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cold environment
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  寒冷环境
    Compulsive cold--water swimming and trauma bylimbbreaking were used to set acute stress models while cold environment of 4--8℃was used to set chronic stress model.
    应激方式分为急性应激(冷水游泳、断肢创伤)与慢性应激(寒冷环境刺激)。
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    Methods The main artery was isolated from Wistar rats and exposed to hypoxia and /or cold environment in vitro. LDH activity was measured using an automatic biochemical analyzer and NOS and SOD activities in blood vessels were analyzed respectively by Griess assay and adrenaline autoxidantion method.
    方法 分离Wistar大鼠主动脉并使其暴露于缺氧或 /和寒冷环境中 ,以全自动生化分析仪测定血管培养液中乳酸脱氢酶 (LDH)活性 ,以Griess化学法观测血管NOS活性 ,以肾上腺素自氧化法测定血管SOD活性。
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  “cold environment”译为未确定词的双语例句
    The results showed that: (1) The A Ⅱ levels inplasma were significantly increased in all three stress--model groups, reaching to900%, 390% and 134% of the control in the swimming group, the limbbroken groupand the cold environment group, respectively.
    结果表明:急性应激动物血中AⅡ剧烈增高,游泳组达对照值的900%,创伤组增至390%,慢性寒冷组增至134%;
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  cold environment
In many applications of piezoelectrically controlled smart structures, the functionality of the system has to be ensured even in an extremely hot and cold environment.
      
Sympathetic denervation of IBAT prevented the effect of the cold environment on GABA content and impaired that on GABA accumulation.
      
Polar organisms should have mechanisms to survive the extremely cold environment.
      
A theoretical study shows that when a thermally thick sample is allowed to radiate instantaneously into a cold environment, then the resulting transient surface temperature depends solely on its hemispherical total emissivity and effusivity.
      
Survival of young and old rats in a cold environment
      
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The brain leucine-enkephalin contents in rats under hot(40 ℃)and cold(0℃)environments,after amphetamine injection and bone fracture were measured byradio immunoassay.A decrease in leucine-enkephalin levels in hypothalamus andstriatum was found in rats under hot and cold environments and after amphetamineinjection,while an increase in leucine-enkephalin levels in striatum and thalamuswas found in bone fractured rats.These results indicate that the changes in enke-phalin levels can not be explained...

The brain leucine-enkephalin contents in rats under hot(40 ℃)and cold(0℃)environments,after amphetamine injection and bone fracture were measured byradio immunoassay.A decrease in leucine-enkephalin levels in hypothalamus andstriatum was found in rats under hot and cold environments and after amphetamineinjection,while an increase in leucine-enkephalin levels in striatum and thalamuswas found in bone fractured rats.These results indicate that the changes in enke-phalin levels can not be explained sorely on the basis of a nonspecific stress response.

本文用放射免疫分析法测定了大鼠在热及冷环境、苯丙胺兴奋和骨折等四种应激状态下脑内亮-脑啡肽(LEK)含量的变化。观察到热及冷环境和苯丙胺兴奋均使下丘脑和纹状体LEK 含量明显下降,而骨折使纹状体和丘脑 LEK 含量明显上升。这一结果说明,在四种不同的应激状态下,脑区 LEK 含量变化的趋向不尽相同。

The changes of content of angiotensin Ⅱ (A Ⅱ) in plasma, anterior hypothala-mus, medulla oblongata, myocardium, vasulature and adrenals during acute andchronic stress were studied in rats. Compulsive cold--water swimming and trauma bylimbbreaking were used to set acute stress models while cold environment of 4--8℃was used to set chronic stress model. The results showed that: (1) The A Ⅱ levels inplasma were significantly increased in all three stress--model groups, reaching to900%, 390% and 134% of the...

