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ventricular contraction
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  心室收缩
     Impaired left ventricular contraction with the mean LV ejection fraction of 40.8%±11.7%, whereas short axis shortening fraction was 17.7%±4.3%.
     左心室收缩功能受损,平均射血分数为40.8%±11.7%,短轴缩短率为17.7%±4.3%。
短句来源
     Study on coordination of ventricular contraction by a phase analysis method in tetralogy of Fallot
     相位分析法对法洛四联症心室收缩协调性的研究
短句来源
     Effects of right atrial and ventricular DDD pacing on cardiac function and ventricular contraction synchrony
     心脏右侧DDD起搏对心功能和心室收缩同步性的影响
短句来源
     Conclusions Left ventricular rotational contraction function in patients with DCM was significantlly impaired,Based on our study,QTVI is a new method for evaluating left ventricular contraction function.
     结论DCM患者的左心室旋转收缩功能显著受损,QTVI技术评价的左心室旋转收缩功能是准确反映左心室收缩功能的超声新指标。
短句来源
     Left ventricular contraction function and ventricular electrical conduction were improved.
     结论:封堵治疗能够有效降低中老年ASD右心内径和肺动脉收缩压,增加左心室内径,改善左心室收缩功能和心室电传导。
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  室收缩
     Conclusion Left ventricular contraction dysfunction occurs in left ventricles after cardiac arrest and resuscitation and this lead to the disorder of indexes of hemodynamics such as the decrease of MABP and the increase of PCWP.
     结论心脏骤停复苏后存在左室收缩功能障碍,导致MABP降低、PCWP增高等血流动力学指标的紊乱。
短句来源
     Result: In comparison with the model group,the function of left ventricular contraction function improved,the area of left ventricular cavity diminished,and proliferation of collagen,content of Ang II and apoptosis percentage of the myocardial cell reduced in all of the treatment groups(P<0.05 or P<0.01).
     结果:各治疗组都改善了心衰大鼠左室收缩功能、减小左室腔面积、抑制胶原增殖、AngⅡ含量和心肌细胞凋亡,与模型组比较有显著差异;
短句来源
     (3)The decrease of right ventricular contraction was global rather than segmental.
     (3)用3DE检测右室节段收缩率,结果表明肺心病患者的右室收缩功能减低为整个心肌收缩力降低而非节段性心肌功能减弱;
短句来源
     Clinical Observation on Improvement of Left Ventricular Contraction in Cardiac Infarction Treated by Gengfukang
     梗复康改善心肌梗塞患者左室收缩功能的临床观察
短句来源
     Acupuncture treatment of silent heart failure and its effect on left ventricular contraction function
     针刺治疗无症状心力衰竭及其对患者左室收缩功能的影响
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  “ventricular contraction”译为未确定词的双语例句
     Results The effective rate of observation group and the control group were 85.7% and 62.5% on the first month,89.8% and 66.7% at the third month on atrial arrhythmias and 86.0% and 61.9 at first month,88.4% and 69.0% at third month on premature ventricular contraction.
     结果观察组与对照组治疗房性心律失常1个月有效率分别为85.7%与62.5%,3个月有效率分别为89.8%与66.7%; 治疗室性期前收缩1个月有效率分别为86.0%与61.9%,3个月有效率分别为88.4%与69.0%;
短句来源
     Method The data for analysis and classification was obtained from MIT-BIH database, including 300 samples each of normal sinus rhythm (NSR), atria premature contraction (APC), premature ventricular contraction (PVC), ventricular tachycardia (VT), ventricular fibrillation (VF) and superventricular tachycardia (SVT).
     方法收集MIT BIHdatabase中正常窦性心律 (NSR)、心房早期收缩 (APC)、心室早期收缩 (PVC)、心室性心动过速 (VT)、心室纤维性颤动 (VF)和室上性心动过速 (SVT)各 30 0例进行分类研究。
短句来源
     Methods Among 54 patients,20 patients were with atrial fibrillation,10 with frequently premature ventricular contraction,4 with frequently premature atrial contraction,3 with supraventricular tachycardia,1 with ventricular tachycardia,14 patients with Ⅰ°AVB,1 with Ⅱ°AVB and 1 with Ⅲ°AVB.All 54 patients were central type foramen secundum ASD.
     方法54例心律失常患者中,心房纤颤20例,频发室性早搏10例,频发房性早搏4例,室上性心动过速3例,室性心动过速1例,Ⅰ°房室传导阻滞(AVB)14例,Ⅱ°、Ⅲ°AVB各1例。
短句来源
     Premature ventricular contraction(PVC), ventricular tachycardia(VT) and ventricular fibrillation( VF) were developed by endothelin 1( ET 1) injected into the coronary ostia at the dose of 900pmol/kg in anesthetized rats.
     本工作在麻醉大鼠冠状动脉口注射内皮素1(ET1)900pmol/kg能引起室性早搏(PVC)、室速(VT)、室颤(VF)等严重心律失常,心律失常评分(AS)为5.6±1.0;
短句来源
     There are left ventricular bands in 28 hearts of 38 cases with premature ventricular contraction.
     在38例室性早搏受检者中,检出左心室条束28例。
短句来源
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  ventricular contraction
This electrical distancing of the leads will potentially improve the mechanics of ventricular contraction and the flexibility of RV-LV optimization.
      
