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neurogenic inflammation
相关语句
  神经原性炎症
     Secretoneurin and neurogenic inflammation
     Secretoneurin和神经原性炎症(英文)
短句来源
     Neurogenic Inflammation of the Visceral Organs Evoked by Electrical Stimulation of Acupoint in Rats
     穴位刺激引起内脏的神经原性炎症反应
短句来源
     Mast cells may participate in neurogenic inflammation(NI) relative to migraine.
     肥大细胞参与神经原性炎症(NI),在偏头痛的发病机制中发挥重要作用。
短句来源
  神经性炎症
     Pratice of The Rat Model of Neurogenic Inflammation in the Study of Medicines for External Application in Traumotology and Orthopedics
     大鼠神经性炎症模型在骨伤科外用药研究中的应用
短句来源
     EFFECT ELECTROACUPUNCTURE ON THE NEUROGENIC INFLAMMATION
     电针对逆向刺激所致神经性炎症的效应
短句来源
     Damage of fine particulate is through oxidant stress, inflammation and neurogenic inflammation and so on, but the detail of the mechanism for them is lacking.
     免疫毒性的作用机制可能通过氧化、炎症刺激和神经性炎症反应等有关 ,但具体致病机制仍不清楚 ,有必要从整体上研究颗粒物的毒性及其作用机制。
短句来源
     Neurogenic inflammation plays an important role in pathogensis of allergic rhinitis.
     神经性炎症在变应性鼻炎发病中发挥了重要作用。
短句来源
  “neurogenic inflammation”译为未确定词的双语例句
     Th1/Th2 cytokine immune imbalance may indirectly induce the airway neurogenic inflammation by regulating the NGF mRNA expression.
     Th1/Th2类细胞因子免疫失衡可能通过调控NGFmRNA表达间接促进其诱导神经源性炎症。
短句来源
     5-hydroxytryptamine1B/1D receptor agonist and valproic acid block dural neurogenic inflammation by different mechanism.
     5-羟色胺1B/1D受体激动剂、丙戊酸是通过各自不同的机制阻滞硬脑膜内的神经源性炎症。
短句来源
     Conclusion LIF and NK-1R were excessively expressed and closely correlated in lungs of the rat asthmatic model,suggesting that LIF may be involved in modulating airway neurogenic inflammation.
     结论哮喘气道存在LIF和NK-1R的过度表达,而且两者存在表达相关性,LIF可能参与了哮喘气道神经源性炎症的调控。
短句来源
     Objective To study the expression of leukemia inhibitory factor(LIF) and neurokinin receptors(NKR) in the lungs of asthmatic rats,and to evaluate the role of LIF in airway neurogenic inflammation.
     目的研究白血病抑制因子(LIF)和神经激肽受体(NKR)在支气管哮喘(简称哮喘)中的表达并分析两者的相关性,以探讨LIF与哮喘神经源性气道炎症的关系。
短句来源
     There were no significant differences between the two groups withrespect to the density of VIP IR positive nerves(t=1 609,P>0 05) The density of SP IR positive nerves was significantly higher in smokers than in non smokers in controls(t<2 119,P<0 05),and was not significant in COPD group(t=0 930,P>0 05) These foundings support a role for SP in neurogenic inflammation and the formation of COPD
     两组间 VIP含量无显著差异 (t=1.6 0 9,P>0 .0 5 ) ; 对照组吸烟者 SP含量显著高于非吸烟者 (t=2 .119,P<0 .0 5 ) ,而 COPD组吸烟者与非吸烟者 SP含量无显著差异 (t=0 .93,P>0 .0 5 )。
短句来源
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  neurogenic inflammation
Neurogenic inflammation (vasodilatation and neurogenic plasma extravasation) is one explanation for the development of headaches and the blood flow changes which occur during migraine headache.
      
Ergot alkaloids have been shown to block neurogenic inflammation via a C-fibre dependent neuronal mechanism.
      
The efficacy of methysergide may also depend on other 5-HT1-like receptor-mediated actions such as cranial vasoconstriction, and inhibition of cranial vascular neurogenic inflammation.
      
Also, recent evidence supports a role for neurogenic inflammation in the development of complications.
      
Neurogenic inflammation is triggered by airway or esophageal acidi.cation, innate immune cells are affected by acidity, and there are pathways by which the acquired immune system also can be activated by the chemistry of an acidic airway.
      
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Rats anaesthetized with sodium pentobarbitone were used in this experiment. All dorsal and ventral roots below L2 were cut and skin hairs were removed and venous injection of Evans blue (40mg/kg) was made. Continuous electric pulses stimulated left bladder nerve, blue oozes which showed points and/or stripe shape were found and recorded with photography in ipsilateral lower limb skin, while the right lower limb showed no changes.Content of Evans blue was measured and it showed significant difference(P<0. 05)with...