The changes of content of angiotensin Ⅱ (A Ⅱ) in plasma, anterior hypothala-mus, medulla oblongata, myocardium, vasulature and adrenals during acute andchronic stress were studied in rats. Compulsive cold--water swimming and trauma bylimbbreaking were used to set acute stress models while cold environment of 4--8℃was used to set chronic stress model. The results showed that: (1) The A Ⅱ levels inplasma were significantly increased in all three stress--model groups, reaching to900%, 390% and 134% of the control in the swimming group, the limbbroken groupand the cold environment group, respectively. It's clear that the level of angiotensionⅡ in the acute stress groupe were much higher than those in the chronic stress group.(2) The A Ⅱ leveis of the rat brain, myocardium and blood vessel were also increasedin the stress animals except the limbbroken group. Furthermore, the contents of an-giotensin Ⅱ of the chronic stress animals were significantly higher than those of theacute stress animals in the brain, myocardium and blood vessels, but not in theadrenals. (3)The adrenal A Ⅱ content was significantly higher than that of the controlin all stressed animals. (4) The plasma corticosterone was also significantly increasedover the control level in both the acute and chronic stress groups. These results suggestthat circulating angiotensin Ⅱ and tissue angiotensin Ⅱ may play a role during the de-velopment of acute and chronic stress, respectively.

本实验观察了三种应激情况下,大鼠血浆、下丘脑、延髓、心肌、血管及肾上腺组织血管紧张素Ⅱ(AⅡ)含量,以及血浆皮质酮含量的变化。应激方式分为急性应激(冷水游泳、断肢创伤)与慢性应激(寒冷环境刺激)。结果表明:急性应激动物血中AⅡ剧烈增高,游泳组达对照值的900%,创伤组增至390%,慢性寒冷组增至134%;而组织AⅡ除肾上腺外,则以慢性寒冷组增加最明显,游泳组次之,创伤组无明显变化。血浆皮质酮各组均显著增高,分别达对照组的380%,382%,250%。作者认为循环AⅡ与组织AⅡ含量的变化,可能分别在急性与慢性应激过程中发挥重要的作用,似应列入应激激素。

Objective: To study the changes of mitochondrial function in hepatocytes during acute cold exposure. Methods: Forty male SD rats were randomized into 4 groups. Group 1 served as the control group, and rats of group 2 and 3 were treated in the temperature of -10℃ for 15 and 60 min, respectively. Group 4 was treated in the temperature of -10℃ for 60 min and in the room temperature for 15 min. Immediately after treatment, livers were collected for examination of the fluidity of submitochondrial membrane in hepatocytes,...

Objective: To study the changes of mitochondrial function in hepatocytes during acute cold exposure. Methods: Forty male SD rats were randomized into 4 groups. Group 1 served as the control group, and rats of group 2 and 3 were treated in the temperature of -10℃ for 15 and 60 min, respectively. Group 4 was treated in the temperature of -10℃ for 60 min and in the room temperature for 15 min. Immediately after treatment, livers were collected for examination of the fluidity of submitochondrial membrane in hepatocytes, the activity of H + transporting ATPase and its sensitivity to oligomycin. Results: When rats were exposed in cold environment to 15 min, the fluidity of submitochondrial membrane, the activity of H + transporting ATPase and its sensitivity to oligomycin was lower than those of the control [(0.250±0.005) vs (0.257± 0.005 ), (7.67±1.00) vs (6.59±1.00) μmol/(min·mg), (98.83±1.44) vs (96.23±1.10)%]. When rats were exposed in cold environment to 60 min, there were significant reduction in the three indices. When rats recovered in room temperature for 15 min, the three indices still remained lower. Conclusions: The above mentioned findings indicated that acute cold exposure might lead to changes in the activity of H + transporting ATPase in hepatocellular mitochondria. The cause of these changes might relate to the reduction of membrane fluidity in hepatocellular mitochondria by cold stress.

目的:为进一步探讨急性冷暴过程中肝线粒体功能的变化.方法:观察雄性大鼠在室温(18℃),冷暴(-10℃)15min,60min和60min后室温15min时肝亚线粒体膜流动性,H+转运ATP酶活性及其对寡霉素敏感性的变化.结果:大鼠在冷暴15min时,膜流动性即开始降低(0.250±0.005对0.257±0.005),H+转运ATP酶活性及其对寡霉素的敏感性也明显下降[(7.67±1.00)对(6.59±1.00)μmol/(min·mg),(98.93±1.44)对(96.23±1.10)%],在冷暴60min时,三项指标下降更明显,室温恢复15min后,三项指标仍保持在较低水平.结论:冷应激可以导致肝线粒体H+转运ATP酶活性的降低,降低的原因可能是冷应激导致的肝线粒体内膜流动性下降,引起了H+转运ATP酶结构的变化,进而影响酶的活性.

 
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