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cadmium treatment
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  镉处理
    The mechanism responsible for the morphological change of the endoplasmic reticulum after cadmium treatment and the possible toxicologic effect were discussed.
    对镉处理后内质网形态改变的机理,及其可能的细胞效应,进行了讨论。
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  镉处理
    The mechanism responsible for the morphological change of the endoplasmic reticulum after cadmium treatment and the possible toxicologic effect were discussed.
    对镉处理后内质网形态改变的机理,及其可能的细胞效应,进行了讨论。
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  “cadmium treatment”译为未确定词的双语例句
    Conclusion After the cadmium treatment the detoxification mechanism of the freshwater crab's hepatopancreas cells lost,the structural and functional integrality of the mostly organelles,such as the nucleous,the mitochondria and the rough endoplasmic reticulum,were destroyed,so that the normal physiological function of hepatopancreas cells was affected.
    结论 镉使长江华溪蟹肝胰腺细胞的解毒功能丧失 ,破坏了肝胰腺细胞的细胞核、线粒体、粗面内质网等主要细胞器的结构与功能的完整性 ,影响了肝胰腺细胞的正常生理机能。
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  cadmium treatment
Out of 380 protein spots detected on 2-D gel using Coomassie staining, 11 spots were changed significantly following cadmium treatment.
      
Alterations of the protein expression patterns following 10 μM cadmium treatment were analyzed on 2-dimensional gels.
      
As in higher organisms, two major forms of metallothionein appeared to be present in the liver following cadmium treatment.
      
Induction of metallothionein and methallothionein mRNA in rainbow-trout liver following cadmium treatment
      
Apoptotic chromatin changes in chromatin structure after cadmium treatment were seen as large extensive disruptions, holes in the nuclear membrane and stickiness of incompletely folded chromosomes.
      
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With transmission electron microscopy,the endoplasmic reticulum of the parathyroid chief cells in normal and cadmium-treated golden hamsters was observed. After 1 month treatment of cadmium, there were dilated and whorl-shaped rough endoplasmic reticulum in the chief cells. After 3 and 6 months treatment of cadmium, there were concentric membranous body and paired endoplasmic reticulum in the chief cells. The mechanism responsible for the morphological change of the endoplasmic reticulum after cadmium treatment...

With transmission electron microscopy,the endoplasmic reticulum of the parathyroid chief cells in normal and cadmium-treated golden hamsters was observed. After 1 month treatment of cadmium, there were dilated and whorl-shaped rough endoplasmic reticulum in the chief cells. After 3 and 6 months treatment of cadmium, there were concentric membranous body and paired endoplasmic reticulum in the chief cells. The mechanism responsible for the morphological change of the endoplasmic reticulum after cadmium treatment and the possible toxicologic effect were discussed.

用透射电镜对正常和镉处理后金黄地鼠甲状旁腺主细胞的内质网,进行了形态学观察。结果表明:镉处理1个月后,在甲状旁腺主细胞内,见有扩张的和呈环状排列的粗面内质网;镉处理3和6个月后,甲状旁腺主细胞内见有膜性同心层状小体和成对内质网。对镉处理后内质网形态改变的机理,及其可能的细胞效应,进行了讨论。

Objective After an experimental exposure to low dosage of Cd 2+(39*!μmol/L) for 30 days,the changes of Cd 2+ on ultrastructure of the hepatopancreas cells of the freshwater crab (Sinopotamon yangtsekiense♂)at three different times were studied. Methods Sixty freshwater crabs(♂)were divided into two groups:control(n=30) and test(n=30)groups.The test(n=30) group was exposed to low dosage of Cd 2+(39*!μmol/L)while the control crabs were in water without cadmium.During the 5*!d,15*!d,30*!d,the ultrastructure...

Objective After an experimental exposure to low dosage of Cd 2+(39*!μmol/L) for 30 days,the changes of Cd 2+ on ultrastructure of the hepatopancreas cells of the freshwater crab (Sinopotamon yangtsekiense♂)at three different times were studied. Methods Sixty freshwater crabs(♂)were divided into two groups:control(n=30) and test(n=30)groups.The test(n=30) group was exposed to low dosage of Cd 2+(39*!μmol/L)while the control crabs were in water without cadmium.During the 5*!d,15*!d,30*!d,the ultrastructure of the hepatopancreas cells was observed by using transmission electro-microscopy(TEM). Results In the course of the experiment,the nuclear membrane changed from spreading all over the place to disintegrated,and the penetration of the mitochondrial outer membrane and inner membrane was changed.The mitochondria showed swelling and vacuolating.Mitochondrial cristae partly disintegrated,then fully disintegrated.Besides,the Cd 2+ exposure affected the gross form and the structure of the rough endoplasmic reticulum(RER).At first,the RER became the different sized vesicles,then,the ribosomes on the little vesicles began to fall,and finally,the RER turned to the concentric round lamellar structures.But the smooth endoplasmic reticulum(SER)increased markedly.In addition,the numbers and types of lysosome were increased in the mean time and some lysosomes became void vesicles,the autophagosomes or the myeloid bodies(MD).The microvilli showed partly falling and vacuolating.Conclusion After the cadmium treatment the detoxification mechanism of the freshwater crab's hepatopancreas cells lost,the structural and functional integrality of the mostly organelles,such as the nucleous,the mitochondria and the rough endoplasmic reticulum,were destroyed,so that the normal physiological function of hepatopancreas cells was affected.

目的 观察低浓度镉 (39μmol L)处理长江华溪蟹 (Sinopotamonyangtsekiense)后 ,其肝胰腺细胞超微结构的变化。 方法 实验组 30只雄蟹培养在浓度为 39μmol L的氯化镉 (CdCl2 )溶液中 ;对照组 30只雄蟹培养在已放置 2d的自来水中 ,分别在镉处理后 5d、15d、30d三个不同时间段活体解剖雄蟹 ,电镜下观察肝胰腺细胞超微结构。 结果 镉对肝胰腺细胞内的主要细胞器影响表现为 :核膜从弥散状到最后解体 ;线粒体膜通透性改变 ,肿胀变形 ,嵴减少或消失 ,最后完全空泡化 ;粗面内质网先裂解为大小不同的小泡 ,尔后小泡上的核糖体开始脱落 ,直至变成同心圆状板层结构 ;出现大量滑面内质网 ;溶酶体数量随镉处理时间的延长而增多 ,形成空泡、自噬体或髓样体 ;微绒毛出现脱落和部分空泡化。 结论 镉使长江华溪蟹肝胰腺细胞的解毒功能丧失 ,破坏了肝胰腺细胞的细胞核、线粒体、粗面内质网等主要细胞器的结构与功能的完整性 ,影响了肝胰腺细胞的正常生理机能。

 
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