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cirrhosis from hepatitis
相关语句
  肝炎后肝硬化
     Study on the expression of HBV DNA in the liver tissue of posthepatic cirrhosis from hepatitis virus B by means of gene chips
     乙型肝炎后肝硬化患者肝组织中HBcAg、HBV DNA表达及基因芯片检测
短句来源
     Methods 68 cases with alcoholic cirrhosis were retrospectively analyzed,with cirrhosis from hepatitis B as control.
     方法以同期住院的乙型肝炎后肝硬化为对照,对68例酒精性肝硬化患者临床资料进行回顾性分析。
短句来源
     Conclusions: Decoction of Intenerate of liver and spleen has statistical significance in improving the patients of cirrhosis from hepatitis clinical symptoms and can improve liver functions, repair liver damage and thus enhance the patients' living quality.
     结论:加用软肝缩脾汤治疗肝炎后肝硬化,对于改善患者的临床症状,改善肝功能,修复肝损伤,提高患者的生活质量优于单纯使用西药治疗。
短句来源
     Clinical Observation of "Blood-Activating and Liver-Softening Decoction" in Treating Cirrhosis from Hepatitis
     活血软肝汤治疗肝炎后肝硬化临床观察
短句来源
     Conclusion HBcAg and HBV DNA in the liver tissues of the patients with posthepatic cirrhosis from hepatitis virus B showed a high expression rate. This is beliveed to be related with the active liver lesion. The difference between the rates of positive results of HBV DNA determined by gene chip and by in situ molecule hybridization was not significant( P >0 05).
     结论 乙型肝炎后肝硬化患者肝组织中有较高的HBV复制率 ,HBcAg及HBVDNA高表达与肝组织病变活动有关 ,基因芯片对肝组织中HBVDNA检出率与原位分子杂交结果无显著差异 ,P >0 0 5。
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  “cirrhosis from hepatitis”译为未确定词的双语例句
     METHODS: SP immunohistochemical tech- nique was performed to detect the expression of PCNA and Ki-67 in 58 liver specimens from patients with cirrhosis from hepatitis B (n = 24) and hepatitis C (n = 22), and chronic hepatitis (n = 12), and the labeling index (LI) of the positive cells was also determined.
     方法:选择58例(乙肝肝硬化24例、丙肝肝硬化22例、慢性肝炎12例)肝脏标本,做H-E染色观察肝组织的病理改变; 利用天狼红染色切片检测肝组织内胶原纤维的面密度;
短句来源
  相似匹配句对
     Bacterial infection in hepatitis B cirrhosis
     乙型肝炎肝硬化的细菌感染
短句来源
     Clinical Analysis on Post-hepatitis Cirrhosis
     肝炎后肝硬化115例临床分析
短句来源
     Hepatitis B Virus
     乙型肝炎病毒
短句来源
     Clinical Observation of Lamivudine for Decompensated Cirrhosis Resulting from Chronic Hepatitis B
     拉米夫定治疗乙型肝炎肝硬化失代偿期的临床观察
短句来源
     Ischaemia hepatitis
     缺血性肝炎
短句来源
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  cirrhosis from hepatitis
Patients with renal disease may have cirrhosis from hepatitis viruses, nephrotic syndrome, or both.
      


Objective To study the preparation of the gene chips for cases of diagnosed hepatitis virus.HBVDNA and HBcAg in the liver tissues of 99 patients with posthepatic cirrhosis were determined by gene chips,immunocytochemistry and in situ molecule hybridization technique.Then respective comparisons were made for the advantages and disadvantages of the diagnostic methods.Methods The gene chip was prepared by the HBV DNA microarrays with spotting...

Objective To study the preparation of the gene chips for cases of diagnosed hepatitis virus.HBVDNA and HBcAg in the liver tissues of 99 patients with posthepatic cirrhosis were determined by gene chips,immunocytochemistry and in situ molecule hybridization technique.Then respective comparisons were made for the advantages and disadvantages of the diagnostic methods.Methods The gene chip was prepared by the HBV DNA microarrays with spotting hybridization (PCR) of target genes put on the special lattice glass.To study the expression of HBV DNA in the liver tissues of the 99 patients with posthepatic cirrhosis from hepatitis virus B with gene chips,immunocytochemistry and molecule hybridization technique.Results There were 67 samples with HBcAg possitive and 53 samples with HBV DNA positive diagnosed by immunocytochemistry and in situ molecule hybridization technique.46 samples having positive results were determined by gene chips.Among them,32 samples were normal when tested by the three methods.The lesions of the liver tissues were active while the HBcAg and HBV DNA showed high expression in the liver tissues.Conclusion HBcAg and HBV DNA in the liver tissues of the patients with posthepatic cirrhosis from hepatitis virus B showed a high expression rate.This is beliveed to be related with the active liver lesion.The difference between the rates of positive results of HBV DNA determined by gene chip and by in situ molecule hybridization was not significant( P >0 05).

