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   hippocampus 在 神经病学 分类中 的翻译结果: 查询用时:0.178秒
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hippocampus
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  海马
    Study of the Role of GJB1、GAD、GIRK1 in Self-acceleration and Self-protection Mechanisms in the Hippocampus of Temporal Lobe Epilepsy Rat
    GJB1、GAD、GIRK1在颞叶癫痫大鼠海马内源性促痫与抗痫机制中作用的研究
短句来源
    Role of L-Type Calcium Channels in Delayed Neuronal Death of Hippocampus after Transient Forebrain Ischemia
    L-型钙通道在脑缺血后海马神经元迟发性死亡中的作用
短句来源
    Expression of Activin βA in the Mouse Hippocampus after Seizures Induced by Pilocarpine and the Protective Effect of Recombinant Human Activin A
    癫痫发作小鼠模型海马活化素βA的表达及重组人活化素A的保护作用
短句来源
    Experimental Study of Effects and Related Mechanism of Infrasound on Rat Hippocampus
    次声对大鼠海马部分作用效应和相关机制实验研究
短句来源
    Study on the Seizure-induced Mitochondrion-linked Apoptotic Pathways of Neurons in Hippocampus
    癫痫发作致海马神经元凋亡的线粒体通路机制研究
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  海马组织
    Results The phosphorylation level of ERK1 in somatosensory cortex increased significantly (P<0.05) after ischemic preconditioning, while no significant changes in ERK2 and that of ERK1/2 in hippocampus.
    结果经脑IPC大鼠躯体感觉皮层内ERK1的磷酸化水平(激活程度)增高(P<0·05); 大脑躯体感觉皮层内ERK2及海马组织内ERK1/ERK2的磷酸化水平无明显变化。
短句来源
    No significant changes in ERK1/2 protein expression were found both in somatosensory cortex and hippocampus after ischemic preconditioning.
    大脑躯体感觉皮层和海马组织内ERK1/ERK2经脑IPC后蛋白表达量均无明显变化。
短句来源
    The changes of striatum,cerebral cortex and hippocampus Glu content on experimental Parkinson's disease
    实验性帕金森病纹状体、大脑皮质、海马组织Glu含量的变化
短句来源
    C-fos induction in hippocampus after neonatal hypoxia-ischemia
    缺氧缺血后海马组织c-Fos基因表达与细胞凋亡
短句来源
    Expression of Caspase-3 in the hippocampus of pentylenetetrazole-kindled rats
    Caspase-3在戊四氮点燃大鼠海马组织中的表达
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  海马区
    Results The expression of protein Bcl-2 was found in different regions of gerbil hippocampus 3d after HBOC. It was the highest in CA_3 region,and next in CA_4,CA_2,and CA_1 regions (P<0.01) in order.
    结果沙土鼠在发生短时氧惊厥后3d,氧惊厥组海马各区神经元Bcl-2蛋白表达阳性,CA_3区表达最强,其次为CA_4、CA_2、CA_1区,而正常对照组及压力对照组动物海马区未见阳性细胞。
短句来源
    In butylphthalide group, there was a significant expression up-regulation to BDNF, NGF, BDNF mRNA and NGF mRNA in the peripheral around infarction and cornu ammonis or hippocampus area (P<0.05). However in the infarction area, the expressions of BDNF, NGF, BDNF mRNA and NGF mRNA had no significantly statistical difference (P>0.05).
