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diabetic family
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  糖尿病家族
     ④Influencing risk factors of diabetes were mainly: Diabetic family history, body-mass index, waist-to-hip ratio, high SBP, high triacylglycerol and physical work, their OR values were 3.049,1.456,1.798,2.895,2.525 and 1.671 respectively.
     ④影响糖尿病患病的危险因素主要有:糖尿病家族史、体质量指数、腰臀比、高收缩压、高三酰甘油,体力活动,其OR值分别为3.049,1.456,1.798,2.895,2.525,1.671。
短句来源
     Rates of diabetes in patients with diabetic family history was 23.13 % and 25.63 % in IGT, which were signigicantly higher than those in patients without diabetic family history, which were 7.02 % and 13.28 % respectively.
     有糖尿病家族史者其糖尿病患病率为23.13%,糖耐量低减为25.63%,均明显高于无家族史者7.02%与13.28%。
短句来源
     Multivariable analysis showed that the age, gender, degree of education, diabetic family history, history of diabetes fall ill, manual labor strength, mental work strength and systolic pressure values were the correlative factors, their odd ratios (ORs) were 1.239, 0.990, 1.223, 2.752, 1.897, 1.373, 0.916, 0.884 and 0.996, respectively.
     多因素分析显示:性别、年龄、文化程度、糖尿病家族史、糖尿病患病史、体力劳动强度、脑力劳动强度、收缩压与糖尿病知识知晓率密切相关,其比值比分别为1.239,0.990,1.223,2.752,1.897,1.373,0.916,0.884和0.996。
短句来源
     ④ The abnormality of glycometabolism was aggravated with the increased detection rates of hypertension, diabetic family history and past increase of blood glucose (χ2=17.968 3, 19.954 0, 12.635 5, P < 0.01).
     ④糖代谢异常情况随着高血压、糖尿病家族史、曾有血糖升高检出率的增高而加重(χ2=17.9683,19.9540,12.6355,P<0.01),少体力活动和高血脂人群以空腹血糖受损最多见。
短句来源
     Conclusion Diabetic family historty,family history of cancer,BMI>26,WHR≥0.9 and thigh circumference>60? cm were associated with higher risk for the development of GDM.
     结论有糖尿病家族史、父母患恶性肿瘤、孕前BMI>26、WHR≥0.9、首诊股围>60 cm是GDM发生的危险因素。
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  dm家族
     But the rate of diabetic family history and insulin sensitivity index (ISI) were lower in AS group than in non-AS group(P<0.05).
     有DM家族史的比率及胰岛素敏感性指数 (ISI)低于非AS组 (P <0 .0 5 )。
短句来源
     34% patients of DM had diabetic family history.
     34%DM患者有明确的DM家族史。
短句来源
     (9) patient history (diabetic family history,hypertension history or hepatopathy;0-no, 1-yes);
     (9)病史(DM家族史、高血压病史和肝病史,0-无,1-有);
短句来源
     Diabetic family history was lower than Japanese ( P <0.01) but drinkers were more than Japanese ( P <0.01).
     DM家族史中国人组低于日本人组 (P<0 .0 1)。
短句来源
     Methods:Seven adolescence women with PCOS whose father or mother was diabetic(test group) and 28 cases of adolescence women with PCOS without diabetic family history(control group) were investigated.
     方法:选择父亲或母亲被确诊患DM的7例青春期PCOS患者为实验组,以无DM家族史的28例青春期PCOS患者为对照组。
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  糖尿病家系