目的 用免疫组织化学法、原位分子杂交法、肝炎基因诊断芯片检测 99例乙型肝炎后肝硬化患者的肝组织 ,观察HBcAg和HBVDNA在肝组织中的表达 ,比较各种方法优缺点。方法 将PCR扩增的HBVDNA探针用点样仪点于玻片介质上 ,处理后制成基因芯片 ,收集肝炎后肝硬化组织标本 99份 ,分别用原位分子杂交法 ,基因芯片检测HBVDNA ,免疫组织化学法检测HBcAg。 结果 HBcAg阳性 67例 (67.7% ) ,原位分子杂交HBVDNA阳性 53例 (53 .5 % ) ,基因芯片检测阳性46例 (46 .5 % )。 32例三种方法检测HBcAg及HBVDNA均阴性。HBcAg颗粒和HBVDNA颗粒在肝组织中呈浆膜型高表达分布 ,其肝组织病变是活动的。结论 乙型肝炎后肝硬化患者肝组织中有较高的HBV复制率 ,HBcAg及HBVDNA高表达与肝组织病变活动有关 ,基因芯片对肝组织中HBVDNA检出率与原位分子杂交结果无显著差异 ,P >0 0 5。

AIM: To investigate the expression and significances of proliferating cell nuclear antigen (PCNA) and Ki-67 antigen in liver tissues of cirrhotic patients. METHODS: SP immunohistochemical tech- nique was performed to detect the expression of PCNA and Ki-67 in 58 liver specimens from patients with cirrhosis from hepatitis B (n = 24) and hepatitis C (n = 22), and chronic hepatitis (n = 12), and the labeling index (LI) of the positive cells was also determined. Sirius red staining was used to...

AIM: To investigate the expression and significances of proliferating cell nuclear antigen (PCNA) and Ki-67 antigen in liver tissues of cirrhotic patients. METHODS: SP immunohistochemical tech- nique was performed to detect the expression of PCNA and Ki-67 in 58 liver specimens from patients with cirrhosis from hepatitis B (n = 24) and hepatitis C (n = 22), and chronic hepatitis (n = 12), and the labeling index (LI) of the positive cells was also determined. Sirius red staining was used to determine the area density of colla- gen in liver tissues. RESULTS: The expression of PCNA and Ki-67 LI was significantly higher in chronic hepatitis than that in cirrhosis (34.67% ± 8.6% vs 10.38% ± 3.76%, P < 0.001; 2.81% ± 0.51% vs 1.69% ± 1.03%, P < 0.001), and moreover, they were markedly higher in hepatitis C-induced cirrhosis than those in hepatitis B-induced one (13.12% ± 1.42% vs 6.32% ± 2.18%, P < 0.001; 2.48% ± 0.54% vs 0.95% ± 0.77%, P < 0.001). The area density ofcollagenous fiber was significantly increased in cirrhosis than that in chronic hepatitis (12.0 ± 3.1 vs 1.40 ± 1.0, P < 0.001), but there was no differ- ence between the two types of cirrhosis (P > 0.05). The area density of collagenous fiber had no sig- nificant correlation with the LI values of PCNA and Ki-6 in cirrhosis (P > 0.05). CONCLUSION: The proliferation rate of liver cells is lower in cirrhosis than that in chronic hepatitis, but it is higher in cirrhosis from hepa- titis C than that in the one from hepatitis B. There is no significant correlation between the area density of collagenous fiber and the expres- sion of PCNA and Ki-67.

目的:探讨乙型肝炎和丙型肝炎肝硬化患者肝组织中PCNA和Ki-67抗原的表达及意义.方法:选择58例(乙肝肝硬化24例、丙肝肝硬化22例、慢性肝炎12例)肝脏标本,做H-E染色观察肝组织的病理改变;利用天狼红染色切片检测肝组织内胶原纤维的面密度;采用免疫组织化学SP法观察肝组织中PCNA和Ki-67抗原的表达,并检测阳性细胞的标记指数(labelingindex,LI).结果:肝硬化胶原纤维的面密度百分比与慢性肝炎比较(12.2%±3.1%vs1.40%±1.0%,P<0.001)明显增高,乙型与丙型肝炎肝硬化胶原纤维的面密度百分比无明显差异(P>0.05);慢性肝炎与肝硬化比较PCNA和Ki-67LI(34.67%±8.6%vs10.38%±3.76%,2.81%±0.51%vs1.69%±1.03%,均P<0.001)明显增高;丙肝肝硬化与乙肝肝硬化比较PCNA和Ki-67LI(13.12%±1.42%vs6.32%±2.18%,2.48%±0.54%vs0.95%±0.77%,均P<0.001)均明显增高;肝硬化胶原纤维的面密度与PCNA和Ki-6LI之间无相关性(P>0.05).结论:肝硬化组织中肝细...

