effectiveness in clinical practice. It was found that if there appeared no effective results in a maximum dose for 2 weeks or a maintenance dose for 1~3 months, it had better to stop the therapy for a period of time and then to restore the use of amidarone because some effect of this drug possibly took place again when readministration of amidarone. The authors also pointed out that some side-effects of amidarone could be observed during the treatemen, particularly the disturbance of thyroid functions. Therefore, it seemed important to exclude the potential dysfunction of thyroid glands caused by amidarone therapy in children with cardiac arrhythmias.

乙胺碘呋酮为治疗小儿心律失常较有效的药物,用于快率性心律失常,特别是近期疗效卓著,故宜用于小儿部分严重心律失常而须迅速控制者。我们认为本药的用法,宜在足量应用2周后,再维持疗程1~3个月。如无效,即使延长疗程亦无裨益。宁可停药一段时间后重新用药,仍可望获得疗效。作者还指出,治疗期间有一定的副作用,尤可致甲状腺功能的紊乱,故提出用药前须谨慎排除潜在的甲状腺功能紊乱,以免进一步诱发。附:乙胺碘呋酮引起甲状腺功能絮乱2例报告

 
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