助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   tumor necrosis α 的翻译结果: 查询用时:0.142秒
图标索引 在分类学科中查询
所有学科
肿瘤学
眼科与耳鼻咽喉科
呼吸系统疾病
更多类别查询

图标索引 历史查询
 

tumor necrosis α
相关语句
  肿瘤坏死因子α
     Objective The prognosis in critical patients valuably evaluated by acute physiological and chronic health evaluation Ⅱ(APACHE Ⅱ), nitric oxide (NO), tumor necrosis α (TNF-α).
     目的探讨急性生理和慢性健康评分Ⅱ(APACHE Ⅱ)和血清一氧化氮(NO)、肿瘤坏死因子α (TNF-α)相关联对危重患者预后的评估价值。
     To examine the change of serum tumor necrosis α(TNF α),interleukins 1β(IL 1β) and IL 6 in patients with congestive heart failure(CHF) and the effect of amiodarone on the prodution of the cytokines in vitro.
     检测充血性心力衰竭 (CHF)患者血清肿瘤坏死因子 α(TNF α)、白细胞介素 1β(IL 1β)和IL 6的变化 ,以及胺碘酮对培养的正常人及CHF患者单核细胞 (PBMC)分泌上述细胞因子的影响 ,以明确上述细胞因子在CHF发生过程中的作用 ,以及胺碘酮作用于CHF的免疫学机制。
短句来源
     Objective To investigate the clinical significance of tumor necrosis α (TNFα) and soluble interleukin 2 receptor (sIL-2R)in patients with acute leukemia (AL).
     目的 为探讨急性白血病 (AL)患者血清肿瘤坏死因子α(TNF -α)和可溶性白细胞介素 2受体 (SIL -2R)水平及其临床意义。
短句来源
  “tumor necrosis α”译为未确定词的双语例句
     [WT5”HZ]Results [WT5”BZ]Almost no inflammatory cytokines were detected in sham group, CLP produced a significant rise in serum TNF α (tumor necrosis α), IL 1, IL 6 (interleukin 1,6) in control group, IL 10 reduced the rise of inflammatory cytokines significantly.
     各组大鼠分别于术后 3小时及 9小时处死 ,取下腔静脉血检测炎性细胞因子水平。 结果 IL 10注入腹腔能使腹腔感染和早期脓毒血症炎性细胞因子水平明显下降 (P <0 .0 1)。
短句来源
  相似匹配句对
     ④Tumor necrosis and ulceration;
     ④肿瘤坏死和溃疡;
短句来源
     ③Tumor incompletely necrosis.
     ③肿瘤不完全坏死。
短句来源
     Tumor necrosis factor-α in gallbladder and gallstone
     胆囊结石及癌变过程中肿瘤坏死因子的变化
短句来源
     Tumor necrosis factor-α and diabetic retinopathy
     肿瘤坏死因子-α与糖尿病视网膜病变关系的研究进展
短句来源
     Psoriasis induced by tumor necrosis factor-α inhibitor
     肿瘤坏死因子-α抑制剂诱发银屑病
短句来源
查询“tumor necrosis α”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
没有找到相关例句


Objective To explore the effect of IL 10 on inflammatory inhibition of intraabdominal infection and sepsis in mice models. Methods Forty eight SD mice were divided equally into 4 groups namely Group 1 underwent laparotomy; Group 2 received cecal ligation plus punctures(CLP) in which normal saline was injected once every 3 hrs; Group 3 injected CLP and IL 10 intraperitoneally and Group 4 IL 10 was injected 1 hr before and every 3 hrs following CLP. The four groups were equally divided into two...

Objective To explore the effect of IL 10 on inflammatory inhibition of intraabdominal infection and sepsis in mice models. Methods Forty eight SD mice were divided equally into 4 groups namely Group 1 underwent laparotomy; Group 2 received cecal ligation plus punctures(CLP) in which normal saline was injected once every 3 hrs; Group 3 injected CLP and IL 10 intraperitoneally and Group 4 IL 10 was injected 1 hr before and every 3 hrs following CLP. The four groups were equally divided into two batches and sacrificed at 3 and 9hr after CLP. The blood cytokines were measured , and the liver, lung and intestine specimen were examined microscopically . Results The cytokines were not detected in group 1, while TNF α (tumor necrosis α), IL 1 and IL 6(interleukine 1,6) in the serum of the other 3 groups were increased by CLP. IL 10 caused the cytokines decreasing to some degree and attenuated the visceral inflammatory reaction. Conclusion IL 10 could inhibit the release of cytokine and suppress inflammatory reaction in mice models. It may act as an anti inflammation modulator.