     A COMBINATION OF MITOCHONDRIAL tRNA leu(UUR) AND ND1 GENE MUTATIONS IN A DIABETIC FAMILY
     线粒体DNA tRNA~(leu(UUR))和 ND1双重突变──糖尿病家系报道
短句来源
     Changes of insulin secretion function and blood lipid in type 2 diabetic family
     2型糖尿病家系胰岛功能和血脂代谢的改变
短句来源
     High-risk factors in the diabetic high risk group of type 2 diabetic family of Sinkiang Uygur nationality
     新疆维吾尔族2型糖尿病家系中糖尿病高危人群高危因素的研究
短句来源
     Changes of the First Phase Insulin Secretion in First Degree Relatives of Type 2 Diabetic Family with Normal Glucose Tolerance
     2型糖尿病家系非糖尿病一级亲属第一时相胰岛素的变化
短句来源
     Objective:To investigate the changes of first phase insulin secretion in first degree relatives of type 2 diabetic family with normal glucose tolerance.
     目的:探讨2型糖尿病家系非糖尿病一级亲属在糖耐量正常时胰岛素第一时相分泌情况。
短句来源
  “diabetic family”译为未确定词的双语例句
     Investigation of A-Behaviour about a Diabetic Family
     一糖尿病家族的A型行为调查
短句来源
     Methods: From April, 2003 to June, 2005, outpatients or inpatients with type 2 diabetes mellitus (T2DM) who had family history, were chosen as probands from the first and second affiliated hospital of Dalian Medical University, Central Hospital, Friendship Hospital, 305 people were received from 91 type 2 diabetic family (probands 91 people, and first-degree relatives 214 people), and 76 spouses of probands were recruited at the same time, which totally was 381 people.
     方法:2003年4月~2005年6月期间,选取大连医科大学附属一院、二院、中心医院、友谊医院门诊及住院有家族史的2型糖尿病患者为先证者,收集了91个家系,共305例(先证者91例,一级亲属214例),同时获得先证者的配偶76例,总计调查381例。
短句来源
     Objective:To investigate the degree of insulin resistance(IR) in adolescence women with polycystic ovary syndrome(PCOS) from diabetic family and the effects of metformin treatment on them.
     目的:探讨糖尿病(DM)家族青春期多囊卵巢综合征(PCOS)胰岛素抵抗(IR)的程度及应用二甲双胍治疗的效果。
短句来源
     Conclusion It is suggested that leptin didn't play an important role in the development of DM for pedigree members who had diabetic family history and increased serum leptin might be one of the risk factors related to type 2 DM.
     结论 :leptin在中国河南汉族DM家系人群中可能不是主要致病原因 ,血清leptin增高可能是 2型DM的致病原因之一。
短句来源
     METHODS: From June 1998 to November 2000, 183 pedigrees, who were the diabetic outpatients and inpatients with diabetic family history in the First Affiliated Hospital, Chongqing University of Medical Sciences, or diabetic patients diabetic family history participated in the health education in Chongqing Diabetes Center, were involved in the study, their family members of 3 generations were investigated with the uniform familial inventory.
     方法:以1998-06/2000-11在重庆医科大学附属第一医院门诊、住院及参加重庆糖尿病中心健康教育的患者中有家族史的糖尿病患者为先证者,采用统一的家系调查表进行3代成员的调查。
短句来源
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  diabetic family
Mutation of HNF-1β gene causing the substitution S36F was found in two subjects of an early onset diabetic family.
      