Thus, in patients following myocardial infarction, not only impaired baroreceptor function but also altered left ventricular contraction behavior contributes to the phenomenon of heart rate turbulence.
      
To be enrolled in these studies, patients were required to have a premature ventricular contraction (PVC) rate of at least 30/hr on a baseline 24-hour ambulatory ECG.
      
TPEN reduced the transient elevation in left ventricular contraction p >amp;lt; 0.05), but did not prevent the subsequent decrease in cardiac function.
      
Perfusion with post-ischemic hepatic effluent caused a transient 15-min increase in left ventricular contraction and coronary flow (p >amp;lt; 0.05), followed by a decrease in cardiac function at one hour.
      
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A total of 23 adult rabbits were used in this study. Six were used as control andthe remaining 17 rabbits were injected with adrenaline to produce cardiac lesion. Theseanimals were sacrificed alternatively either by medullary-shock or clacium block after 1,3, 5, 7, 10, 14 and 21 days of experiment. The left ventricle was excised and fixed inCarnoy fixative, embedded in paraffin, stained with either H-E or PAS (McManus)method. Slides of saliva digestion and PAS staining were used for the identificationof glycogen....

A total of 23 adult rabbits were used in this study. Six were used as control andthe remaining 17 rabbits were injected with adrenaline to produce cardiac lesion. Theseanimals were sacrificed alternatively either by medullary-shock or clacium block after 1,3, 5, 7, 10, 14 and 21 days of experiment. The left ventricle was excised and fixed inCarnoy fixative, embedded in paraffin, stained with either H-E or PAS (McManus)method. Slides of saliva digestion and PAS staining were used for the identificationof glycogen. The following points of conclusion were drawn: 1. More glycogen was present in the left ventricle of animals killed by calciumblock than those killed by medulla-shock. 2. The glycogen content in the outer and inner myocardial layers of the normalanimal killed by calcium block was identical and that of the middle layer was lesser.Since these myocardial layers perform different functions in ventricular contraction, theauthors suggest that the glycogen content of the different normal myocardial layers wasassociated with its function. 3. Medulla-shock causes anoxia of the heart. There were severe exhaustion ofglycogen in the outer layer during anoxia. The influence of anoxia was restricted to theintersinusal region (the parasinusal region was less influenced) of the middle layer. Theinfluence of anoxia to the inner layer was minimal. These phenomena were interpretedas due to the different blood sources of each layer. 4. In the myocardium of the shocked animals, glycogen was constantly stagnatedat the following places: the perisinusal fibers of the middle layer, the lateral boundariesof the papillary muscle, and the inner layer of the myocardium. These phenomena fur-ther proved the relationship between the glycogen distribution and the blood supply. 5. The perilesion deposition of glycogen due to adrenaline was not influenced bythe process of anoxia in any layer of the myocardium. The abnormal glycogen disap-peared coincidentally with other cytoplasmic material during degeneration. So, it wasbelieved that this glycogen probably was combined with the protein. The range ofabnormal glycogen distribution was in accordance with the severity of the lesion. 6. Within the first 5 days of our experiment, the focal lesion had not yet form-ed, the injury of individual fiber was variable and so did glycogen distribution. Afterthe 14th experimental day, the lesion was replaced by scar tissue and the perilesion car-diac fibers reappeared to normal. Thus, there was no abnormal glycogen deposit in thatregion. Only between the 5th and 14th experimental days, the abnormal glycogen de-position was distinct.

