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inflammation media
相关语句
  炎症介质
     3.The inflammation media TNF-α、IL-6 and IL-10 in circular blood were reduced by applying HVHF.
     3. HVHF 减少循环血中炎症介质(如TNF-α、IL-6 和IL-10)含量;
短句来源
     Conclusion:Type 2 diabetics had higher level inflammation media.
     结论 :2型糖尿病病人存在高水平炎症介质 ,即有炎症反应存在。
短句来源
     Changes in Serum Inflammation Media and Peripheral WBC in Type 2 Diabetes
     2型糖尿病血清炎症介质水平及血白细胞变化
短句来源
     Objective To study the relationship between microalbuminuria and inflammation media of C-reactive protein(CRP) and interleukin-6(IL-6) in type 2 diabetes mellitus,and to explore the clinical significance.
     目的研究T2DM尿微量白蛋白水平与炎症介质C-反应蛋白(CRP)和白介素-6(IL-6)之间的关系,并探讨其临床意义。
短句来源
     [Conclusion] The inducible activation of NF-κB starts ICAM-1 expression after ischemia reperfusion in rat liver transplantation; PNS can inhibit the transcription and expression of inflammation media and protect the liver graft.
     结论大鼠移植肝缺血再灌注时刺激NF-κB的活化,启动ICAM-1的表达参与肝脏缺血-再灌注损伤的发生过程,三七总皂甙能显著降低再灌注时供肝组织中炎症介质的转录表达,并有效改善供肝的再灌注损伤。
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  “inflammation media”译为未确定词的双语例句
     Objective Smoking effects on vascular endothelial dilatation function and on inflammation media such as interleukin-6 (IL-6), tumor necrosis factor-α(TNF-α) and C-reactive protein (CRP) were to be evaluated in healthy young adults and in passive smokers.
     目的探讨被动吸烟对健康青年人血管内皮舒张功能和白细胞介素6(IL-6)、肿瘤坏死因子-α(TNF-α)、C反应蛋白(CRP)等炎性介质的影响。
短句来源
     Objective:To investigate the changes in concentration of serum inflammation media (CRP?TNF-α?IL-6) and peripheral WBC counter in type 2 diabetes .
     目的 :研究 2型糖尿病患者炎症因子C反应蛋白 (CRP)、肿瘤坏死因子α(TNF α)、IL 6浓度及外周血白细胞水平变化。
短句来源
     Passive smoking effects on vascular endothelial dilatation function and inflammation media in healthy young people
     被动吸烟对血管内皮舒张功能及炎性介质的影响
短句来源
     lt participate in regulating the express of lots of cytokines and inflammation media,and is closely associated with inflammatory disease.
     它是有关炎症病理过程中许多炎性介质表达调控的关键性转录因子,在启动、放大和延续炎症反应过程中起中枢性调节作用。
短句来源
     Conclusion Inflammation media was involved the develop process of DN,hsCRP was a risk factor, his level suggessed lesion degree of DN.
     结论在DN的发展过程中有炎症因素参与,hsCRP为一危险因子,其水平可提示DN的损害程度。
短句来源
  相似匹配句对
     Media
     媒介
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     MEDIA
     媒体话题
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     Inflammation
     炎症(1)
短句来源
     Effects of acupuncture on inflammatory media in the patient of chronic inflammation
     针刺对慢性炎症疾病中炎症介质的影响
短句来源
     Phospholipase D and Inflammation
     磷脂酶D和炎症的关系
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Objective:To investigate the changes in concentration of serum inflammation media (CRP?TNF-α?IL-6) and peripheral WBC counter in type 2 diabetes .Methods:Selected 30 normal individuals,30 hypertension individuals and 45 type 2 diabetics to test their hs-CRP? TNF-α?IL-6 and WBC.Result:Inflammation media of hypertension group were higher than controls(CRP? TNF-α,P<0.05), and in type 2 diabetics they were significantly higher than hypertension individuals(P<0.01).Conclusion:Type 2 diabetics had higher...

Objective:To investigate the changes in concentration of serum inflammation media (CRP?TNF-α?IL-6) and peripheral WBC counter in type 2 diabetes .Methods:Selected 30 normal individuals,30 hypertension individuals and 45 type 2 diabetics to test their hs-CRP? TNF-α?IL-6 and WBC.Result:Inflammation media of hypertension group were higher than controls(CRP? TNF-α,P<0.05), and in type 2 diabetics they were significantly higher than hypertension individuals(P<0.01).Conclusion:Type 2 diabetics had higher level inflammation media.