Rats anaesthetized with sodium pentobarbitone were used in this experiment. All dorsal and ventral roots below L2 were cut and skin hairs were removed and venous injection of Evans blue (40mg/kg) was made. Continuous electric pulses stimulated left bladder nerve, blue oozes which showed points and/or stripe shape were found and recorded with photography in ipsilateral lower limb skin, while the right lower limb showed no changes.Content of Evans blue was measured and it showed significant difference(P<0. 05)with the same area of the right lower limb and back and abdominal region.The study indicated that stimulating the bladder nerve could cause dilatation of cutaneous blood vessels and extravasation of plasma ipsilaterally.This neurogenic inflammation was induced through long viscerosomatic axonal reflex. The long axonal reflex is a part of acupoints and meridian activities and ralated to mechanism of referred pain. This study provided a data to clarify the essence of meridian and the mechanism of referred pain.

实验用大白鼠,戍巴比妥钠腹腔麻醉,切断L2以下背腹根,脱去L1以下全部皮毛,静脉注射Evans蓝(40mg/kg),连续电刺激左侧膀胱神经,在同侧下肢皮肤及皮下可呈现点条状分布的蓝色渗出,而对侧则无,照相记录。并测定其皮肤及皮下Evans蓝含量,发现其与对侧相同区域、背部及腹部有显著性差异(P<0.05)。以上结果说明:刺激膀胱神经引起的同侧下肢皮肤血管扩张、血浆外渗等神经源性炎症反应是通过内脏躯体长轴突反射引起的。长轴突反射活动是穴位及经络活动的一部分,并参与牵涉痛机理的形成。为阐明经络生理实质及牵涉痛机理提供了可靠的证据。

Mast cells may participate in neurogenic inflammation(NI) relative to migraine. The effects of Tianrong acupoint therapy on unilateral degranulation of mast cells in rat dura mater following elcetrical stimulation of trigeminal ganglion were evaluated by transmission electron microscopy. We found that Tianrong acuPOint injection with saline and Quchi acuPoint injection with prednisolonum inhibited effectively the degranulation of mast cells in rat dura mater induced by electrical stimulation of trigeminal...

Mast cells may participate in neurogenic inflammation(NI) relative to migraine. The effects of Tianrong acupoint therapy on unilateral degranulation of mast cells in rat dura mater following elcetrical stimulation of trigeminal ganglion were evaluated by transmission electron microscopy. We found that Tianrong acuPOint injection with saline and Quchi acuPoint injection with prednisolonum inhibited effectively the degranulation of mast cells in rat dura mater induced by electrical stimulation of trigeminal ganglion, but it was much weaker than T1anrong acupoint injection with prednisolonum. The results provided the evidence that Tianrong acupoint therapy could inhibit the NI on the affected side.

肥大细胞参与神经原性炎症(NI),在偏头痛的发病机制中发挥重要作用。本研究采用透视电镜技术观察‘天容’穴治疗对电刺激三叉神经节诱发的硬脑膜肥大细胞脱颗粒的影响。结果发现:“天容”穴注射强的松龙可显著抑制电刺激三叉神经节所致的硬脑膜肥大细胞脱颗粒,而“天容”穴注射生理盐水和“曲池”穴注射强的松龙仅有部分抑制作用。提示抑制硬脑膜NI是“天容”穴治疗偏头痛的作用机制之一。

Chymase is a kind of serine proteinase,mainly exists in secretory granules of mast cell and extracellular Interstitium.The mature enzyme is a glycoprotein comprising 226 amino acid residues,with molecular weight of 3 0kD.It can be inhibited by serine proteinase inhibitors but not by angiotensinⅠconverting enzyme inhibitors.The cDNA and genomic DNA of human chymase clond and sequenced.Chymase has close relations with neurogenic inflammation,extracellular matrix catabolism,control of vasoactive peptide metabolism,and...

Chymase is a kind of serine proteinase,mainly exists in secretory granules of mast cell and extracellular Interstitium.The mature enzyme is a glycoprotein comprising 226 amino acid residues,with molecular weight of 3 0kD.It can be inhibited by serine proteinase inhibitors but not by angiotensinⅠconverting enzyme inhibitors.The cDNA and genomic DNA of human chymase clond and sequenced.Chymase has close relations with neurogenic inflammation,extracellular matrix catabolism,control of vasoactive peptide metabolism,and so on.It plays an important role in angiotensin Ⅱformation in human heart.

糜酶是一类丝氨酸蛋白酶,主要存在于肥大细胞的分泌颗粒和细胞间质。成熟的酶为一糖蛋白,由226个氨基酸组成,分子量为30kD,其酶活力能被丝氨酸蛋白酶抑制剂所抑制,而不受血管紧张素Ⅰ转换酶抑制剂的影响。糜酶的cDNA和基因组DNA均已被克隆和测序。该酶和神经炎症反应、血管活性肽代谢和细胞外基质代谢等有密切联系,在心脏血管紧张素Ⅱ形成中起重要作用。

 
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