    丁基苯酞治疗组BDNF和NGF蛋白BDNF mRNA和NGF mRNA在梗死区周围和海马区表达均高于对照组,差异有统计学意义(P<0.05),在梗死区差异均无统计学意义(P>0.05)。
短句来源
    The results of MRS imaging showed that in the normal hippocampus region the peak of NAA was erectly high, and the peaks of Cho, Cr and MI were relatively low; MI/Cr radio of the left side was 0.33 ± 0.13 and the right side was 0.28 ± 0.14; the metabolism was normal.
    磁共振波谱成像显示,正常海马区N-乙酰天冬氨酸峰高耸,胆碱复合物、肌酸以及肌醇峰相对较低,肌醇/肌酸比值左侧为0.33±0.13,右侧0.28±0.14,代谢正常;
短句来源
    In the hippocampus region of non-demented Parkison disease patients, MI peak was obviously high, MI/Cr radio of the left side was 0.53 ± 0.30 and the right side was 0.39 ± 0.27, they were all higher than those in the controls (P < 0.01, left; P < 0.05, right).
    非痴呆性帕金森病组患者海马区肌醇峰明显增高,肌醇/肌酸比值左侧为0.53±0.30,右侧0.39±0.27,高于正常对照组(P左<0.01,P右<0.05)。
短句来源
    The expression of Nogo-A appeared peak in the cortical area and striatal area after reperfusion for 6 to 12 hours, which was significantly increased in hippocampus neurons.
    缺血再灌注6~12h皮质区及纹状体区Nogo-A表达达高峰,海马区表达明显增加。
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  “hippocampus”译为未确定词的双语例句
    Electrical Stimulation to the Hippocampus of Rats to Found Animal Models and Detection the Expression of Gst Gene with Gene Cmos Chip Technique in Intractable Epilepsy
    电刺激大鼠癫痫模型及基因芯片检测谷胱苷肽-巯-转移酶基因的表达
短句来源
    CHANGES OF GABA_A RECEPTOR γ_2 SUBUNIT IN HIPPOCAMPUS OF CHRONIC EPILEPTIC RATS──A STUDY OF IN SITU HIBRIDIZATION HISTOCHEMISTRY
    慢性癫痫大鼠海马内GABA_A受体γ_2亚单位的改变──原位杂交组织化学研究
短句来源
    Compared with that of 10.0 g/m~3 group,the percentages increased significantly only in 4hr group at hippocampus.
    但与10.0 g/m~3剂量组比较,仅4 h组有明显增加,差异有统计学意义(P<0.05)。
短句来源
    Objective To investigate the mechanism of melatonin (MT) in preventing L-Glu-induced seizures by studying its effect on glutamate (Glu), GluR2, γ-aminobutyric-acid (GABA) , and GABRA1 in rat hippocampus.
    目的探讨褪黑素(melatonin,MT)对谷氨酸(glutamate,Glu)致痫大鼠海马内Glu及GluR2、γ-氨基丁酸(γ-aminobutyric-acid,GABA)及其受体GABRA1水平的影响,进而研究褪黑素的抑痫作用机制。
短句来源
    Objective To investigate the significance of changes of expression of microtubule-associated protein-2 (MAP-2) in dentate gyrus of hippocampus in rat models of cerebral ischemia-reperfusion injury and the possible protective effect of hypothermia.
    目的:观察大鼠脑缺血再灌注损伤后海马齿状回微管相关蛋白(MAP-2)变化规律及其意义;
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  hippocampus
The rCBF of the right frontal lobe and hippocampus was also detected by the PerifluxPF model laser Doppler flowmetry, and the expressions of c-fos or c-jun or Bcl-2 and Bax were also measured by immune histochemistry S-P method accordingly.
      