目的 :研究 2型糖尿病患者炎症因子C反应蛋白 (CRP)、肿瘤坏死因子α(TNF α)、IL 6浓度及外周血白细胞水平变化。方法 :选择健康成人 30例 ,高血压病人 30例 ,2型糖尿病病人 4 5例 ,测定各组人群超敏C反应蛋白 (Hs CRP)、TNF α、IL 6、白细胞 (WBC)水平 ,观察炎症介质在各组人群中的差异。结果 :高血压组和 2型糖尿病组的血清炎症介质水平较基础值均有升高 ,而糖尿病组血清炎症介质水平上升更明显 ,与另两组差异有显著性。结论 :2型糖尿病病人存在高水平炎症介质 ,即有炎症反应存在。

Objective To investigate the effects of escharectomy during burn shock stage on sCD14 and complicated liver injury in patients with burns. Methods We compared the levels of lipopolysaccharide(LPS), sCD14, tumor necrosis factor-α(TNF-α), aspartate aminotransferase(AST) and alanine aminotransferase(ALT) in serum of patients with different therapy in two groups. Results All the markers mentioned above in patients with escharectomy were lower than those of patients with regular therapy, and positive correlation...

Objective To investigate the effects of escharectomy during burn shock stage on sCD14 and complicated liver injury in patients with burns. Methods We compared the levels of lipopolysaccharide(LPS), sCD14, tumor necrosis factor-α(TNF-α), aspartate aminotransferase(AST) and alanine aminotransferase(ALT) in serum of patients with different therapy in two groups. Results All the markers mentioned above in patients with escharectomy were lower than those of patients with regular therapy, and positive correlation was found between sCD14 and other markers. Conclusion Our data suggest that there is an important pathway among LPS, sCD14 and TNF-α, which might lead to liver injury in severely burned patients, and by which escharectomy control the release of main inflammation media and relieve the live injury to some extent.

目的 研究休克期切痂对严重烧伤后可溶性脂多糖受体CD14 (sCD14 )和肝损害的影响 ,并探索其可能的内在机制。方法 收集休克期切痂组和非休克期切痂组的大面积烧伤病人血标本 ,检测内毒素 (LPS)、sCD14和肿瘤坏死因子 (TNF-α)含量 ,同时测定血清谷丙转氨酶 (ALT)和谷草转氨酶 (AST)的水平。结果 休克期切痂组的上述指标均明显低于非休克期切痂组 (P <0 .0 5 ) ,sCD14和各项指标呈正相关。结论 严重烧伤后LPS、sCD14和TNF -α途径是并发肝功能损害的一条重要途径 ,休克期切痂可能通过该途径在一定程度上控制早期主要炎症介质的释放 ,降低烧伤后肝损害的程度。

[Objective]To investigate The effect of panax notoginseng saponins(PNS) on expression nuclear factor-κB(NF-κB) and ICAM-1 after ischemia reperfusion in liver transplantation. Rat orthotopic liver transplantation was performed according to Kamada's technique and divided randomly into three groups: NS control group (N), PNS preconditioning group (P) and sham-operated groups (S). The blood and liver samples were studied 2, 6 and 24 h after ischemia reperfusion. The expression level of NF-κB and ICAM-1 was elevated...

[Objective]To investigate The effect of panax notoginseng saponins(PNS) on expression nuclear factor-κB(NF-κB) and ICAM-1 after ischemia reperfusion in liver transplantation. Rat orthotopic liver transplantation was performed according to Kamada's technique and divided randomly into three groups: NS control group (N), PNS preconditioning group (P) and sham-operated groups (S). The blood and liver samples were studied 2, 6 and 24 h after ischemia reperfusion. The expression level of NF-κB and ICAM-1 was elevated by immunohistochemistry, the filtration of poly morphonulcear neutriphils (PMNs) was measured with MPO enzyme maker method. The activities of ALT and AST in blood were also detected. The activation of NF-κB in liver graft increased significantly 2 h after reperfusion and then decreased; After reperfusion 2 h, the expression level of ICAM-1 increased significantly, reached peak at 6 h and had a positive relationship to the filtration of PMNs and the injury of liver graft. But P group was lower significantly than N group (P <0.05). [Conclusion] The inducible activation of NF-κB starts ICAM-1 expression after ischemia reperfusion in rat liver transplantation; PNS can inhibit the transcription and expression of inflammation media and protect the liver graft.

目的研究三七总皂甙(PNS)对大鼠移植肝缺血再灌注损伤中核因子-κB、ICAM-1表达的影响。方法改良Kamada法建立同种大鼠原位肝脏移植模型,将动物模型随机分为3组:生理盐水对照组(N组)、三七总皂甙预处理组(P组)和假手术组(S组)。3组分别于供肝再灌注后2、6和24h时相点,用免疫组化检测NF-κB及ICAM-1的表达;用髓过氧化酶法(MPO)定量测定肝组织中中性粒细胞浸润,并抽血检测血清丙氨酸转氨酶(ALT)和天冬氨酸转氨酶(AST)水平。结果N组供肝再灌注后2hNF-κB的活性显著升高,之后逐渐下降;肝组织ICAM-1的表达在再灌注后2h开始增高,6h达到高峰,与中性粒细胞的浸润增加和肝损伤有相关性。但P组明显低于N组(P<0.05)。结论大鼠移植肝缺血再灌注时刺激NF-κB的活化,启动ICAM-1的表达参与肝脏缺血-再灌注损伤的发生过程,三七总皂甙能显著降低再灌注时供肝组织中炎症介质的转录表达,并有效改善供肝的再灌注损伤。

 
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