Both patients had no diabetic family history, but had been treated with prednisone for 4 and 8 months, receiving total doses of 6.6 and 10.8 g, respectively.
      
Sera of 406 individuals, 174 Type 1 (insulin-dependent) diabetic patients, 125 non-diabetic family members and 107 unrelated control subjects, were screened for the presence of antibodies against glycated albumin.
      
Five year evolution of insulin secretion in five non-diabetic family members who have inherited the V393I mutation.
      
Clinical and biological data of diabetic and non-diabetic family members who carry the mutation are summarized in Table II.
      


Objective:To understand the relationship between the charecteristics,the complications of NIDDM and the mutant allele of β3AR gene(Arg64) in Chinese.Methods:The presence of the allele Arg64 in 152 Chinese with NIDDM was investigated.The polymorphism of β3AR gene were determined by PCRRFLP.Results:There were 7 Arg64 homozygotes、38 heterozygotes and 107 Trp homozygotes among 152 cases of NIDDM,that is,the frequency of Arg64 allele was 017(52/304).The onset ages of diabetes were 443,469,496 years old respectively in 3 groups.6 patients had diabetic foot gangrene in 45 cases with at least one allele of Arg64(133%),only 2 cases had diabetic foot gangrene in 107 Trp64homozygotes(19%).The odd ratio was 808, Fisher test P<001.The onset age of NIDDM in patients with at least one alleles of Arg64 was significantly younger than those without it(P<005 after adjusted sex,age,diabetic family history and the highest body mass index); the highest BMI has no difference between these 2 groups.This results showed the frequency of Arg64 in Chinese with NIDDM was significantly higher than that of Finns,lower than that of Pima Indians and not different from that of Japanese with NIDDM. Conclussion: Arg64 allele of β3AR gene is associated with the earlier onset of NIDDM in Chinese and it could be one of the susceptible genes of diabetic gangrene. To reduce this severe complications, it could be valuable to offer prophylactic measures to the diabetics especially to those who have the Arg64 allele. ...

Objective:To understand the relationship between the charecteristics,the complications of NIDDM and the mutant allele of β3AR gene(Arg64) in Chinese.Methods:The presence of the allele Arg64 in 152 Chinese with NIDDM was investigated.The polymorphism of β3AR gene were determined by PCRRFLP.Results:There were 7 Arg64 homozygotes、38 heterozygotes and 107 Trp homozygotes among 152 cases of NIDDM,that is,the frequency of Arg64 allele was 017(52/304).The onset ages of diabetes were 443,469,496 years old respectively in 3 groups.6 patients had diabetic foot gangrene in 45 cases with at least one allele of Arg64(133%),only 2 cases had diabetic foot gangrene in 107 Trp64homozygotes(19%).The odd ratio was 808, Fisher test P<001.The onset age of NIDDM in patients with at least one alleles of Arg64 was significantly younger than those without it(P<005 after adjusted sex,age,diabetic family history and the highest body mass index); the highest BMI has no difference between these 2 groups.This results showed the frequency of Arg64 in Chinese with NIDDM was significantly higher than that of Finns,lower than that of Pima Indians and not different from that of Japanese with NIDDM. Conclussion: Arg64 allele of β3AR gene is associated with the earlier onset of NIDDM in Chinese and it could be one of the susceptible genes of diabetic gangrene. To reduce this severe complications, it could be valuable to offer prophylactic measures to the diabetics especially to those who have the Arg64 allele. 

目的:探讨β3肾上腺素能受体(β3AR)基因64位点的色氨酸(Trp)密码子被精氨酸(Arg)置换与胰岛素抵抗及代谢综合征的关系。方法:利用PCR扩增包含64位密码子的该基因片段,内切酶BstNI消化电泳确定64位点基因型。结果:152名中国NIDDM患者的Arg等位基因频率为017,高于芬兰人,低于Pima印第安人,与日本人NIDDM人群基因频率相近。45例携带Arg等位基因和107例Trp纯合子的NIDDM患者中分别有6例和2例发生糖尿病足坏疽,前者糖尿病足坏疽危险性8倍于后者(P<001)。β3AR基因64位密码子Trp被Arg置换的患者糖尿病发病年龄有年轻化的趋势,Arg纯合子、杂合子及Trp纯合子的糖尿病发病年龄分别是443岁、465岁和496岁。结论:NIDDM发病年龄与β3AR基因的Arg64替代有关,并在世界上首次提出β3ARArg64等位基因可能是糖尿病足坏疽的风险基因

Objective: To determine the influence of diabetic family history on diagnostic age of NIDDM (non-insulin-dependent diabetic mellitus) patients, and the different effects of parental hereditary backgrounds on the NIDDM incidence among their offspring. Methods: 199 NIDDM patients with family history (HZ+) and 273 individuals without family history (HZ-) were used to compare the difference in age of diagnosis, and the parental influence on the prevalence of NIDDM in offspring. Results:...