目的 利用动物实验方法探讨白细胞介素 10 (interleukine 10 ,IL 10 )在腹腔感染和脓毒症时有无抑制炎症的作用。方法  48只SD大鼠随机分为 4组 ,每组 12只。第 1组单纯腹腔切开 ;第 2组腹腔切开后行盲肠结扎和穿刺 (cecalligationandpunctures,CLP) ;第 3组 ,术前 1h腹腔内注入IL 10 15 0 0 0U ,CLP术后再每 3h分别注入IL 10 15 0 0 0U ;第 4组 ,术前不用药 ,CLP术后每 3h分别每次注入IL 10 15 0 0 0U。各组大鼠分 2次在术后 3h和 9h处死 ,取下腔静脉血测炎性细胞因子水平及肝、肺、肠组织作病理切片检查。结果 CLP能使大鼠血中炎性细胞因子水平升高 ,IL 10注入腹腔降低了早期脓毒症时血中这些炎性细胞因子水平 ,并在一定程度上减轻了内脏组织的炎症改变。结论 IL 10对大鼠腹腔感染和早期脓毒症时的炎症有一定的抑制作用 ,提示它可能是有效的抗炎性物质之一。

HT6”,5”] [WT5”BZ] [WT5”HZ]Objective [WT5”BZ]To explore the effect of IL 10 on the inhibition of early proinflammatory cytokine release in intraabdominal infection and early sepsis. [WT5”HZ]Methods [WT5”BZ]Forty eight SD rats were randomized into 4 groups, 12 in each group, ①sham operation group, ②control group, ③prophylactic group, ④therapeutic group. Group 1 underwent laparotomy only, group 2 received laparotomy and cecal ligation plus punctures (CLP) with saline injected once every 3 hrs, group 3...

HT6”,5”] [WT5”BZ] [WT5”HZ]Objective [WT5”BZ]To explore the effect of IL 10 on the inhibition of early proinflammatory cytokine release in intraabdominal infection and early sepsis. [WT5”HZ]Methods [WT5”BZ]Forty eight SD rats were randomized into 4 groups, 12 in each group, ①sham operation group, ②control group, ③prophylactic group, ④therapeutic group. Group 1 underwent laparotomy only, group 2 received laparotomy and cecal ligation plus punctures (CLP) with saline injected once every 3 hrs, group 3 underwent CLP and IL 10 injection intraperitoneally 1 hr before surgery and once every 3 hrs following operation, group 4 received CLP and IL 10 injection once every 3 hrs after operation. At 3 and 9 hr points, rats were sacrificed and blood samples were taken for measurement of inflammatory cytokines. [WT5”HZ]Results [WT5”BZ]Almost no inflammatory cytokines were detected in sham group, CLP produced a significant rise in serum TNF α (tumor necrosis α), IL 1, IL 6 (interleukin 1,6) in control group, IL 10 reduced the rise of inflammatory cytokines significantly. [WT5”HZ]Conclusion [WT5”,6BZ]IL 10 could inhibit the early inflammatory cytokine release in rat model of sepsis. Suggesting it may attenuate the severity of inflammation. [HT6,5”] [WT5”HZ]

目的 探讨白细胞介素 10有无降低腹腔感染和早期脓毒血症炎性细胞因子的作用。方法  48只SD大鼠随机分为 4组 ,每组 12只。第 1组 :单纯腹腔切开 (shamoperation) ;第 2组 :腹腔切开后行盲肠结扎和穿刺 (cecalligationandpunctures ,CLP) ,腹腔注入生理盐水 2ml;第 3组 :术前 1小时腹腔内注入IL 10 15 0 0 0U ,CLP术后再每 3小时分别注入IL 10 15 0 0 0U ;第 4组 :术前不用药 ,CLP术后每 3小时分别注入IL 10 15 0 0 0U。各组大鼠分别于术后 3小时及 9小时处死 ,取下腔静脉血检测炎性细胞因子水平。结果 IL 10注入腹腔能使腹腔感染和早期脓毒血症炎性细胞因子水平明显下降 (P <0 .0 1)。结论 IL 10能降低腹腔感染和早期脓毒血症炎性细胞因子水平 ,对炎症有一定抑制作用

To examine the change of serum tumor necrosis α(TNF α),interleukins 1β(IL 1β) and IL 6 in patients with congestive heart failure(CHF) and the effect of amiodarone on the prodution of the cytokines in vitro.Vein blood of both healthy volunteers( n =20),and CHF patients( n =20) were obtained.Serum TNF α,IL 1β and IL 6 were measured by an Enzyme linked immunosorbent assay(ELISA).Peripheral blood mononuclear cell(PBMC) were obtained from both the control and the patients group.PBMC...

