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acute myocardial ischemic injury
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  “acute myocardial ischemic injury”译为未确定词的双语例句
     Aim To investigate the role and mechanism of neutrophil infiltralion in acute myocardial ischemic injury. The hydroxyurea was used for to observe the cardioprotective effect.
     目的 探讨急性心肌缺血时,中性粒细胞(PMN)浸润在缺血性心肌损伤中的作用及其作用机制,并针对其发生机制,给予药物(羟基脲)干预,观察羟基脲对缺血心肌的保护效应。
短句来源
     Conclusion:The acute myocardial ischemic injury and sudden death of heart might have relationship with apoptosis of cardic myocytes.
     结论:急性心肌缺血性损伤与心源性猝死和心肌细胞凋亡有关。
短句来源
     CGRP May Take Part in the Effect of Electroacupuncture in Resisting Acute Myocardial Ischemic Injury
     降钙素基因相关肽参与电针抗急性心肌缺血损伤的实验研究
短句来源
     These results indicate that electric stimulation of LC region is benefi-cial to the recovery of experimental acute myocardial ischemic injury,the possibleunderlying is discussed.
     蓝斑核埋藏电极组的恢复时间为44.5±9.97分,与蓝斑核区电刺激组相比,其差异均非常显著(P<0.01)。 这些结果说明蓝斑核区的刺激对大鼠急性心肌缺血性模型的恢复有促进作用。
短句来源
     By histochemical means, we observed the effects of reperfusion on succinate dehydrogenase(SDH)activity after acute myocardial ischemic injury in rabbits.
     本文以组织化学方法探讨家兔心肌缺血不同时间后再灌注对琥珀酸脱氢酶(SDH)活性的影响。
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     Mitochondrion and myocardial ischemic-reperfusion injury
     线粒体与心肌缺血-再灌注损伤
短句来源
     Aim:To study the effects of captopril on acute myocardial ischemic reperfusion injury.
     目的:在兔急性心肌缺血再灌注损伤模型上观察开博通对再灌注损伤心肌的影响。
短句来源
     The protective effect of captopril on ischemic reperfused injury in acute myocardial infarction patients
     卡托普利对急性心肌梗塞患者缺血再灌注损伤的保护作用
短句来源
     EFFECT OF ISCHEMIC POSTCONDITIONING ON MYOCARDIAL BIOCHEMICAL INDEXES AFTER ACUTE ISCHEMIC INJURY
     缺血后处理对急性缺血再灌注心肌生化指标的影响
短句来源
     The Protection of Borneol in Acute Myocardial Injury in Rats
     冰片对大鼠急性心肌缺血损伤的保护作用
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  acute myocardial ischemic injury
It was shown that the reconstructed myocardial resistance and water content correlate dynamically with the process of the development of acute myocardial ischemic injury.
      


The effect due to implanted electrode and electric stimulation at the regionof locus ceruleus nucleus(LC)on acute myocardial ischemic injuries was studiedin 20 rats.The acute myocardial ischemic model was produced by intravenousinjection of pituitrin,ECG limb lead T_Ⅱ,S-T_Ⅱ and heart rate were monitored.It was found that the recovery period of ischemic ECG was 67±12.7 min in thecontrol group which was shortened to 44.5±10.0 min in the group with mereimplantation of the electrode.When LC was stimulated the recovery...

The effect due to implanted electrode and electric stimulation at the regionof locus ceruleus nucleus(LC)on acute myocardial ischemic injuries was studiedin 20 rats.The acute myocardial ischemic model was produced by intravenousinjection of pituitrin,ECG limb lead T_Ⅱ,S-T_Ⅱ and heart rate were monitored.It was found that the recovery period of ischemic ECG was 67±12.7 min in thecontrol group which was shortened to 44.5±10.0 min in the group with mereimplantation of the electrode.When LC was stimulated the recovery time wasshortened to 18.8±8.3 min after electric stimulation(3V,square pulses 40Hzfor 60s).These results indicate that electric stimulation of LC region is benefi-cial to the recovery of experimental acute myocardial ischemic injury,the possibleunderlying is discussed.

本实验在大鼠尾静脉注射垂体后叶素造成急性心肌缺血性损伤,以心电图肢体导联 S-T_Ⅱ段和 T_Ⅱ以及心率为指标,观察了电刺激蓝斑核区对大鼠急性心肌缺血性损伤的影响。实验结果表明蓝斑核区电刺激组的缺血性心电图恢复时间与对照组相比显著提前,前者为18.8±8.31分,后者为67±12.74分(P<0.01)。脑非蓝斑核区电刺激组的缺血性心电图恢复时间为69.2±6.8分。蓝斑核埋藏电极组的恢复时间为44.5±9.97分,与蓝斑核区电刺激组相比,其差异均非常显著(P<0.01)。这些结果说明蓝斑核区的刺激对大鼠急性心肌缺血性模型的恢复有促进作用。

By histochemical means, we observed the effects of reperfusion on succinate dehydrogenase(SDH)activity after acute myocardial ischemic injury in rabbits. Results showed that SDH activity was normal when the reperfusion was instituted after myocardial ischemia for 15 minutes. However, if the reperfusion was carried out after myocardial ischemia lasted over 45 minutes, the SDH activity was significantly lowered. It suggested that the development of reperfusion injury be closely related...

