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botulinum toxin
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  肉毒毒素
     After follow up for 6~36 months, the recurrence rate of botulinum toxin A group and haloperidol group was 63.6%(14/22) and 95.0% (19/20) (χ 2=4.76, P<0.05).
     在 6~ 36个月的随访期间 ,A型肉毒毒素治疗组和氟哌啶醇对照组的复发率分别为 6 3 6 %(14 / 2 2 )与 95 0 % (19/ 2 0 ) (χ2 =4 76 ,P <0 0 5 )。
短句来源
     The minimal detectable concentration of type B toxin was 16 ng/ml ( 4 LD50/ml), and the threshold of cross-reaction with type A botulinum toxin was 32ug/ml (300 LD50/ml ) with a specific index of 2000 or over.
     可检出B型肉毒毒素16ng/ml(4LD_(50)/ml) A型大于32μg/ml(大于301LD_(50)/ml),特异性比值大于2,000;
短句来源
     Botulinum toxin A in the treatment of dystonia:a clinical analysis of 409 cases
     A型肉毒毒素治疗肌张力障碍:409例疗效分析
短句来源
     A clinical study on Botulinum toxin A in treatment for spasticity in spinal cord injury
     A型肉毒毒素治疗脊髓损伤痉挛状态的临床研究
短句来源
     Inhibition of rhinorrhea with botulinum toxin type A and the expression of VIP on nasal mucosa in allergic rhinitis rat
     A型肉毒毒素抑制大鼠变应性鼻炎鼻溢及VIP在鼻腔粘膜的免疫表达
短句来源
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  肉毒杆菌毒素
     A clinical study on the treatment of congenital nystagmus by using of botulinum toxin A
     A型肉毒杆菌毒素治疗先天性眼球震颤临床研究
短句来源
     Gene Sequence and Analysis of the Clostridium Botulinum Toxin of Type A
     A型肉毒杆菌毒素的基因测序与分析
短句来源
     Clinical observation of therapeutic effects of botulinum toxin type A on 235 hemifacial spasm patients
     A型肉毒杆菌毒素治疗面肌痉挛235例临床研究
短句来源
     Clinical study of botulinum toxin type A in treating hemifacial spasm
     A型肉毒杆菌毒素治疗面肌痉挛的临床研究
短句来源
     Methods: a total of 68 individuals were enrolled in the clinical study. The Botulinum Toxin A was locally injected into the involved muscles. The dosage for each injection was 1~2 ng/mL(2.5~50 U/mL).
     方法 :应用A型肉毒杆菌毒素局部肌肉注射治疗共 6 8例 ,药物浓度为 0 1~ 0 2ng/mL(2 5~ 50U/mL)。
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  内毒毒素
     Inhibition of Terminal Sprouting Triggered by Botulinum Toxin Type A in Rats Poised with Acrylamide
     丙烯酰胺抑制A型内毒毒素肌注后神经芽生的实验研究
短句来源
  “botulinum toxin”译为未确定词的双语例句
     Objective To detect the expression quantity and To investigate signal transduction of ras-related C3 botulinum toxin substrate,Rac1 and T lymphoma invasion and metastasis inducing factor 1,Tiam 1 in gastric cancer tissues、tissues around the cancer andgastric benign lesion tissues,and To investigate its effect in gastric cancer metastatic.
     目的研究T淋巴瘤侵袭转移诱导因子1(Tlymphoma invasion and metastasis inducingfactor 1,Tiam1)与Ras相关的C3肉毒素底物1(ras-related C3 botulinumtoxin substrate,Rac 1)信号转导及检测其在胃癌组织、癌旁组织、胃良性病变组织中的量值水平,进一步探讨其在胃癌转移中的作用。
短句来源
     Method: Injecting the botulinum toxin type A into the hypertrophied masseter of 25 cases patients,50 U dosage was injected in per-side.
     方法:采用A型肉毒素(botulinum toxin type-A,BTX-A)对25例咬肌肥大的患者进行治疗,每一侧注射剂量为50U。
短句来源
     The children in group A were treated by Botulinum toxin A(BTX-A). The dosage was 55-150IU(average dosage 80.4 IU).
     A组患儿38例,应用BTX—A治疗,用量为55—150IU,平均80.4IU。
短句来源
     Results After Botulinum toxin A injection,the effect last for 18.92 ±6.12 weeks in patients with hemifacial spasm,12.04±6.35 weeks in patients with blepharospam.
     结果 药效作用时间为 :面肌痉挛 18.92± 6 .12周 ,眼睑痉挛 12 .0 4± 6 .35周。
短句来源
     Objective To investigate the relationship between the expression of T lymphoma invasion and (metastasis) inducing factor 1 (Tiam1), ras-related C3 botulinum toxin substrate 1 (Rac1) with the invasion and metastasis of gastric cancer.
     目的 探讨T淋巴瘤侵袭转移诱导因子 1 (Tiam1 )和Ras相关的C3肉毒素底物 1 (Rac1 )表达与胃癌侵袭、转移间的关系。
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  botulinum toxin
It is to be assumed that the facial paresis resulted from the toxin related disturbance of acetylcholine release at the motor endplates, comparable to the effect of botulinum toxin.
      
Polyradiculoneuritis following botulinum toxin therapy
      
The development of Guillain-Barré syndrome is reported in a patient, who had previously received botulinum toxin type A injections into both orbicularis oculi muscles to treat idiopathic blepharospasm.
      
Treatment of cervical dystonia hand spasms and laryngeal dystonia with botulinum toxin
      
One hundred and twenty-six patients with different forms of focal dystonia (89 with cervical dystonia, 12 with hand cramps and 25 with laryngeal dystonia) were treated with localised injections of botulinum toxin.
      