Objective: To determine the influence of diabetic family history on diagnostic age of NIDDM (non-insulin-dependent diabetic mellitus) patients, and the different effects of parental hereditary backgrounds on the NIDDM incidence among their offspring. Methods: 199 NIDDM patients with family history (HZ+) and 273 individuals without family history (HZ-) were used to compare the difference in age of diagnosis, and the parental influence on the prevalence of NIDDM in offspring. Results: (1) The age of diagnosis of NIDDM patients with family history was much younger than that of those without family history (46.2 vs 52.2 yr, P<0.01). (2) There were more maternal diabetic patients than paternal ones (36.7% vs 19.1%). (3) Parental diabetic history played the most important role in the onset of NIDDM. There were no interactions between different hereditary backgrounds. (4) The order of hereditary effect was as follows: mother>father> second degree relatives > silbings. Conclusion: (1) The NIDDM patients' age of diagnosis with hereditary influence is much younger than that of those without family history. (2) Maternal hereditary effect is greater than the paternal one. Each hereditary factor acts independently.

目的:探讨糖尿病家族史对非胰岛素依赖型糖尿病(NIDDM)诊断年龄的影响以及患有糖尿病的父母对子女NIDDM发病影响的差别。方法:对199例有家族史及273例无家族史的NIDDM患者诊断年龄、父母亲NIDDM发病情况等临床特点进行统计学处理。结果:(1)有糖尿病家族史的NIDDM患者诊断年龄明显早于无家族史者(46.2岁vs52.2岁,P<0.01);(2)家族中母亲患糖尿病者多于父亲(36.7%vs19.1%,P<0.01);(3)父母糖尿病史是NIDDM的重要危险因素,它们的作用相互独立且相加;(4)各种遗传背景对NIDDM发病的影响顺序为:母亲>父亲>二级亲属>同胞。结论:(1)有遗传倾向的NIDDM患者的诊断年龄提前;(2)母亲对NIDDM遗传的影响大于父亲,父、母亲的作用相互独立。

Objective Mitochondrial tRNA Leu(UUR) gene nt3243 A→G point mutation is one of the diabetic genes. This study aims to know the prevalence of the mutation in type 1 diabetes mellitus and whether serum autoimmune insulitis marker coexits with the mutation. Methods The mutation had been screened from 116 unrelated patients with type 1 diabetes mellitus by ApaI digestion of product of polymerase chain reaction amplification. Glutamic acid decarboxylase (GAD) antibody was detected in 82 cases. Results 1 case...

Objective Mitochondrial tRNA Leu(UUR) gene nt3243 A→G point mutation is one of the diabetic genes. This study aims to know the prevalence of the mutation in type 1 diabetes mellitus and whether serum autoimmune insulitis marker coexits with the mutation. Methods The mutation had been screened from 116 unrelated patients with type 1 diabetes mellitus by ApaI digestion of product of polymerase chain reaction amplification. Glutamic acid decarboxylase (GAD) antibody was detected in 82 cases. Results 1 case had the mutation (0.86%) but negative GAD antibody. None of the 48 patients with positive GAD antibody and none of the 41 patients with slowly progressive type 1 diabetes mellitus had the mutation. The diabetic family members demonstrated different onset types but shared the characteristic of insulin defficiency. Conclusion Our limited data suggested that diabetes mellitus caused by mitochondrial tRNA Leu(UUR) nt3243 A→G mutation was a distinct subtype and the phenotype of insulin dependent (or requiring) might not relate with autoimmune pancreatic isletis.

目的 线粒体t R N A Leu( U U R) 基因3243 位点 A→ G 突变是糖尿病的致病基因之一。本研究是为了了解该基因的突变在中国1 型糖尿病患者中的情况以及与自身免疫导致1 型糖尿病有无关联。方法 对116 例随机收集的1 型糖尿病患者用聚合酶链反应限制性内切酶消化作该点突变的筛选;82 例患者同时进行了谷氨酸脱羧酶( G A D) 抗体的测定。结果 发现1 例该点突变(0 .86 % ) ,48 例 G A D 抗体阳性(58 .5 % ) 。线粒体t R N A Leu( U U R) 基因突变携带者 G A D 抗体阴性,其家系成员糖尿病有不同的发病方式,但均表现为胰岛素缺乏。结论 线粒体t R N A Leu( U U R) 基因异常所致糖尿病表现为胰岛素缺乏可能与自身免疫胰岛炎无关,而是一种独特的糖尿病亚型。

 
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