By histochemical means, we observed the effects of reperfusion on succinate dehydrogenase(SDH)activity after acute myocardial ischemic injury in rabbits. Results showed that SDH activity was normal when the reperfusion was instituted after myocardial ischemia for 15 minutes. However, if the reperfusion was carried out after myocardial ischemia lasted over 45 minutes, the SDH activity was significantly lowered. It suggested that the development of reperfusion injury be closely related to the duration of myocardial ischemia.

本文以组织化学方法探讨家兔心肌缺血不同时间后再灌注对琥珀酸脱氢酶(SDH)活性的影响。结果表明,冠脉结扎15分钟后再灌注,SDH活性接近正常,冠脉结扎45分钟后再灌注,SDH活性明显降低。这说明了再灌注损伤的出现与缺血的时间长短有关。

Objective: To study the expression of some apoptosis related genes in AMI and the influences of aloperine and propranolol. The relationship between apoptosis related genes and AMI were also investigated. Methods: AMI rat model was induced by ligation of the anterior branch of the left coronary artery. All animals were divided into four groups: sham operation group, occlusion group, aloperine group (20 mg·kg -1 ) and propranolol group (2 mg·kg -1 ). ECG lead Ⅱ was recorded. The level of serum creatine...

Objective: To study the expression of some apoptosis related genes in AMI and the influences of aloperine and propranolol. The relationship between apoptosis related genes and AMI were also investigated. Methods: AMI rat model was induced by ligation of the anterior branch of the left coronary artery. All animals were divided into four groups: sham operation group, occlusion group, aloperine group (20 mg·kg -1 ) and propranolol group (2 mg·kg -1 ). ECG lead Ⅱ was recorded. The level of serum creatine kinase (CK) activity, myocardial malondialdehyde (MDA) content and myocardial superoxide dismutase (SOD) activity were also determined. The myocardial expression of apoptosis related genes: Fas, CPP32, Bcl 2 and Bax were detected with RT PCR. Statistical significance in multiple comparisons was determined by One Way ANOVA. Results: Coronary occlusion of the rats led to significent ischemic changes of ECG, and increased the level of serum CK activity, myocardial MDA content and SOD activity. The myocardial expression of Fas, CPP32 and Bcl 2 were increased by coronary occlusion. But had no significance was detected in Bax expression. Aloperine and propranolol attenuated the ischemic changes of ECG, reduced the level of serum CK activity, myocardial MDA content and SOD activity. Both drugs also reduced the myocardial expression of Fas, CPP32 and Bcl 2, but no significance with Bax expression. Conclusion: The acute myocardial ischemic injury induced by ligation of the anterior branch of the left coronary artery may have relationship with apoptosis related genes: Fas, CPP32 and Bcl 2 family.

目的:研究大鼠冠状动脉结扎时心肌组织中凋亡相关基因的表达状况与急性心肌缺血的关系。方法:结扎大鼠左冠状动脉前降支建立急性心肌缺血模型。描记Ⅱ导心电图;检测血清肌酸激酶(creatine kinase,CK) 活性、心肌组织丙二醛(malondialdehyde, MDA)含量和超氧化物歧化酶(superoxide dismutase,SOD) 活性;RTPCR 法检测凋亡相关基因Fas、CPP32 、Bcl2 和Bax 在心肌组织的相对表达量。观察伪结扎组、结扎组、苦豆碱(aloperine,Alo)给药组(20 mg·kg- 1)、普萘洛尔(propranolol,Prop)给药组(2 mg·kg-1) 对上述各指标的影响。采用单因素方差分析(One Way ANOVA) 对结果进行统计处理。结果:结扎大鼠左冠状动脉前降支可导致心电图明显的缺血性改变及血清CK 活性显著升高;心肌组织的MDA 含量和SOD 活性显著增加;缺血心肌组织中Fas、CPP32 和Bcl2基因的表达显著增加( P<0 .001),Bax 变化不明显( P< 0.05) 。Alo 和Prop 能明显缓解心电图的缺血性改变并降低血...

目的:研究大鼠冠状动脉结扎时心肌组织中凋亡相关基因的表达状况与急性心肌缺血的关系。方法:结扎大鼠左冠状动脉前降支建立急性心肌缺血模型。描记Ⅱ导心电图;检测血清肌酸激酶(creatine kinase,CK) 活性、心肌组织丙二醛(malondialdehyde, MDA)含量和超氧化物歧化酶(superoxide dismutase,SOD) 活性;RTPCR 法检测凋亡相关基因Fas、CPP32 、Bcl2 和Bax 在心肌组织的相对表达量。观察伪结扎组、结扎组、苦豆碱(aloperine,Alo)给药组(20 mg·kg- 1)、普萘洛尔(propranolol,Prop)给药组(2 mg·kg-1) 对上述各指标的影响。采用单因素方差分析(One Way ANOVA) 对结果进行统计处理。结果:结扎大鼠左冠状动脉前降支可导致心电图明显的缺血性改变及血清CK 活性显著升高;心肌组织的MDA 含量和SOD 活性显著增加;缺血心肌组织中Fas、CPP32 和Bcl2基因的表达显著增加( P<0 .001),Bax 变化不明显( P< 0.05) 。Alo 和Prop 能明显缓解心电图的缺血性改变并降低血清CK 活性;减少心肌组织的

 
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