To examine the change of serum tumor necrosis α(TNF α),interleukins 1β(IL 1β) and IL 6 in patients with congestive heart failure(CHF) and the effect of amiodarone on the prodution of the cytokines in vitro.Vein blood of both healthy volunteers( n =20),and CHF patients( n =20) were obtained.Serum TNF α,IL 1β and IL 6 were measured by an Enzyme linked immunosorbent assay(ELISA).Peripheral blood mononuclear cell(PBMC) were obtained from both the control and the patients group.PBMC were cultured with 0,0.1,1,and 10 μmol/l of amiodarone in the presence of lipopolysaccharide.After 24 hours' incubation,TNF α,IL 1β and IL 6 were measured in the culture supernatants by an ELISA.Results:Serum TNF α,IL 1β and IL 6 were significantly higher than that of the control group( P< 0.001),and the higher the heart failure degree,the higher the levels of the cytokines( P< 0.001).TNF α positively correlate both with IL 1β( r=0.9684,P< 0.001) and IL 6( r=0.9768,P< 0.001).The production of TNF α,IL 1β and IL 6 were greatly inhibited by amiodarone in both groups in a concentration depended manner.Conclusion:①TNF α,IL 1β and IL 6,may play an important role in the pathogenesis of CHF;②The effect of improvement of heart function of amiodarone may be caused by the inhibition of the production of some certain cytokines.

检测充血性心力衰竭 (CHF)患者血清肿瘤坏死因子 α(TNF α)、白细胞介素 1β(IL 1β)和IL 6的变化 ,以及胺碘酮对培养的正常人及CHF患者单核细胞 (PBMC)分泌上述细胞因子的影响 ,以明确上述细胞因子在CHF发生过程中的作用 ,以及胺碘酮作用于CHF的免疫学机制。取 2 0例正常人和 2 0例Ⅲ~Ⅳ级的CHF患者静脉血 :①测其血清TNF α、IL 1β和IL 6含量 ;②离心取PBMC ,分别加入胺碘酮和脂多糖 (LPS)等 ,使胺碘酮的终浓度为 0 ,0 .1,1和10 μmol/ml进行培养 ,经 2 4h孵化后 ,取培养液上清 ,用酶联免疫吸附法 (ELISA)测培养上清中TNF α、IL 1β和IL 6。结果 :CHF患者血清TNF α、IL 1β和IL 6明显高于对照组 (P均 <0 .0 0 1) ,并随心衰程度的加重而增加 (P <0 .0 0 1)。TNF α与IL 1β和IL 6呈正相关 (r=0 .96 84,0 .9786 ;P均 <0 .0 0 1)。胺碘酮对两组PBMC分泌TNF α、IL 1β和IL 6均具有抑制作用 ,并呈剂量依赖性。结论 :①细胞因子T...

检测充血性心力衰竭 (CHF)患者血清肿瘤坏死因子 α(TNF α)、白细胞介素 1β(IL 1β)和IL 6的变化 ,以及胺碘酮对培养的正常人及CHF患者单核细胞 (PBMC)分泌上述细胞因子的影响 ,以明确上述细胞因子在CHF发生过程中的作用 ,以及胺碘酮作用于CHF的免疫学机制。取 2 0例正常人和 2 0例Ⅲ~Ⅳ级的CHF患者静脉血 :①测其血清TNF α、IL 1β和IL 6含量 ;②离心取PBMC ,分别加入胺碘酮和脂多糖 (LPS)等 ,使胺碘酮的终浓度为 0 ,0 .1,1和10 μmol/ml进行培养 ,经 2 4h孵化后 ,取培养液上清 ,用酶联免疫吸附法 (ELISA)测培养上清中TNF α、IL 1β和IL 6。结果 :CHF患者血清TNF α、IL 1β和IL 6明显高于对照组 (P均 <0 .0 0 1) ,并随心衰程度的加重而增加 (P <0 .0 0 1)。TNF α与IL 1β和IL 6呈正相关 (r=0 .96 84,0 .9786 ;P均 <0 .0 0 1)。胺碘酮对两组PBMC分泌TNF α、IL 1β和IL 6均具有抑制作用 ,并呈剂量依赖性。结论 :①细胞因子TNF α、IL 1β和IL 6参与CHF的发生 ;②胺碘酮可能通过抑制细胞因子的产生 ,对心力衰竭患者产生改善心功能作用。

 
<< 更多相关文摘    
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关tumor necrosis α的内容
在知识搜索中查有关tumor necrosis α的内容
在数字搜索中查有关tumor necrosis α的内容
在概念知识元中查有关tumor necrosis α的内容
在学术趋势中查有关tumor necrosis α的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社