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cerebral ischemic reperfusion damage
相关语句
  脑缺血再灌注损伤
     Conclusion:EAA,oxygen free radicals are closely related with the mechanism of cerebral ischemic reperfusion damage.
     结论 :EAA、氧自由基参于脑缺血再灌注损伤机制
短句来源
     In GM1treated group, the changes significantly improved (P <0. 01 ). The results suggested that EAA excitotoxin is closely related with the cerebral ischemic reperfusion damage.
     表明脑缺血再灌注损伤与EAA的过度释放和氧自由基的产生密切相关,而GM1对大鼠脑缺血具有保护作用(P<0.01)。
短句来源
     Results Taurine had the effect of anti free radical on cerebral ischemic reperfusion damage, decreasing ET and protecting brain cellfunction. There was abvious difference in comparing with non meclication group.
     结果 四项检测方法从不同方面证实牛磺酸对脑缺血再灌注损伤有抗自由基,稳定细胞膜,降低内皮素,保护脑组织细胞等抗脑缺血再灌注损伤的脑保护作用,与未用牛磺酸组有显著差别。
短句来源
     Objective: To investigate the neuroprojective effects of GM1 on rat global cerebral ischemic reperfusion damage.
     目的 :探讨单唾液酸四己糖神经节苷酯 (GM1)在脑缺血再灌注损伤中的神经保护作用及其机制。
短句来源
     The topamax protective effect on cerebral ischemic reperfusion damage in gerbils
     妥泰对沙土鼠脑缺血再灌注损伤保护作用的实验研究
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  脑缺血再灌流损伤
     Conclusion BDE may have protect effect on cerebral ischemic reperfusion damage by decreasing Nos activity.
     结论 BDE抑制神经元NOS活性 ,对脑缺血再灌流损伤具有脑保护作用。
短句来源
  “cerebral ischemic reperfusion damage”译为未确定词的双语例句
     Objective :To ovserve the excitatory amino acid(EAA), oxygen free radicals change on cerebral ischemic reperfusion ,and study the mechanism of cerebral ischemic reperfusion damage.
     目的 :观察脑缺血再灌注过程中兴奋性氨基酸 (Excitatory amino acid,EAA)、氧自由基的变化 ,研究探索脑缺血再灌注损伤的机制。
短句来源
     The GM_1 effect to abnormal of calcium balance,oxygen free radicals metabolism and pathologic changes on rat global cerebral ischemic reperfusion damage
     GM_1对大鼠全脑缺血再灌注中钙平衡紊乱、氧自由基代谢异常以及病理损伤的影响
短句来源
     Effects of GM1 on lactate and glucose on rat global cerebral ischemic reperfusion damage
     GM1对大鼠脑缺血再灌注损伤的乳酸及葡萄糖含量的影响
短句来源
     Conclusion GM 1 could improve abnormal of calcium balance,oxygen free radicals metabolism in cerebral ischemic reperfusion damage,and reduced its pathologic injury.
     结论 GM1 能改善脑缺血再灌注中钙平衡紊乱和氧自由基代谢异常,减轻脑缺血再灌注病理损伤。
短句来源
     Experimental observation on relation of excitatory amino acid,oxygen free radicals and cerebral ischemic reperfusion damage
     兴奋性氨基酸、氧自由基与缺血再灌注脑损伤关系的实验观察
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Objective: To study GM1 protective effect of cerebral ischemia reperfusion damage.Methods: Contents of hippocampus excitatory amino acid (EAA), Calmodulin (CaM ), Malondiadehyde(MDA)were measured respectively at sham group, ischemia 30 min reperfusion 60 min group treated byGM1 (10mg/kg, IP) and by normal Sodium (NS) Results: The hippocampus EAA contents weresignificantly lower in ischemia reperfusion group compared with sham group (P<0. 01), and CaM, MDA,Contents increased significantly than sham group's, the...

Objective: To study GM1 protective effect of cerebral ischemia reperfusion damage.Methods: Contents of hippocampus excitatory amino acid (EAA), Calmodulin (CaM ), Malondiadehyde(MDA)were measured respectively at sham group, ischemia 30 min reperfusion 60 min group treated byGM1 (10mg/kg, IP) and by normal Sodium (NS) Results: The hippocampus EAA contents weresignificantly lower in ischemia reperfusion group compared with sham group (P<0. 01), and CaM, MDA,Contents increased significantly than sham group's, the changes of EAA, CaM, MDA Contents wereimproved in GM1-treated group. Conclusion: GM1 Can mediate EAA excitatoxin, calcium abnormalbalance and excess production of oxygen free radicals in cerebral ischemic reperfusion damage, act on brainPretective effect.

