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pancreatic regeneration
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     Plasma CCK concentration of each group is Px(-) group (28.22±2.45) pg/mL, Px+CCK-RA group (53.62±4.49) pg/mL and Px group (62.85± 4.33)pg/mL by radioimmunoassay, the results discovered significant difference (F=171.8, P <0.01). [Conclusions] Endogenous CCK is one of the essential factors for pancreatic regeneration;
     Px(-)组中血清CCK的水平为(28.22±2.45)pg/mL,Px+CCK-RA组为(53.62±4.49)pg/mL,Px组为(62.85±4.33)pg/mL,3组比较差异有显著性(F=171.8,P<0.01)。
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  相似匹配句对
     Regeneration and Reorganization
     再生与重组
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     Liver Regeneration
     肝再生
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     PANCREATIC SCHISTOSOMIASIS
     胰血吸虫病
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     PANCREATIC ENCEPHALOPATHY
     胰性脑病——附9例报告
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     The Studying for Genesis and Development as well as Regeneration of Pancreatic Islet in Related Genes
     胰岛发生、发育和再生及相关基因的研究
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  pancreatic regeneration
In the present study we have investigated the alteration of GABA receptors in the brain stem of rats during pancreatic regeneration.
      
Decreased GABAA Receptor Function in the Brain Stem during Pancreatic Regeneration in Rats
      
Thus, from our results it is suggested that the central α1-adrenergic receptors have a functional role in the pancreatic regeneration mediated through the sympathetic pathway.
      
Competition analysis in cerebral cortex and brain stem showed a shift in affinity during pancreatic regeneration.
      
The purpose of this study was to investigate the role of brain α1-adrenergic receptor binding in the rat model of pancreatic regeneration using 60-70% pancreatectomy.
      
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[Objective] To study effect of cholecystokinin-A recaptor antagonist on experimental pancreas regeneration model. [Methods] 24 rats were divided into 3 groups, as sham operation Px(-), using CCK-RA antagonist with pancreastectomy Px+CCK-RA, pancreastectomy without using CCK-RA antagonist Px by random method. After 5 days, we detected the samples wet weight of parabiliary segment from the duodenum and cell proliferation index by microscope observation using bromodeoxyuridine (BrdU) labeling,cell proliferation...

[Objective] To study effect of cholecystokinin-A recaptor antagonist on experimental pancreas regeneration model. [Methods] 24 rats were divided into 3 groups, as sham operation Px(-), using CCK-RA antagonist with pancreastectomy Px+CCK-RA, pancreastectomy without using CCK-RA antagonist Px by random method. After 5 days, we detected the samples wet weight of parabiliary segment from the duodenum and cell proliferation index by microscope observation using bromodeoxyuridine (BrdU) labeling,cell proliferation index(LI) and the plasma CCK concentrations were measured. [Results] 24 rats were detected, wet weight of parabiliary segment from the duodenum in Px(-) group is (86.98±3.51) mg, Px+CCK-RA group (92.46±6.67) mg, Px group (210.72±7.39) mg, compared each group have revealed significant difference (F=772.9, P <0.01). In Px(-) group, the LI in acinar cells was approximately 1%, but in Px and Px+CCK-RA group are about 10% and 5%; Plasma CCK concentration of each group is Px(-) group (28.22±2.45) pg/mL, Px+CCK-RA group (53.62±4.49) pg/mL and Px group (62.85± 4.33)pg/mL by radioimmunoassay, the results discovered significant difference (F=171.8, P <0.01). [Conclusions] Endogenous CCK is one of the essential factors for pancreatic regeneration; secretion of them is promoted by partial pancreatectomy. But its effect are inhibited by CCK-RA, the molecular mechanism by CCK secretion is promoted remains to be elucidated.

目的研究胆囊收缩素A受体拮抗剂(CCK-RA)在实验性胰腺再生模型中的作用。方法全部24只大鼠,随机分成3组,Wistar大鼠分为假手术Px(-)、手术加使用CCK-RA受体完全拮抗剂Px+CCK-RA和手术但不使用CCK-RA受体完全拮抗剂组Px,5d后测定它们环胆管下端部分湿重、免疫组化观察不同部位的细胞摄取溴脱氧尿苷以及放射免疫方法血清CCK的浓度。结果Px(-)组环胆管下端部分湿重为(86.98±3.51)mg,Px+CCK-RA组为(92.46±6.67)mg,Px组为(210.72±7.39)mg,3组比较差异有显著性(F=772.9,P<0.01);免疫组化观察不同部位的细胞摄取溴脱氧尿苷的永生化指数Px(-)组为1%,Px组为10%,Px+CCK-RA组为5%;Px(-)组中血清CCK的水平为(28.22±2.45)pg/mL,Px+CCK-RA组为(53.62±4.49)pg/mL,Px组为(62.85±4.33)pg/mL,3组比较差异有显著性(F=171.8,P<0.01)。结论内源性CCK是胰腺再生的基本要素之一,胰腺部分切除可以促进其分泌,这些作用可以被CCK-RA遏制,有关分子机制...

目的研究胆囊收缩素A受体拮抗剂(CCK-RA)在实验性胰腺再生模型中的作用。方法全部24只大鼠,随机分成3组,Wistar大鼠分为假手术Px(-)、手术加使用CCK-RA受体完全拮抗剂Px+CCK-RA和手术但不使用CCK-RA受体完全拮抗剂组Px,5d后测定它们环胆管下端部分湿重、免疫组化观察不同部位的细胞摄取溴脱氧尿苷以及放射免疫方法血清CCK的浓度。结果Px(-)组环胆管下端部分湿重为(86.98±3.51)mg,Px+CCK-RA组为(92.46±6.67)mg,Px组为(210.72±7.39)mg,3组比较差异有显著性(F=772.9,P<0.01);免疫组化观察不同部位的细胞摄取溴脱氧尿苷的永生化指数Px(-)组为1%,Px组为10%,Px+CCK-RA组为5%;Px(-)组中血清CCK的水平为(28.22±2.45)pg/mL,Px+CCK-RA组为(53.62±4.49)pg/mL,Px组为(62.85±4.33)pg/mL,3组比较差异有显著性(F=171.8,P<0.01)。结论内源性CCK是胰腺再生的基本要素之一,胰腺部分切除可以促进其分泌,这些作用可以被CCK-RA遏制,有关分子机制有待进一步阐明。

 
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