作者应用肾上腺素产生实验性心肌病变。从钙杀死及捶死的正常及实验的家兔心肌PAS及H.E染片的观察中,作者得出下列几点结论: 1,钙杀死的动物较之捶死动物的心肌含有更多的糖原。 2,钙杀死动物心肌内层与外层糖原含量相等而其功能皆为缩短心室纵径;司心室横径收缩的心肌中层糖原较少。因此作者认为心肌各层糖原含量与其功能有关。 3,乏氧对心肌糖原的影响以外层最显著,心肌中层的血窦间地区(不包括血窦旁地区)糖原消耗较多,而乏氧对心肌内层亦有一定影响。作者认为以上差别是与其血液来源相关。 4,在捶死动物中,血窦旁及乳头肌两侧以及心肌内层皆保留糖原一事再次证明糖原与血液供应的关系。 5,心肌异常糖原不受乏氧影响,它可能是与蛋白貭结合的糖原。 6,5—14天的病灶周旁异常糖原较多。14天以上的病灶结瘢修复,其周旁纤维康复,无异常糖原。在5天以内的材料中,病灶尚未形成,病变纤维周旁的纤维因受损与恢复程度不同故而糖原分布极不恒定。

Dynamic observations were made With systolic time interval determination on 28 cases of acute myocardial infarction at the first-day. 3-4th days, 7-8th days, 13-14th days, 28-30th days and three-month intervals after onset of the disease. A diminution of QS_2I and LVET, prolongation of PEPI and elevation of P/L ratio value had been found in AMI, as compared with normal individuals. These indices were regarded to reflect the left ventricular contraction performance, stroke-volume and the alteration of ventricular...

Dynamic observations were made With systolic time interval determination on 28 cases of acute myocardial infarction at the first-day. 3-4th days, 7-8th days, 13-14th days, 28-30th days and three-month intervals after onset of the disease. A diminution of QS_2I and LVET, prolongation of PEPI and elevation of P/L ratio value had been found in AMI, as compared with normal individuals. These indices were regarded to reflect the left ventricular contraction performance, stroke-volume and the alteration of ventricular compliance.It has a guiding significance for evaluating the cardiac performance and the prognosis of AMI. An increase of sympathetic activity has been observed in most patients of AMI.Its influence must be taken into consideration in evaluating the meaning of each index alteration.

对28例急性心肌梗塞患者于第一月内和三月后进行六次动态观察,与对照组比较,急性心肌梗塞患者的QS_2I和LVET缩短,PEPI延长,P/L比值升高;这些指标可反映左室心肌收缩功能、心搏出量、心肌顺应性改变,对估价心功能和预测预后有指导意义。大多数患者有交感神经系统活动性增嵩,在判断各项指标变化时,应考虑其影响。

In this study we used the method of electrical-induced ventricular fibrillation (VF) to evaluate quantitatively the effect of ventricular contraction on segmtntal coronary arterial resistance.Experiments were done on 10 anesthetized,open-chest dogs.The circumflex coronary artery was pump-perfused at constant flow.Pressure was measured at the tip of the perfus-ion sannula sealed in the circumflex artery,in an apical branch of the circumflex artery and in thoracic aorta.Electrocardiogram was recorded.When...

In this study we used the method of electrical-induced ventricular fibrillation (VF) to evaluate quantitatively the effect of ventricular contraction on segmtntal coronary arterial resistance.Experiments were done on 10 anesthetized,open-chest dogs.The circumflex coronary artery was pump-perfused at constant flow.Pressure was measured at the tip of the perfus-ion sannula sealed in the circumflex artery,in an apical branch of the circumflex artery and in thoracic aorta.Electrocardiogram was recorded.When VF was induced,total coronary arterial resistance reduced from 2.20±0.77(M±SD ) to 1.67±0.71mmHg·min/ml (P<0.001),averagely 24% of control.Small coronary arterial resistance in the myocardiuri reduced from 1.94±0.78 to 1.44±0.76mm Hg·min/ml (P<0.001),averagely 26% of control However,epicardial large coronary arterial resistance did not change significantly,from 0.26±0.11 to 0.23±0.11mm Hg·min/ml.It is concluded that the ventricular contraction mainly affects the resistanre of small coronary vessels in the myocardium and does not have a significant effect on the resistance of epicardial large coronary vessels.

本实验用电致心室纤颤(VF)的方法,定量评定心室收缩对冠状动脉节段阻力的作用。在10条麻醉开胸犬,冠状动脉左旋支恒流灌注,分别测量主动脉压、左旋支灌注压及其远端冠状动脉压,记录心电图。VF时,冠状动脉总阻力(R_T)由VF前的2.20±0.77(均数±标准差)下降至1.67±0.71mmHg·min/ml(P<0.001),R_T平均减低24%;心肌内小冠状动脉阻力(R_S)由1.94±0.78下降至1.44±0.76mmHg·min/ml(P<0.001),R_s平均减低26%;然而,心外膜大冠状动脉阻力(R_L)并无显著改变,由0.26±0.11变为0.23±0.11mmHg·min/ml。实验证明,心室收缩主要影响R_S,对R_L无明显影响。

 
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