目的:观察单唾液酸四已糖神经节苷胸(GM1)对脑缺血再灌注损伤的保护作用。方法:采用四血管闭塞(4VO)全脑缺血再灌注动物模型,测定假手术组,缺血30min再灌注60minGM1;处理组(10mg/kg,缺血5min腹腔注射),缺血30min再灌注60min生理盐水(NS)处理组鼠脑海马组织兴奋性氨基酸(ExcitatoryAminoAcid,EAA),钙调素(Calmodulin,CaM),丙二醛(Malondiadehyde,MDA)的含量。结果:脑缺血再灌注组海马组织EAA含量显著性降低(P〈0.01),而CaM、MDA则显著性升高(P<0.01),GM1处理组海马EAA、CaM、MDA含量同假术术组比较没有显著性差异(P>0.05)。结论:GM1能阻抑脑缺血再灌注损伤中EAA的过度释放和/或重摄取障碍,细胞内外钙平衡紊乱,氧自由基产生过多等病理生理过程,发挥脑保护作用。

Objective To study the effect of taurine on rats with cerebral ischemic reperfusion damage. Methods Taurine group(G 3)、non medication group(G 2) and control group(G 1) were established respectively. The endothelin (ET)、 Lipo superoxide in the brain tissue and the ratio of wet dry brain tissue in weight were measured in each group,and cerebral histological optical microscope was applied correspondingly. Results Taurine had the effect of anti free radical on cerebral ischemic reperfusion...

Objective To study the effect of taurine on rats with cerebral ischemic reperfusion damage. Methods Taurine group(G 3)、non medication group(G 2) and control group(G 1) were established respectively. The endothelin (ET)、 Lipo superoxide in the brain tissue and the ratio of wet dry brain tissue in weight were measured in each group,and cerebral histological optical microscope was applied correspondingly. Results Taurine had the effect of anti free radical on cerebral ischemic reperfusion damage, decreasing ET and protecting brain cellfunction. There was abvious difference in comparing with non meclication group. Conclusions Our study of taurine could supply laboratory evidence for treating cerebral ischemic disease.

目的 为了探讨牛磺酸对脑缺血再灌注损伤的作用。 方法 建立沙土鼠脑缺血再灌注模型进行实验。设立实验组G3( 注射牛磺酸组) 、实验组G2( 不用任何药物组) 及对照组G1 。处死动物后,即时分别检测各组血浆内皮素、脑组织脂质过氧化物、脑组织湿/ 干重比值、脑组织学光镜检查等。 结果 四项检测方法从不同方面证实牛磺酸对脑缺血再灌注损伤有抗自由基,稳定细胞膜,降低内皮素,保护脑组织细胞等抗脑缺血再灌注损伤的脑保护作用,与未用牛磺酸组有显著差别。 结论 为临床应用牛磺酸治疗脑缺血缺氧及缺血性脑血管病提供了实验依据

Objective To study the effect of GM 1 to the abnormal of calcium balance,oxygen free radicals metabolism and pathologic changes on rat global cerebral ischemic reperfusion damage.Methods With rat global cerebral ischemia and reperfusion model (4VO), we measured the rat hippocampic mitochondria calcium (MCa),calmodulin (CaM),malondiadehyde (MDA) contents and the pathologic changes of hippocampic CA 1 district in the sham group,cerebral ischemia for 30 min and reperfusion for...

Objective To study the effect of GM 1 to the abnormal of calcium balance,oxygen free radicals metabolism and pathologic changes on rat global cerebral ischemic reperfusion damage.Methods With rat global cerebral ischemia and reperfusion model (4VO), we measured the rat hippocampic mitochondria calcium (MCa),calmodulin (CaM),malondiadehyde (MDA) contents and the pathologic changes of hippocampic CA 1 district in the sham group,cerebral ischemia for 30 min and reperfusion for 60 min group treated by normal sodium (NS) and GM 1 group,cerebral ischemia for 30 min and reperfusion for 60 min.Results The hippocampic MCa,CaM,MDA contents were significantly higher in NS group (P< 0.01 ),the hippocampic CA 1 district neuron showed severe ischemic damage.The changes of biochemistry and pathology had more remarkable improvement in GM 1 group than NS one.Conclusion GM 1 could improve abnormal of calcium balance,oxygen free radicals metabolism in cerebral ischemic reperfusion damage,and reduced its pathologic injury.

目的 研究单唾液酸四已糖神经节苷脂(GM1)对大鼠全脑缺血再灌注中钙平衡紊乱、氧自由基代谢异常以及病理损伤的影响。方法 运用大鼠全脑缺血再灌注模型(4VO),观察假手术组、脑缺血30 分钟再灌注60 分钟生理盐水(NS)处理组(NS组)和缺血30 分钟再灌注60 分钟GM1 处理组(GM1 组)的脑海马组织线粒体钙(MCa)、钙调素(CaM)、丙二醛(MDA)及海马CA1 区病理改变。结果 NS组海马组织MCa、CaM、MDA含量显著升高(P< 0.01),海马CA1 区神经元呈较严重缺血损伤性改变,而GM1 组较NS组生化和病理变化明显改善。结论 GM1 能改善脑缺血再灌注中钙平衡紊乱和氧自由基代谢异常,减轻脑缺血再灌注病理损伤。

 
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