目的:探讨乙型肝炎和丙型肝炎肝硬化患者肝组织中PCNA和Ki-67抗原的表达及意义.方法:选择58例(乙肝肝硬化24例、丙肝肝硬化22例、慢性肝炎12例)肝脏标本,做H-E染色观察肝组织的病理改变;利用天狼红染色切片检测肝组织内胶原纤维的面密度;采用免疫组织化学SP法观察肝组织中PCNA和Ki-67抗原的表达,并检测阳性细胞的标记指数(labelingindex,LI).结果:肝硬化胶原纤维的面密度百分比与慢性肝炎比较(12.2%±3.1%vs1.40%±1.0%,P<0.001)明显增高,乙型与丙型肝炎肝硬化胶原纤维的面密度百分比无明显差异(P>0.05);慢性肝炎与肝硬化比较PCNA和Ki-67LI(34.67%±8.6%vs10.38%±3.76%,2.81%±0.51%vs1.69%±1.03%,均P<0.001)明显增高;丙肝肝硬化与乙肝肝硬化比较PCNA和Ki-67LI(13.12%±1.42%vs6.32%±2.18%,2.48%±0.54%vs0.95%±0.77%,均P<0.001)均明显增高;肝硬化胶原纤维的面密度与PCNA和Ki-6LI之间无相关性(P>0.05).结论:肝硬化组织中肝细胞增殖指数比慢性肝炎低,但丙型肝炎肝硬化的肝细胞增殖指数比乙型肝炎肝硬化高;PCNA和Ki-67LI与肝硬化胶原纤维面密度无相关性.

Objective To study clinical features and its diagnosis and treatment of alcoholic cirrhosis.Methods 68 cases with alcoholic cirrhosis were retrospectively analyzed,with cirrhosis from hepatitis B as control.Results Alcoholic cirrhosis accounted for 8.5% of in-patient cirrhosis in the correspomding period,mean age being(53.5±14.6),and with more than 135g alcohol intake per day which persisted over 10 years.Such signs significantly increased as dark complexion,liver palms,spider naevi,hepatomegaly,ynecomastia,hypertrophy...

Objective To study clinical features and its diagnosis and treatment of alcoholic cirrhosis.Methods 68 cases with alcoholic cirrhosis were retrospectively analyzed,with cirrhosis from hepatitis B as control.Results Alcoholic cirrhosis accounted for 8.5% of in-patient cirrhosis in the correspomding period,mean age being(53.5±14.6),and with more than 135g alcohol intake per day which persisted over 10 years.Such signs significantly increased as dark complexion,liver palms,spider naevi,hepatomegaly,ynecomastia,hypertrophy of parotid gland,γ-glutamyltansferase(GGT),ratio of aspartate aminotransferase(AST) to alanine aminotransferase(ALT),mean corpuscular volume(MCV),uric acid(UA),serum ferritin(SF) rose apparently,usually combined with hepatogenic diabetes,peptic ulcer,hyperuricemia,hyperlipemia.Peculiar complication were alcohol withdrawal syndrome,gort,gouty arthritis,aseptic necrosis of femur.Among them,53 cases(77.9%) improved clinically,9 cases(13.2%) showed no effect,6 cases(8.8%) died.Conclusions Occurrence of dark complexion,liver palms,spider naevi,hepatomegaly,ynecomastia,hypertrophy of parotid gland and test of GGT,AST/ALT,MCV,UA,SF are helpful to clinic and differentiation diagnosis of alcoholic cirrhosis,abstinence are not able to prevent its development.Long-term therapy aiming at endotoxin toxemia might improve prognosis of alcoholic cirrhosis.

目的探讨酒精性肝硬化的临床特点及诊治方法。方法以同期住院的乙型肝炎后肝硬化为对照,对68例酒精性肝硬化患者临床资料进行回顾性分析。结果酒精性肝硬化占同期住院肝硬化总数的8.5%,发病年龄平均(53.5±14.6)岁;日平均饮酒折合乙醇量>135 g,持续10年以上;酒精性肝硬化中面色黝黑、肝掌、蜘蛛痣、肝大、男性乳房发育、腮腺肿大等体征明显且多见,γ-谷氨酰基转移酶(GGT)、AST异常及肝原性糖尿病、消化性溃疡、高尿酸血症、高脂血症较常见,可出现戒酒综合征、痛风、痛风性关节炎和股骨头坏死等并发症;临床好转53例(77.9%),无效9例(13.2%),死亡6例(8.8%)。结论面色黝黑、肝掌、蜘蛛痣、肝大、男性乳房发育、腮腺肿大等体征、并发症的发生情况及血液生化检查结果,有助于酒精性肝硬化的临床诊断和鉴别诊断;戒酒不能阻止酒精性肝硬化病情进展;长期针对内毒素血症的治疗,有可能改善酒精性肝硬化的预后。

 
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