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The experiments were made on guinea pigs. The results obtained demonstrate that after crushing the nerve the regeneration of the motor nerve endings in muscle poisoned by botulinum toxin can proceed with the same effectiveness as in unpoisoned muscle. At the same time they show that causing the poisoned nerve endings to degenerate and then regenerate may be employed as a means of accelerating the recovery of the poisoned muscle; for example, the very slow spontaneous recovery usually shown by a poisoned...

The experiments were made on guinea pigs. The results obtained demonstrate that after crushing the nerve the regeneration of the motor nerve endings in muscle poisoned by botulinum toxin can proceed with the same effectiveness as in unpoisoned muscle. At the same time they show that causing the poisoned nerve endings to degenerate and then regenerate may be employed as a means of accelerating the recovery of the poisoned muscle; for example, the very slow spontaneous recovery usually shown by a poisoned muscle requiring about a year for completion, can be shortened to about 11/2 months by crushing the nerve. The suggestion is made that the slow spontaneous recovery iust mentioned is probably the result of a process of slow spontaneous tissue renewal constantly occurring in the motor nerve endings. It is also pointed out that the experimental finding reported here constitutes a new piece of corroborative evidence for the conclusion that the site of action of botulinum toxin is confined to the motor nerve endings.

实驗在豚鼠进行,結果揭示,夹毁神经后运动神经末梢的再生在肉毒杆菌毒素中毒的肌肉与在正常肌肉完全一样地有效。用夹毁神经的办法,可以比较迅速地解除肉毒中毒肌肉的麻痹。本文结果,亦对肉毒的作用地点问题提供了新的佐証。

After botulinum poisoning, the "slow" and "fast" muscle fibres of the chick exhibit opposite trophic reactions. This was first demonstrated with the pure "slow" muscle, anterior latissimus dorsi (ALD), and the pure "fast" muscle, posterior latissimus dorsi (PLD). After botulinum injection, for periods from 1 to 8 weeks studied, ALD regularly showed hypertrophy, while PLD manifested the usual atrophy. That the hypertrophy of ALD was really brought about in some specific way by botulinum toxin and was not...

After botulinum poisoning, the "slow" and "fast" muscle fibres of the chick exhibit opposite trophic reactions. This was first demonstrated with the pure "slow" muscle, anterior latissimus dorsi (ALD), and the pure "fast" muscle, posterior latissimus dorsi (PLD). After botulinum injection, for periods from 1 to 8 weeks studied, ALD regularly showed hypertrophy, while PLD manifested the usual atrophy. That the hypertrophy of ALD was really brought about in some specific way by botulinum toxin and was not due to incidental stretching, was ascertained in experiments, in which any gross difference in the operation of mechanical factors on ALD on the poisoned and on the control side was excluded. Additional evidence for the contrasting trophicreac tions of the two types of muscles to botulinum toxin was provided by observations on the sartorius muscle. Histological studies of this muscle following botulinum poisoning revealed the presence of both hypertrophied and atrophied fibres, corresponding to the fact that the chick sartorius muscle contains both "slow" and "fast" fibres. The effects of botulinum toxin on the chick muscles described in this paper form an exact parallel to the effects of denervation described by us in a previous report. It is remarkable that the novel phenomenon of post-denervation muscular hypertrophy in chick could also be exactly reproduced by botulinum poisoning.

肉毒杆菌毒素象去神经一样,在鸡的“慢”肌和“快”肌引起形成鮮明对比的营养性变化。在肉毒中毒后1—8周純“慢”肌前背阔肌无例外地发生肥大,而純“快”肌后背阔肌則出现通常的萎縮。混合肌肉缝匠肌中毒后,其中“慢”和“快”两种纤维,相应地出现肥大和萎縮两种相反的营养性反应。

The recovery effects of 4-aminopyridine,guanidine,tetraethylammonium andamphotericin B on the neuromuscular transmission blocked by type A botulinumtoxin were investigated at the isolated phrenic diaphragm preparation of rat.Itwas found that the spike of motor nerve terminal abolished by botulinum toxinreappeared after application of 4-aminopyridine(2.5×10~(-6))or guanidine(3 mM)to the bathing medium.The amplitude of the spike as well as that of the endplate potential restored progressively.Consequently,the...

The recovery effects of 4-aminopyridine,guanidine,tetraethylammonium andamphotericin B on the neuromuscular transmission blocked by type A botulinumtoxin were investigated at the isolated phrenic diaphragm preparation of rat.Itwas found that the spike of motor nerve terminal abolished by botulinum toxinreappeared after application of 4-aminopyridine(2.5×10~(-6))or guanidine(3 mM)to the bathing medium.The amplitude of the spike as well as that of the endplate potential restored progressively.Consequently,the contraction of nerve-muscle preparation to indirect stimulus also reappeared.The effect of tetraethylam-monium(3 mM)was similar to that of 4-aminopyridine and guanidine but not sopotent as the latters.Amphotericin B(1×10~(-5)),in the presence of 6 mM of Ca~(++),could increase the mean quantum content of the end plate potential but could notrestore the spike of the nerve terminal abolished by botulinum toxin.

A型肉毒杆菌毒素中毒大白鼠的膈神经膈肌标本,随着毒素作用的发展,细胞外记录的神经末梢动作电位与终板电位的平均振幅逐渐下降,直至完全消失,神经冲动不再能到达神经末梢。4-氨基吡啶、盐酸胍可恢复运动神经末梢传导冲动的能力,增加终板电位的量子含量,解除肉毒素产生的传递阻遏,恢复肌肉对间接刺激的收缩反应,而二性霉素对末梢电活动已消失的接头则无恢复作用。在上述资料基础上,对内毒素的作用机制和4-氨基吡啶等的抗肉毒效应进行了讨论。

 
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