Results showed that the rCBF of the right frontal lobe and hippocampus in the operation group was significantly lower than that in the false operation group (P >amp;lt; 0.05).
      
In conclusion, rCBF decrease can impair the learning and memory function in rats, which may be related to the increase of the expression ratio of c-fos or c-jun or Bcl-2 or Bax in the frontal cortex and hippocampus.
      
Proliferative Activity of Matrix Cells in the Lateral Ventricles and Granule Cells in the Dentate Gyrus of the Rat Hippocampus u
      
The stem and progenitor cells, as well as neuro- and gliogenesis, are most explored in the hippocampus and olfactory bulb.
      
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Glucose utilization in the cerebrum was studied after ligation of the right com-mon carotid artery for 1 to 3 hrs in gerbils weighing about 60 g. A total of 60animals was subjected to carotid ligation of which 15 developed neurological signsof brain injury (turning in circles, splaying or seizure). The onset of neurologicalsymptoms varied considerably among individual animals, but could be correlatedwith the absence of collateral circulation by latex tracing of the cerebral vessels,and by the reduction of glucose...

Glucose utilization in the cerebrum was studied after ligation of the right com-mon carotid artery for 1 to 3 hrs in gerbils weighing about 60 g. A total of 60animals was subjected to carotid ligation of which 15 developed neurological signsof brain injury (turning in circles, splaying or seizure). The onset of neurologicalsymptoms varied considerably among individual animals, but could be correlatedwith the absence of collateral circulation by latex tracing of the cerebral vessels,and by the reduction of glucose utilization in the lesioned hemisphere. Those gerbilswhich failed to develop symptoms were found to have collateral circulation and nodifference in glucose utilization between the lesioned and intact hemispheres. Theanimals were killed at 3 hrs, 24 hrs and 4 days postligaton. Each animal wasinjected intraperitoneally with 2-deoxy-D-glucose(1uC/10g body weight)45 minutesprior to sacrifice. Serial coronal sections were exposed to X-ray films for 7 daysand the autoradiograms were read with a digital densitometer with a 0.1min aperture.The following brain regions were examined: frontal, parietal, occipital corti-ces, dorsal hippocampus and caudate nucleus. Glucose utilization in various brainregions was inferred from the optical density (O. D.) on the X-ray flims, and theO. D. of the lesioned hemisphere was expressed as a percentage of that of the intact(control) hemisphere. At 3 hrs postligation, glucose utilization on the ischemichemisphere was reduced to less than 50% of the control, with the frontal cortexand caudate nucleus showing the greatest reduction. At 24 hrs postligation, glucoseutilization showed a small increase in all brain regions examined, and by day 4,it had returned to about 90% of control values in the parietal and occipital corti-ces and the hippocampus. The glucose utilization of the frontal cortex and caudate-nucleus however, still lagged much behind. These data indicate considerable varia-tion among different brain regions to ischemic insult and also the rate of recovery.These metabolic changes are being correlated with morphological investiga-tions.

本文摘要报告了结扎右侧颈总动脉后,导致同侧大脑半球缺血的实验动物的缺血侧大脑对放射性14碳2—D脱氧葡萄糖的利用情况。实验资料表明,大脑不同区域对缺血的反应性及以后的恢复情形有着明显的差别。

The concentration of glucocorticoid, mineralocorticoid,estrogen and progestin receptors has been determined in brain and in various brain tumors. The appearance of various intracranial neoplasms has been examined with computed tomography (CT) and related to the effect of steroid treatment and to receptor levels.The results can be summarized as follows: 1 . Glucocorticoid Receptor in Brain1 ) The glucocorticoid receptor proteins in cytosol from human and rat brain are similar both with regard to protein structure...

The concentration of glucocorticoid, mineralocorticoid,estrogen and progestin receptors has been determined in brain and in various brain tumors. The appearance of various intracranial neoplasms has been examined with computed tomography (CT) and related to the effect of steroid treatment and to receptor levels.The results can be summarized as follows: 1 . Glucocorticoid Receptor in Brain1 ) The glucocorticoid receptor proteins in cytosol from human and rat brain are similar both with regard to protein structure and hormone specificity. 2 ) The average concentration of glucocortic'oid receptor in various intracranial les-ions was as follows; cerebral metastases > meningiomas > acoustic neuromas > gliomas>peritumoral brain tissue>contused brain tissue. The clinical response to dexamethasone in brain edema associated with various intracranial lesions is to some extent parallel to the GR concentration in these lesions.3 ) The study on the relationship between CT findings and glucocorticoid receptorconcentrations in various intracranial tumors showed that some correlation existed between the glucocorticoid receptor level and CT contrast enhancement. This indicates that glucocorticoid receptor interaction is of importance in the regulation of the vascular permeability of tumors.4 ) The effect of dexamethasone treatment on brain edema was evaluated by CT in14 patients with various intracranial tumors.The degree of reduction of edema volume (cerebral metastases>gliomas>meningiomas)following steroid treatment paralleled the clinical response of dexamethasone. Ⅱ. Mineralocorticoid Receptor in Brain1 ) Rat kidney and rat hippocampus contain a mineralocorticoid receptor with similar properties in these two tissues which can be distinctly separated from the glucocorticoid receptor. 2 ) The order of tumor types with respect to average concentration of mineralocorticoid receptor was gliomas>neuromas>metastases >meningiomas. Glial cellshave a high concentration of intranuclear sodium, and it was found that gliomasalso have high levels of mineralocorticoid receptors. This indicates that the mineralocorticoid receptor may play a role in the cause and process of cytotoxic brain edema.Ⅲ.Estrogen and progestin Recentors in BrainFifteen of sixteen (94%) and nine of eleven (82%) meningiomas had detectable amounts of estrogen and progestin receptors, respectively. Except for one case of acoustic neuroma, other types of intracranial tumors did not contain detectable amounts of estrogen receptors.These results indicate that the sex steroid receptor profile of meningiomas is unique among intracranial neoplasms. The findings give further support to the view that selective endocrine therapy can be considered as an alternative treatment in case of meningioma,especially in poor-risk patients. In conclusion the study has extended the knowledge of steroid hormone receptors in brain tissue and neoplasms and indicates that glucocorticoid and mineralocorticoid receptors may play roles in the cause of and treatment of vasogenic edema and cytotoxic edema, respectively, whereas meningioma probably could be subjected to selective endocrine therapy.

甾体激素受体在脑内的生理意义与机理所知尚少,本文应用受体放射分析技术与现代生物化学分析技术(如等电聚焦电泳,超离心技术)对鼠脑与人脑内的糖皮质激素受体(GR)、盐皮质激素受体(MR)、雌激素受体(ER)与孕激素受体(PR)的化学特性、含量检测及其临床意义作了比较系统的研究。尤其侧重在人脑肿瘤中GR,MRER和PR与脑水肿的关系和对肿瘤治疗及预后方面所起的作用。 概括地说,人脑与脑肿瘤中的GR与血管源性脑水肿的治疗和转归有密切关联;根据CT观察,进一步证实血管源性脑水肿主要原因是病变区血管的通透性呈病理性增高。糖皮质激素由于能降低和改善血管通透性,对脑水肿起到积极的治疗作用。颅内肿瘤中GR的多少与治疗因肿瘤引起的血管源性脑水肿的效果成正比。人脑与脑肿瘤,尤其是胶质细胞瘤中的MR推论与缺血性脑水肿的形成与发展有密切联系。而脑膜瘤象乳癌一样,瘤组织中含有ER及PR的阳性率分别为94%与82%;说明脑膜瘤是一种与雌激素关系密切的肿瘤,为今后对这类肿瘤的内分泌治疗开辟了道路。

To observe the effect of ischemia on glucose utilization of the cerebrum, and to correlate the ultrastructural alterations with the metabolic changes mongobian gerbils with 60-80g body weight were selected as animal models. 47 and 50 animals for EM and 2-DG respectively. At 3 and 24hrs after the reflow of blood all the elements of the brain were moderate to severe edema, especially the processes of the astrocyte about the perivascular area. Mitochondriae were dilated. On 4 to 14 days postligation of the blood...

To observe the effect of ischemia on glucose utilization of the cerebrum, and to correlate the ultrastructural alterations with the metabolic changes mongobian gerbils with 60-80g body weight were selected as animal models. 47 and 50 animals for EM and 2-DG respectively. At 3 and 24hrs after the reflow of blood all the elements of the brain were moderate to severe edema, especially the processes of the astrocyte about the perivascular area. Mitochondriae were dilated. On 4 to 14 days postligation of the blood vessel, wide spread degeneration were evident. Many neurons were indifferent stages of chromatolysis, axonal reactions, presynaptic terminals and dendrites degenerations, Glial cells, such as astrocytes contained large amounts of glycogen and large numbers of gliofilaments were accumulated. Edema was limited to the perivascular part. Microglia were numerous. On the 21-28 days of survival period, recovery of morphologic integrity was indicated. The recovering cells contained clusters of lysosomes, small hissl bodies, and irregular outlines of nuclear and plasma membranes. Cerebral edema was greatly reduced. Although a small number of neurons exhibited advanced chromatolytic changes, an indication of "delayed cell death" 2-DG observation: At 3 and 24 hrs postligation, 2-DG uptake on the hemisphere of ischemia decreased to about 30% of the intact side. On 4 and 7 days of survival time glucose uptake of the ischemia hemisphere increased about 50-80%. But, the frontal cortex and caudate nucleus lagged consistently behind.On 14 and 21 days postligation, the frontal cortex, caudate nucleus and hippocampus reached to about 70% of control values in 2-DG uptake. The other brain regions recovered to about 80-90% of the control values. The long-term investigations are underway to monitor the recovery process.

结扎蒙古沙土鼠颈总动脉,造成脑缺血模型,松解后,观察存活期14天、21天、28天的实验动物的大脑形态学和代谢功能的改变。结果显示大脑皮质、尾状核、海马和豆状核壳等部位对缺血损害最为敏感。存活4天~21天的动物对2-DG的摄取明显回升并缓慢恢复,血流恢复后28天的脑组织,其神经元结构出现不同阶段的恢复。

 
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